Walz Final Material Flashcards
Primary causative agent of liver abscess in cattle?
Fusobacterium necrophorum
Two potential causes for liver abscess (sources of infection)
Rumenitis (secondary to lactic acidosis)
Navel infection (neonates)
Are liver abscesses routinely treated?
No–poor prognosis & difficult for antibiotics to penetrate abscesses
Two ways to prevent rumenitis?
slowly transition over to grain diets
provide access to long-stem roughage
Which groups of cattle are at increased risk for hepatic lipidosis?
Postpartum dairy cows
Overconditioned beef cows
Lipid accumulation in hepatocytes occurs due to?
increased levels of circulating NEFAs
*NOT due to low apolipoprotein
IV glucose and insulin are indicated treatments for which disease? What do they do?
Hepatic lipidosis
Insulin–inhibits lipolysis
Glucose–decreases hormone sensitive lipase activity
Concerning infections with flukes, which spp. is more likely to get:
1) acute infections
2) chronic infections
1) sheep
2) cattle
Which fluke cannot be detected in feces? Why?
F. maga
Walls itself off–infection doesn’t become patent
Which stage of flukes are targeted by:
1) albendazole
2) Clorsulon
3) Ivermec + Clorsulon
1) adults only
2) adults & late immatures
3) adults only (lower concentration of Clorsulon)
Hepatotoxic plants inhibit the animal’s ability to clear which compound? What does it lead to?
Phylloerythrin
Secondary photosensitization
Permanent dental formula for cattle
2(0/4, 3/3, 3/3)= 32 teeth
Causative agent for:
1) Wooden tongue
2) Lumpy jaw
1) Actinobacillus lignieresii
2) Actinomyces bovis
The lesions of wooden tongue infect the _____ and cause what type of inflammation?
Soft tissue & cause granulomatous inflammation (granulomas at base of tongue)
Wooden tongue and lumpy jaw can both be treated with?
Sodium iodide–watch for toxicity
Lumpy jaw occurs when the bacteria gains access to the ____
bone (most commonly the horizontal ramus of mandible)
T/F: BOTH Actinobacillus lignieresii & Actinomyces bovis are normal inhabitants of the oral cavity
TRUE
Give two causative agents for stomatitis in cattle (1 viral, 1 bacterial)
Viral–Bovine papular stomatitis
Bacterial–F. necrophorum (oral necrobacillosis)
Which clinical sign differentiates Bovine papular stomatitis from FMD & mucosal disease(BVDV)?
LACK of lesions on feet
Which spp. are affected by vesicular stomatitis?
Pigs, sheep/goats, cattle, horses
The 3 primary hosts of foot & mouth disease and their purpose
Sheep–maintenance host
Pigs–amplifiers
Cattle–indicators
Which species does NOT get FMD?
Horses!
T/F: FMD cannot be distinguished from vesicular stomatitis clinically
True!
A common cause of choke (esophageal obstruction) &4 places it can occur
rapid ingestion of grain (not enough saliva)
1) throat latch (pharyngeal-esophagus junction)
2) base of heart
3) in front of cardia
4) thoracic inlet
4 Ruminal contraction patterns:
1) primary contraction
2) secondary contraction (eructation)
3) rumination
4) esophageal groove closure
Hypermotility of the rumen is commonly associated with
vagal indigestion
T/F: Chloride levels in a normal rumen should be high
FALSE–chloride is made in abomasum and should be LOW in rumen
The following clinical signs are associated with what disease:
- papple contour of abdomne, intermittent anorexia with weight loss
- presence of stems in feces
- L-shaped rumen felt on rectal palpation
Vagal indigestion
For which type(s) of vagal indigestion would you expect rumen chloride to be elevated?
Type 3 & 4
*high chloride indicates abomasal reflux