TMJ Flashcards
What type of joint is the TMJ?
Ginglymoarthrodial joint with translational movement in the superior joint space and rotational movement in the inferior joint space
What is the capsular ligament?
-Also known as the joint capsule
-Surrounds the joint
-Attaches to the temporal bone and surrounds the condylar head/neck circumferentially
-Lined by synovium which provides nutrition and immunosurveillance and lubricates the joint
What are the other ligaments of the TMJ?
-Collateral ligaments
-Temporomandibular ligament
-Sphenomandibular ligament
-Stylomandibular ligaments
What is the articular disk of the TMJ?
-Fibrocartilage disc made of 3 zones (anterior, intermediate and posterior)
-Posterior to the disk is the retrodiscal tissues which are highly vascular and innervated
How is movement of the TMJ determined?
Muscles of mastication (masseter, lateral pterygoid, medial pterygoid, and temporalis) as well as inframandibular accessory muscles
What is the vascular supply to the TMJ?
-Branches of superficial temporal, maxillary, and masseteric arteries
What is the nerve supply of the TMJ?
-Branches of auriculotemporal
-Contributions from masseteric and posterior deep temporal nerve
What is myofascial pain dysfunction?
-Non-articular TMJ disorder
-Presents as a dull regional masticatory myalgia
-Worsens with function and can lead to decreased ROM
-Can involve muscles of mastication and any combo of supra/infra mandibular muscle groups
-Most common TMJ disorder
What are possible etiologies of myofascial pain dysfunction?
-Parafunctional habits (bruxism, nail biting, clenching, gum chewing)
-Life stressors
-Apertognathia or OJ greater than 6 mm
-Lack of posterior dentition leading to muscle hyperactivity
What are clinical manifestations of myofascial pain dysfunction?
-Jaw tenderness at muscles of mastication
-Wear facets
-Sore teeth
-Decreased ROM
-Buccal exostoses
-Pain at side of face, diffuse pain
What is degenerative joint disease?
-Chronic inflammatory arthritis withint he TMJ resulting in degradation of articular cartilage with remodeling of the subchondral bone
What is the etiology of osteoarthritis?
-Non-inflammatory degenerative joint disease
-Due to an imbalance between catabolic and anabolic processes
-Leads to expression of catabolic cytokines (TNF-a, IL-1, IL-6)
-This initiates liberation of collagenases and proteases that result in degradation of the articular cartilage
-OA can be preceded by internal derangement and trauma. Can also develop in patients that have had orthognathic surgery
What is the etiology of inflammatory arthritis?
-Joint destruction due to an inflammatory arthritic process
-Includes rheumatoid arthritis, juvenile rheumatoid arthritis, psoriatic arthritis, gout, pseudogout, ankylosing spondylitis, reactive arthritis
What is the treatment of degenerative joint disease?
-Depends on extent and the level of life disruption. May include medications, PT, or steroids, or disease modifying drugs
-For mild cases failing conservative treatments, consider arthrocentesis and arthroscopic procedures
-More advanced cases may require arthroplasty or TJR
What is the definition of internal derangement of the TMJ?
-Disorder of the TMJ in which the articular disk is in an abnormal position as it relates to the condyle and fossa when the teeth are in occlusion. Malposition of the disk may lead to pain, instability, decreased ROM, abnormal mobility
What are the etiologies of internal derangment?
-Trauma
-Joint laxity
-Parafunctional habits
-Altered joint lubrication system
-Anchored disk (disk adhesion)
-MPD
How is internal derangement diagnosed?
-Clinical exam: Decreased MIO, deviation, deflection, palpable clicks, crepitus. Preauricular pain
-Radiographic: MRI T1 and T2. Disk usually in anteromedial vector. May see osseous changes and abnormal contours
-Disk displacement with reduction: Pt opens mouth with accompanying click (disk is in correct position when open, once closed you hear another click and disk is again anterior)
-Disk displacement without reduction: Pt attempts to open but the condyle cannot pass over the posterior band of disk. Results in decreased opening, deflection t o ipsilateral side and decreased excursion
Describe Wilkes stage I.
Clinical: Painless clicking, no pain or locking.
Radiographic: ADD noted, disk contour normal w/o osseous changes.
Surgical: Normal disk noted and displaced anteriomedially
Describe Wilkes stage II.
Clinical: Occasional painful clicking with intermittent locking
Radiographic: ADD with reduction on opening. Mild disk deformity w/o osseous changes
Surgical: The disk appears thickened and displaced anteriomedially
Describe Wilkes stage III.
Clinical: Frequent painful clicking with severe limitation in ROM. Pt with joint tenderness
Radiographic: ADD w/o reduction. Moderate disk deformity, no osseous changes
Surgical: Disk is deformed and displaced anteromedially. Adhesions may be appreciated
Describe Wilkes stage IV.
Clinical: Restricted ROM with chronic pain and joint crepitus
Radiographic: ADD w/o reduction. Marked osseous changes
Surgical: Perforated disk with osseous changes of the condylar head/fossa
Describe Wilkes stage V
Clinical: Joint pain and crepitus
Radiographic: Disk displaced, marked deformity with severe osseous changes
Surgical: Disk is perforated. Severe osseous changes of the condylar head and fossa
What is the treatment for internal derangement?
-Conservative treatment first line
-Intra-articular injections with local anesthetic/steroid mixture
-Those unresponsive would benefit from arthrocentesis w/ or w/o arthroscopy, arthroplasty with repositioning, or meniscectomy w/ or w/o graft/replacement or modified condylotomy
-Post-op management: Physical therapy/ROM exercises
What are extra-articular causes of hypomobility (pseudo-ankylosis)?
-Muscle fibrosis secondary to radiation, myofascial pain, tumors, infection, hysteric trismus, myositis ossificans, fractures of the condyle, zygomatic arch, coronoid process