Tooth Eruption and Exfoliation Flashcards

1
Q

The primary and permanent dentitions develop
— as the jaws increase in size
from infancy to adolescence.

A

simultaneously

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2
Q

Jaws of the infant accommodate development of —

deciduous teeth

A

20

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3
Q

At 4-5 yrs of age, the jaws accommodate

A

20 erupted

teeth and 28 developing teeth

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4
Q

The mixed dentition stage, —yrs of age, involves
the concomitant exfoliation of deciduous teeth and
eruption of permanent teeth

A

8-12

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5
Q

Tooth eruption is defined as the

A

movement of a
tooth from its site of development within the
alveolar process to its functional position in
the oral cavity

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6
Q

Tooth eruption is divided into

three developmental stages:

A

 Pre-eruptive phase
 Pre-functional eruptive phase
 Functional phase (post-eruptive phase)

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7
Q

Pre-eruptive tooth movement results from: (3)

A

 Growth of jaws
 Growth of tooth buds
 Remodeling of the walls of the bony crypt
(e.g., mesial migration results when there is resorption
of the mesial wall with concomitant apposition of bone
on the distal wall)

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8
Q

Over time, the jaws increase in: (4)

A

 Length of jaw (anterior-posterior)
 Width of jaw (coronal plane)
 Alveolar ridge height
 Buccal-lingual width of the alveolus

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9
Q

Pre-eruptive Phase
Initially the deciduous teeth develop within the jaw and
have considerable room. However, the individual tooth
germs grow rapidly and due to jaw length, they become

A

crowded in the anterior region. As the jaws increase in
length, the crowding is alleviated by migration of teeth
distally which allows a more even distribution.

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10
Q

With increased jaw size, the developing teeth move (2)

A

outward (facially) and towards the oral cavity (upward or downward).

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11
Q

Because of jaw length, the permanent developing molars have
angled inclinations prior to and during eruption and achieve
vertical alignment only when

A

jaw length is sufficient to allow

it.

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12
Q

Root Growth

A

 Elongation of the roots in relation to stability of the

fundus of the socket

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13
Q

Vascular Pressure

A

 Increased hydrostatic pressures in the apical dental sac

or periodontal ligament

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14
Q

Selective Bone Deposition and Resorption

A

 Coronal bone resorption concomitant with bone apposition

in the fundus area

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15
Q

Pulpal Pressure

A

 Tissue pressure differential in the pulp compared to

the PDL

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16
Q

Periodontal and Gingival Fiber Ligaments

A

 Cells (myofibroblasts) exert traction on the tooth through

the collagen network and cell-to-cell contacts

17
Q

Of the numerous causes of tooth eruption, the

most frequently cited are (3)

A

root growth, alveolar
bone remodeling, and periodontal ligament
formation.

18
Q

Other factors that influence tooth eruption include: (2)

A

 Parathyroid hormone (influences mineralization and
resorption of roots)
 MMPs produced by fibroblasts, osteoclasts, macrophages

19
Q
Gubernacular canal (a.k.a. gubernacular
cord or eruptive pathway):
A

 A strand of connective tissue that contains
remnants of dental lamina epithelium
(derived from the successional lamina)

20
Q

The rate of eruption is not constant but best
described as a “burst of eruption” that
averages about

A

3 mm every 3 months

21
Q

Occlusal Surface Wear: (2)

A

 Oblique fiber groups of the PDL continually pull the tooth
into occlusion as enamel is abraded.
 Apical cementum deposition also serves as a
compensatory eruptive mechanism to continual occlusal
abrasion.

22
Q

Proximal Wear (Mesial Drift):

A

 Mesial inclination of teeth in full contact will yield an
anterior force vector and when coupled with the pull of the
transseptal fibers, results in mesial drift.

23
Q

Cell mediated tooth resorption involves the

“—”

A

odontoclast

24
Q

Derived from monocytes that exit capillaries to
become connective tissue macrophages which, in
turn,

A

fuse together to form a (syncytum) large
multinucleated giant cell that functions like an
osteoclast.

25
Q

Odontoclasts degrade both the

A

collagenous and
non-collagenous matrix of cementum and dentin
(lysosomal enzymes) and the hydroxyapatite
mineral phase (acids).

26
Q

Fibroclast

A

 Specialized fibroblast-like cells are thought to
destroy the collagen fibers of the PDL associated
with the resorbing tooth root.

27
Q

The finding of apoptotic cell death in the resorbing

PDL suggests that shedding of teeth is a

A

programmed event.

28
Q

The finding of apoptotic cell death in the resorbing
PDL suggests that shedding of teeth is a
programmed event.
• Support for this theory is found in studies of tooth
eruption in monozygotic twins, which indicates that

A

tooth
eruption and shedding is determined by genetic factors
(approximately 80% genetic).