Venous Ulcers Flashcards
Etiology
Acquired valvular dysfunction, especially of the communicating system
Venous hypertension leads to fibrin cuff formation around small vessels leading to decreased nutrient diffusion
Venous Ulcer risk factors
trauma DVT pregnancy obesity clotting disorder family history varicose vein
Presentation of Venous Ulcer
good pulses (may be obstructed by edema) usually on lower extremities, not including feet shallow, partial or full thickiness minimal necrosis little pain (pain due to edema possible) brawny edema hemosiderin staining moderate to heavy drainage "wood-like" tissue over gaiter area (right above) inverted champagne bottle deformity maceration common
lipodermatosclerosis
brawny edema with hardening and induration
hyperpigmentation of skin
fibrosis
eczema like skin changes
Edema scal
1+ mild, 1/4 inch pitting
2+ moderate 1/2 inch pitting under 15s
3+ severe 1 inch pitting for 15-30s
4+ very severe >1 inch pitting for >30s
Doppler US
examines venous system
Resting test: supine, listening for spontaneous sounds
Augmentation test: squeeze distal to probe to enhance doppler system
Reflux test: squeeze proximal for signal to disappear
Care of VLU
debridement: needed for re-epithelialization
cleansing
dressings: maintain temp/pH, protect, keep moist
antibiotics
pressure redistribute
Management of chronic wounds
Treat underlying condition optimize blood glucose control adequate nutrition revascularization pain management infection control
Barriers to healing a VLU
presence of unresponsive or senscent cells
inflammatory environment (MMP’s wreak havoc)
deficient or unavailable growth factors
presence of bacteria
VLU plan of care
consider sx compression (after ABI) exercise and walking elevation debridement wound care for every 2 hrs standing, elevate for 30 min
Compression
for ABI’s over .6
30-40mmHg needed to treat
TED hose 15mmHg
ACE wrap: 8-12 mmHg
Prophylactic: 20-30 mmHg
base of toes to knee, distal to proximal with overlap of 25-50%. used with ABI of >.6
change 1/2 times/week.
use caution with CHF
Med/Surg options for VLU
- Ligation: tying off
- vein stripping: resection of perforating veins/vericosities
- Sclerotherapy: injection to fibrose dysfunctional veins
- SPES interruption of perforating veins.
Unna boot
overlap 25-50%
add kerlix and ACE or Coban to maintain compression
MUST BE AMBULATORY
dont stretch with application
OTC/ Custom compression hosiery
Replaced every mo. measure for in morning must be worn completely variable compression NOT Ted hose
Mixed vascular ulcers
About 20% of venous ulcers have a significant arterial insufficiency as well.
High compression not indicated for these patients