Unit 2 Flashcards
Serena Williams, an international tennis star underwent a series of serious health problems. It started in Fall, 2010 when she cut her foot on a piece of glass at a restaurant. She underwent 2 operations to repair the foot. She had limited movement for 20 weeks first with a cast, followed by a walking boot for 10 weeks. In February, 2011 she was hospitalized for pulmonary embolisms (PE) in both lungs.
Which part of Vichow’s triad put Serena at risk for a DVT and PE?
vascular wall injury
The most obvious answer is vascular wall injury due to the trauma that incurred that led to her injury and subsequent surgery. Surgery alone causes vascular wall injury. You could also argue for venous stasis due to the immobility that she had for the 20 weeks when she was in a cast.
Symptom of a DVT?
erythema at the area of the thrombosis
yes
Symptom of a DVT?
swelling in one leg
yes
Symptom of a DVT?
severe pain (10 out of 10)
no
Symptom of a DVT?
Temp of 100 F
no
Symptom of a DVT?
maculopapular rash at the area of thrombosis
no
WBC count of 11,000 cells/mm3
yes
What is the most serious risk for a patient with a DVT?
pulmonary embolism
Patients with a DVT can have all of the items listed (pain, blood thinners, another DVT and a PE). The one with the biggest consequence is a PE, because it can be deadly if is large enough or if there are many PEs. A PE interferes with the ability for gas exchange in the lungs and can result in death.
Which of the following patients has the highest risk of developing a DVT?
A pt who has a history of having a DVT
True or False
A person with a PE can experience extreme shortness of breath, tachycardia, chest pain and a feeling of anxiety.
True
A patient has to have a DVT first before a PE develops. A PE is a DVT that has traveled (emboli) to the pulmonary arteries.
Symptoms of a PE include chest pain, fast heart rate, shortness of breathe, feeling of anxiety (or impending doom) and can be deadly if the pulmonary artery occlusion is large enough.
We treat patients with a DVT so that a PE is prevented. (We treat both DVT and PE.)
A way to prevent a DVT is through ambulation.
Normal or hypertensive?
89 year old with a BP of 182/90
hypertensive
Normal or hypertensive?
18 year old with BP of 118/72
normal
Normal or hypertensive?
72 year old with BP of 145/92
Hypertensive
Normal or hypertensive?
65 year old with BP of 145/92
normal
The formula for arterial blood pressure is ____________X peripheral vascular resistance.
Cardiac output
Cardiac output X PVR= arterial pressure. You can take it a step further an look at what makes up cardiac output and what influences PVR.
Modifiable Hypertension risk factors
Tobacco use, obesity, diet low in potassium, excessive alcohol use, oral contraceptive pill use
Emily Sparks is a 39-year old female who has end stage renal disease (CKD stage 5) and is on dialysis. She was recently diagnosed with HTN. The nurse recognizes that she most likely has what type of HTN?
secondary
Emily most likely has secondary HTN as the end stage kidney disease can lead to HTN since the kidney is not functioning and can not eliminate wastes like it should.
True or False
Dysfunction of the sympathetic nervous system, RAAS (renin, angiotensin, aldosterone system) adducin and naturietic peptides can result in increased peripheral vascular resistance and increased blood volume; two main causes of sustained hypertension.
True
True or False
HTN can lead to blindness
True
HTN can lead to retinopathy, which can eventually lead to blindness.
True or False
Kidney failure can cause HTN. HTN does not cause kidney failure.
False
Kidney failure can cause HTN because of fluid volume overload and HTN can cause nephropathy and increased pressure in the kidney causing dysfunction in the glomerulus and difficulty with filtration.
Sustained hypertension for years can lead to ___________in the heart.
myocardial infraction, hypertrophy of left ventricle, heart failure, and angina
Put the following in order from least pathogenic (1) to the most pathogenic (4).
- fibrous plaque
- fatty streak
- foam cells
- complicated lesion
3,2,1,4
The foam cells turn into fatty streaks, which develop into fibrous plaques, which have the potential to become a complicated lesion if the plaque ruptures.
