Training adaptations to strength/power - muscle 3 Flashcards

1
Q

Define muscle hyperplasia

A

Longitudinal splitting and lateral budding been seen.

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2
Q

Powerlifters have reported to have more muscle fibres. TRUE or FALSE?

A

TRUE

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3
Q

What change/feature from hyperplasia in the single muscle fibre is seen with a biopsy?

A

“Feathering”

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4
Q

In humans, is hyperplasia or hypertrophy more important?

A

Hypertrophy

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5
Q

Skeletal muscles are dynamic tissues whos cells do not remain as a fixed population throughout life. TRUE or FALSE

A

TRUE

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6
Q

How do muscles alter their phenotypic profile?

A

Undergo regeneration and remodeling to diverse functional demands.

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7
Q

What do satellite cells do?

A

Donate material for the change to nucleus, so that new tissue has more e.g strength.

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8
Q

Activation of muscle via specific types and intensities of long-term use stimulates what type of cell to proliferate and differentiate?

A

Dormant Myogenic stem cells

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9
Q

List the fast twitch fibre’s in order from highest force rating to lowest.

A

IIx, IIax, IIa, IIac, IIc.

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10
Q

List the slow twitch fibre’s in order from highest force rating to lowest (lowest endurance to highest endurance)

A

Ic, I.

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11
Q

What muscle fibre transition happens with resistance training?

A

Transition from IIx to IIa - More force to be produced over time.

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12
Q

What is a positive and a negative of type IIx fibre?

A

Positive = stronger. Negative = High fatigability renders less useful, and recruited last.

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13
Q

TRUE or FALSE? Detraining causes a pattern back to IIx fibres.

A

TRUE

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14
Q

Is there evidence of transitions between types I and II?

A

NO

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15
Q

What determines a muscles functional capacity?

A

The ratio of fibre types in the muscle.

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16
Q

What athletes have a larger % of type 1 fibres?

A

Endurance athletes

17
Q

What athletes have a larger % of type II fibres?

A

Strength/power athletes

18
Q

What muscular adaptations do you see in the first 6 weeks of resistance training?

A

Increased:

  • myofibrillar volume
  • cytoplasmic density
  • sarcoplasmic reticulum and t-tubule density
  • sodium/potassium ATPase activity.
19
Q

What other muscular adaptation is seen from sprint training?

A

enhances calcium release

20
Q

What happens to the mitochondrial density as an adaptation to training?

A

Reduces

21
Q

Why do we see a decreased capillary density with heavy training?

A

Due to muscle hypertrophy. Same amount but being spread amongst greater CSA.

22
Q

What sort of training will increase capillary density?

A

High volume

23
Q

What happens to the buffering capacity, the high energy phosphate stores and muscle glycogen as an adaptation to resistance training?

A

All increase.

24
Q

TRUE or FALSE? a muscular adaptation includes changes in muscle substrate content and enzyme activity.

A

TRUE

25
Q

By how much does resistance training increase pennation angle of the muscle?

A

2-5 degrees

26
Q

Why does the pennation angle increase?

A

hypertrophy causes increased CSA and thickens fibre.

27
Q

If angle increases too much this may limit force production, why is this?

A

Fibres are not pulling in line with tendon. - hence extreme hypertrophy may actually limit force.

28
Q

Which enzymes are enhanced from resistance training?

A
  • Adenylate Kinase (2ATP from 2ADP)
  • Creatine Kinase
  • Muscle glycogen stores
  • PFK
29
Q

TRUE or False? Heavy resistance training increases ATP-PC enzymes?

A

FALSE - it does not.

30
Q

Why are ATP-PC enzymes not stimulatesd?

A

Very short duration sets (<6s).

31
Q

What happens to the concentration of enzymes per unit muscle due to hypertrophy?

A

Reduces - watered down.

32
Q

Why are aerobic enzymes needed in resistance exercise?

A

For recovery and strength endurance.

33
Q

What sort of training increases aerobic enzymes?

A

High volume, short rest periods and moderate intensity.