pain science nxtgen Flashcards

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1
Q

How does the IASP define pain

A

an unpleasant sensory and emotional experience associated with actual or ….
….potential tissue damage, or desired in terms such as damage

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2
Q

The purpose of pain

A
  • produce a with drawl effect (physical and mental)
  • protection
  • resolution
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3
Q

How does the the body evaluates pain

A
  • sensory information
  • prior experience
  • stress regulation
  • social cues
  • future expectations
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4
Q

What is it important to understand to functional of pain related to potential tissue damage

A
  • it can be the basis of unexplainable pain responses
  • phantom limb
  • unexplainable weakness
  • CRPS
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5
Q

How can you use the examples of hunger, thirst and vision to help explain the potential aspect of pain

A
  • hunger thirst and vision don’t always tell an accurate story
  • you can be very hungry or thirst and have no need for food or water
  • you vision can be fooled
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6
Q

How is habitation used in the treatment of chronic pain

A

the pain response can be modified by gradual introduction of the pain stimulus

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7
Q

What cognitive-evaluative variable impact pain processing in the neuromatrix model

A
  • tonic: cultural learning, past experience, personal variability
  • phasic: attention, expectation, anxiety, depression
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8
Q

what are the sensory discriminative inputs of sensory processing according to the neuromatrix

A

cutaneous, visceral and musculoskeletal inputs

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9
Q

What are the outputs of the pain response in the occurring to the neuromatrix model

A
  1. pain perception - sensory, affective and cognitive
  2. actions program - involuntary and voluntary
  3. stress regulation - cortisol, noradrenaline levels, endorphins levels, immune response
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10
Q

Smart 2012 identified three classification of pain response when looking at low back pain

A
  1. nociceptive
  2. peripheral neuropathic - disease or injury directly to the nerve
  3. central sensitization
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11
Q

what is causalgia

A

IASP 0 a syndrome of sustained burning pain, allodynia and hyperpathia after traumatic nerve lesion, often combined with vasomotor and sudomotor dysfunction and later trophic changes

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