Test 2 Flashcards
Worms in the order Strongylida:
- Ancylostoma caninum (SF: Ancylostomatoidea)
- Cyathostomes (SF: Strongyloidea)
- Haemonchus Contortus (SF: Trichostrongyloidea)
- Parelaphostrongylus tenius (SF: Metastrongyloidea)
- Trichostrongylus Axei
- hyostrongylus rubidis
- Strongylus Vulgaris
- oesophagostomum
Ancylostoma Caninum
Common name: Order: SF: F: Lifecycle: D or indire.
CN: Dog Hookworm O: Strongylida SF: ancylostomatoidea F: Ancylostomidae LC: Direct (skin pen., transmammary)
Cyathostomes
Common Name: Order: SF: F: LC:
CN: Small Strongyles O: Strongylida SF: Strongyloidea F: Strongylidae LC: Direct (ingestion)
Haemonchus Contortus
Common Name: order: SF: F: Life Cycle:
CN: (Hair Worms) Barber's Pole, Candy Cane Stomach Worm O: Strongylida SF: Trichostrongyloidea F: Trichostrongylidae LC: Direct (ingestion)
Parelaphostrongylus Tenuis
Common Name: O: SF: F: Lifecycle:
CN: (Lung worms) Meningeal or Brain Worm O: Strongylida SF: Metastrongyloidea F: NONE LC: Indirect (ingests snail; lymphatic-tracheal)
Ascaris Lumbricoides
Common Name: order: SF: F: Life Cycle:
CN: Giant Human Roundworm O: Ascaridida SF: Ascaroidea F: Ascarididae LC: Direct (ingestion)
Ascaris Suum
Common Name: order: SF: F: Life Cycle:
CN: Swine Roundworm Order: Ascaridida SF: Ascaroidea F: Ascarididae LC: Direct (ingestion)
Toxocara Canis
Common Name: order: SF: F: Life Cycle:
Dog Roundworm O: Ascaridida SF: Ascaroidea F: Ascarididae LC: Direct (Ingestion, transplacental, rarely transmammary)
Worms in the Order: Ascaridida
- Ascaris Lumbricoides (Giant Human Roundworm)
- Ascaris Suum (Swine Roundworm)
- Toxocara Canis (Dog Roundworm)
- Parascaris Equorum (Horse Roundworm)
Enterobius Vermicularis
Common Name: order: SF: F: Life Cycle:
CN: Human Pinworm O: Oxyurida SF: Oxyuroidea F: NONE LC: Direct (hand/mouth or aerosol trans. of eggs)
Oxyuris Equi
Common Name: order: SF: F: Life Cycle:
CN: Horse Pinworm O: Oxyurida SF: Oxyuroidea F: NONE LC: Direct (horses chew on wood, pick up eggs, where others rubbed off from their perianal area)
Worms in the order Oxyurida, SF: Oxyroidea
- Enterobius Vermicularis (human Pinworm)
- Oxyuris equi (Horse Pinworm)
Physaloptera rara
Common Name: order: SF: F: Life Cycle:
CN: N/A O: Spirurida SF: Spiruroidea F: NONE LC: Indirect (ingesting beetles)
Habronema Muscae
Common Name: order: SF: F: Life Cycle:
CN: n/a O: Spirurida SF: Spiruroidea F: NONE LC: Indirect (muscae domestica fly places deposits on horses lips, water, feed or the horse eats the fly); fly deposits in wound leading to cutaneous migrans
worms in the order: Spirurida
What Superfamilies are they in?
