Tone,Mobility,Force Generation Flashcards

1
Q

What are positive signs of UMN lesion?

A
  • Hyperreflexia
  • Spasticity
  • Pathological Reflexes
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2
Q

Negative signs of UMN lesion?

A
  • Paresis( v force generated)
  • Loss of fractionation
  • abnormal motor recruitment
  • Obligatory synergy
  • Decreased coordination
  • abnormal spatial/temporal movements.
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3
Q

A light tone in muscle AT REST

A

tone.

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4
Q

Decerebrate vs decorticate rigidity?

A

decerebrate=extension.

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5
Q

What are the interventions for hypertonia?

A

air casting
serial casting
splinting.

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6
Q

Complete absence of muscle tone?

when does it happen?

where is it most common?

A

Flaccidity/hypotonia

Immediately after CVA(Stage 1)

most common in UE

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7
Q

what does the length of flaccidity relate to?

A

Prognosis.

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8
Q

What are some effects of hypotonia?

A

shoulder sublet

genu recurvatum

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9
Q

What are interventions for hypotonia?

A

Estim
mirror training
Neurofacilitation.
positioning to prevent sublet

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10
Q

Velocity dependent response of muscle to Passive stretching

A

spasticity.

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11
Q

Spasticity resistance increases as ____

in what direction?

A

speed and rhythm of movement increase.

uni/bidirectional

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12
Q

a series of involuntary, rhythmic,muscular contractions and relaxations.

A

clonus.

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13
Q

What are possible reasons for clonus?

A

hyperactive stretch reflex
increased mn excitability/decreased inhinition
nerve signal delay
cletus?

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14
Q

What are hypotheses for spasticity?

A

decreased inhibition of alpha motor neuron**

or stimulation of hypersensitive fusimotor system.

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15
Q

What is modified ash worth tested in and what are 2 speeds?

A

supine

entire ROM slow then fast.

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16
Q

Grades for modified ash worth

A
0= normal tone
1=catch and release
1+ catch and resistance through half rom
2=increase in tone and resistance through most rom
3= passive movement difficult
4=rigid.
17
Q

What is reliability of ash worth scale?

A

good intrarater

bad inter-rater

18
Q

What are influences of spasticity?

A
Tonic labyrinthine change in positions
postural exertion/mental
pain/discomfort
infection/inflammation
handling
19
Q

What are complications of Spasticity?

A
  • dec strength/fx
  • inactivity
  • contracture/deformity
  • tissue breakdown
  • discomfort
  • impaired posture
20
Q

What are the effects of Spasticity on MS

A
  • trunk asymetry
  • inversion spasticity
  • finger deformity
  • heterotopic ossifications
21
Q

What are the advantages of spasticity?

A

assist with posture control/ADLs

gastroc stabilize knee and ham stabilize trunk

22
Q

What are inhibiting techniques of spasticity?

A

slow rocking

  • counter rotation
  • Deep tendon pressure
  • Prolonged stretch
  • positioning.
  • reflex inhibition position
  • jt mobs and ST
  • Casting
23
Q

2 types of casting?

A

inhibitive: restore normal.
Serail: long duration low load stretch.

24
Q

What are pharmacological interventions?

A

ORal meds

injectable meds/nerve block

25
Q

What are the oral meds given?

A
Baclofen
Diazepam
Dantrolene
Tizanidine
Clonidine
26
Q

what are injectable meds/nerve

A

Phenol

Botulinum toxin

27
Q

What are surgical interventions for Spasticity?

A

Selective Dorsal Rhizotomy
Intrathecal Baclofen Therapy
tendon lengthening

28
Q

What is selective dorsal Rhizotomy?

A

knocking out sensory info in spindle. Helps to grade amount of input

29
Q

What is intrathecal Baclofen?

A

Medication given directed to spinal cord. given at lower dose.

30
Q

abnormal coactivation of muscle groups

A

synergies.

happen together when proximal muscle group loaded

31
Q

UE flexion lesion?

A
elevation
retraction
abd
elbow flexion
sup
radial deviation
32
Q

LE extension?

A
hip ext
add
ir
knee ext
pf
inversion
33
Q

Interventions for synergy?

A

NDT
arrom/strengthen out of synergy
facilitate opposition synergy
load isometrics out of synergy.

34
Q

Why is there impaired force generation after stroke?

A
  • decreased motor units/rate of firing/recruitment
  • learned nonuse
  • atrophy/ type 2 fibers fire like type 1
  • Disordered pattern of activity
  • Passive restraints
  • increaased fatigue.
35
Q

Brunnstrom stages of recovery

A

1: flaccid
2: spasiticty/synergy emerge
3: spasticity is highest/moev in pattern
4: Most movement in synergy
5: more isolated movement
6: No spasticity/ coordination emerges
7: normal

36
Q

A patient with hemiplegia with full passive elbow ROM is unable to lift a book and place it on a table with his hemiplegic UE that is within arm’s reach. Patient is able to place the empty hemiplegic hand on the shelf easily. What is the MOST likely cause?

A

limited elbow ext. 2ndary to flexion synergy

37
Q

What is the rationale for administering the Modified Ashworth Scale with the patient in supine for all muscle groups?

A

to standardize influence of vestibular inputs on spasticity

38
Q

What PT technique would you use to decrease the influence of this structure on spasticity?

A

Tendon pressure

39
Q

What surgical technique would you use to decrease the influence of this structure on spasticity?

A

dorsal rhisotomy