U3 AOS2 Flashcards

1
Q

what are the fuels and what system are they used by

A

-fat- aerobic energy system
- cp- ATP-CP system
carbohydrate-

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2
Q

cross over concept

A
  • when resting preferred fuel is fats
    -when we increase int, more carbs r used
  • when enrgy supplied by fats and carbs is equal= cross over point
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3
Q

cp fuel

A
  • limited quantities
  • energy from breakdown of phosphocreatine
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3
Q

fats

A

-take longer to break down
- higher oxygen cost
- preferred fuel source at rest

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4
Q

cabs

A
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5
Q

why don’t we use fats all the time

A
  • transport of free fatty acids to the muscle is slow
  • breakdown of free fatty acids requires greater oxygen than glucose

when intensity increases we need more energy faster and the time we have to produce energy decreases

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6
Q

fat oxidization and glycogen sparing

A
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7
Q

anaerobic glycolysis

A

-produces energy by partially breaking down glucose anaerobically
glycogen breaks to glucose into pyruvic acid, insufficient oxygen its turned into lactic acid not as fast as ATP cp but still fast high intensity exercise (100 VO2 max or > 85% max hr)
- carbs used, if glucose is depleted this system can no longer produce energy
- fast rate
- fatigues after aprox 75 due to metabollic by products (hydrogen ions)- active recovery required to remove by products

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8
Q

what’s interplay

A

all 3 energy systems work together to supply energy and resynthesis atp

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9
Q

aerobic energy system

A

-oxygen required
- activities below 90% VO2 max and below 80% HR
- carbs is mainly used to fuel this system, however fats are used at a lower intensity
- if glucose and fats r depleted the system cant produce energy
- process is slow and produces heat( decrease temp, too much = fatigue, heat is transferred to skin through redistribution of blood flow), water ( mito makes as a part of krebs and etc) and co2 (oxygen to resynthesis atp) as by product
- heavily relied on for endurance sports

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10
Q

intermittent vs continuous

A
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11
Q

acute vs chronic

A

a- short term, going for a run
c- long term, training not exercise

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12
Q

muscular acute responses

A
  • i recruitment of motor units
    -i body temp
  • d intermuscular substrate levels
  • i metabolic by products
  • i blood flow to working muscles
  • i oxygen uptake and consumption (aVO2 diff)
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13
Q

increased recruitment of motor units

A
  • as intensity i amount of force dev in a working muscle i
  • your brain either
  • i the number of motor units activated
  • i the frequency of messages sent to activate motor units
    (small motor before large motor units)
    ( greater fiber recruitment= greater muscle force)
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14
Q

increased respiratory rate

A

number of breaths taken in a min

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15
Q

increased ventilation

A

volume of air breathed in per min
- rr X i tv = iv

16
Q

increased tidal volume

A

how much air is inspired and expired in 1 breath

17
Q

increased pulmonary diffusion

A

the transfer of oxygen from the alveoli (lungs ) to the capillaries (blood)

18
Q

fatigue

A

exercise induced reduction in the ability of a muscle to produce force and power
- slow down less explosive

19
Q

factors that affect rate of fatigue

A
  • type of activity (continuous, intermittent )
  • muscle fiber type
  • type of contraction- eccentric, concentric isometric
    -intensity- sub, max, high int
  • duration
    -level of fitness
20
Q

sites of fatigue

A

cns

21
Q

FATN

A
22
Q

atp-cp fuel dep

A
  • if cp is depleted then an athlete is forced to use glucose to maintain high int
  • glucose is slower and doesn’t provide energy as fast, muscle doesn’t produce as much force or power
23
Q

LIP

A

lactic acid is continuously produced and removed
lip- ability to balance removal and production of lactic acid
on a graph its the final point where balance is withheld, it represents the max intensity athletes can still be working at aerobically without any build up of lactic acid
large build up = fatigue occurs at a faster rate

24
Q

accumulation of metabolic by- products

A
  • increase in ADP level, reduce force by slowing relaxation of muscle
  • caused by blocking calcium to leave muscle
  • an increase in pi reduces muscular force by stopping calcium being released into muscle cell
  • stopping contraction as it doesn’t allow bridges to attach to actin microfilament
25
Q

decrease in energy substrate

A
26
Q

increased metabolic by products

A
  • lactate production increases as exercise int increases
    -before lip body is either oxidising it for energy production or converting to glucose/glycogen
  • once intensity i and passes lip body is unable to clear the lactate as quickly as its being produced
27
Q

increased body temp (acute response)

A
  • heat is a by product of breaking down atp to adp
  • greater enrgy=greater heat
  • body stimulates sweat glands to produce sweat (cool body) as well as i blood flow to skin
28
Q

increased blood flow

A
29
Q

respiratory acute responses

A
  • i respiratory rate
    -i tidal volume
    -i ventilation
    -i diffusion
30
Q

increased rr,tv and ve

A
31
Q

increase in diffusion (respiratory)

A
  • gas exchange in the lungs at the alveolar capillary interface and in the muscle
  • diffusion of gases always happens in areas of high pressure
  • i diffusion allows greater amounts of o2 and co2 to be exchanged during exercise
  • o2 concentration is high (high pressure) in the alveoli therefore oxygen diffuses into blood stream
  • co2 conc is high in blood (from muscle) and diffused into the alveoli
32
Q

v02

A
  • oxygen uptake- oxygen that can be upatken and used
33
Q

vo2 max, absolute and relative

A
  • max amount of oxygen that can be taken up, transported and utalised per min
    ab- actual volume of o2 being consumed by the muscles per min (l/min)

r- takes into account body weight or composition (ml/kg/min)

34
Q

relationship between o2 consumption and exercise intensity

A

oxygen has a linear relationship with intensity. as int increases so does o2 consumption/demand

35
Q
A