Skin and Soft tissue Flashcards

1
Q

inflammatory cells in the wall of hair follicle (no red patches of skin)

A

folliculitis

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2
Q

folliculitis risk factors

A

MC cause - shaving frequent/poor technique

also obesity, occlusive, hot temps, diabetes, abx, immunosuppression

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3
Q

folliculitis common pathogens

A

staph

pseudomonas (hot tub)

yeast (occasionally)

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4
Q

folliculitis treatment

A

gentle cleansing and hot compress

don’t pop

can use topical or oral abx (anti fungal if yeast) - Keflex, clindamycin, bactrim

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5
Q

non necrotizing inflammation of skin and subcutaneous tissue that doesn’t involve fasica/muscle

A

cellulitis

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6
Q

risks for development of cellulitis

A

untreated and infected breakdown of epithelium (laceration, bite, puncture)

may not have obvious portal of entry

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7
Q

cellulitis infecting pathogens

A

strep pyogenes or staph aureus

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8
Q

cellulitis s/s

A

localized swelling, tenderness, and erythema

slow developing

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9
Q

cellulitis tx

A

if there is no purulence or drainage = strep = dicloxacillin, amoxicillin, cephalexin

purulent = staph = bactrim or doxy

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10
Q

acute infection of upper dermis and superficial lymphatics

sharply demarcated border, systemic symptoms

A

erysipelas

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11
Q

erysipelas infecting pathogens

A

strep species

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12
Q

erysipelas ss

A

raised, well demarcated red region with raised border

acute onset, systemic symptoms

No purulence

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13
Q

risk factors for skin infection

A

elderly

DM

venous stasis and PAD

chronic liver and kidney disease

chronic steroids/immunodeficiency

IVDA

cancer

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14
Q

imp. aspects of skin infection

A

trauma

exposure

chronic disease and level of control

recent ABX use

prior pathogen

immunization status

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15
Q

skin infection exam

A

erythema, pain, swelling, warmth

asses for LAD and toxicity

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16
Q

outpatient cellulitis management

A

Abx

non-prulent (dicloxacillin, amoxicillin, cephalexin)

purulent cellulitis (bactrim, doxycycline)

5-10 days, f/w 48-72hrs

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17
Q

inpatient cellulitis antibiotic therapy

uncomplicated

A

cefazolin (Cephlex IV)

can use oxacillin or naficillin

MSSA coverage

18
Q

inpatient cellulitis antibiotic therapy

cellulitis with purulence

A

drainage, vancomycin

also ceftaroline, daptomycin

19
Q

inpatient cellulitis antibiotic therapy

aquatic environment

A

3rd/4th gen cephalosporin (ceftazidime, daptomycin)

or quinalone (Ciprofloxacin, levofloxacin)

20
Q

types of ulcers

A
  1. pressure ulcers
  2. stasis ulcers
  3. diabetic ulcers
21
Q

pressure ulcers

A

occurs at weight bearing sites

where skin is hypoxic and necrotic bc mechanical pressure > perfusion pressure

22
Q

stasis ulcers

A

poor circulation causes hypoxia and edema that predisposes to skin breakdown

23
Q

diabetic ulcers

A

occurs on foot and lower leg

caused by neurological factors (neuropathy, glucose toxic), vascular factors (Venous stasis/PAD), metabolic factors

24
Q

Stage 1 ulcer

A

pressure wound

push and reveals no blanching

25
stage 2 ulcer
dermis loss but no fat exposure raised edges with surrounding redness
26
stage 3 ulcer
visible globular fat molds
27
stage 4
can see all layers of dermis, skin visible tendons
28
DM ulcers treatment
wound care hyperbaric oxygen therapy surgical Treatment (debridement, revascularization) if infected inpt- Unyson, zosyn outpt- cephalexin, diclozacillin, bactrim + augmentin
29
deep infection of subcutaneous tissue that rapidly spreads along fascial planes
necrotizing fasciitis
30
necrotizing fasciitis pathogen
caused by B-hemolytic strep or staph often poly microbial
31
necrotizing fasciitis | risk factors
recent surgery or trauma DM and cirrhosis
32
necrotizing fasciitis presentation
initial injury is trivial but patient will be in significant pain w/o cellulitis dis proportionate pain for injury, clinical condition will deteriorate anesthesia of the skin
33
necrotizing fasciitis | diagnostic test
finger test anesthesia of skin, make 2cm incision, probe with finger passes directly thru SubQ tissue w/o muscle
34
necrotizing fasciitis care
immediate or debridement of all necrotic tissue
35
animal bites most often occur
late on summer afternoons most animals are known to victim
36
animal bites dog, tx
typically force injury so crush component may not be immediately apparent staph, strep, eikenella, pasteurella polymicrobial bite
37
animal bites cat tx
scratches or punctures likely to become infected pastuerella, actinomycetes, propionibacterium, bactericides
38
animal bite treatment wound
copious irrigation, debridement of dead tissue, exploration of the wound primary closure if they are cleaned if not completely cleaned secondary intuition
39
animal bite DOC
augmentin prophylactic 3-5 days, 10+ days
40
clenched fist injury
high risk of infection tendon moves into hand an infection gets deep into the skin
41
human bite treatments
excellent cleansing with irrigation and debridement ABX prophylaxis for all human bite wounds w/Augmentin, Moxifloxicin, or Unasyn
42
human bit wounds should not be closed:
obvious infection >12 hrs old puncture wounds or wounds to hand