Dentoalveolar Flashcards

1
Q

What is the classification system of third molars?

A

Pell and Gregory:
A-C (relation to second molar), 1-3 (relation to ascending ramus)
A: Third molar occlusal plan in line with 2nd molar
B: Third molar between occlusal plane and cervical junction
C: Below Cervical jntion
1: MD diameter anterior to ramus
2: Half lf crown covered by ramus
3: Tooth completely in ramus

Winter’s:
Based on angle between occlusal plane and longintudinal axis of third molar
Inverted: <0, very rare
Horizontal: 0-30 (10%)
Mesioangular: 31-60 (most common 45%)
Vertical: 61-90 (40%)
Distoangular: >90 (5%)

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2
Q

What are the indications of third molar removal?

A

Pericoronitis (most common reason)
Ortho, pericoronal pathology, caries, fracture, unexplained pain (5% removal helps), overlying prosthesis, periodontal disease

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3
Q

What is the anatomy of the IAN in relation to 3rd molar removal and the incidence of injury?

A

-1% injury incidence
-Generally nerve located buccal and apical

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4
Q

What is the anatomy of the lingual nerve in relation to 3rd molar removal and incidence of injury?

A

-0.6-2% incidence
-Average 2.8mm below crest and 2.5mm medial to lingual cortex
-4.6-21% of lingual nerves at or above crest of bone
-22% reported at lingual plate of bone
-Turns towards tongue at region of first/second molars

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5
Q

At what age does the risk of complications increase in 3rd molar removal?

A

25

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6
Q

What radiographic signs describe intamacy of IAN with roots of 3rd molar? (Rood’s Criteria)

A

-Darkening of root
-Deflection of root
-Narrowing of root
-Bifid root apex
-Diversion of canal
-Narrowing of canal
Interruption in white line of canal

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7
Q

What is alveolar osteitis?

A

-Incidence between 1-30% (subjective criteria)
-Seen at POD 3-7
-Theory: Fibrinolytic activity leads to break down of clot
-Risk factors: Tobacco smoke, increased age, pericoronitis, birth control, female gender, inexperienced surgeon, inadequate irrigation, increased medical co-morbidities

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8
Q

When can a root tip be left in place?

A

-Non-infected
-Small (<2mm)
-Risk of surgery outweighs benefit

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9
Q

What is the incidence of bleeding with 3rd molar surgery?

A

.2-5.8%
-Must rule out coagulopathies

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10
Q

How does gelfoam work?

A

-Absorbable gelatin spone
-Matrix for blood clot formation
-Gelatin made from purified porcine skin
-May cause excessive granuloma or fibrosis

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11
Q

How does Avitene work?

A

-Microfibrillar collagen
-Mechanically broken down bovine collagen
-Aggregates platelets onto fibrils and acts as a matrix for blood clot formation

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12
Q

How does HemCon/Chitoflex work?

A

-Chitosan dressing
-Polysaccharide from shellfish
-Positively charged to attract erythrocytes
-Acts as a scaffold for clotting
-Dissolves in 48h

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13
Q

How does thrombin work?

A

-Promotes clot formation through activated bovine prothrombin
-Activates factors IIA
-Acts as a serine protease converting fibrinogen to fibin

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14
Q

How does surgicel work?

A

-Oxidized regenerated methylcellulose
-Binds platelets
-Negative pH is bacteriostatic and precipitates fibrin
-More effective than gelatin sponge
-Aids in pressure hemostasis
-Causes prolonged healing,
-Can be neurotoxic (acidic environment)

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14
Q

How does a collaplug work?

A

-Cross-linked collagen
-Promotes platelet aggregation

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14
Q

How does a teabag help with hemostasis?

A

-Tanin serves as a vasoconstrictor

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15
Q

How does Amicar work (aminocaproic acid)?

A

-Stabilizes clot by inhibiting plasmin

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16
Q

How does TXA work (5% tranexamic acid)?

A

-Antifibrinolytic, inhibits conversion of plasminogen into plasmin

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17
Q

What is the management for displacement of a root into the sinus?

A

-Most commonly palatal root of maxillary 1st molar
-Take PA to verify position
-Attempt to suctioning into sinus to remove
-Pack sinus with xeroform gauze and pull out in one stroke
-Preform antral lavage
-Have patient block opposite nostril and blow nose
-Enlarge opening and explore

-fragments 3 mm or less that are not infected can be left in place (inform patient)

-Removal with Caldwell-Luc approach

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18
Q

How are OACs managed?