True or False
When a patient says “I was told I have 4 vessel occlusion” he is referring to atherosclerosis in his coronary arteries.
True
A plaque from atherosclerosis can result in:
1) progressive narrowing of the vessel resulting in occlusion (like in this example)
2) sudden vessel obstruction from plaque hemorrhage or rupture.
3) thrombois and formation of emboli
4) aneurysm formation caused by weak blood vessel walls.
A patient complains of angina whenever he takes his morning walk with the dog. He tells his provider that his pain in his chest always come on when he reaches “Elm St”. The provider recognizes what is the underlying pathology in this situation?
Stable angina
This situation is characteristic of stable angina, which would be related to a fibrous plaque which is causing a decreased diameter in the blood vessel. It is predictable as the demand on the heart increases, the pain appears.. Stable angina is relieved by rest and/or nitroglycerin.
John Jacob Jingleheimer Smidht is a 68 year old male. He presents to the outpatient clinic with complaints of occasional chest pain. He is concerned because his Dad died from heart problems at an early age. He has a history of hypertension and “sugar diabetes” but doesn’t take any medications for them because he doesn’t feel like he needs them. He smokes 1/2 pack a day of cigarettes and has since he was 12 years old. “Hard to quit, when I’ve been doing it for so long”. He was told he needs to watch what he eats as his cholesterol was high the last time it was checked. On physical exam, you note that he is considered “obese” based on his body mass index and he has a waist circumference of 44 inches.
What risk factors does John have for coronary artery disease?
Smoking, cigarettes, family history, diabetes mellitus, high cholesterol
John has a lot of risk factors (smoking, family history, DM, high cholesterol). He does have a 44 inch waist. Most men with a large waist are “apple” shaped. Apple shaped is a higher risk factor for cardiovascular disease than the typical pear shape of women. I know we haven’t discussed this yet—teachable moment.
True or False
A foam cell is an oxidized LDL (low density lipoprotein) that is consumed by a macrophage. This occurs early in the process of atherosclerosis.
True
Which of the following are some core components to atherosclerosis?
Narrowed blood vessels from the buildup of plaque
Behavioral and Genetic factors that lead to endothelial injury
High LDL levels. The LDLs become oxidized.
Plaque disruption can lead to a thrombus or an emboli which could mean a heart attack.
True or False
Atherosclerosis occurs only in the coronary arteries of the heart.
False
Atherosclerosis can occur in every blood vessel in the body. Common places are the abdominal aorta, coronary arteries, femoral and popliteal arteries (leads to peripheral arterial disease), carotid arteries and the arteries in the brain.
It is the same process, it just occurs in different part of the body.
When a patient develops peripheral arterial disease, are some expected findings?
history of dyslipidemia
shiny, pale skin
reports of intermittent claudication
PAD (peripheral arterial disease) is atherosclerosis in the peripheral arteries of the body.
Risk factors are the same for PAD as they are for coronary artery disease (CAD).
Symptoms include intermittent claudication, diminished or absent pulses, coolness of skin, pallor, and parathesias.
If a person assigned female at birth has angina, how might they present?
Extreme fatigue, heavy pain in the chest and down the left arm, shortness of breath, and nausea and heartburn
Typical angina presents as heavy type pain that is steady and centered in the precordial or substernal area with radiation to jaw, shoulder or arm. Anginal equivalents is a description of non-typical presentations of angina which includes dyspnea, diaphoresis, feeling faint/dizzy, extreme fatigue and heartburn.
Those assigned female at birth can present with either.
Put the following in the order from the least serious (1) to the most serious (4).
- STEMI
- unstable angina
- non-STEMI
- stable angina
4,2,3,1
stable angina–unstable angina—non-STEMI (non Q wave MI)——-STEMI (Q wave MI)
A patient arrives in the emergency room with chest pain and is diagnosed with a STEMI. Which of the following findings are expected?
increased cardiac enzyme (troponin), EKG changes with ST elevation and eventually a Q wave, and necrosis of the myocardium transmural (from endocardium to epicardium)
After _____________[x] minutes without oxygen, the cellular damage to the heart is irreversible.
