CVA impairments pt. 1 Flashcards

1
Q

stage 1 of motor recovery

A
  • period of flaccidity immediately following the acute episode
  • no movement of the limbs can be elicited
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2
Q

stage 2 motor recovery

A
  • basic limb synergies
  • minimal voluntary movement responses
  • spasticity develops
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3
Q

stage 3 motor recovery

A
  • pt gains voluntary control of the movement synergies
  • full range of all synergy components does not develop
  • spasticity has further increased
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4
Q

stage 4 motor recovery

A
  • some movement combinations that do not follow the paths of either synergy are mastered
  • spasticity declines
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5
Q

stage 5 motor recovery

A
  • more difficult movement combos are learned as synergies lose their dominance
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6
Q

stage 6 motor recovery

A
  • disappearance of spasticity
  • individual joint movements and coordination approaches normal
  • normal motor fxn restored
  • recovery can plateau at any stage
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7
Q

what plays a significant role in a pt’s progression through the stages of motor recovery

A
  • initial weakness
  • presence of spasticity
  • cognitive deficits
  • access to rehab
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8
Q

what is recommended before beginning exercise program post-CVA

A
  • graded exercise testing
  • ECG monitoring
  • MI protocol
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9
Q

what is the MI protocol for exercise

A
  • sub-max protocols
  • peak HR 120 bpm or 70% age predicted HRmax
  • BP <250/115
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10
Q

if unable to do graded exercise testing:

A
  • light to moderate exercise
  • increase training fq, duration or both to compensate for reduced intensity
  • close monitoring of HR, BP
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11
Q

dysdiadochokinesia

A

impaired ability to perform rapid alternating movements

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12
Q

dysmetria

A

inability to judge distance or range of movement

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13
Q

dysynergia

A
  • fragmented movement patterns

- movement occur in sequence of component parts rather than a single & coordinated cmooth output

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14
Q

asynergia

A
  • loss of ability to associate muscles together for complex movements
  • more severe form dyssynergia
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15
Q

rebound phenomenon

A

inability to rapidly and sufficiently halt movement of a body part after a strong isometric force

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16
Q

tremor

A

unintentional oscillatory movement

17
Q

ataxia

A
  • uncoordinated movement that manifests when voluntary movements are attempted
  • results in difficulties with fluidity/timing, accuracy and speed of movements
18
Q

if you have impairments in coordination..

A
  • cerebellar pathology
  • basal ganglia pathology
  • disruption of dorsal column-medial lemniscus and associated structures
19
Q

cerebellum potential exam findings

A

IPSILATERAL

  • trunk, limb, and/or gait ataxia
  • dysmetria, dyssynergia, dysdiadochokinesia
  • balance
  • oculomotor deficits
  • lack of check reflex
  • mild hypotonia
  • intentional tremor
  • slurred speech (dysarthria)
  • significant difficulties with motor learning
20
Q

basal ganglia potential exam findings

A

CONTRALATERAL

  • trunk, limb, and/or gait ataxia
  • dysmetria, dyssynergia, dysdiadochokinesia
  • balance
  • spasticity (rigidity)
  • resting and intentional tremor
  • difficulty intiating movements
  • slowed movements, smaller movements
  • considerable strength deficits
21
Q

dorsal column potential exam findings

A

CONTRALATERAL

    • trunk, limb, and/or gait ataxia
  • dysmetria, dyssynergia, dysdiadochokinesia
  • balance
  • abnormal sensory exam –> proprioception
  • unlikely to see tremor
22
Q

tone

A

resistance to passive stretch

23
Q

acute UMN injuries results in ..

what is the cause and duration

A
  • temporary hypotonia
  • cerebral or spinal shock
  • variable
24
Q

when do you get spasticity with UMN injuries

A
  • subacute –> chronic

- does go down over time