A

-3-6 mm: Place gel foam and close with figure-eight suture
->6mm: May require tension free primary closure, exicision of fistulous tract and inversion into the sinus. Consider buccal fat pad closure, buccal finger flap or tongue flap

-Sinus precautions 2 weeks, decongestants, antibiotics that cover sinus flora, no blowing nose

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19
Q

What is the management of displacing a root into the submandibular/sublingual space?

A

-Lingual cortex thins out in more posterior region
-Displacement often inferior to mylohyoid muscle

-First attempt to milk root back through cortical hole via manipulation
-Attempt at a lingual flap extended anterior to premolar with an incision to detach mylohyoid muscle
-Allow 6 weeks for fibrosis
-Get a CT scan to localize root
-May require transcutaneous approach via submandibular incision

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20
Q

What is the management for displacement of a root into the infratemporal space?

A

-Likely due to lack of retractor protection with excessive force/poor visualization
-Most likely lateral and inferior to pterygoid plate
-May attempt to manipulate tooth back manually
-Extend incision and retrieve with hemostat
-Allow 4-6 weeks to allow for fibrosis, obtain CT scan and use a spinal needle to identify, diessect along needle length
-Reported to preform a hemicoronal incision

-If no functional deficit and asymptomatic, may leave in place

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21
Q

What is the management for displacement of a root into the IAN canal?

A

-Retrieval attempts may lead to nerve damage
-Single attempt with suction should be attempted
-If root is not infected and no neurologic abnormality, consider leavign in place

-If sensory complication, must retrieve
-CT scan should be taken to ensure location
-Can approach by unroofing extraction site, lateral window intraoral or submandibular incision

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22
Q

What is the management for aspiration of a foreign object during dentoalveolar procedure?

A

-Heimlich maneuver may be attempted
-If under GA, deepen level of sedation and attempt visualization and removal with Magills

-If no respiratory distress, likely ingested
-Obtain abdominal and CXR to rule out

-Assume aspiration, place on right side and in trendelenburg, watch for signs of hypoxia and respiratory distress. ER for removal

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23
Q

What is the incidence and typical patient demographics of impacted maxillary canines?

A

-2% incidence (0.4% mandibular canines)
-2:1 female ratio
-Canines normally erupt between 11 and 12 years of age
-Labial impaction due to arch length discrepancies
-Palatal impaction seen with peg or missing laterals

-Genetic theory: Genetic disposition
-Guidance theory: Max canine erupts along lateral incisors, malformation or lack of lateral leads to failure of canine

24
Q

What are aspects of your clinical exam pertinent to impacted canines?

A

-Pay attention to bulging of area
-Evaluate gingival health and quality (thick vs thin, presence of gingivitis, quantify amount of keratinized tissue (may affect approach)
-Evaluate presence and position of lateral incisor
-Examine tonsils and adenoids (sedation considerations)
-Mobile teeth (aspiration risk)
-Ortho treatment (Where will chain be secured, is there adequate space for a canine ~7.5 mm)

25
Q

Describe your radiographic work-up of an impacted canine?

A

-CBCT

-Clark’s SLOB rule (same lingual, opposite buccal)

-Pan: If horizontal/larger and out of focus likely palatal. Labial often vertical

26
Q

What ortho considerations are needed pre-op prior to an expose and bond?

A

-Create room for canine before expose and bond
-stabilize teeth with full thickness passive wire to allow anchorage
-Need about 7.5 mm of space
-The more perpendicular the canine is to the lateral, more likely tooth needs to be extracted instead of E&B

27
Q

Describe the interceptive technique for impacted maxillary canine.

A

-Extract primary canine before age 11 if not palpable on buccal. 91% success. Success drops to 64% if crown is mesial to midline of lateral incisor

28
Q

Describe the apically positioned flap technique for impacted maxillary canine.

A

-For labial impaction (and not displaced mesial or distal)
-Maintains keratinized gingiva
-Use if less than 3 mm of keratinized gingiva is expected after open window technique
-Don’t use for canines high in alveolus (thick palatal bone can push canine buccally and create dehiscence. Use closed technique

-Flap is Mesial-distal width of tooth
-Remove bone over surgical margin and remove follicular remnants
-Reposition flap apically at cervical margin
-Acid etch (30 seconds with 30% phosphoric acid), irrigate thoroughly with water
-Know system you use (primer and bonding agent)
-I use OrthoSource cuspid bond primer and adhesive (one drop of primer, adhesive over gold mesh and cure on tooth x40 seconds)
-Suture keratinized tissue apically around CEJ of tooth

29
Q

What is the open exposure technique for impacted canines? (Window technique)

A

-Crown is uncovered and left exposed
-Ortho bracket may or may not be used
-Window of overlying gingiva is removed or reflected
-Tooth may spontaneously erupt or site can be packed with periodontal packing (left 2-3 weeks)

30
Q

What is the closed technique for expose of impacted canines?

