vascular Flashcards

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1
Q

Risk factors for PVD

A
Diabetes
smoking
HBP
Obesity
hyperlipidemia
clotting factors
family history
lack of activity
over 65
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2
Q

common sources of eboli

A

chronic ulcerative colitis
IV drug use
aortic thrombi that embolize
thrombi within the heart that embolize

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3
Q

signs and symptoms of emboli

A

decreased sensation

  • edema and muscle pain
  • paleness, decreased or absent pulses, coolness to the touch, weakness, spasm
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4
Q

what is buerger’s disease

A
  • inflammation of small.medium arteries and veins
  • leads to bollackage of arteries of the lower legs and arms
  • symptoms: color changes, temp changes, paresthesia, density to cold, thrombophlebitis, ulcerations
  • SMOKERS, young men age 20-40
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5
Q

what is peripheral material occlusive disease

A
  • stenosis of artery walls
  • common disease greater than 50
  • men 5x greater than women
  • fat and cholesterol congregate on arterial wall
  • symptoms include leg pain worse with exercise and decreases with rest, numbness at rest, muscle pain, loss of hair, paleness cyanosis, weak.absent pose, difficulty walking
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6
Q

How do the symptoms of ischemic rest pain present

A
  • pain unrelated to exercise or exertion
  • pain in the distal forefoot over the met heads
  • aggravating factors - foot elevated in bed out of depend position
  • relieving factor - dependent position to assist blood flow
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7
Q

What is Raynaud

A
  • bilateral vasospasm seen in hands and toes
  • women 20-40
  • trophic changes can occur including shiny and frail skin
  • aggravated by cold, emotional swings and anxiety
  • BILATERAL and progress from white (no flow), blue (vasodilatation), red (blood returns), normal
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8
Q

What is polyarteritis Nodosa

A
  • autoimmune disease causing necrotizing vasculitis of medium and small arteries
  • adult men with hep B
  • symptoms HTN, kidney failure, nausea, vomiting, abdominal pain, seizures, AMS HA, weakness, numbers, rashes
  • may affect heart, brain, muscles, liver skin, kidney
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9
Q

what are the different anatomical groups of veins in the leg

A
  1. subcutaneous/superfiscial
  2. deep/intramuscular
  3. perforating/communicating
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10
Q

what are the major veins of the leg

A
  • great saphenous (femoral triangle down the medial side of entire leg)
  • small saphenous - down the middle of the posterior lower leg
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11
Q

what is thrombophlebitis

A
  • thrombotic occlusion prevent ing outflow of venous system
  • can be deep or superficial
    superficial - redness, warmth, TTP, edema, no serious circulatory effects
    Deep- significant edema, inflammation
  • causes - trauma, muscle strain, blood dycrasias, prolong bed rest or reduced venous out flow
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12
Q

what is the most common place of stasis ulcers

A
  • working muscle have to shut the blood to the superficial veins due to increase IMP
  • if the superficial system can’t handle the blood you get metabolic failure of the local system
  • progressive disorder associated with chronic pooling of blood in the extremity
  • medial malleolus is the most common site
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13
Q

what arteries are you concerned about with humeral shaft fractures

A

axillary and brachial

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14
Q

Why are vascular complications such a high concern with knee and elbow dislocations

A

there is only one vessel going to the lower half of the limb

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15
Q

DVT rate post op without profolactice

A

50-60% - best thing is to get up and walk

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16
Q

What is the Wells criteria for DVT

A

algorithm for detecting DVT

17
Q

what is a false aneurysm

A
  • full thickness tear that is contained by the adventia or adjacent connective tissue of the artery
  • most common finding is a palpable pulsating mass at the site of former trauma
  • requires diagnostic testing with angiography or US
    requires surgery to fix
18
Q

What do doppler vascular tests tell you

A
  • speed and direction of blood flow
19
Q

What are normal ABI

A
Systolic ankle pressure / systolic arm pressure
1-1.4 - normal 
0.9-1 - acceptable
0.8 - some disease
less than 0.8 needs referral
20
Q

what is pulsed volume recording

A

measures changes in the volume of an extremity that occurs as blood flow to the extremity increases and decreases

  • normal - wave form is a steep up slope, sharp peak and a concave down curve, down slop with a possible “dicrotic notch” indicating the closing of the aortic valve
  • abnormal - decrease slope of the upslope, rounded peak and possible reversal of the downslope to a convex curve
  • pathology is infront of one of the 4 measuring cuffs
21
Q

arterial injury occurs in what percentage of long bone fractures

A

0.6-3% with middle and distal femur the most common site

22
Q

what tests would help you differentiate an arterial disruption versus a compartment syndrome

A
  1. capillary refill and distal pulses
    - arterial disruption they should be absent
    - compartment syndrome they are often present
23
Q

what are hard signs of arterial injury

A
  • pulsatile external bleeding
  • enlarging hematoma
  • absent dital pulses
  • bruit with auscultation
  • grossly ischemic limb (6Ps)
24
Q

what are the signs of ischemia

A

6 Ps

  • pain
  • paralysis
  • pallor
  • paresthesia
  • pulselessness
  • poikilotherm (coolness)
25
Q

what is a bruit and how should you assess it

A

the abnormal sound generated by turbulent flow of blood in an artery due to either an area of partial obstruction; or a localized high rate of blood flow through an unobstructed artery

  • must auscultate
  • Most occur only in systole, so the bruit is intermittent and its frequency dependent on the heart rate
  • Anything increasing the blood flow velocity such as fever, anemia, hyperthyroidism, or physical exertion, can increase the amplitude of the bruit.
26
Q

what are the compartment of the leg

A
  • anterior
  • lateral
  • superficial posterior
  • deep posterior
27
Q

what is a major complication associated with compartment syndrome

A

REPROFUSION INJURY - as circulation is restored metabolic byproducts causes production of the oxygen free radical superoxide that damages the tissues and this requires medical management to prevent damage

28
Q

what are the layers of the artery

A
  • inner - intima
  • media
  • outer - adventia
29
Q

What are the most common arterial diseases

A
  1. emboli
  2. thromboangiitis obliterans AKA Beurgher’s disease
  3. peripheral arterial occlusive disease
  4. raynauds
  5. hand vibration syndrome
  6. polyarterieties nodosa
30
Q

what are the common venous pathologies

A
  1. thrombophlebitis (superficial and deep [DVT])
  2. chronic venous insufficiency
  3. varicose vein
  4. stasis ulcers
31
Q

what are the stages of lymphedema

A

stage 1 - spontaneous reversal with pitting edema
stage 2 - non pitting edema
stage 3 - irreversible edema with hardening of affected area

32
Q

what is a arterial venous fistula

A
  1. direct communication of the arterial and venous systems, skip the capillaries
  2. The problem result from the low-hesitance flow circuit
    - increase strain on the heart from increased cardiac output
    - venous return is impaired secondary to increased demand on the system
    - limb ischemia secondary to skipping of the capillaries
33
Q

how does chronic arterial ischemia present

A
  • muscle atrophy
  • thick down curving nails
  • skin is shinning and hairless
  • dependent rubor - hyperemic (red) appearance of the skin in dependent position
  • extremity edema from keeping the extremity in a dependent position to help with blood flow
  • usually the distal toes
34
Q

how does chronic venous insufficiency present

A
  • skin is pigmented and brawny

- ulceration in the ankle and distal calf, usually sparing the toes