20
A client has been diagnosed as having unstable angina without an MI.
The nurse expects the symptoms of ______based on what pathological occurrence ______ ? The nurse expects ______ of cardiac enzymes like troponin.
chest pain at risk or minimal exertion
Plaque disruption that has caused occlusion of a blood vessel.
no elevation
When a myocardial infarction occurs, what has happened?
necrosis of cardiac tissue
Ischemic death of cardiac tissue or necrosis of cardiac tissue occurs with an MI. That part of the cardiac muscle becomes noncontractile, necrotic and scarred leading to further problems.
Which of the following are symptoms of an MI?
Intense feeling of anxiety
heavy, constricting chest pain
nausea
suddenly feeling sweaty
shortness of breath
What happens to the heart after an MI?
After 2-3 months scar tissue has formed causing contraction and conduction issues.
There are a lot of complications immediately from an MI (chest pain, acidosis, electrolyte imbalances, contraction dysfunction, dysrhythmias and increase catecholemine affects).
After the MI, physiologically there are changes:
6 hours–blue and swollen (think hypoxia)
48 hours–grey with yellow streaks, neutrophils move in
8-10 days-granulation tissue
2-3 months–scar tissue and ventricular remodeling.
In heart failure, the heart is likely to be ______, so that the diameter of the heart ______ .
overstretched, decreases
The body tries to “fix” the problem of heart failure by compensatory mechanisms. How does the body compensate?
Increased heart rate
increased sympathetic nerve impulses
release of renin and aldosterone
The body tries a lot of compensatory mechanisms without any improvement. Strategies include: activating RAAS (water and Na retention), increased HR, increased peripheral vascular resistance.
Systolic heart failure, the heart has a problem with _____ . As a result, the ejection fraction ______ . A normal ejection fraction (EF) is 65%.
ejection, decreases
What is an example of high output heart failure?
Severe anemia
High output heat failure is not very common. Examples include severe anemia and thyrotoxicosis. Low output heart failure is much more common.
Left or right heart failure symptom?
cough of pink frothy sputum
left
Left or right heart failure symptom?
2+ ankle edema
right
Left or right heart failure symptom?
shortness of breath
left
Left or right heart failure symptom?
paroxysmal nocturnal dyspnea
left
Left or right heart failure symptom?
anorexia
right
Left or right heart failure symptom?
weight gain of 3 lbs overnight
right
Left or right heart failure symptom?
orthopnea
left
Left or right heart failure symptom?
dyspnea on exertion
left
Left or right heart failure symptom?
hepatomegaly
right
Which two pieces of information would be most important to know about a patient being admitted with heart failure?
BNP levels and ejection fraction
BNP is used for diagnosis of heart failure. The higher the number the worse the clinical situation. A BNP of 500 is diagnostic.
The EF of a patient tells you how decompensated their heart function is and gives a good idea of severity of the illness.
True or False
A person who develops heart failure may have symptoms that are more indicative of failure on one side (right or left) but eventually will develop symptoms of both right and left sided heart failure.
True
Which symptoms would the nurse expect to see in a patient with predominately right-sided heart failure?
loss of appetite or getting full easily
swollen feet and ankles
Signs of systemic congestion are much more common with right-sided heart failure and can include lower extremity edema, organ congestion (hepatomegaly) which can lead to abdominal fullness and anorexia and also jugular vein distention.
Pulmonary symptoms are much more common with left-sided heart failure (shortness of breath, PND, orthopnea, pink frothy sputum, crackles in the lungs).
After an automobile accident a client who is unconscious and exhibiting decerebrate posturing is brought to the emergency department. When assessing the client, what does the nurse expect to observe?
Hyperextension of both the upper and lower extremities
Decorticate: inward
flexion of the arms, wrists, fingers with adduction of the upper extremities, internal rotation and plantar flexion of the LE.
Decerebrate: outward
results from increased muscle excitability. Rigidity of the arms with the palms of the hands turned away from the body and the stiffly extended legs with plantar flexion of the feet.
Both are POOR PROGNOSTIC SIGNS—usually happens in stage IV.
