week 1 :endrocrine review Flashcards

1
Q

Cushing syndrome : alterations for diagnosis

A

risk of infections
alterations of nutrition
disturbed body image
risk of skin integrity

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2
Q

cushing syndrome : what is it ?

A

excess glucocorticoids –> corticosteroids

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3
Q

how do we prevent/inteventions for cushing syndrome

A

reduce corticosteroids:
find underlying issues : tumour = remove
too much - suppress - medication = ketoconodazole

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4
Q

risk factors for cushing syndrome :

A

prednisone users
adrenal tumours
women 20-40 years olds

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5
Q

expected findings for cushing syndrome :

A

centripenal fat - buffalo hump , purple striae, htn/moon face

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6
Q

what are the diagnoses we use for cushing syndrome :
what do we teach out patient in terms of education :

A

ct/mri

patient education : avoid extreme temperatures, infection/being too stress

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7
Q

cushing syndrome : nursing care

A

VS/glucose/daily wt
assess for any infection
monitor for thromboembolic events/pulmonary embolic
assess for any pain/loss of function

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8
Q

what are the complications of cushing syndrome

A

hyperglycemia
loss of collagin
muscle wasting
loss of bone matrix- osteoporosis and back pain

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9
Q

what are the signs of infection for cushing syndrome that we should look out for ?

A

drainage/extreme warmth

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10
Q

what are the nutrition management for cushing syndrome

A

high protein, high potassium , high calcium
low carbs , low fat, low sodium
these help correct the effect of excess corticosteroids

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11
Q

it is important to attain wt appropriate for the ht of the patient when it comes down to nutrition management of a Cushing syndrome patient.

A

yes this is true

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12
Q

true or false. it is important to maintain a low calorie diet that meets nutritional needs

A

yes this is true

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13
Q

what type of device do we use for a Cushing syndrome patient ?

A

use device of the bed ( sheep skin to protect the patient )

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14
Q

what is hypothyrodism ?

A

inadequate amount of circulating t3/t4 hormones. decrease in metabollic rate.

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15
Q

what are the alterations in diagnosis when it comes down to hypothyrodism

A

1: thyroid gland dysfunction
2 : anterior pituary, target cell dysfunction
3: hypothalamus not producting tr factor ( tertiary ( rare ) not enough tsh )

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16
Q

health promotion/disease prevention

A

long drug therapy should not be stopped abrupt
diet should include iodine/selenium/zinc

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17
Q

what are the risk factors for hypothyrodsim

A

female- older than 60 years , exposure to radiation in the neck prior to surgery
- inadequate iodine intake/autoimmune disease

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18
Q

what is the expected findings for hypothyrodism
early findings vs. late findings

A

anemic- iron deficiency
early findings - irritability/fatigue.cold intolerance/constipation/wt gain/depression

late fings : low heart rate,rr,bp,dysrythmias,dry skin,peural effusion

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19
Q

what is pleural effusion

A

too much fluid between the layers of your pleura

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20
Q

what are the hormone levels for hypothyrodism

A

high tsh
low t4
high TPO
serum urine/acth/cotisol - normal

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21
Q

true or false. monitor cardiac changes
- take meds on empty stomach when it comes to hypothyrodism

A

true

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22
Q

what are the diagnostic tool for hypothyrodism

A

xray/ct scan/mri

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23
Q

what are the safety consideration for hypothyroidism

A

fall risk
altered metabolism
mental changes
loss of conciousness
immobility due to depression

