Sleep disorders Flashcards

1
Q

How long is short term insomnia?

A

A few days or weeks

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2
Q

How long is long term insomnia?

A

More than 4 weeks

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3
Q

What’s primary insomnia?

A

Not directly caused by another condition

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4
Q

What’s secondary insomnia?

A

Due to an underlying cause

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5
Q

What’s the study for teenage insomnia?

A

Roberts et al (2008) found its a major problem. 4000 Ps aged 11 and 17 from Texas. 25% had symptoms of insomnia, 5% said it affected their life

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6
Q

Evaluate primary vs secondary insomnia

A

Its important to be able to see which condition to treat. Ohayon and Roth found out of 15000 Europeans insomnia often proceeds mood disorders

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7
Q

What’s are the 4 consequences of insomnia?

A

Cognitive impairment, psychological disturbance, accidents, immune system

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8
Q

Study for the effects of insomnia on accidents?

A

Arendt found depriving someone of even 3 more hours sleep than normal had the same effects on driving as alcohol. There are 1500 deaths a year from sleep deprivation and driving.

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9
Q

Study for the effects of insomnia on the immune system

A

Savard et al found fewer immune cells in insomniacs. However could also be due to stress (Keicolt-Glaser)

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10
Q

Who devised the 3p model for insomnia

A

Spielman (1991)

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11
Q

3p model for insomnia: 1

A

Predisposing factors - Watson found 50% due to genetics. Could also be physical factors e.g. hyperarousal

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12
Q

3p model for insomnia: 2

A

precipitating factors - triggering events

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13
Q

3p model for insomnia: 3

A

Perpetuating factors - maintaining factors

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14
Q

Real-world application for insomnia

A

Using attribution theory peoples belief that they are going to have a bad sleep can be changed. Storms (1970) gave Ps a pill which caused ‘arousal’ or ‘sedation’. Those who had the first pill fell asleep faster because they attributed their arousal to the pill.

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15
Q

What are the 4 explanations for narcolepsy?

A

Psychological, REM, HLA, hypocretin

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16
Q

Narcolepsy: psychological explanation?

A

Lehman (1943) suggested sudden attacks of sleepiness cause sexual fantasies

17
Q

Narcolepsy: REM explanation

A

Cataplexy usually happens in REM

18
Q

Narcolepsy: HLA explanation

A

Honda et al (1983) increased frequency of one type of HLA (found in white blood cells) in narcoleptic. Found in 90% of narcoleptic with cataplexy.

19
Q

Narcolepsy: hypocretin explanation

A

These regulate sleep and wakefulness, usually there are 10,000-20,000 hypocretin-producing cells but in narcoleptic many of these are missing.

20
Q

Narcolepsy: REM evaluation

A

Vogel (1960) found in narcoleptics REM happened at the beginning of sleep rather than the end

21
Q

Narcolepsy: HLA evaluation

A

Not found in all narcoleptic, and can be found in non-narcoleptic people

22
Q

Narcolepsy: Hypocretin evaluation dog study

A

Lin et al (1999) found a mutation of chromosome 12 in narcoleptic dogs which disrupts hypocretin. Also found in humans

23
Q

Narcolepsy: Hypocretin evaluation causes

A

Minot (1998) found its unlikely to be genetic, more due to stress or illness.

24
Q

Narcolepsy: real-life application

A

Hyprocretin molecule is fragile and can’t yet be used for a treatment

25
Q

Sleep walking: 3 A01 points for sleep walking

A

Incomplete arousal, risk factors, childhood

26
Q

Sleep walking: what brain waves suggest incomplete arousal

A
Delta waves (SWS)
Beta waves (awake)
27
Q

Sleep walking: who studied the reason this happens in childhood

A

Olivier (2008) the system which usually inhibits motor activity during SWS is not developed in some children and adults