- Physaloptera rara (Spiruroidea)
- Habronema Muscae (Spiruroidea)
- Draschia (Spiruroidea)
- Dirofilaria Immitis (Filaroidea)
Trichuris Vulpis
Common Name: order: SF: F: Life Cycle:
CN: Whipworm O: Enoplida SF: Trichinelloidea F: Trichuridae LC: Direct (ingestion of L1, infectious, eggs)
Trichinella Spiralis
Common Name: order: SF: F: Life Cycle:
CN: Human Whipworm Order: Enoplida SF: Trichinelloidea F: Trichinellidae LC: Direct (ingestion of infectious cyst)
Strongylus Vulgaris
Common Name: order: SF: F: Life Cycle:
CN: Large Strongyles O: Strongylida SF: Strongyloidea F: Strongylidae LC: Direct
Different Genera of Cyathostomes
- Cyathostomum
- Cylicodontophorus
- Cylicostephanus
- Gyalocephalus
- Poteriostomum
Trichostrongylus Axei
Common Name: order: SF: F: Life Cycle:
CN: stomach parasite of rum. horses, swine, humans O: Strongylida SF: Trichostrongyloidea F: Trichostrongylidae LC: Direct (ingestion)
Parascaris Equorum
Common Name: order: SF: F: Life Cycle:
CN: Horse Roundworm O: Ascaridida SF: Ascaroidea F: Ascarididae LC: Direct
Dirofilaria Immitis
Common Name: order: SF: F: Life Cycle:
CN: Heart Worm O: Spirurida SF: Filaroidea F: NONE LC: Indirect (mosquito bite)
Strongyloides Stercoralis
Common name: Order SF F Indirect or Direct LC
Common name: Intestinal Thread worm O: Rhabditida SF: Rhabditoidea F: Rhabdidae LC: Direct (skin pen., ingestion and mucous membr.pen., transmammary)
Nematomorpha.
Common Name:. ssp?. Morph:. Lifecycle trend. Clinical importance:.
CN: Horse Hair Worm.
SSP: Gordius.
Knotted adult worms.
Lives in water and looks like horse hair; katydid (arthropods) infected by these, worm is released when in aquatic environment (or animals stomachs if they eat them).
CI: no clinical importance but client may become worried if they see their animals vomit the worms out.
Nematodes:.
Dioecious or Monoecious.
more to come.
Dioecious (require 2 individuals of separate sex).
Strongyloides spp.
- Males and females parasitic?.
- What is the infective stage, explain how infection occurs.
- How long is Prepatent period.
- When are hypobiotic larva activated.
- Clinical Signs.
- How does auto-infection happen.
- only females are parasitic.
- L3 is infective - skin/mucosal membrane penetration, transmammary; skin tracheal migration, molting in lungs, coughing may be clinical sign.
- PPP: 2-3 wks.
- activated at parturition and passed transmammary in horse/cow (not dog)
- Pneumonitis and diarrhea.
- immune suppressed individual may develop auto-infection by L1 moving to perianal area developing into L3 and crawl back in anus to reinfect the host.
Strongyloides Stercoralis
- Where is adult worm found.
- Hosts.
- Esophagus Type.
- Describe Cycle of egg.
- zoonotic?.
- how to diagnose.
-
- small intestine mucosa.
- DOGS, Humans/primates.
- long, filariform esophagus.
- eggs hatch, become L1 (Rhabditform esophagus) which are passed in feces. Develop to L3 (infective) or adult in feces.
- should be considered zoonotic
- dx: direct observation of L1 eggs in feces in saline.
Ancylostoma Caninum
Common name:. Location in host:. Morph. of adult, sucks bld or no?. Describe Life phases. What type of Migration. Blood suckers?. Esophagus type: Clinical Signs. anthelminics effective?. Prepatent period. zoonotic?.
CN: Dog Hookworm.
L: small intestine.
M: Large buccal cavity w/3 teeth, voracious blood sucker.
LF: eggs hatch in enviro, feed on bact, molt to L3 and then leave feces. MUST be warm, moist environment (NOT AMARILLO)
Migration:
- Skin-tracheal (adults in intestines, in naive hosts)
- Skin-aortic (adults in tissues, in resistant hosts)
- L4 and adults are voracious blood suckers
- Major clinical sign is anemia, only get disease when infected the first time, build up immunity after that.
- anthelminics effective in all but hypobiotic larva.
- PPP:
- 2-3 wks transmammary
- 4-5 wks skin (longer in resistant host).
- zoonotic: can pen. human skin. can’t get to intestines but do cutaneous migrans and make you itch.
Esophagus type: clubbed
Strongylus Vulgaris.
host:.
migration type.
clinical sign.
low/high susceptibility.
Host: Horses.
MT: migrate through cranial mesenteric artery.
CS: common cause of equine colic.
