TBI Intro: Secondary Complications Flashcards

1
Q

paroxysmal autonomic instability and dystonia (PAID)

A
  • “sympathetic storming”
  • severe medical instability
  • everything is heightened
  • episodes unprovoked, last for hours or end abruptly
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2
Q

clinical S&S of PAID

A
  • tachypnea
  • tachycardia
  • HTN
  • diaphoresis
  • decorticate/decerebate posturing
  • agitation
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3
Q

how do you diagnosis PAID

A

thru observation

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4
Q

management for PAID

A
  • symptom management (morphine, fentanyl)

- “ride out the storm”

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5
Q

without appropriate treatment for PAID what happens

A

increased morbidity

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6
Q

heterotopic ossification

A

formation of abnormal bone growth around joint tissue

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7
Q

how many cases of TBI experience heterotopic ossification and what increases the risk

A
  • 10-20%

- increased risk if TBI polytrauma injury results in fx near joint lines

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8
Q

what is heterotopic ossification most common in

A

large joints of the body

hips, knees, shoulders, elbows

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9
Q

what is the cause of heterotopic ossification

A

unknown, associated with trauma, immobility and hypertonicity

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10
Q

onset of heterotopic ossification

A

4-12 weeks after injury

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11
Q

clinical presentation of heterotopic ossification

A
  • initial signs are loss of ROM (HARD END FEEL) and pain in the joint area
  • local erythmea, pain with movement, swelling, warm to touch
  • severe HO may result in vascular and/or nerve compression
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12
Q

treatment for HO

A
  • PROM and stretching –> maintaining
  • medications
  • surgical excision
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13
Q

what medications help with HO

A
  • didronel: preventative

- anti-inflammatory

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14
Q

when do u get a surgical excision following HO

A

> 1.5 yr after injury

want the bone to mature & finish growing before removal

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15
Q

what other complications are secondary to TBI

A
  • GI (50%)
  • genitourinary (45%)
  • respiratory (34%)
  • CV (32%)
  • dermatological (21%)
  • urinary bowel incontinence
  • hydrocephalus
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16
Q

what clinical manifestations are associated with TBI

A
  • altered level of consciousness/impaired arousal
  • cognitive and behavioral impairments
  • neuromuscular impairments
  • autonomic dysfunction
  • sensory abnormalities
  • CN damage
  • vestibular deficits
  • secondary complications
17
Q

secondary complications

A

ICP management

seizure reduction or prophylaxis

18
Q

what can increased ICP lead to

A

can compress brain tissue

decrease perfusion to brain tissues

19
Q

normal ICP

A

5-10 mm Hg

20
Q

abnormal ICP

A

20 mm Hg caution @ 15

21
Q

activities that increase ICP

A
full supine or trendelenburg 
cervical flexion
percussion & vibration
valsalva - coughing, sneezing, holding breath, etc.
exertional activities
22
Q

S&S increased ICP

A
decreased responsiveness
impaired consciousness
severe HA
vomiting
irritability
papilledema
increased d BP
decreased HR
23
Q

treatment for increased ICP

A

careful monitoring
pharmacological agents
ventricular peritoneal shunting

if permanent correction is needed

24
Q

when can PT treat after a seizure

A

24 hrs seizure free

25
Q

events that trigger seizures

A
stress
poor nutrition
electrolyte imbalance
missed mediations or drug use 
flickering lights
infxn
anxiety 
mobility
26
Q

common S&S for seizures

A

focal or diffuse twitching, jerking, stiffness
+/- loss of consciousness
absent staring

27
Q

interventions for seizures

A

continuous electroencephalogram

medications