Sports Injuries Unique to Young Athletes Flashcards

1
Q

apophysis

A

where muscle-tendon unit attaches to bone

provides contour and shape without adding length

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2
Q

What part of the bone structures are the “weakest links” in the musculoskeletal chain in children?

A

ossification centers

more vulnerable to injury than ligaments, tendons, and muscles

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3
Q

osteochondroses (apophysitis)

A

overuse injury

inflammation and irritation at an apophysis or physis due to excessive stress

inflammation results form mechanical pressure

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4
Q

acute injuries in children

A

physeal fractures

apophyseal avulsion fractures

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5
Q

What is the spectrum of pathology in osteochondrosis?

A

cartilage swelling

irregular ossification

tendon thickening

separation

fragmentation of cortical bone

delayed closure

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6
Q

What is the mechanism of injury in osteochondrosis?

A

mis-use due to:

improper technique

equipment poorly-fitted or worn out

change in playing surface

overuse:

rapid increase in volume or intensity of training

inadequate time for rest and recovery

adult-directed training vs. kid-directed free play

sports specialization at early age

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7
Q

What are the risk factors of osteochondrosis?

A

growth spurt

tight muscles

certain variants of normal anatomic alignment

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8
Q

What are the effects of a growth sport on the bones?

A

period of high metabolic activity at growth center - cells become more vulnerable to stress

bones grow faster than muscle-tendon units - results in tight muscle-tendon units and increase tension on apophyses where they attach

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9
Q

What are the common sites of osteochondroses?

A

lower extremity:

calcaneus (Sever’s Disease)

tibial tuberosity (Osgood-Schlatter Disease)

upper extremity:

medical epichondyle of elbow (Little League Elbow)

proximal humeral physis (Little League Shoulder)

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10
Q

Sever’s Disease

A

calcaneal osteochondrosis

peaks at age 8-11

bilateral up to 61%

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11
Q

What are the mechanisms of Sever’s Disease?

A

tension from Achilles and plantar fascia

direct impact pressure

most common sports are soccer and basketball

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12
Q

What are the risk factors for Sever’s Disease?

A

inappropriate shoes

tight calf muscles - less than 10 degrees of ankle dorsiflexion

subtalar overpronation

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13
Q

What is the common presentation of Sever’s Disease?

A

history of heel pain and worse with activity

physical exam shows pain with medio-lateral compression of heel

may find risk factor’s such as tight heel cords or subtalar overpronation

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14
Q

What is the treatment of Sever’s Disease?

A

rest from sports

ice massage

analgesics rarely necessary - never use before activity

calf muscle stretching - hold each ofr 30 seconds 1-2 times a day

heal cups

molded shoe inserts if overpronation is present

when severe, may need crutches until weightbearing is painfree

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15
Q

What is the prognosis of Sever’s Disease?

A

usually resolves in 2-8 weeks with proper treatment

no significant long-term effects

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16
Q

What is the occurrence of Osgood-Schlatter Diseaes (OSD)?

A

osteochondrosis of tibial tuberosity

girls aged 8-13

boys aged 10-15

21% of athletic adolescents

common in running, jumping sports

4.5% of non-athletes

bilateral in 20%

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17
Q

What are the risk factors of Osgood-Schlatter Disease?

A

right quadriceps and hip flexors

external tibial rotation:

when tibial tubercle sits lateral to the center of patella

increases traction stress from patellar tendon

patella alta - patella sits higher than normal

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18
Q

What is the clinical presentation of Osgood-Schlatter DIsease?

A

history of pain, swelling at tibial tubercle

worsens with activity, improves with rest

tenderness at tibial tubercle

pain with resisted knee extension

often have tight quadricepts and hip flexors

thomas test and/or ely test positive

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19
Q

What are common findings in x-rays in Osgood-Schlatter Disease?

A

not required for diagnosis

typically normal

may see soft tissue swelling

useful to rule out other pathology

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20
Q

What is the treatment for Osgood-Schlatter Disease?

A

rest from sports

ice massage

quad and hip flexor stretching - hold each for 30 seconds 1-2 times a day

analgesics rarely necessary - never use before activity

patellar tendon strap may help

severe cases may require immobilization - long leg cast or knee immobilizer brace for 3-4 weeks

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21
Q

What is the prognosis of Osgood-Schlatter Disease?

A

usually resolves with proper rest and treatment - may take 12-24 months to run its course

22
Q

What are the complications of Osgood-Schlatter Disease?

A

persistent painful ossicle - may require surgical excision for relief of symptoms

tibial tubercle aversion - rare and risk of avulsion does not warrant restriction from sports for those with OSD

23
Q

When should individuals return to play after Sever’s and OSD?

A

may continue play if pain occurs only after activity and resolves by the next day

should not return to play if pain occurs with daily activities or if pain occurs during sports activity

no analgesics prior to exercise - may mask pain which is a sign of worsening injury

24
Q

What is little league elbow?

