Trans 012 Disturbances In Vision Flashcards

1
Q

Where does Retinal detachment occur?

A

Detachment occurs in the subretinal space (between RPE layer and rods & cones

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2
Q

Types of Retinal detachment

A

Non rhegmatogenous: fraction and exudative

Rhegmatogenous.

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3
Q

What type of Retinal detachment

 There is a leak in the blood vessels
• Breakdown of integrity of the vascular system of choroid and
/or retina
o Eclampsia&pre-eclampsia
o Tumorsofchoroidandretina
o Intraocular inflammation: choroidal effusion
▪ Vogt Koyang Harada Disease o Collagen Disease

A

Exudative detachment

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4
Q

 May humila sa retina kaya umangat
• Ischemic retinal disease causing fibroproliferative membrane
formation
o Diabetic Retinopathy
o Central Retinal Vein Occlusion (CRVO), Branch Retinal
Vein Occlusion (BRVO)
• Intraocular inflammation causing membrane formation
• Rhegmatogenous retinal detachment with proliferative
vietreoretinopathy (traction membranes)

A

TRACTION DETACHMENT

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5
Q

What retinal detachment has this symptom Symptoms:
o Blurry wavy vision, visual fields cuts, floaters and flashes
preceding blurry vision, NO PAIN, absence of symptoms is possible

• Pathophysiology
o Presenceofretinalbreak

o Theremaybevitreoustractiontotheretinalbreak
o Accumulation of fluid between RPE and photoreceptors

A

Take note: Pag nasa taas ang butas, retinal detachment is faster rhegmatogenous Retinal detachment

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6
Q

StageI:mildtomoderatearteriolarnarrowingorsclerosis
o Stage II: Moderate to marked arteriolar narrowing, with
focal or generalized narrowing, exaggerated light reflex,
and AV crossing changes
o Stage III: the above PLUS cottonwool spots, hard
exudates, retinal hemorrhages, extensive microvascular
changes, retinal edema
o StageIV:theabovePLUSdiscedema

What staging?

A

Keith Wagener Barker Staging of Hypertensive Retinopathy

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7
Q

W’hat stage in
Barker staging has

AV crossing changes

A

Stage 2

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8
Q

W’hat stage in

Barker staging has Cotton wool spots

A

Stage 3

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9
Q

W’hat stage in

Barker staging has disk edema

A

Stage 4

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10
Q

Av nicking versus venous elevation versus venue deviation

A

If an artery which is a thick muscular coat crosses a vein plus the effect of hypertension the changes in the wall will be harder. It will cause narrowing of the tips of the vein “pencil point” AV nicking
 The vein cross to the hard artery, the vein will be elevated. Venous elevation
 If the artery cross the vein it might delineate the path of the vein. Venous deviation

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11
Q

Deflection of retinal vein as it crosses the

arteriole.

A

Salus’ sign

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12
Q

Tapering of the retinal vein on either side

of the AV crossing.

A

Gunn’s sign

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13
Q

BankingoftheretinalveindistaltotheAV

crossing.

A

Bonnet’s sign

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14
Q

Causes
o Hypertension, arteriosclerosis, diabetes mellitus,
inflammation, collagen disease, hyperviscosity
syndromes
• Symptoms/signs
o Sudden painless blurring of vision, afferent pupillary defects

what disease?

A

Central Retinal vein occlusion

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15
Q

Treatment of the central Retinal vein occlusion if there is swelling of the macula

A

if there is swelling of the macula we inject

Anti-VEGF(vascular endothelial growth factor) medication

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16
Q

Causes:
o Emboli, thrombi, hemorrhage under an atherosclerotic
plaque, inflammation, spasm, dissecting carotid artery aneurysm, hypotension, hypertensive arterial necrosis, prolonged pressure on the globe
• One of the only 2 TRUE ophthalmologic emergencies!  The other one is chemical burns of the cornea
• Treatment:
o Loweringeyepressurestat,toimproveocularbloodflow  How to be resolve within 5 minutes

 Can also have a branch occlusion

A

CENTRAL RETINAL ARTERY OCCLUSION

17
Q

Risk factors of having diabetic retinopathy

A
Risk Factors
o DurationofDiabetes
o Glycemic control
o Age
o Type of diabetes
o Family Hx of DM
o Blood pressure
o Serum lipids
o Nutritional Factors/Obesity o Clotting Factors
o Renal Disease
18
Q

Types of diabetic retinopathy

A

Non proliterative and proliterative

19
Q

Mild, Moderate, Severe, Very Severe microaneurysms, retinal hemorrhages and hard and soft exudates, venous beading, IRMAs

Symptoms:
o Variablevisualdeficits/floaters
o Normal20/20visionpossible

A

Non proliterative

20
Q

Early, High Risk
o Retinal and disc neovascularization (NVE & NVD),
vitreous and pre-retinal hemorrhages, fibrovascular membranes

A

Proliferative Dr

21
Q

If there is macular

Edema, will there be loss of vision?

22
Q

induces regression of new vessels and reduces the
incidence of severe visual loss from proliferative diabetic
retinopathy by 50%. Several thousand regularly spaced laser burns are
applied throughout the retina outside the vascular arcades to reduce the angiogenic stimulus from ischemic areas

A

Pan-retinal photo-coagulation (prp)

23
Q

Treatment for macular edema

A

• Intravitrel anti-VEGF injections

24
Q

Reason for proliferation in diabetic retinopathy

A

Lack of oxygen

25
The most common hereditary retinal dystrophy
RETINITIS PIGMENTOSA
26
``` Clinical Manifestations • Nyctalopia or night blindness • Problems with peripheral vision • Deteriorating vision • Prolonged dark adaptation • Contracted visual fields • Abnormal ERG (Electroretinogram) ``` what disease?
RETINITIS PIGMENTOSA
27
Classic triad of retinitis pigmentosa
classic triad of findings comprises of bone-spicule retinal pigmentation, arteriolar attenuation, and ‘waxy’ disc pallor.
28
most common malignancy in childhood
Retinoblastoma | 12-18 months to 5 years old
29
2 types retinoblastoma
hereditary and sporadic
30
Most common presenting sign retinoblastoma
leukocoria
31
abnormality noted in the “retinoblastoma gene” at the q14 | locus
Ch 13
32
The most accepted treatment now for retinoblastoma
chemotherapy and debulking chemotherapy.
33
Innermost layer of the choroid
Bruch'S membrane
34
occlusive microvasculopathy characterized by multiple retinal white areas around the optic nerve head and fovea with paravascular clearing which may be associated with intraretinal hemorrhages.
PURSTCHER’S RETINOPATHY
35
It involves the choroid, Bruch membrane and retinal pigment epithelium  Direct ruptures are located anteriorly at the site of impact and run parallel with the ora serrata
Choroidal rupture