The Cardiovascular System Flashcards
Characteristics of Normal Sinus Rhythm
Normal P-wave
PR Interval between 0.12 - 0.20 seconds
QRS < 0.12 seconds
Rate: 60 - 100bpm
Regular in rhythm
Characteristics of Sinus Bradycardia
Normal P-wave
PR Interval between 0.12 - 0.20 seconds
QRS <0.12 seconds
Rate <60bpm
Regular in rhythm
Characteristics of Sinus Tachycardia
Normal P-wave
PR interval > .20 seconds
Rate > 100 bpm
Regular in rhythym
Causes of Sinus Tachycardia
caffeine, exercise, fever, pain, hypotension, volume depletion
Treatment for Sinus Tachycardia
fix the cause
First Degree Heart Block Characteristics
Normal P-wave
PR interval <0.12 seconds
Rate: 60 - 100bpm
Regularity
Prolonged impulse travel
Second Degree Heart Block (Mobitz type 1) Characteristics
Abnormal P-waves
Prolonged PR interval with drops
QRS < 0.12 seconds
Rate: 60 - 100bpm
regularity
Cause of Second Degree Heart Block
fix the cause; if asymptomatic, no treatment is required. If asymptomatic, pacing is required.
Second Degree Heart Block (Mobitz type 2) Characteristics
Abnormal P waves (1 p-wave per 2 QRS)
PR interval between 0.12 - 0.20
QRS < 0.12
Rate >100bpm
Regularity
Cause of Mobitz type 2 heart block
MI; Ischemia
Treatment of Mobitz Type 2 Heart Block
treat the cause; pacing
Third Degree Heart Block Characteristics
Normal P-wave (inconsistent)
PR interval varies
Rate <60bpm
irregular
Cause of Third Degree Heart Block
Myocardial damage; MI; heart valve disease; Rheumatic fever; sarcoidosis
Heart Block Rhyme
If the R is far from P, its a first degree
longer, longer, and drop, then you have a Wincheback (2nd degree, type 1). If some P’s dont get through, you have a Mobitz type 2. If P’s and Q’s dont agree, you have type three.
Longterm Treatment for Arrythmias
Pacemaker
Pacemaker Teaching
Do’s:
- keep pacemaker card on person
- bath and shower 48 hrs post-op
- safe to use appliances
- notify airport security
Don’ts:
- apply pressure over generator
- wear tight clothing
- get lead wires wet
- get an MRI
Atrial Fibrillation
arrhythmia characterized by funny P-waves
Causes of Atrial Fibrillation
HF, heart disease, MI, pericarditis, and CHF
Treatment for Atrial Fibrillation
Fix the cause
Cardioversion
Antiarrythmics
Amiodarone
Beta Blockers
Metoprolol
CCB’s (Diltiazem)
Atrial Flutter
irregular, saw-tooth like P-waves
Supraventricular Tachycardia Characteristics
Irregular P-waves
PR Interval is immeasurable
QRS < 0.12 seconds
Rate: 150 - 250 bpm
Regular
Causes of SVT
caffeine, HF, fatigue, hypoxia, altered pacemaker
Treatment of SVT
Fix the cause
Cardioversion
Adenosine
Ventricular Tachycardia
characterized by no atrial p-waves and a wide QRS complex with a regular rhythm.
Treatment for Ventricular Tachycardia
fix the cause
if there is a pulse, cardio version
No pulse, defibrillation
Epinephrine
Ventricular Fibrillation Characteristics
Fibrillation of the ventricles
irregular
usually no pulse
Cause Ventricular Fibrillation
MI; ischemia; hypoxia; acidosis; hypokalemia; hypotension
Treatment of Ventricular Fibrillation
Fix the cause
CPR
Defibrillation
Epinephrine
Asystole
Flat-lined; caused by hypothermia, v-fib, a-fib, acidosis, hypoxia, overdose
Treatment: CPR and epinephrine
Preload
amount of blood returning to the right side of the heart
Afterload
pressure against. which the left ventricle must pump to eject blood.
Compliance
how easily the heart muscle expands when filled with blood
contractility
strength of contraction of the heart muscle
Stroke Volume
Volume pumped out of ventricles with each contraction
Cardiac Output
important to tissue perfusion and end-organ function; CO = HR X SV
Signs and Symptoms of Poor Cardiac Output
decreased LOC; chest pain; weak peripheral pulses; SOB; crackles; rales; cool and clammy skin; mottled skin; decreased urine output.
Causes of decreased CO
bradycardia, arrhythmias, hypotension, MI, cardiac disease.
Causes of Increased CO
increased blood volume, tachycardia, drugs (ACE inhibitors, ARBs, Nitrates, Inotropes).
Enalapril
Pharm. Class: ACE inhibitors
Examples: Captopril, Enalapril, Lisinopril
Action: blocks conversion of angiotensin 1 and angiotensin 2, increases renin levels, and decreases aldosterone, leading to vasodilation.
Nursing Considerations: dry cough (discontinue the drug), monitor BP; contraindicated during pregnancy.
Losartan
Pharm. Class: ARB
Indication: HTN, DM, neuropathy, CHF
Action: inhibits vasoconstrictive properties of angiotensin II.
Nursing Considerations: monitor BP, fluid levels, renal/liver status; contraindicated in pregnancy.
Amlodipine
Pharm Class: CCBs
Indication: HTN, angina, A-fib, A-flutter
Action: blocks transport of calcium into muscles, inhibiting excitation and contraction, causing peripheral vasodilation.
Nursing Considerations: avoid grapefruit; blocks enzyme involved in metabolizing calcium channel blockers, causing their levels of increase; monitor bp (OH); can cause gingival hyperplasia (teach patients to use a soft toothbrush).
