Thompson Flashcards

1
Q

What happens when the kidneys crump

A
  • electrolyte imbalances (phos get high, potassium high, calcium gets LOW)
  • anemia
  • blood pressure irregularities
  • MEDS HANG AROUND
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2
Q

JNC 8 Recommendations

A
  • > 60 years = 150/90
  • < 60 years = 140/90
  • > 18 years with CKD = 140/90
  • > 18 with diabetes = 140/90
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3
Q

Risk factors of HTN

A
  • AGE
  • smoking
  • DM
  • high lipids
  • inactivity/weight
  • family history
  • race
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4
Q

secondary causes of HTN

A
  • sleep apnea
  • primary hyperaldosteronism (low K, HTN resistant)
    • DIAGNOSE WITH HIGH ALDOSTERONE/RENIN RATIO (there K will often be LOW)
    • Consider in patients with resistant HTN and low potassium
  • CRF
  • thyroid
  • renal vascular disease
  • pheochromocytoma
  • cushings or steroid therapy
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5
Q

when to suspect secondary causes

A
  • age extremes (very young or old)
  • severity
  • lack of family history
  • does not respont to tx
  • sleep apnea
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6
Q

Beta blockers

A
  • Cause dizzy/syncope, fatigue, LOW HR, impotence, makes working out difficult
  • very effective; always used in MI or CAD
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7
Q

ACE inhibitors

A
  • Side effects
    • increases K
    • Angioedema (asian/blacks are more likely)
    • cough
    • increase Creatinine
      • can increase 30% before stop or big w/u
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8
Q

HTN take away medications

A
  • WHites = ACE/ARB, thiazides
  • Blacks = aoid ACE
  • Diabetics/CRF = ACE
  • Post MI/CVA = beta blocker/ace
  • CHF = beta blocker + ACE, plus thiazise plus spiranolactone
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9
Q

HTN emergencies

A
  • Malignant Hypertension
  • Any increase in BP that results in ACUTE END ORGAN DAMAGE
    • encephalopathy, stroke, CHF, aortic dissection, MI, ARF
    • Nosebleed
  • STILL WANT TO LOWER THE BP SLOWLY
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10
Q

Suspect RAS???

A
  • Doesn’t respond to reatment
  • intermittent claudication
  • sudden worsening of HTN
  • image abnormality
  • ACE leads to worsening creatinine
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