Troubleshooting Small Animal Anesthesia Flashcards
What are the normal systolic and diastolic blood pressures seen in anesthetized patients? What is indicative of hypotension and hypertension?
- SYSTOLIC = 100-130 mmHg
- DIASTOLIC = 60-100 mmHg
(mean = 80-120 mmHg) - HYPOTENSION = SAP <80 mmHg, MAP <60 mmHg
- HYPERTENSION = SAP >130 mmHg, MAP >120 mmHg
What is the most common consequence of hypotension? What is over 15 minutes of hypotension associated with?
poor perfusion of the brain, heart, kidneys, GIT, and liver
long-term damage to kidneys
What is arterial blood pressure a product of? Why is it so important?
CO and systemic vascular resistance
critical determinant of tissue/organ perfusion and oxygen delivery
If a patient is chronically hypertensive, how is hypotension determination changed?
> 20-40% reduction from awake baseline
In what ways can the surgeon cause hypotension while performing their surgery?
- hemorrhage
- vascular compression (vena cava)
What 5 patient factors should be checked when hypotension is present?
- HR and rhythym, pulse quality, CRT
- oxygenation, ventilation, anesthesia machine integrity
- anesthetic depth
- bradycardia, profound tachycardia
- cardiac arrest
What anesthetic machinery should be checked with hypotension? How is it adjusted?
oscillometric or Doppler
- check BP cuff placement, size, and tightness
- change position of cuff
- try another machine
What are the 2 most common ways to troubleshoot hypotension?
- decrease inhalant concentration and increase or add injectable drugs
- treat bradycardia
In what 2 ways is bradycardia treated in patients with hypotension?
- bolus fluids +/- hypertonic saline +/- blood products or colloids depending on comorbidities (DON’T keep bolusing if there is no improvement)
- administer inotropes or pressors - dopamine, dobutamine, vasopressin, norepinephrine, epinephrine (emergency)
What are the 2 phases of Dexmedetomidine affects? How does this alter ways of troubleshooting its effects?
- initial 45 mins - vasoconstriction causes hypertension with reflex bradycardia
- after 45 mins - central sympathetic nervous system depression leads to hypotension and continued bradycardia
treating bradycardia (atropine, glycopyrrolate) within phase 1 would cause increased HR in the face of high afterload = increased myocardial work
- wait until phase 2, where hypotension can be resolved by improving HR
What is the normal arterial CO2 and ETCO2 in normal patients?
35-45 mmHg
> 50 mmHg
In what 7 ways should a patient with hypoventilation be checked?
- HR and rhythm, pulse quality, CRT
- anesthetic dept - jaw tone, palpebral reflex, response to surgery)
- check oxygen supply, machine, flow meter rate, and machine integrity
- check ETT for kinks or obstruction
- check ETCO2 and waveform quality
- check quality of spontaneous ventilation
- ventilate by hand and assess compliance
What 4 actions should be considered in patients experiencing hypoventilation?
- assisted or mechanical ventilation
- decreasing anesthetic depth
- reducing respiratory depressant drugs
- reversal
What 5 things should be ruled out in patients experiencing hypoventilation?
- airway obstruction
- asthma or chronic airway disease
- excessive dead space (pulmonary or equipment)
- external compression of chest impending excursion (tight chest wrap)
- increased RR = high inspired CO2
What anesthetic depth is associated with hypoventilation?
too deep or paralyzed
What is ventilation defined by? Why is ETCO2 commonly lower than arterial CO2?
CO2 levels
physiological dead space
What is the primary motivator for breathing? How does anesthetics affect this?
arterial CO2
depress ventilatory response to CO2 at sub-anesthetic doses
What 3 things does the measurement of ETCO2 depend on?
- production of CO2 - metabolism
- delivery of CO2 to lungs - CO
- removal of CO2 - ventilation