Propanolol
Pharm Class: antiarrythmic
Indication: HTN, angina, MI, cardiomyopathy, ETOH withdrawal, and anxiety.
Action: blocks beta 1 and beta 2 receptors, slowing the heart.
Nursing Considerations: do not discontinue abruptly; can mask signs of hypoglycemias (monitor BG); caution with asthma and COPD (may cause bronchospasm).
Adenosine
Pharm Class: Antiarrythmic
Indications: SVT
Action: Slows conduction through AV node, interrupting re-entry pathways through the AV node, restoring normal sinus rhythm.
Nursing Considerations: there will be a period of asystole after admin; warn client that I will feel like getting kicked in the chest; will flat line on monitor; rapid push or will not be effective; extreme caution with asthmatics.
Vasopressors
Increases BP and causes vasoconstriction
Drugs: norepinephrine, epinephrine, vasopressin, phenylphrine.
Normal Blood Pressure
120/80 mmHg
Pre-Hypertensive
120-129/80mmHg
Stage 1 HTN
> 130/>80 mmHg
Hypertensive Crisis
> 180/>120mmHg
Signs and Symptoms: vision changes, headaches, dizziness, epistaxis, SOB, angina
Risk Factors: stress, smoking, caffeine, high sodium intake, family history, African American descent, advanced age, obesity, hyperlipidemia, CAD
Complications: stoke, MI, RF, HF, vision loss.
Nursing Education for Hypertension
Medications:
- ACE inhibitors
- Beta Blockers
- CCBs
- Beta Blockers
Diet:
- DASH
- low sodium
-avoid caffeine and alcohol
- weight loss
- smoking cessation
- less sitting; more walking
Chronic Stable Angina
Chronic disease caused by narrowing of the arteries and plaque build up. There are periods of increased schema to the heart. Decreased blood flow to the heart leads to decreased O2 delivery, and schema of tissues. Ischemia causes chest pain.
Causes: venous and arterial dilation, decreasing after load, increasing CO.
Nursing Considerations: Nitroglycerine may be given S/L; 1 pill q 5 min x3 doses; do not swallow; keep bottle in cool, dark place; headache is expected; ECG after treatment; causes decreased BP.
Education: decrease workload on the heart by rest, not overeating, no smoking and avoiding extreme temperatures.
Unstable Angina (Acute Coronary Syndrome)
stable angina can progress to unstable angina, and subsequently MI if untreated. There is decreased blood flow to the heart, leading to schema and necrosis of the tissue. Goal is to treat quickly and limit damage to the heart.
Assessment: dull, midsternal chest pain; crushing pain; radiating to left arm and jaw; epigastric discomfort; fatigue; SOB; vomiting.
Treatment: if STEMI, send to Cath lab within 90 mins for PCI; give morphine for pain; administer O2 only if saturation is below 92%; nitroglycerine, aspirin.
Education: quit smoking, diet low in salt and cholesterol, weight loss by walking.
Heart Failure
inability of the heart to pump enough blood to meet the body’s demands for oxygen. There are two types: right sided and left sided heart failure; often occurs as complication to another disease; #1 cause is hypertension.
Causes: hypertension, cardiomyopathy, endocarditis, MI.
Treatment: decrease the workload of the heart
- ACE inhibitors: decrease afterload
- ARBs: decrease blood pressure and increases cardiac output
- Diuretics: reduce edema within the body and supports decrease in blood pressure.
Education: take diuretics in the am; monitor electrolytes with diuretics; low sodium diet; elevate HOB; daily weights; report weight gain of 2-3 lbs in 24 hrs*
Left Sided HF
the heart is unable to move blood forward to the rest of the body. The blood backs up into the lungs.
S & S: pulmonary congestion, dyspnea, cough, wet lung sounds (crackles), blood tinged sputum, murmur, orthopnea, tachycardia, cyanotic, confused.
Treatment: decrease the workload of the heart
- ACE inhibitors: decrease afterload
- ARBs: decrease blood pressure and increases cardiac output
- Diuretics: reduce edema within the body and supports decrease in blood pressure.
Right sided HF
the right side of the heart cannot move blood forward to the lungs. Blood backs up in the body.
S & S: JVD, dependent edema, hepatomegaly, spenomegaly, ascites, weight gain, fatigue, and anorexia.
Treatment: decrease the workload of the heart
- ACE inhibitors: decrease afterload
- ARBs: decrease blood pressure and increases cardiac output
- Diuretics: reduce edema within the body and supports decrease in blood pressure.
Peripheral Arterial Disease
Blood flow to lower extremities is impaired
S & S: pallor; hairlessness; eschar in wounds; dangle legs when pain occurs; pain stops with rest; absent pedal pulses.
Peripheral Vascular Disease
Blood flow back to the heart is affected
S & S: brown discolouration of skin; edema; pedal pulses present; focus on proper wound care.
Aortic Aneurysm
dilation of the aorta, typically caused by atherosclerosis, HTN, smoking, and family history.
S & S: abdominal/back pain that is sharp; severe pain; SOB; trouble swallowing
Considerations: dont palpate the mass*
Air Embolism
Air bubble enters the vein or artery; rare; may be complication of surgical procedure (placement of CVAD/arterial catheter)
Treatment: Durant’s Manuever
- Left lateral trandelenburg
- Prevents air from lodging in lungs
Shock
A state where the vital organs are not receiving enough oxygen. Lack of oxygen causes organ damage and forces cells to use anaerobic metabolism to produce energy, producing elevated lactate. Can have ineffective blood flow back to the heart, vasculature damage, or volume deficits.
1. Cardiogenic
2. Distributive
3. Hypovolemic