Topic 7B : Exercise Flashcards

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1
Q

state the name and location of the two nodes involved in heart contraction

A

sinoatrial node : within the wall of the right atrium

atrioventricular node : near lower end of right atrium in the wall that separates the two atria

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2
Q

cardiac muscle as myogenic

A

it can contract and relax without receiving signals from neuroens

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3
Q

explain how heartbeats are initiated and coordinated

A

SAN sets the rhythm of the heartbeat by sending out regular waves of electrical activity to the atrial walls causing both atria to contract at the same time.

A band of non conducting collagen tissues prevents the waves of electrical impulses being passed directly from the atria to the ventricles so the eaves are transferred from SAN to AVN instead.

AVN passes waves of electrical activity to the bundle of his, but there is a slight delay before AVN reacts to make sure ventricles contract after the atria have emptied.

the bundle of his is a group of muscle fibres responsible for conducting the waves of electrical activity to the finer muscle fibres in the right and left ventricles walls called purkyne fibres.

they carry out the waves of electrical activity into muscular walls of the right and left ventricles causing them to contract simultaneously from the bottom up.

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4
Q

electrocardiograph

A

a machine that records the electrical activity of the heart

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5
Q

formula for cardiac output

A

stroke volume (V) x heart rate (R)

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6
Q

tachycardia

A

heartbeat too fast (120 beats per minute)

its okay during exercise but at rest it shows that the heart is not pumping blood efficiently.

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7
Q

bradycardia

A

heartbeat too slow (60 beats per minute) at rest

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8
Q

ectopic heartbeat

A

extra heartbeat caused by earlier contraction of atria or ventricles than in previous heartbeats

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9
Q

fibrillation

A

irregular heartbeat due to atria/ventricles completely loosing their rhythm and stop contracting properly.

it can result in anything from chest pain and fainting to lack of pulse and death.

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10
Q

what happens to your heart and breathing rate when you exercise

A

heart rate increases to deliver oxygen and glucose to the muscles faster and remove extra carbon dioxide produced by increased rate of respiration in the muscle cells.

breathing rate increases to obtain more oxygen and to get rid of more carbon dioxide.

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11
Q

what controls rate of breathing in medulla oblongata

A

two ventilation centres called inspiratory and the expiratory

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12
Q

how does the inspiratory centre control breathing rate

A

inspiratory centre in medulla oblongata sends nerve impulses to the intercostal and diaphragm muscles to make them contract.

this increases the volume of the lungs lowering the pressure in the lungs.

air enters the lungs due to pressure differences between lungs and outside.

as lungs inflate, stretch receptors in the lungs are stimulated, stretch receptors send nerve impulses back to medulla oblongata which inhibit the action of the inspiratory centre.

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13
Q

how does the expiratory centre control breathing rate

A

it sends impulses to the diaphragm and intercostal muscles to relax which causes lungs to deflate expelling air.

stretch receptors become inactive

inspiratory centre is not longer inhibited so cycle starts again.

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14
Q

how does exercise trigger an increase in breathing rate by decreasing blood pH

A

level of carbon dioxide in blood increases which decreases blood pH.

chemoreceptors in medulla oblongata, apetic bodies and carotid bodies are sensitive to changes in blood pH.

if chemoreceptors detect decrease in blood pH they send nerve impulses to medulla oblongata which sends more frequent nerve impulses to the intercostal muscles and diaphragm and this increases the rate and depth of breathing.

this causes gaseous exchange to speed up, carbon dioxide level drops and extra carbon dioxide is supplied for muscles and the pH returns to normal and breathing rate decreases.

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15
Q

aortic bodies

A

clusters of cells in the aorta

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16
Q

carotid bodies

A

clusters of cells in the carotid arteries

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17
Q

carotid artery

A

carries blood to brain

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18
Q

ventilation rate

A

volume of air breathed in or out in a period of time eg a minute

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19
Q

how does exercise affect ventilation rate

A

it increases because breathing rate and depth increase

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20
Q

baroreceptors

A

pressure receptors in the aortic and carotid bodies which are stimulated by high and low blood pressure.

