Sexual Behavior Flashcards

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1
Q

What are the advantages of sexual reproduction? Why do we have sex instead of asexual reproduction?

A

+Decrease pathogens and limit mutations

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2
Q

How many sets of chromosomes do we have?

How many sets of autosomes?

How many sets of sex chromosomes?

A

23 sets of chromosomes
22 sets of autosomes
1 set of sex chromosome

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3
Q

What type of sex chromosome (genotypes) are females vs. males? (XX vs. XY)

A
Females = XX
Males = XY
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4
Q

Which chromosome determines sex?

X or Y?

A

Y - in a vacuum, we can say males determine sex of baby

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5
Q

Up to the ___ week, males and females have undifferentiated primordial gonads.

What happens after this week?

A

Up to the 6th week…

If the cell contains a Y-chromosome, at about the 6th week, the sex-determining region of the Y chromosome (SRY) gene is expressed in MALES.

When the SRY gene is expressed, it encodes the Testes Determining Factor, and turns the gonad tissues into Testes.

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6
Q

When the SRY gene is expressed, what does it encode?

A

It encodes the Testes determining factor.

It turns the gonads into Testes.

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7
Q

What happens once the gonads have been determined as either Testes or Ovaries?

A

If Testes, it produces testosterone.

If Ovaries, they do NOTHING.

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8
Q

What are the 2 systems that make up the internal sex organs?

A
  1. Wolffian system - Male, develops into epididymis and the seminal vesicles (ball sack).
  2. Mullerian system - Female, develops into fimbria, fallopian tubes, uterus, inner 2/3 vagina
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9
Q

What happens after the gonadal tissue develops into testes?

*2 things that have to happen in order to develop into a male…

A

2 things will happen:

  1. It starts to produce testosterone. That testosterone will stimulate the Wolffian system to develop.
  2. It will also secrete ANTI-MULLERIAN hormone to suppress the Mullerian system.
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10
Q

What happens if you don’t have the anti-mullerian hormone but have testosterone, what system will you get?

A

You get development of both systems for internal organs.

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11
Q

What happens if you have anti-mullerian hormone, but don’t have testosterone, what system will you get?

A

You will get no internal sex organs.

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12
Q

Describe the chromosomal malfunction of Turner’s syndrome.

What type of sex organs do they have/not have?

A

Condition that results from either an incomplete pairing of gametes, or pairing of X with O gamete - O gametes have no sex chromosomes (XO).

There is no gonads (No Y) or ovaries (need both XX).

However, Mullerian system develops so the internal and external sex organs are female.

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13
Q

In the absence of any hormones, what’s the default form?

A

Females - nature favors the female.

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14
Q

Can ppl with Turner’s syndrome get pregnant?

A

They cannot have biological children bc they don’t have gonads - but they CAN carry babies to term with embryo implants and hormone supplementation bc they have the internal (uterus) and external organs.

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15
Q

What type of genotype do Klinefelter syndrome patients have?

What type of internal and external sex characteristics?

A

XXY - due to the SRY gene of the Y chromosome, they develop testes.

Internal and External = Male characteristics

They do sometimes have feminine characteristics (pubic hair, fat distribution)

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16
Q

What type of genotype do Jacob’s syndrome patients have?

How did they get that genotype?

What type of internal and external sex characteristics?

What are some of non-sexual symptoms of Jacob’s syndrome?

A

XYY

Due to problems during fertilization - There’s 2 Y’s instead of 2 X’s…

Produce Testes Determining Factor due to their Y chromosome - Internal & External male characteristics.

Acne, tallness, lean… fertility issues.

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17
Q

What were inmates with XXY or Jacob’s syndrome in jail for?

Why does this matter?

A

They were in jail for things like theft… it was mostly NON-VIOLENT crimes - this disproved the theory that an extra Y chrom. made them more aggressive (due to excess testosterone).

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18
Q

What type of genotype do Androgen-Insensitivity Syndrome (AIS) patients have?

What type of internal and external sex characteristics?

A

XY genotype - BUT defective Testosterone receptors.

XY = Gonads become Testes and secrete Testosterone and Anti-Mullerian hormone BUT bc of the defective Testosterone receptors, there is NO development of the Wolffian system. The testes don’t descend. - AIS - They HAVE NO INTERNAL SEXUAL ORGANS.

Since Wolffian system is not stimulated, it withers away.

Anti-Mullerian hormone IS recognized and suppressed-so there is no Internal sex organs, but externally develop female sex organs since androgens are not recognized- default to female form.