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24
Q

what are the lab test for hypothyrodism

A

t3
t4
thyroid deficiencies
cholesterol
hgb
hct
rbc

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25
suddenly stopping levothyroxine can lead to what?
can lead to decrease hr, bp, rr , co, coma, hypoxia
26
what is the worst thing that could happen with hypothyrodism
myxedema coma - life threathening , impacted by stress
27
recall that constipation is one of the things that could occur with hypothyrodism , why is that
decrease in gastrointestinal motility
27
what is hyperthyroidism :
hyperactivity of the thyroid gland , with sustained increase in synthesis and release of thyroid hormones
28
what is the alterations in diagnosis of hyperthyrodism :
activity intolerance impaired nutrional status risk of impaired electrolyte balance
29
what is the health promotion/disease prevention
avoiding caffeine reducing stress smoking cessation
30
what is the risk factors for hyperthyrodism
women>men 20-40 years old family history of thyroid disease of autoimmune disease
31
what is the expected findings for hyperthyrodism
goitre bruits exopthlamus ( protusion of eyeballs from orbits, usually bilateral)
32
what could happen after thyroidectomy ?
airway obstruction, after thyroid surgery is an emergency, keep 02, suction/tracheostomy
33
what is exophathalmus
potential corneal injury related to dryness + irritation
34
nursing care for hyperthyrodism
monitor for dysrthimias ensure 02 give iv fluids - replace fluid and electrolyte losses from n and v calm and quiet assisting exercise involving muscle groups
35
what is the diet we should give hyperthyrodism
high protein, high calorie hypermetabolism tatchy , dyspnea, dysarthmias, diaphoresis, pallor, high bp
36
what are the laboratory test we should look upon when it comes down to hyperthyrodism
tsh levels t3 and t4 levels thyroid antibody levels
37
what is the diagnostic procedures
raiu test thyroid scan thyroid ultrasound
38
what are some interventions we could do with hyperthyroidism
surgical therpahy subtotal ( thyroidectomy ) nutritional diet ( high calorie ) radioactie iodine therpahy
39
complications for hyperthyrodism
acute thyrotoxicosis ( life emergency ) released into the circulation -afib -osteoporosis
40
what should the position for a hyperthyroidism patient be ? what should u do before surgery ( thyroidectomy )? what should u encourage the pt ?
have a pt sit upright as much as possible to promote fluid drainage from periorbital area before surgery - show pt how to support head manually while turning in bed to minimize stress on suture line encourage regular exercise
41
what is diabetes mellitus
partial or complete metabolic deficiency of insulin by destruction of pancreatic beta- type 1 arises when body fails to use insulin theraphy - type 2
42
alterations in heath when it comes down to diabetes mellitus
ineffective health management readiness to enhance nutrition --> hyperglycemia ( 3 Ps, weakness lethargy, malaise, blurring of vision, H/A ) risk of injury risk of peripheral injury ( neurovascular dysfunction )
43
what are the risk factors for dm
genetics can predispose type 1 toxins and viruses can predispose an individual to diabetes leading to type 1 obesity/ physical inactivity/ triglycerides>250/ htn - insulin resistance
44
when do we restrict exercise for dm
more than 14 mmol ( especially ketones present ) -- bodys requirement or 128 hz tuning fork for baseline findings -- comprehensive findings foot assesment
45
what is the health promotion and disease prevention
bg levels/exercise regimen/diet pattern
46
what is the laboratory test for diabetes mellitus
8 hr fasting blood glucose 126 mg/ml randome glucose of 200 mg/ml hb a1c > glycosated hgb oral glucose tolerance test
47
true or false. it is good self monitored blood glucose, before meals and bedtime
true it is good
48
nursing care for dm :
monitor vs/bg/in and out/skin integrity/healing wounds/sensory alterations/dietary patterns/exercise patterns
49
what is the complications when it comes down to dm
dka ( diabetic ketoacidosis ) this is life threatening ( ketones )
50
what is addison's disease?
decrease production of adrenal hormones, decrease in cortisol production initially followed by aldosterone
51
recall that addison's disease : decrease production of adrenal hormones, decrease in cortisol production initially followed by aldosterone both of which will what ?
increase acth and msh hormones due to the loss of negative feedback inhibition
52
true or false. addison disease can be prevented
false it cannot be prevented
53
risk factors for addison's disease
type 1 diabetes pernicious anemia testicular dysfunction graves disease mysastenia gravis
54
what is the expected findings for addisions
fatigue extreme weakness unexplained wt loss muscle aches dizziness fainting changes in bp or hr abdominal pain n and v
55
what are the laboratory test for Addison disease
cortisol acth aldosterone renin electrolytes renin bun creatine glucose crh
56
what is the diagnostic tests for addison's disease
abdominal ct xray
57
what is the safety consdierations for addison's disease
risk infection risk fluid imbalance risk for electrolyte imbalance
58
what is the nursing care for addison's disease
monitor i and o administer iv monitor electrolytes administer meds
59
what should we teach the patient for addison's
high sodium high caloric low potassium monitor fluids/med compliance
60
what are the complications for addison's disease
severe dehydration severe hypotension shock vomitting diarrhea extreme muscle weakness h/a extreme sleepiness coma death