A

osteochondrosis of medial epicondyle

affects up to 50% of pitchers between 9-14 years old

caused by excessive traction stress on medial epicondyle apophysis with pitching motion

25
When does traction stress peak during phases of throwing?
traction stress peaks during late cocking and acceleration
26
What is the mechanism of injury in little league elbow?
traction stress from flexor-pronator muscles and valgus force on elbow
27
What are the risk factors of little league elbow?
lots of pitching throwing breaking pitches before skeletal maturity - require more flexor-pronator muscle action and sliders rincrease risk for elbow pain by 86% lack of pitching experience poor conditioning and muscle strength
28
What is the clinical presentation of little league elbow?
history of gradual onset of pain with pitching, recent increase in pitching volume, and usually no pain with throwing, batting, or AD:'s tender at medial epicondyle may have pain with valgus stress to elbow may have pain with reisted wrist flexion or pronation may lack full elbow extension
29
What are findings on x-rays of little league elbow?
not required for diagnosis may show widening, irregular ossification, and fragmentation
30
What is the treatment of little league elbow?
rest from pitching for 2-4 weeks until physis is non-tender rehabilitation for 4-6 weeks - stretch flexor/pronator muscles and strengthen shoulder, core/hip muscles interval throwing program for 6-8 weeks that may begin when athlete has full ROM and strength, start at half distance and power and incrementally increase
31
What is the prognosis of little league elbow?
most heal with conservative treatment long-term problems are rare indadequate rest/treatment can lead to failure of physeal closure, chornic pain from non-union, and acute avulsion fracture
32
What is the mechanism of little league shoulder?
osteochondrosis of proximal humeral physis due to excessive rotational stress, 35% of pitchers are aged 9-14 seen in other overhand sports such as tennis and volleyball
33
What are the risk factors of little league shoulder?
lots of pitching throwing breaking pitches before skeletal maturity lack of pitching experience poor conditioning and muscle strength
34
What is the clinical presentation of little league shoulder?
history of shoulder pain with pitching may also have pain with regular throwing and ADLs physical exam is tender at proximal humeral physis may have painful arc of motion weakness and pain with resisted shoulder abduction and external rotation
35
What is the treatment of little league shoulder?
**rest from pitching (4-6 wks)** - until physis is non-tender and ROM is full and pain-free and strength testing does not generate pain **rehabilitation (4-6 wks)** - stretch and strengthen shoulder, core/hip muscles **interval throwing program (6-8 wks)** - may begin when athlete has full ROM and strength, start at 50% distance and power and incrementally increase
36
What are the most important factors for preventing pitching injuries?
don't pitch through pain or fatigue follow guidelines for pitch count limits
37
What are apophyseal avulasion fractures?
separation of apophysis from main bone pelvis is most common site 90% occur in boys 80-90% are sport-related peak incidence is at 14-18 years of age
38
What is the mechanism of apophyseal avulsion fractures?
sudden, forceful eccentric muscle contraction
39
What is the clinical presentation of avulsion fractures?
sudden forceful contraction of muscle pulls apophysis away from adjacent bone athletes report a painful "pop" commonly unable to bear weight after injury upon physical exam: tender at apophysis, typically no swelling/bruising, muscle usuall ynot tender, pain with passive stretch and resisted muscle test
40
What are common x-ray findings in avulsion fractures?
damage of the ischial tuberosity - 30-50% of pelvic avulsions, due to forceful contraction of hamstrings anterior inferior iliac spine - 20% of pelvic avulsions, due to forceful contraction of rectus femoris iliac crest - 2-11% of pelvic avulsions, due to forceful contraction of abdominal obliques
41
What is the treatment of avulsion fractures?
majority respond to non-operative treatment displacement \> 2cm may require surgical fixation **rest (1-4 weeks)** - goal is to releive tension on injured apophysis, requires crutches until weightbearing is pain-free **stretching (1-3 weeks)** - may begin when apophysis is non-tender, gentle actuve stretching involved muscle group **rehabilitation (2-4 weeks)** - progressive resistance exercises, proprioception training, jogging and sport-specific activities
42
What is the prognosis of avulsion fractures?
generally good **ischial tuberosity avulsions**: longest recovery time (3-4 months) up to 68% result in non-union 75% have difficulty returning to sports 25% dropped out of sports altogether
43
What is spondylolysis?
stress freacture at pars interarticularis most common at L4 and L5
44
What is spondylolishtesis?
when upper vertebra slides forward can occur if spondylolysis is bilateral can also be congenital
45
What is the clinical presentation of spondylolysis/listhesis?
symptoms of low back pain with lumber extension, loading pain usually relieved with flexion PE findings reveal positive Stork test - pain with single leg hyperextension tender at affected vertebra
46
What are common imaging modalities for spondylolysis/listhesis?
xrays (AP, lateral, obliques) - may see fx (dog collar) or slip, may be normal SPECT scan - 100% sensitive MRI - sensitivity improving with newer techniques (ex. saggital STIR)
47
What is the treatment for spondylolysis/listhesis?
custom-fitted rigid brace until pain-free ROM physical therapy - progressive core strengthening, first 4 weeks exercise with neutral spine and then slowly increase ROM if pain-free no sports for at least 3 months and pain-free ROM, adequate core strength
48
What are the risk factors for back pain?
common muscle imbalances in gymnasts increase risk for back pain
49
What is the Thomas test? What does it measure?
touch the knee to the chest measures hip flexor flexibility if thigh lifts off table when opposite hip is maximally flexed, hip flexor is tight
50
What is the Trendelenberg test and what does it measure?
the patient is asked to stand unassisted on each leg in turn, whilst the examiner's fingers are placed on the anterior superior iliac spines. The foot on the contralateral side is elevated from the floor by bending at the knee estimates gluteus medius strength
51
What are some ways to prevent back injury?
core/gluteal strengthening stretch hip flexors, erector spinae