Thompson Flashcards
1
Q
What happens when the kidneys crump
A
- electrolyte imbalances (phos get high, potassium high, calcium gets LOW)
- anemia
- blood pressure irregularities
- MEDS HANG AROUND
2
Q
JNC 8 Recommendations
A
- > 60 years = 150/90
- < 60 years = 140/90
- > 18 years with CKD = 140/90
- > 18 with diabetes = 140/90
3
Q
Risk factors of HTN
A
- AGE
- smoking
- DM
- high lipids
- inactivity/weight
- family history
- race
4
Q
secondary causes of HTN
A
- sleep apnea
- primary hyperaldosteronism (low K, HTN resistant)
- DIAGNOSE WITH HIGH ALDOSTERONE/RENIN RATIO (there K will often be LOW)
- Consider in patients with resistant HTN and low potassium
- CRF
- thyroid
- renal vascular disease
- pheochromocytoma
- cushings or steroid therapy
5
Q
when to suspect secondary causes
A
- age extremes (very young or old)
- severity
- lack of family history
- does not respont to tx
- sleep apnea
6
Q
Beta blockers
A
- Cause dizzy/syncope, fatigue, LOW HR, impotence, makes working out difficult
- very effective; always used in MI or CAD
7
Q
ACE inhibitors
A
- Side effects
- increases K
- Angioedema (asian/blacks are more likely)
- cough
- increase Creatinine
- can increase 30% before stop or big w/u
8
Q
HTN take away medications
A
- WHites = ACE/ARB, thiazides
- Blacks = aoid ACE
- Diabetics/CRF = ACE
- Post MI/CVA = beta blocker/ace
- CHF = beta blocker + ACE, plus thiazise plus spiranolactone
9
Q
HTN emergencies
A
- Malignant Hypertension
- Any increase in BP that results in ACUTE END ORGAN DAMAGE
- encephalopathy, stroke, CHF, aortic dissection, MI, ARF
- Nosebleed
- STILL WANT TO LOWER THE BP SLOWLY
10
Q
Suspect RAS???
A
- Doesn’t respond to reatment
- intermittent claudication
- sudden worsening of HTN
- image abnormality
- ACE leads to worsening creatinine