Task 4 - classical conditioning Flashcards

1
Q

Unconditioned stimulus

A

-biologically significant stimulus that elicits a natural reflective response

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2
Q

Unconditioned response

A

Natural reflex elicited by US

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3
Q

Conditioned Stimulus

A

-cue that was previously neutral but that through training becomes associated with US

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4
Q

Conditioned response

A

-learned response to CS that has been paired with US

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5
Q

Aversive /appetitive conditioning

A
  • US is unpleasant or negative event

- US is a positive event

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6
Q

Conditioned compensatory response

A
  • results in tolerance
  • decrease in reaction to a drug (or something else) , so that larger doses are required to have a similar effect
  • occurs primarily in body system that has mechanism for homeostasis (tendency to gravitate towards state of equilibrium)
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7
Q

Extinction

A

-reducing a learned response to a stimulus , by stopping to pair that stimulus with a reward/punishment

  • > association that became ‘inhibited’ following extinction will not be lost in its entirety
  • > in fact, learned response is not gone, just unexpressed
  • extinction is not forgetting!
  • extinction = overwriting/replacing
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8
Q

Compound conditioning

A
  • Simultaneously conditioning of two cues , usually presented at same time
  • two cues compete against each other
  • neither produces as much learning as it would have ( if trained alone)
  • compound cues share predictive value
  • > learning is faster with compound cues
  • learning rate is faster -> steep learning curve
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9
Q

Overshadowing

A

-effect seen in compound conditioning when a more salient cue within a compound acquires more association strength than less salient cue

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10
Q

Acquisition

A
  • the CR gets stronger gradually

- if CS proves to be predictive of US then CR follows CS, also without US n

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11
Q

Tolerance

A

-compensatory response in preparation to US , in an attempt to maintain a balanced state (homeostasis)

Evidence:
-heart rate drops when awaiting an adrenaline injection, diminishing the heart rising effect of adrenaline

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12
Q

Blocking

A

-a stimulus can only become an CS if it brings ne predictive value

Evidence:

  • light and sound are followed by a shock
  • > both stare the predictive power
  • if only light is followed by the shock and sound is added later
  • > sound will not add value to prediction

No Error = No learning

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13
Q

Rescola - Wagner Model general idea

A
  • if you are surprised, you are learning
  • US occurs unexpectedly + positive error ( E) -> increase in association of CS and US

-US does not occur as expected = negative E -> decrease in association of CS and US

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14
Q

Prediction Error

A

-determines how much learning (change in association) occurs

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15
Q

Error - correction - rule

A
  • with trials of learning the prediction error gets reduced

- learning occurs incrementally

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16
Q

Formula prediction error

A

Actual US - Expected US

17
Q

Formula Expected - US

A

(Learning rate * predition error) + previous Exp US

18
Q

Problem - latent inhibition

A
  • rescorla Wagner model can’t explain latent inhibition
  • > prior exposure to a CS retards later learning of the CS-US association during acquisition training

-> model makes false prediction that pre-exposed group should be no different from control group

19
Q

US -Modulation theory

A
  • Solar-Wagner model

- > how unexpected is US given preceding CS

20
Q

CS - Modulation theory

A
  • Mackintosh

- which CS is attended determines which becomes associated with US

21
Q

Cerebellar Cortex

A
  • granule cells
  • parallel fibers
  • Purkinje cells -> location of learning
  • climbing fibers
22
Q

Cerebellar deep nuclei

A
  • mossy fibers
  • interpositus -> output
  • climbing fibers
23
Q

Brainstem

A
  • Pontine nuclei
  • inferior olive

-> Inputs

24
Q

Steps conditioning

A

Purkinje cell inhibits interpositus (as normal)

  • > CS (tone, light)
  • > reduced Purkinje cell activation
  • > releases interpositus form inhibition
  • > interpositus triggers eye muscle before standard reflex pathway is triggered by US sensory input
  • > after conditioning the CS pathway is faster
25
Q

Cellular perspective in Purkinje cell

A

26
Q

Rescorla Wagner model - 3 assumptions

A

1) a cue (CS) has a weight (predictive value) that predicts the US
2) in compound cues, the weiht is spread over cues
3) learning corresponds to an increase in cue weight (accumulated over trials) and a corresponding reduction in prediction error

27
Q

Additional learning after removal of one of two compound cues

A

-predictive value of the remaining cue should be 50 % and new learning should occur until you reach full level of learning again

28
Q

Cerebellar function in motor performance (and motor learning)

A

-compares motor plans with feedback on the execution of movements based on sensory feedback

29
Q

Canonical modulators circuit cerebellum

A

30
Q

Cerebellar pathology

A

..

31
Q

Optogenetics

A

….

32
Q

Optogenetics - use

A
  • alleviating epilepsy by optogenetic stimulation of Purkinje cells
  • deep brain stimulation ( Parkinson’s, depression)
  • treatment of heart arithmia’s
  • modification of traumatic memory, treatment of addictions etc.