Tendinopathies of UE Flashcards
Tendons
Connect Mm to bone
Tendinopathies often fall in the category of
Cumulative Trauma (CTI) or Repetitive Strain Injuries (RSIs)
* Common work related injuries
* Can occur in anyone
* Can affect tendons and or Nn (nerve issues usually
due to the inflammation and swelling
Other causes of Tendinopathy
Falling on an area
Trauma induced tendinopathy
Poor blood flow
Poor posture and mechanics
Poor proximal stability and core strength decreased ability to maintain a good safe posture leading to added stress on the tendons
* Tendonitis or nerve issue = always correct posture
Tendon pain
“itis” (inflammation)
“osis” (scar tissues) - fibrosis of the tendon that has been injured more than once and laid down scar tissue (tendinosis)
“algia” (nerve irritation)
Could have a combination of all the above, which makes it hard to treat
Tendonitis
Inflammation process. Inflammatory cells found in tissue.
Tendinosis
Degenerative process with fibrosis, decreased circulation. Fibrosis found in the tissue.
Tendonalgia
“Pain” neurogenic in nature. Free nerve ending are irritated and signaling “pain” to the brain. Neurotransmitters and inflammatory markers are present (immune system signals and sends those out)
*Doctors often call it tendonitis but does not mean you take that at face value, so you must ask ?'s to figure out if it is "itis" "osis" "algia".
Tenosynovitis
The synovial sheath the tendon runs through is also inflamed
Combination
Various processes occurring simultaneously
Tendinopathy Secondary to an RSI
- Repetitive use especially against resistance
- Repetitive sheering of tissue across a bone
- Extreme postures for prolonged period of time
Pulls on the origin and insertion sites - Carrying resistance when Mm are stretched
Lateral Epicondylitis (Tennis Elbow)
Very common
Very challenging diagnosis (is it inflammation = acute or scar tissue = chronic????
Originally is an “itis”
Starts as inflammation of the tendon fibers that attach the forearm extensor Mm to the lateral epicondyle
Routine use of the arm causing shearing of the ECRB over the radial head especially with the elbow extended or an injury to this area may stress or damage the muscle attachment
During healing the tendons often get stuck in fibroplasia (the process of forming fibrosis tissue)
The tissue has a failure to thrive.
Calcification, rupture and additional fibrosis follows.
Shearing over radial head and stress to muscle attachments
First stage wants to lay down collagen = fibrosis
Usually caught after the inflammatory stage
Difficult to alleviate symptoms if the condition is chronic
Treatment is different for acute vs chronic
Over 23 ailments that can cause lateral elbow pain
Cervical Radiculopathy =
(C5/C6) narrowing of foramen (compression)
Proximal Neurovascular entrapment of lateral ante brachial cutaneous nerve
Arthritis
Tumors
Radial Tunnel/PIN entrapment = big one that mimics or coexists with tennis elbow (Posterior Interosseus Nerve entrapment)
Distal biceps insertion at radial tuberosity
Triceps insertion at olecranon
Shoulder tendonitis
Carpal Tunnel Syndrom
Trigger points
Loose Bodies (bone fragments (often see with falls, do X-ray
Anatomy
Common Extensor Origin:
* ECRB = biggest, primary structure involved in LE and
has a poor blood supply at origin
* EDC = next biggest, the EDC of long finger combines with
the ECRB tendon to insert at the lat epicondyle
poor posture and body mechanics while typing
and playing piano. Excessive use of EDC instead
of floating digits can lead to LE
* ECRL
* ECU
- ECRB = origin: lat epicondyle of humerus
ins: base of MC 3
actions: ext and radial deviation of carpus
nerve: deep radial N
* active during sup and pro - EDC = origin: lat epicondyle of humerus
ins: extensor expansions of dig 2-4 and distal
tendon of extensor digits minimize
actions: extends carpus and dig 2-5
nerve: deep radial N - ECRL = origin: lat supracondylar ridge of humerus
ins: base of MC 2
actions: ext and radial deviation of carpus
nerve: radial N - ECU = origin: lat epicondyle of humerus and post
border of ulna
ins: base of MC 5
actions: ext and ulnar deviation of carpus
When pt have been casted, after cast is removed they have a tendency to want to use the EDC to extend the wrist = put a pen or pencil in their hand and then have them extend the wrist this will force them to use their ECRB and ECRL
Lateral Epicondylitis
NOT usually an inflammatory conditions
Tissue death due to:
* Microscopic tears of ECRB and EDC
* Calcifications = muscle started hardening and turning
into bone
* Excessive granulation = lots of scar tissue
* Alteration in neuropeptides = things that are not
supposed to be there
* Avascular areas = scar tissue
* Adhesions = things are stuck
* Mucoid Degeneration
Immuno-reactive neurogenic component to lateral epicondylitis
Presence of Neuropeptides/Neurokinin 1-receptors, Substance P (SP) and calcitonin gene related peptide (CGRP)
Glutamate = good when it is supposed to be there but toxic if not supposed to be there. Toxic to nerve tissue and eats away at it, therefore free nerve endings increase sensitivity and pain.