A client is having a brain attack (CVA) is brought to the emergency department. The vital signs are P 78, R 16, BP 120/80. The change in this client’s vital signs that indicates increasing intracranial pressure (ICP) requiring notification of the practitioner is:
Pulse 50, Resp 20, BP 140/40
In stage 3, as the brain is beginning decompensation, vital signs start to change. Remember Cushing’s Triad: Increased systolic BP (up to 270 mmHg) with widened pulse pressure; irregular respirations and bradycardia. Widened pulse pressure is the difference between the systolic BP and the diastolic BP. Bradycardia occurs in a compensatory response because of the extremely elevated BP.
Initially after a brain attack (CVA), a client’s pupils are equal and reactive to light. Later the nurse assesses that the right pupil is reacting more slowly than the left and the systolic blood pressure is beginning to increase. The nurse concludes that these signs are suggestive of:
Increased intracranial pressure
In stage 3, pupillary changes start to occur. Initially the pupils are slower to react or react differently from each other. They then progress to pinpoint size. In stage 4, one pupil becomes dilated and one is pinpoint then eventually both are dilated (usually called fixed and blown by practitioners). Systolic BP starts to rise in stage 3.
The nurse is caring for a client with increased intracranial pressure. Which of the following assessment findings should the nurse report immediately?
Absence of pupillary response
Absence of pupillary response is always an abnormal finding and in someone with suspected IICP, this should be reported immediately. Vomiting tends to happen early, but it is usually not accompanied by nausea and tends to be projectile in nature.
When increased intracranial pressure occurs in the brain, what is the relationship between ICP and arterial pressure?
ICP = arterial pressure
impacts blood flow and the exchange of oxygen. This first starts to happen in stage 3.
Which of the following is options describe normal statements regarding intracranial pressure?
The first state of increased ICP, the body is able to compensate by displacing CSF and if often not even noticable.
Normal ICP is less than 15 mmgHg (5-15 mmHg is normal for testing purposes).
A decrease in level of consciousness is the earliest and most reliable sign of increased intracranial pressure.
The body compensates first by displacing CSF and is often not even noticeable.
During ICP, there is massive vasoconstriction to the point where the ICP equals the arterial pressure. Remember there is a increased blood pressure with a widened pulse pressure. Example 180/60 blood pressure.
A client with a head injury is admitted to the nursing unit. The following is found on physical assessment:
BP 180/50, pulse 48 beats per minute. Client opens eyes but does not respond verbally. The pupils are slow to respond to light. The breathing is irregular in pattern and the client has decorticate posturing.
What is this client most at risk for?
Brain Herniation
This patient is very advanced in displaying signs of IICP and is very high risk of brain herniation if it has not happened already. The patient is in stage 3 and looks like they are moving toward stage 4 where herniation occurs.
A ____ is an acute focal neurological deficit from an interruption of blood flow in a cerebral vessel due to thrombi or emboli or bleeding into the brain.
Stroke
A TIA can be described as a brief period of inadequate cerebral blood flow that has neurological deficits that usually resolve within hours (lasts no longer than 24 hours) and can be caused by platelets accumulating at the site of a thrombosis or from vasospasm.
The nurse is admitting a client diagnosed to have a CVA involving left-brain damage. The nurse is told that the patient has aphasia. What does this mean?
Difficulty either understanding what is said to them or difficulty in speaking
Which of the following factors put someone at increased risk for having a cerebral vascular accident (CVA)?
African Americans
Blood pressure readings consistently running 160/90 - 180/90
Alcohol use disorder
Afib
diabetes mellitus
What pathological changes occur with a stroke?
creation of a necrotic core surrounded by a prenumbra
cerebral edema
creation of lactic acid from anaerobic metabolism
A client is admitted with a hemorrhagic stoke. What does this mean?
The client had a cerebral artery rupture that led to bleeding
What is a common cause of an embolic stroke?
Afib
True or False
A person who has a seizure disorder always has convulsions. True or False
False
People with convulsions have seizures, but not all seizures have motor activity like convulsions.