A

-Used when teeth are not in position to allow for repositioning of flap after crown exposed
-For palatal impaction that is not close to the alveolar ridge

-Local, full thickness flap, expose tooth crown to CEJ
-Etch with 30% phosphoric acid 30s
-Ensure hemostasis
-Primer/bonding agent
-Orthosouce primer and adhesive, apply 1 drop of primer and spread, apply adhesive to gold chain and light cure 40s
-Test chain
-Secure chain to bracket
-Flap sutured back to place
-Ortho traction after 1 week of tissue healing

31
Q

How do you manage a situation in which an impacted tooth fails to erupt after an expose and bond?

A

-Re-explore area and check for ankylosis (percuss tooth and check for a high pitched sound)
-Luxate tooth may aid in mobility
-May consider segmental osteotomy or corticotomies

-Consider extraction of tooth (leave space for dental implant, premolar substitution, autotransplantation

32
Q

What are other complications of expose and bond?

A

-Resorption of tooth: Occurs from aggressive exposure, stop orthodontic movement and re-eval, consider extraction

-Lack of attached gingiva: Occurs from poor quality of gingival mucosa, over-aggressive tissue removal and inappropriate treatment selection. May need a connective tissue graft

-Bracket detachment: Occurs from ankylosis, disruption in path of guidance, over-aggressive movement, poor bonding. Re-exposure of tooth, check for ankylosis. Consider reattachment (consider polish with pumice or diamond burr to aide in bonding. Do not use ligature!)

33
Q

What can happen if you remove bone past CEJ during expose and bond?

A

-Root resorption
-Ankylosis
-Perio disease

34
Q

What is the correct terminology in describing nerve anatomy?

A

-Endoneurium: Connective tissue sheath that surrounds group of fibers forming a fascicle

Perineurium: Connective tissue surround a bunch of fascicles within a nerve

Epineurium: Outermost layer of a peripheral nerve, surrounding multiple fascicles and blood vessels

35
Q

What is the incidence of IAN and lingual nerve injury from 3rd molar surgery?

A

-Lingual nerve: 0.04-0.6%
-IAN: 0.1-1%

-IAN more likely to self resolve
-33% chance of persistent nerve impairment of IAN after 1 year s/p BSSO

36
Q

What is the Seddon classification?

A

Based on histology
-Neuropraxias: Focal segmental demyelination without disruption of axon continuity
-Axonotmesis: Anatomical interruption of the axon with either none or partial interruption of connective tissue framework
-Neurotmesis: Complete transection of a nerve

37
Q

What is Sunderland’s classification of nerve injuries?

A

Type 1: Neuropraxia. Temperary disturbance in nerve conduction. No treatment. Typically recovery spontaneously in up to 3 months

Type 2: Axonotmesis. Loss of continuity. Endoneurium and perineurium intact. Surgery only if foreign body. Possible spontaneous recovery at 2-4 months

Type 3: Axonotmesis. Endoneurium ad axonal loss of continuity, perineurium. Severe crush or chemical injury. Treat with microsurgery if no improvement for 3-4 months. Possible partial spontaneous recovery

Type 4: Axonotmesis. Loss of endoneurium, perineurium. Epineurium intact. Extreme crush. Microsurgery if no improvement 3-4 months

Type 5: Neurotmesis. Complete transection. Requires microsurgery

Type 6: Neuroma. Requires microsurgery

38
Q

What is the term for when you have pain from a non-painful stimulus?

A

Allodynia

39
Q

What is the definition of anesthesia?

A

Absence of any sensation

40
Q

What is anesthesia dolorosa?

A

Deafferentation pain is pain felt in an area, which is completely anesthetic to touch

41
Q

What is hyperalgesia?

A

Increased response to stimulus that is normally painful

42
Q

What is hyperpathia?

A

-Prolonged pain following a repetitive noxious stimulus that lingers beyond expected uration

43
Q

What is hypoalgesia?

A

Diminished response to a normally painful stimulus

44
Q

What is hypoesthesia?

A

Decreased sensitivity to stimulation

45
Q

What is the definition of paresthesia?

A

Abnormal sensation (not unpleasent)

46
Q

What is tinel sign?

A

Tingling or ‘pins and needles’ sensation. Elicited by tapping on distribution of the nerve

47
Q

What is Wallerian degeneration?

A

Distal degeneration of the axon and its myelin sheath after injury, may result from passive wasting of the distal axonal fragment due to lack of nutrient supply

48
Q

How is a nerve injury graded/evaluated?