Evaluation Components
- History how long and how many times have they had LE
(<12 weeks = acute) (>12 weeks = chronic) - Cervical screening- Dermatomes and Myotomes
- Visual Analog scale (unidementional measurement of
pain intensity - Grip strength (elbow flex and ext if not in a lot of pain)
- Pinch strength
- Palpate for tenderness:
radial head
lateral epicondyle
radial tunnel-crepitus, 2-5cm distal and ant to
LE. Radial tunnel 4
finger widths and dorsal
over supinator - Arcade of Frohse = where radial nerve bifurcates
(dives deep) and can get
trapped - If posterior interoseus nerve gets pinned, it can look like
LE - Can have both radial tunnel and lateral epicondylitis
(LE = tennis elbow and arcade of frohse = radial tunnel) - If long finger extension test causes pain = ask where
the pain is, LE = TE and arcade = RT - Resisted wrist extension, supination (Cozen’s Test)
pain = ask where and if pain at rest is greater than
5 do not do resistance training - ROM/Muscle tension with composite stretch
progressively check positional progress and can use as a
treatment tech, always stretch if non-painful - Patient Rated Functional Assessment: PREE, DASH
- Muscle strength if not in a great deal of pain
Grip Strength Tension Test
Indiana Hand Center
Should be seated
1) Elbow flexed
2) Elbow extended, wrist neutral and full pronation
Places tension on the origin (stretching and grip)
Positive = decrease in strength, probably TE
Negative = no decrease in strength, not TE
3) More challenging is extend arm horizontal to floor
Treatment Interventions
- Always proximal to distal, correct posture (rhomboids,
post delt) and strengthen coreForward shoulder and int rotation puts tension on
lateral elbowExt rotation = puts force on medial elbow (Tommy
Johns surgery) - Determine whether condition is inflammatory or chronic
Inflammatory Tendonitis
- Rest
- Ice massage or cold packs
- Cortisone injections
- Iontophoresis using Dexamethasone ( do not do
if they have had a cortisone shot) - Stretching exercises = as long as it is done progressively
it should be pain free
full stretch for ECRB = arm extended, hand in fist with
ulnar deviation - Wrist support = only decreases firing of extensors
7% - Tennis elbow strap = only works with power gripping
wear 2 inches below LE, if worn wrong could compress
radial nerve, good for placebo effect
Chronic Tennis Elbow
- Most of what we see
- Scar tissue needs blood flow and remodeling (use heat)
- Exercise (graded eccentric exercises (help with
concentric and let them do eccentric, want to remodel
and elongate scar tissue, more reps less weight - Best way to warm up tissue is active exercise, arm bike
focused on extension - Progressive stretching
- Joint mobilization (loosen up capsule)
- Modalities-Heat focus (hot packs)
- Strengthen core and correct posture
Acute or Chronic Interventions
Long arm elbow splint for night wear, placing arm at 35 - 40 degrees of flexion to prevent extreme flexion which also places stress on the extensor origin
About 6 weeks if tolerated
Physical Agents Modalities (PAMS)
- Ultrasound/Phonophoresis
used to administer meds (dexamethasone)
transcutaneously but now docs do cortisone
injections (with cortisone may raise blood sugar
if they have diabetes be aware) - Cryotherapy
- Heat
- High volt Iastem
- TENS
Exercise
Controlled and close to body, working toward farther away
Instrument Assisted Soft Tissue Mobilization
Scar tissue massage through skin
Kinesiotape
Opening over the arcade of frohse = decompress radial nerve, relaxing extensors
Alternative Modalities
Acupuncture = Short term pain relief (24 hrs)
Extracorporeal Shock Wave therapy: very expensive, cortisone found to be more effective
Low Level Laser Therapy = no evidence except when used in combination with PT
Cortisone injection
Occasionally work
Dependent on timing of injection
May have an effect on the sensory nerve response
Cortisone injection can lead to cell death and atrophy and inhibits collagen synthesis
Autologous Blood Injections/Protein Rich Plasma (PRP)
Induce healing by providing cellular mediators