UE function Flashcards

1
Q

What is the enablement model?

A
Define patient's roles (participation)
What skills (activities) are needed
What resources does the patient have? What is lacking?
Set goals related to functional recovery: control of arm movements changes depending on the goal of the task
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2
Q

What are factors at play during sensory motor processing?

A

Constraints of individual
type of task
Specific environmental constraints

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3
Q

What are UE functional roles?

A
Balance/arm swing
Pointing/gesturing
Weight bearing
Reaching
Grasping
Manipulation
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4
Q

What are subsystems for reaching, grasping, manipulating?

A

Musculoskeletal

Neural: internal representations, higher level processes, sensory processes, motor processes

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5
Q

What part of nervous system controls reach and grasp?

A

Primary motor cortex
Premotor cortex
Posterior parietal lobe
Cerebellum

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6
Q

What is amplitude of head movement when coordinating eye, head, trunk when looking at something?

A

When head movement is necessary to look at an object the amplitude of head movement is usually about 60-75% of the distance to the target

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7
Q

What sensory systems are involved in normal UE function?

A

Somatosensory input

Visual feedback

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8
Q

What would you expect from a patient who has a ventral stream lesion?

A

Inaccurate in estimating size of object but they can still reach for it.
Intact action, altered perception

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9
Q

What are the motor systems involved in normal UE function?

A

Multiple degrees of freedom
Reach/grasp: under distinct neural control
ROM, strength, tone, coordination
Posture: scapula position, grasp patterns
Eye movement: saccade
Head, trunk movement
Transport hand: role of thumb, shape of hand opening
Postural support

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10
Q

What does the bell shaped velocity profile look like for point vs. grasp?

A

Movement duration of reaching is longer than pointing
Preparing to grasp object: acceleration of reaching is shorter than duration
Pointing: acceleration is longer than the deceleration (high velocity when finishing)

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11
Q

What are aspects of coordinating grasp?

A

Close on object by moving fingers (thumb stabilizes)
Sensory info on characteristics of object: weight, firmness, shape, slickness
Grasp patterns

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12
Q

What is manipulation?

A

Interaction with environment: writing, typing, dressing, opening/closing an object, eating, throwing, counting money, turning page

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13
Q

What are theories of neural control of reach and grasp?

A

Fitt’s law: the more difficult the task, longer it takes to move
Speed-accuracy trade off: contrains of visual system

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14
Q

What are UE dysfunctions (impairments, abnormalities)?

A

Impairments in: vision, perception, sensation, proprioception
Abnormalities in: tone, voluntary movement (strength, coordination)

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15
Q

What are causes of reach dysfunction?

A

Timing problems
Impaired inter limb coordination: elbow, shoulder (degrees of freedom)
Proximal weakness

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16
Q

What are different grasp dysfunctions?

A
Anticipatory hand shape
Grip force
Precision grip
Premature finger closure
Slow release
17
Q

What are characteristics of neuro recovery?

A

Proximal vs. distal motor control
Unilateral vs. bilateral function
Driven by active movement, task goals
Learned non use post CVA

18
Q

What are 3 parts of assessment for UE?

A

Impairments (hand grip force, pinch force), observation, functional performance scale

19
Q

What are some different functional performance scales?

A
Motor activity log
Wolf motor function test
Chedoke-McMaster stroke/hand and arm inventory
Box and block test
Arm reaction arm test
Nine hole peg test
20
Q

What is an effect of stroke on the shoulder?

A

Pain in 70-84% of patients
Local, end range pain develops into diffuse, pervasive pain
Effect on posture, learned non use

21
Q

T/F: shoulder subluxation can occur post CVA?

A

True

22
Q

What are some causes of shoulder pain post CVA?

A
RTC muscles: paralysis, incoordination or weakness
Adhesive capsulitis
Scapular position
Scapulo-humeral rhythm
Repeated trauma
23
Q

What are complex regional pain syndrome symptoms post CVA?

A

Chronic pain affecting paretic arm or leg
10-25% post CVA
Hand tenderness, hypersensitivity, swelling, warm, red, glossy skin
Monitor for early signs

24
Q

What are treatment ideas for shoulder impairments post CVA?

A
Depends on cause
Prevent subluxation
Soft tissue restrictions
Air splint
Weight bearing
25
Q

What is treatment for reduced scapular mobility?

A

Soft tissue scapular mobilizations
Side lying
Add active movements/PNF diagonals
Soft tissue/capsular tightness may also be present in elbow, wrist, finger joints

26
Q

What are considerations for treating hand impairments?

A

Edema: multiple causes
Impaired grasp and release function
Alien hand syndrome

27
Q

What is treatment for hand impairments?

A

Prevent/treat edema
Facilitate or inhibit flexor/extensor muscles
Hand positioning splints

28
Q

What is task oriented training for UE treatment?

A

Treat impairments
Retrain strategy: eye head coordination, reach, grasp/release, manipulation
Restore function
Connect to patient goals, familiar activities
unilateral vs bilateral tasks

29
Q

What are deficits of uninvolved UE and why do we see these?

A

Deficits: coordination, timing/speed, grip strength
Why: bilateral cortical control, component of corticospinal tract that does not decussate, cognitive deficits, visual perceptual deficits

30
Q

What is constraint induced movement therapy (CIMT)?

A

overcome learned non use in stroke
Shows changes in brain mapping
Restrict less affected limb during waking hours of 14 days
Improved motor and function : maintained 2 years post study

31
Q

What are issues with CIMT?

A

Patient compliance with protocol, safety, criteria for minimal motor function, reimbursement