Treating Surgical GI Dz Flashcards
What is the most common surgical approach taken for equine abdominal exploratories?
(Ventral midline in dorsal recumbency)
Why is it difficult to evacuate the stomach during surgery, resulting in horses needing to be woken up and treated medically?
(Bc you cannot exteriorize the stomach which means you cannot pack it off well enough from the abdominal cavity to prevent leakage and horses just need to look at a picture of a septic abdomen and they’ll get one)
What is the typical signalment and presentation associated with an ascarid impaction?
(Foals and weanlings 2-24 months in age, will appear unthrifty, impacting usually occurs after anthelmintic treatment so a hx of recent deworming)
Why is the prognosis for ascarid impactions guarded?
(Bc you have two options, milk the ascarids into the cecum which can cause bruising of the serosa leading to enteritis, adhesions formation, abscess formation or you can perform an R&A which can lead to contraction of the bowel lumen since the impaction is typically in the small intestines which already has a small diameter)
Where do non-strangulating lesions of the equine small intestines most commonly occur?
(Ileum)
What do you expect to see on ultrasound when there is an ileal impaction?
(Distended small intestines)
What is used in exploratory surgeries to decrease the chance of adhesions from irritation of the serosal surface?
(Carboxymethylcellulose aka belly jelly)
(T/F) Ileal impactions can be treated medically at first and then surgically as needed depending on the response to medical treatment.
(T)
Compare and contrast type 1 and 2 cecal impaction.
(Type 1 is associated with normal function of the cecum that fills with firm ingesta that eventually causes an impaction, type 2 is associated with cecal dysfunction that results in decreased to no cecal outflow into the right ventral colon)
Why is type 1 cecal impaction called a “silent killer”?
(Bc the horses cecum fills from the apex to the base (bc the apex is ventral and that’s how gravity works), horse will still be eating and defecating until the cecum fills up enough to block flow into the right ventral colon, cecum is usually very full and close to rupture at that time → if cecum ruptures it is a death sentence)
How do you assess if medical treatment of a cecal impaction is successful?
(Serial rectal examinations to palpate the cecum and feel if the impaction is changing at all)
Compare and contrast the surgical treatments for type 1 vs type 2 cecal impactions.
(Type 1 just needs a typhlotomy to evacuate the cecal contents, type 2 will be a typhlotomy with the addition of either a partial bypass or a cecocolic anastomosis)
Where do large colon impactions occur most commonly? Two answers.
(In the left ventral colon at the entrance to the pelvic flexure OR in the right dorsal colon at the entrance to the transverse colon)
How are large colon impactions typically diagnosed?
(Rectal palpation)
(T/F) Medical management of large colon impactions is typically successful.
(T, entails IV fluids +/- oral fluids, laxatives, and analgesics, if unresponsive → surgery)
Which portion of the large colon is the best option for an enterotomy and why?
(Pelvic flexure and because it can be removed from the abdominal cavity the most i.e. best approach for keep crap from getting into the abdominal cavity to cause septic peritonitis and adhesions)