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21
Q

what effect do neurotransmitters released from sympathetic neurones have on heart rate

A

they get the body ready for action and increase the heart rate during exercise

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22
Q

what effect do neurotransmitters released from parasympathetic neurones have on heart rate

A

they calm the body down and decrease the heart rate after exercise

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23
Q

stroke volume

A

volume of blood pumped by one ventricle each time it contracts

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24
Q

cardiac output

A

total volume of blood pumped by a ventricle every minute

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25
Q

how does the heart responds to high blood pressure

A

baroreceptors detect high blood pressure

impulses are send to cardiovascular control centre which sends impulses along parasympathetic neurones which secrete acetylcholine that would bind to receptors on the SAN.

the SAN fires impulses less frequently to slow heart rate and reduce blood pressure back to normal

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26
Q

how does the heart responds to low blood pressure

A

baroreceptors detect low blood pressure

impulses sent to cardiovascular control centre which sends impulses along sympathetic neurones which secrete noradrenaline that would bind on receptors of SAN.

the SAN fires impulses more frequently to increase heart rate and bring blood pressure back to normal.

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27
Q

how does the heart respond to high blood oxygen, low carbon dioxide or high pH levels

A

chemoreceptors detect chemical changes in the blood.

impulses are sent to the cardiovascular control centre which sends impulses along parasympathetic neurones which secrete acetylcholine that binds to receptors on SAN.

SAN fires impulses less frequently to decrease heart rate and return all of these to normal levels.

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28
Q

how does the heart respond to low blood oxygen, high carbon dioxide or low pH levels

A

chemoreceptors detect chemical changes in the blood.

impulses are sent to cardiovascular control centre which sends impulses along sympathetic neurones which secrete noradrenaline that binds to receptors on the SAN.

SAN fires impulses more frequently to increase heart rate and return these to normal levels.

29
Q

respiratory minute ventilation

A

the volume of gas breathed in or out in a minute

30
Q

respiratory minute ventilation formula

A

tidal volume x breathing rate

31
Q

oxygen consumption

A

volume of oxygen used by the body often expressed as rate

32
Q

tidal volume

A

the volume of air in each breath usually about 0.4dm

33
Q

what machine can be used to measure ventilation

A

spirometer

34
Q

spirometer

A

machine that gives readings of tidal volume and allow measurement of a person’s breathing rate, oxygen consumption and respiratory minute ventilation

35
Q

homeostasis

A

the maintenance of stable internal environment

36
Q

how does homeostatic systems use negative feedback to reverse a change

A

receptors, a communication system and effectors are involved

receptors detect when level is too high/low and the information is communicated via nervous or hormonal system to effectors.

effectors respond to counteract the change and bringing the level back to normal.

negative feedback keeps things around the normal level eg 0.5 above or below.

37
Q

positive feedback mechanism

A

positive feedback mechanism amplifies a change from the normal level.

effectors respond to further increase the level away from the normal level.

38
Q

when can positive feedback occur

A

when something needs to be activated quickly eg blood clot after injury or when a homeostatic system breaks down

39
Q

does positive feedback mechanism keep the internal environment of a body stable

A

no

40
Q

what are the different mechanisms that reduce body temperature

A

sweating

hairs lie flat

vasolidation

41
Q

what are the different mechanisms that increase body temperature

A

shivering

much less swear

hair stand up

vasoconstriction

hormones

42
Q

how does sweating reduce body temperature

A

more sweat is secreted from sweat glands when the body is too hot.

the water in sweat evaporates from the surface of the skin and takes heat from the body making the skin cool

43
Q

how does hair lying flat reduce body temperature

A

hair provides insulation by trapping air (air is poor conductor of heat).

when its too hot, erector pili muscles relax so the hairs lie flat.

less air is trapped, so the skin is less insulated and heat can be lost more easily

44
Q

how can vasodilation reduce body temperature

A

when its hot, arterioles near the surface of skin dilate.

more blood flows through the capillaries in the surface layers of dermis so more heat is lost from the skin by radiation and the temperature is lowered.