Susc: are now endangered species so lots of horses susceptible if infected.
What worm was the most important in horses in 2015?.
Cyathostomes.
Cyathostomes.
Life cycle:. infective stage:. Prepatent Period:. Clinical Signs:. anthelminics useful?:.
LC: eggs passed in feces, hatch, molt to infective L3 (1 wk-1month in pasture). L3 ascends vegetation in water (dew), horse eats it, moves to mucosa of lg. intestine. (L3/4 can undergo hypobiosis) molts to L4 moves to lumen of lg. intestine, becomes L5
IS: L3.
ppp: 2m-2yrs
CS: Weight loss, diarrhea; clinical dz. primarily in spring in horses under 4 yrs old.
anthelminic resistance= issue on some properties.
Trichostrongylus Axei.
- Host.
- Location in host.
- Importance of worm.
- H: horse, rum, swine, human.
- L: Stomach.
- not important unless you or your livestock have them.
Haemonchus Contortus.
- anthelminics effective:.
- Describe life cycle.
- Texas worms resistant to all enthelminics.
- Development:
- eggs passed in feces, dev. to L3 (1wk. longer when cool)survive in pasture 1-8mo, ingested
- dev. in the abomasum or hypobios in gastric glands
- become L4 and suck blood; adult in 21-28 days.
- hypobios (overwinter), also reactivate at parturition.
Parelaphostrongylus Tenuis
- Type of migration:.
- Direct/Indirect LC and host?.
- Describe Lifecycle.
- Clinical signs.
- Prepatent Period.
Lymphatic Tracheal migration
- Indirect. Snail L1-L3, deer eats snail, L3 intestine to spinal cord, live in venous sinuses, eggs passed to capillaries in lungs and hatch, L1 coughed up and swallowed, passed in feces on slime layer of pellet, penetrates feet of snails.
CS: None in deer, won’t infect cows, ataxia, paraplegia, tetraplegia only in moose, goat, antelope, llama.
PPP: 3 months.
Haemonchus Contortus.
- Morph of adults.
- Clinical and physio. signs.
- M: females red and white striped (red=digestive tract, white = uterus)
- CS: bottle jaw (hypoprotenemia), death, wool break.
- PS: ANEMIA, hypoprotenemia
Ascarididae family:.
Host:. Location in host:. Looks like what other worm:. Morph of adult:. Infective stage:. Direct or Indirect:.
H: Humans. L: small intestines. LL: indistinguishable from Ascaris Suum. Morph: large, white worms with 3 Lips. IS: L2. Direct: some may use paratenic hosts.
Ascaris Lumbricoides.
describe lifcycle:. amt. of eggs by female:. Clinical Signs. % of ppl have it:. Clinical Signs:.
- eggs passed in feces, require 1m. to become infective
L2 ingestion, hepato-tracheal migration, molt to L3 in liver, L4 and adult in small intestine. - female produces thousands of eggs.
CS: Pneumonitis, Eosinophilia
Z: yes, 40% of population has it.
CS: cognitive delays/malnutrition.
PPP: 2 months
Ascaris Suum
- affects old or young swine and where?.
- Describe Lifecycle:.
- clinical and physio. signs in pigs:.
- young swine heavily in crowded, soil environments
- LC: Egg passed in feces, L2 in enviro (lives for yrs), ingest, hepatotracheal migration, adults in small intestine.
- signs: milk spot liver, pneumonitis, ill thrift, weight loss.
Parascaris Equorum.
Size of worm:. Host:. Host Young or Old:. Location in Host:. Migration:. Prepatent Period:. Immune response:.
Size: LARGE; 15-45cm.
H: Young horses.
L: small intestine.
M: hepatic-tracheal migration.
PPP: 65 days, usually no eggs until foal is 3mo.
IR: migration stimulates strong immune response. will only have during 1st infection.
Toxocara Canis.
Host:. Location in host:. Infective Stage:. Describe Lifecycle:. Type of migration:. Prepatent Period:. paratenic hosts and what does it cause:.
H: Puppies.
L: small intestine.
IS: eggs dev. to infective L2 in 1 month. (can last yrs. in enviro), infect direct or paratenic hosts
- less than 5 wks = hepatotracheal migration to small intestine.
