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E-Pathanatomy > Special - Pathogenesis > Flashcards

Special - Pathogenesis Flashcards

1
Q

248

Post Traumatic Focal Inflammatory Reaction of Myocardium
Pathogenesis

A
  • Neutrophils are present for foreign body phagocytosis
  • Continuation of repair process, granulation tissue forms with macrophages and lymphocytes
  • Fibrocytes are also seen (spindle shaped cells) in the same direcion as the normal myocytes (more mature scar tissue)
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2
Q

261

Vasculitis Chronica
Pathogenesis

A
  • During uraemia there is deposititon of calcia salts occurs
  • Chronic inflammatory processes in the arteries tunica mediae
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3
Q

262

Mediocalcinosis (van Kossa)
Pathogenesis

A
  • During uraemia there is deposititon of calcia salts occurs
  • Chronic inflammatory processes in the arteries tunica mediae
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4
Q

6

Emphysema Alveolare Chronicum
Pathogenesis

A
  • Most important is the loss of bronchus wall elasticity causing poorer expirium than inspiriu
  • Air accumulates in the lungs (long inhales and short fast exhales) leading to distention of alveoli and lung inflation ending in rupture
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5
Q

200

Endobronchiolotis Obliterans et peribronchitis nodosa
Pathogenesis

A
  • Obstructive atelectasis areas of round-irregular shape occur and bronchioles become obstructed, filling with granulation tissue
  • This may proliferate into the alveoli causing carnification of the lung
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6
Q

228

Crupous (fibrinous, lobar) Pneumonia and Chronic Interstitial Pneumonia
Pathogenesis

A
  • The pink colour inside alveoli is due to fibrin and exudate, walls are slightly thickend
  • Darkened margin surrounds the pathology and coagulation necrosis also occurs
  • The stages of interstitial pneumonia: congestive stage, red hepatization, and resolution (repair or carnification)
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7
Q

270

Epulis Fibromatosa
Pathogenesis

A
  • Occur in gingiva near teeth as covered, soft, nodular masses with three potential forms: fibromatous, osseous and acanthomatous.
  • Fibromatous and osseous types are benign tumours
  • Acanthomatous type infiltrates and destroys periodontal apparatus
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8
Q

177

Colitis Fibrinosa (Levaditi)
Pathogenesis

A
  • Bacteria colonise large intestine and releases toxins that damage the intestinal lining
  • May penetrate deeper into mucous membrane at the base ofcrypts
  • Transmission primarily occurs through the fecal-oral route
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9
Q

9

Lymphadenitis Acuta Simplex
Pathogenesis

A
  • An infection at a nearby site, bacteria or their products drain into regional lymph nodes, triggering an inflammatory response
  • Leading to lymph node enlargement, as immune cells are recruited to the site of infection
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10
Q

238

Infectious Bursal Disease
Pathogenesis

A
  • Virus is shed in feces, it is highly contagious an the results depend on age and breed of the chicken
  • Subclinical and clinical infection occur
  • Cloacal bursa is swollen, edematous, yellowish/haemorrhagic and muscular haemorrhage in legs is common
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11
Q

27

Cholangitis et Pericholangitis Chronica Hyperplastica (Coccidiosis Hepatis Cunuculorum)
Pathogenesis

A
  • The coccidea hepatis cuniculorum are intracellular parasites, develop within the cytoplasm of epithelial cells, resultins in death of each cell affected
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12
Q

268

Hepatitis Purulenta Acuta
Pathogenesis

A
  • Early stadium of purulent inflammation occurs in hepatic parenchyma
  • Later stadium form neutrophilic granulocyte accumulations (abscesses)
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13
Q

196

Leucosis Lymphadenoidea hepatis
Pathogenesis

A
  • Virus infects, leading to proliferatio of B lymphocytes and the development of lymphosarcoma, affecting various organs, like liver
  • Tumour tissue arises from portal and bile tracts and gradually invades the center of the lobules and may even substitue them
  • After parasitic larvae migration, haemorrhagi tracts may arise in normal liver tissue
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14
Q

164

Glomerulonepritis Chronica
Pathogenesis

A
  • Kidney fibrosis occurs in the chronic process of periglomerulitis chronica
  • End stage healing of damaged tissue leading to glomerulosclerosis
  • Neutrophil aggregation occurs as they move by chemotacis to the site of Ag-Ab complex
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15
Q

259

Nephrocirrhosis
Pathogenesis

A
  • Deposition of calcium salts, primarily calcium oxalate or calcium phosphate, within the renal parenchyma
  • Formation of crystals within the renal tubules impairs normal renal function
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16
Q

250

Nephrocirrhosis (Van Koss)
Pathogenesis

A
  • Deposition of calcium salts, primarily calcium oxalate or calcium phosphate, within the renal parenchyma
  • Formation of crystals within the renal tubules impairs normal renal function
17
Q

206

Meningitis Spinalis Purulenta
Pathogenesis

A
  • The infectious agents trigger an intense inflamatory response in the meninges, leading to accumulation of pus
  • This inflammation can result in spinal cord compression, vascular compromise and neurological symptoms
18
Q

159

Cysticercus ovis (brain)
Pathogenesis

A
  • The death of the parasite result from development of an immune response
  • The necrotizing larvocst is surrounded by giant cells and is accompanied by necrosis and meningiis with eosinophils, lymphocytes and macrophages
  • Anaohylactic reaction may occur as well
19
Q

207

Fibrosarcoma Uteri
Pathogenesis

A
  • Cells have different size and shape of nuclei, and general pleomorphism, malignacy is clear
  • Pathological mitosis signifies rapid tumour growth and increases metastatic potential
20
Q

242

Hypertrophy of Prostata
Pathogenesis

A
  • Division: Acinar with cyst formation due to overproduction of androgens or Fibromuscular due to estrogen overproduction
  • Consequences include infections, urinary bladder obstruction, hydronephrosis and constipation
21
Q

163

Dystrophia Musculorum
Pathogenesis

A
  • Equine paralytic myoglobinuria = energy production in muscle cells is altered, causing severe myopathy, myoglobinuria (dark) and potentially fatal renal faiilure
  • White muscle disease = increased oxidative damage due to a lack of suitable antioxidants (Se and Vit E) against FA oxidation, leading to degenerationof skeletal and cardiac muscle
22
Q

151

Trichinellosis Musculorum
Pathogenesis

A
  • Larvae invade the interstital wall, enter the bloodstream and migrate to skeletal muscles, where they encapsulate as cysts and inflammatory response follows
23
Q

109

Myositis Sarcosporidica
Pathogenesis

A
  • Ingestion of sporulated oocyst leads to sporozoite and cyst formation, causing an inflammatory response (myositis)
24
Q

273

Mastitis Apostematosa
Pathogenesis

A
  • Corpora amylacea is normally present in milk of multiparous cows only, as a result of synthetic and secretory processes
  • Has various staining properties
  • Bacteria enter mammary glands and multiply an immune respone leads to inflammation
25
Q

240

Tumor mixtus mammae
Pathogenesis

A
  • Slow growing tumour that may become malignant
  • Characterized by the presence of both epithelial and stromal components
26
Q

258

Dermatitis Eosinophilica
Pathogenesis

A
  • Mites burrow into the skin to feed, induce an inflammatory response
  • Leading to the formation of crusts and debris within the ear
  • Secondary bacterial infections may occur due to the compromised skin integrity
27
Q

237

Fibropapiloma
Pathogenesis

A
  • Different type of viruses, various manifestations of papillomas
  • Viral transmission occurs through direct contact or contaminates fomites

E-Pathanatomy (10 decks)

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