WEEK 2 Flashcards
Non-blanching petechial rash
N. Meningitidis
APAP antidote
N. Acetylcysteine
Anticholinergic antidote
Physostigmine
Except TCA’s
Benzo antidote
Flumazenil
Cyanide antidote
Na Nitrite
Methanol antidote
Ethanol
Narcotic antidote
Naloxone
Garlic odor on breath
Acute arsenic ingestion
Dig toxicity causes?
Hyperkalemia
Dig reversal agent
Digabind
Pinpoint pupils and resp depression =
Opiate overdose
3 C’s of TCA tox
Cardiac abnormalities
Convulsions
Coma
EKG abnormalities in TCA tox
Wide QRS
Prolonged QT interval
Early signs of APAP tox
N/V
Rotten ages
NItrogen sulfide
ASA Tox
Resp alkalosis
Met acidosis
Normal pH
7.35 - 7.45
Normal CO2
35 - 45
Normal O2
83 - 102
Normal HCO3
22 - 28
Normal Anion Gap
10 - 16
pH: 7.48
CO2: 40
HCO3: 30
Metabolic Alkalosis
pH: 7.45
Co2: 47
HCO3: 29
Metabolic Alkalosis
with
Respiratory Comp
pH: 7.30
CO2: 40
HCO3: 18
Metabolic Acidosis
Fruity odor to breath
DKA
DKA
*** Aggressive fluid therapy with NS *** .1 units/kg insulin per hour Continuous infusion is preferred Potassium repletion Routine bicarb not recommended
Almost all patients in DKA present with glucose above
300 mg/dL
Most will be above 500
HHNS occurs in
Type 2 diabetics
No ketone production because of enough insulin to use glucose in cells.
HHNS syndrome
Severe hyperglycemia > 600
Hyperosmolarity > 315 mOsm/kg
Relative lack of ketonemia (pH >7.3)
HHNS presentation
Usually elderly
AMS
Non-ambulatory
Concomitant infxn