45
Q

how can shivering increase body temperature

A

when its cold, muscles contract in spasms which makes the body shiver and more heat is produced from increased respiration.

46
Q

how can less sweat increase body temperature

A

less sweat is secreted from sweat glands when its cold so amount of heat loss is reduced.

47
Q

how does hair standing up increase body temperature

A

erector pili muscles contract when its cold making the hairs stand up which traps more air and so prevents heat loss.

48
Q

how can hormones increase body temperature

A

the body releases adrenaline and thyroxine which increase metabolism and so more heat is produced.

49
Q

how can vasoconstriction increase body temperature

A

when too cold, arterioles near the surface of the skin contract so less blood flows through the capillaries in the surface layers of dermis which reduced heat loss.

50
Q

which part of the brain controls body temperature

A

hypothalamus

51
Q

explain how does hypothalamus controls body temperature

A

it receives information about temperature from thermoreceptors via sensory neurones

hypothalamus sends impulses along motor neurones to effectors

effectors respond via any of the ways mentioned previously to bring back the body temperature to normal.

52
Q

thermoregulation

A

control of body temperature

53
Q

name two ways hormones can affect the activity of transcription factors

A

by working inside the cell

by working inside the cell membrane

54
Q

explain how peptide hormones affect the activity of transcription factors

A

hormone attaches to receptor in the cell membrane causing functional second messenger to activate enzymes or transcription factors

55
Q

explain how steroid hormones affect the activity of transcription factors

A

hormones bind to receptor inside the cell and hormone-receptor complex acts as a transcription factor switching enzyme synthesis on or off

56
Q

how can transcription be prevented

A

no transcription factor synthesised

transcription factor remains inactive (wrong shape)

repressor molecule binds to promoter region

repressor molecule binds to transcription factor

57
Q

transcriptional factor

A

regulatory protein that stimulates a gene before transcription

it contains sites that can bind to a specific region of the DNA

58
Q

pre-initiation complex

A

RNA polymerase and transcription factor combine in order to initiate transcription

59
Q

keyhole surgery

A

way of doing a surgery without making a large incision (cut) in the skin

60
Q

explain how keyhole surgery works

A

surgeons make a small incision in the patient and insert a tint video camera and specialised medical instruments through it.

61
Q

advantages of keyhole surgery

A

patients loose less blood and have less scarring of the skin due to small incision

patients are in less pain and recover more quickly after because less damage is done

hospital stay is shorter making it easier for the patient to return to normal activities

62
Q

prosthesis

A

artificial device that replaces damaged or missing parts of body

they could replace whole limbs or parts of limbs

63
Q

how can damaged knee joints can be replaced by prosthetic joints

A

a metal device is inserted into knee to replace damaged cartilage and bone

knee joint and the ends of leg bones are replaced to provide smooth knee joint, cushioning in the new joint helps reduce impact on the knee

a knee joint replacement allows people with serious knee problems to move around and participate in low-impact sports

64
Q

anabolic steroids

A

increase strength, speed and stamina by increasing muscle size and allowing athletes to train harder as well as increase aggression.

65
Q

stimulants

A

these speed up reactions, reduce fatigue and increase aggression

66
Q

narcotic analgesics

A

reduce pain so injuries don’t affect performance

67
Q

arguments against using performance-enhancing drugs

A

some are illegal

competition becomes unfair if people take drugs as people gain an advantage by taking them not through hard work

side effects such as high blood pressure and heart problems

athletes may not be fully informed about health risks

68
Q

arguments for using performance-enhancing drugs

A

athletes have the right to make their own decision and if its worth risking for

different athletes have access to different training facilites so drug-free sport isn’t really fair anyway

athletes that want to compete at a higher level may only be able to by using performance-enhancing drugs