Training adaptations to strength/power - muscle 3 Flashcards

1
Q

Define muscle hyperplasia

A

Longitudinal splitting and lateral budding been seen.

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2
Q

Powerlifters have reported to have more muscle fibres. TRUE or FALSE?

A

TRUE

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3
Q

What change/feature from hyperplasia in the single muscle fibre is seen with a biopsy?

A

“Feathering”

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4
Q

In humans, is hyperplasia or hypertrophy more important?

A

Hypertrophy

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5
Q

Skeletal muscles are dynamic tissues whos cells do not remain as a fixed population throughout life. TRUE or FALSE

A

TRUE

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6
Q

How do muscles alter their phenotypic profile?

A

Undergo regeneration and remodeling to diverse functional demands.

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7
Q

What do satellite cells do?

A

Donate material for the change to nucleus, so that new tissue has more e.g strength.

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8
Q

Activation of muscle via specific types and intensities of long-term use stimulates what type of cell to proliferate and differentiate?

A

Dormant Myogenic stem cells

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9
Q

List the fast twitch fibre’s in order from highest force rating to lowest.

A

IIx, IIax, IIa, IIac, IIc.

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10
Q

List the slow twitch fibre’s in order from highest force rating to lowest (lowest endurance to highest endurance)

A

Ic, I.

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11
Q

What muscle fibre transition happens with resistance training?

A

Transition from IIx to IIa - More force to be produced over time.

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12
Q

What is a positive and a negative of type IIx fibre?

A

Positive = stronger. Negative = High fatigability renders less useful, and recruited last.

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13
Q

TRUE or FALSE? Detraining causes a pattern back to IIx fibres.

A

TRUE

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14
Q

Is there evidence of transitions between types I and II?

A

NO

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15
Q

What determines a muscles functional capacity?

A

The ratio of fibre types in the muscle.

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16
Q

What athletes have a larger % of type 1 fibres?

A

Endurance athletes

17
Q

What athletes have a larger % of type II fibres?

A

Strength/power athletes

18
Q

What muscular adaptations do you see in the first 6 weeks of resistance training?

A

Increased:

  • myofibrillar volume
  • cytoplasmic density
  • sarcoplasmic reticulum and t-tubule density
  • sodium/potassium ATPase activity.
19
Q

What other muscular adaptation is seen from sprint training?

A

enhances calcium release

20
Q

What happens to the mitochondrial density as an adaptation to training?

21
Q

Why do we see a decreased capillary density with heavy training?

A

Due to muscle hypertrophy. Same amount but being spread amongst greater CSA.

22
Q

What sort of training will increase capillary density?

A

High volume

23
Q

What happens to the buffering capacity, the high energy phosphate stores and muscle glycogen as an adaptation to resistance training?

A

All increase.

24
Q

TRUE or FALSE? a muscular adaptation includes changes in muscle substrate content and enzyme activity.

25
By how much does resistance training increase pennation angle of the muscle?
2-5 degrees
26
Why does the pennation angle increase?
hypertrophy causes increased CSA and thickens fibre.
27
If angle increases too much this may limit force production, why is this?
Fibres are not pulling in line with tendon. - hence extreme hypertrophy may actually limit force.
28
Which enzymes are enhanced from resistance training?
- Adenylate Kinase (2ATP from 2ADP) - Creatine Kinase - Muscle glycogen stores - PFK
29
TRUE or False? Heavy resistance training increases ATP-PC enzymes?
FALSE - it does not.
30
Why are ATP-PC enzymes not stimulatesd?
Very short duration sets (<6s).
31
What happens to the concentration of enzymes per unit muscle due to hypertrophy?
Reduces - watered down.
32
Why are aerobic enzymes needed in resistance exercise?
For recovery and strength endurance.
33
What sort of training increases aerobic enzymes?
High volume, short rest periods and moderate intensity.