A

Modified Medical Research Council Scale. S0-S4
S0: No sensation
S1: Deep cutaneous pain
S2: Some superficial pain and touch sensation
S2+: Superficial pain and touch plus hyperesthesia
S3: Superficial pain and touch w/o hyperesthesia, 2 point>15 mm
S3+: Same as S3, 2 point 7-15 mm
S4: 2 point 2-6 mm

49
Q

What are the average two-point discrimination distances?

A

IAN generally 4 mm
Lingual generally 3 mm
Anything over 6.5 mm considered abnormal

Lower lip mucosa: 3.5 mm (upper normal 6.5)
Lower lip skin: 5 mm (upper normal 9)
Chin: 9 mm (upper normal 18)
Tongue tip: 3 mm (upper normal 4.5)
Tongue dorsum: 5 mm (upper normal 12

50
Q

What is the ideal timeframe for intervention of a nerve injury?

A

3 months critical for intervention. At 12 months, distal nerve tissue too damaged and recovery unlikely.

Monitor every 2-4 weeks, at 3 months generally do not see further improvement

51
Q

Describe your neurosensory testing.

A

Level A: Spatiotemporal perception. Aa & Ab fibers.
-Cotton swab brush strokes.
-2-point discrimination (4 mm normal, 6.5+ abnormal)
-Stimulus localization with cotton stick (1-3 mm off considered normal)
-If level A normal, no need to do further testing

Level B: Static Light touch
-A-b fibers
-Touch skin with end of cotton tip applicator (should be able to feel w/o skin indentation), use Semmes-Weinstein monofilaments

Level C: Nociception
-A-delta and C fibers
-27 gauge needle without indentation of skin should evoke pain response
-Can delineate A-delta with heated gutta percha vs cold testing.

Diagnostic Nerve blocks: If patient complains of altered sensation/pain, a block can be given to establish if pain is from peripheral nerve vs central. If central, very unlikely to help with surgery

52
Q

What imaging can be completed prior to nerve injury repair?

A

-CBCT or panorex can help give insight on foreign body, retained root, hardware or bony damage.

Does not give insight on nerve integrity

53
Q

What are indications for nerve repair

A

-Observed nerve transection
-Complete post-op anesthesia
-Persistent anesthesia x1 month w/o improvement
-Prescence or development of dysesthesia
-Paresthesia w/o improvement x3 months
-Foreign body in canal
-Pt unable to tolerate hypoesthesia

54
Q

Describe the steps of a nerve repair.

A

-External neurolysis (decompression): First step, exposing nerve from soft tisue bed without disrupting epineurium. May be only procedure if only mild disturbance and no neuroma

-Neuroma excision: Resect 3 mm proximal and distal. Examine fascicles under magnification. Consider frozen sections

-Direct neurorrhaphy: 4-6 circumferential epineural sutures with 7.0-9.0 nylon sutures. Lingual nerve gap of 1 cm and IAN gap of 5 mm possible for direct repair. Minimal tension

-Nerve grafting: Need 25% longer graft than defect due to shrinkage. Sites include sural, greater auricular. May also use Processed allograft (axogen Avance, non-immunolgic alternative that provides scaffold)

-Entubation: Polyglycolic acid conduit that breaks down in 3 months and resorbed by 9 months. Collagen type I tubes best <10 mm

55
Q

What is the associated morbidity with a sural and greater auricular nerves?

A

-Sural: Anesthesia of heal and lateral foot, gait disturbance
-Greater auricular: Anesthesia of lateral neck, posterior mandible and earlobe. Small diameter may require cable graft

56
Q

What is the overall prognosis of nerve repairs?

A

-Overall success rate around 50%
-70% of painful neuromas have improvement
-All require sensory education after surgery
-Hypoesthetic nerve injury better prognosis than hyperesthetic injuries
-Delays >6 months have poorer outcomes

57
Q

What is a coronectomy?

A

Partial tooth removal leaving roots behind to prevent inadvertent IAN damage

58
Q

What are contraindications to coronectomy?

A

-Horizontal impaction with tooth along length of nerve, risk sectioning higher than complete removal
-Inability to access or remove all enamel layer
-Infection of roots
-Plan for distalization of 2nd molar
-Mobility of roots

59
Q

What is the technique for coronectomy?

A

-Removal of all enamel and root remnant 3 mm below alveolar crest of bone

-Post-op antibiotics and primary closure have little effect of success

60
Q

What is the prognosis s/p coronectomy?

A

-Roots migrate 30% of time and can usually be appreciate within first 3 months