- over 5 wks = hepatoaortic migration to tissues.
- trans placental is common.
PPP:
- 3 wks (trans placental)
- 4-5 weeks (Direct)
PH: Kids less than 3 yrs, visceral larval migrans; target organ = retina, also infects liver, lungs, brain, causes eosinophilia
Superfamily: Oxyroidea.
Hosts:.
Morph of adult worms:.
Infective stage:.
Esophagus feature:.
- H: primates, rodents, equids reptiles
- M: females have pointed tails
- IS: L3 infective
- E: posterior bulb on esophagus
Enterobius Vermicularis
How eggs exit:.
When eggs infective:.
Pass between species?.
how to diagnose:.
- female deposits eggs in perianal area w/sticky substance.
- become infective 6 hrs after, up to 3 weeks.
- NOT passed to pets.
- cellophane tape to perianal area, look for lopsided eggs.
Oxyrus Equi.
- eggs exit how:.
- when infective:.
- migration:.
- Location of adults:.
- Pre patent period:.
- How to treat:.
- female lays in stick substance in perianal area.
- eggs infective in 3-5 days, survive in enviro for 30 days.
- no migration
- location: colon/rectum
- PPP: 5 months
- Treat often as re-infestation is common, remove horse from area for at least a month
Trichuroidea.
Features of the adult.
Adult: Long slender stichosome esophagus, small mouth.
Male has a curved posterior with 1 or no spicule.
Trichuridae versus Trichinellidae egg features.
Trichuridae: Thick shelled, single cell, bipolar plugs.
Trichinellidae: Larva released.
The whip of whipworms is the anterior or posterior end of the worm?
Anterior
Trichuris vulpis.
Lifecycle, include time frames.
Egg to L1 in 2 weeks.
L1 (infective) eggs can live in the environment for prolonged periods of time.
After ingested it hatches in SI and the L1 enters the mucosa migrates through mucosa to large intestine to molt into adults in 10 days.
Trichinella spiralis.
Lifecycle with time frames.
Larva are released from the female and migrate through blood and lymphatics to muscle.
Encysts in skeletal muscle cell.
Infective within 2-3 weeks.
Cyst is ingested by a carnivore and L1 develops into an adult in the SI in 2-6 days.
Adult female enters mucosa and lays eggs (15,000/life).
Trichinella spiralis.
Zoonotic potential.
High prevalence in swine via rodents.
Is zoonotic through eating infected pork that is not properly stored and cooked.
Can be fatal to humans.
Spiruroidea.
Adult location in host.
Features of the adult.
Location: Stomach.
Adult: 2 or 4 lips, well demarcated pharynx, Esophagus is muscular on anterior portion and glandular posteriorly, cuticular ornaments.
Male: coiled tail.
Spiruroidea.
Egg features.
Larvated eggs produced.
Physaloptera rara. Hosts. Location of adult in host. Clinical signs. Diagnosis.
DH: Dogs, cats, other large predators.
IH: Beetle
Found in the anterior duodenum with the anterior end embedded in the wall of the stomach.
Diagnosis: recover and ID worm from vomit.
Physaloptera rara.
Lifecycle, include timeframes.
Adult lays eggs.
L1 egg is ingested by beetle larva and worm develops to L3 within beetle.
Infected beetle is ingested.
Develops into adult in the stomach.
Habronema muscae.
Hosts.
Lifecycle, include time frames.
Prepatency.
DH: Equids. IH: Musca domestica. Adult lays eggs into feces. L1 eggs ingested by fly larvae. Worm develops to L3 in fly. L3 deposited on lips, in water, on feed, or fly itself is ingested. Develops to adult in stomach and lives under mucus coat in glandular portion. Prepatency: 2 months.
Habronema versus Physaloptera eggs
Habronema: thin shelled and larvated.
Physaloptera: thick cuticle and larvated.
Explain how cutaneous habronemiasis happens.
Common name.
Common name: Summer sores.
L3 is deposited on abraded skin, conjunctiva, or mucous membranes of genitalia.
Causes a pruritic chronic granulomatous lesion that regresses in winter and presents again in spring.