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19
Q

What do you need to develop external genitalia into male sex pattern?

A

Androgens (testosterone)

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20
Q

If someone fails to menstruate or grow secondary hairs during puberty - what type of syndrome might they have and why?

A

They probably have AIS - androgen-insensitivity syndrome. They do not have internal female sex organs.

The secondary hair factor is controlled by testosterone - and AIS is insensitive to testosterone…

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21
Q

What happened during development inCongenital Adrenal Hyperplasia (CAH)?

What type of internal and external sex characteristics?

*Testosterone’s effect…

A

Heritable condition in which the fetus’s adrenal glands, NOT THE TESTES, release excess androgens.

INTERNAL GENITALIA: The extra testosterone in females, causes Wolffian system to not completely wither away (causing issues with reproduction) - AND gonads are set as ovaries because it developed before the testosterone was expressed by the adrenal glands.

Since there is NO testes and the Anti-Mullerian system is inactive- the Mullerian system develops by itself - PLUS a little dash of Testosterone

External Genitalia = Females can get ambiguous genitals (ex: micropenis, vagina and penis)

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22
Q

How are prenatal effects organized?

A

Prenatal effects of testosterone (no prenatal estrogen) organizes gonadal, internal, and external tissue to develop in certain ways.

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23
Q

What is happening during activation effects during puberty?

A

Activation effects - Not permanent, only see effects when the hormones are high (ex: females with higher estrogen levels will cause them to eat more; etc).

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24
Q

When does secondary sexual characteristics develop?

What are the secondary characteristics

A

During puberty.

\+Increased hairs
\+Fat deposition patterns change; males = trunk, females = butt, thighs, breast
\+Bone growth
\+Males = Muscles
\+Females = Menstrual
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25
Q

How old do females go into puberty now?

What are some explanations as to why females go into puberty earlier now than at 200 years ago?

A

+9 or 10 years old.

+Fat tissues - we are getting fatter earlier, so we can sustain pregnancies.

+RBST, estrogen hormones.

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26
Q

How is puberty started, neurochemically?

What do these chemicals do for females vs. males?

A

+Begins with the hypothalamus releasing the GnRH (gonadotropin-releasing hormone).

+Then the GnRH signals to the Anterior Pituitary gland to produce FSH (follicle-stimulating hormone) and LH (luteinizing hormone).

> Released in BOTH males and females.

+Females = begin menstrual cycles by stimulating ovaries (egg-making machines)

+Males = FSH increases sperm production.

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27
Q

What is FSH and LH’s role in males?

What does their initial production of testosterone do?

A

> FSH and LH increases gamete production (sperm) through increased production of Testosterone.

> Initial production of Testosterone promotes the development of male sec. sex. characteristics, such as muscle mass, external genitalia maturity, regulation of sperm production.

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28
Q

Why would males have a weaker immune system than females?

What does this say about the theory of sex attraction?

A

Testosterone weakens the immune system - It decreases the T and B cells, making males more vulnerable to disease.

The theory of sex attraction states that females are attracted to males with big muscles and strong jaws - They are signals of High Testosterone levels, and the advantages is that he probably has a kick-ass immune system.

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29
Q

Males with strong jaws, deep voices; etc means what for potential mates?

A

He has a high testosterone level - he must have a great immune system because he’s been able to survive this long even with all this testosterone = awesome mate.

30
Q

What is FSH and LH’s role in females?

What does their initial production of testosterone do?

A

The FSH and LH stimulates the ovaries to produce Estradiol.

The initial production of Estradiol promotes sec. sexual characteristics; breast, maturity of uterus, fat distribution; etc.

31
Q

What does Estradiol do for skeletal growth in both male and females?

A

It halts skeletal growth - since males have less estradiol, they’re not affected as much and keep growing.

Females will be the same height as when they first enter puberty.

32
Q

Why would entering into puberty earlier have anything to do with how short females are?

A

Due to release of estradiol by the FSH and LH during puberty, females will stop growing. So the earlier they go into puberty, the shorter they are because Estradiol halts skeletal growth.

33
Q

Concerning 5-Alpha-Reductase Deficiency:

What is the hormone that changes the bi-potential genitalia into the male pattern?

A

It is dihydrotestosterone that stimulates dev. of male genitalia by the way of: 5-alpha-reductase converts testosterone to 5-alpha-dihydrotestosterone.

34
Q

What happens when someone doesn’t produce enough 5-alpha reductase prenatally?

*Internal and external genitalia.

What happens once they hit puberty?

A

During prenatal development, there’s not enough dihydrotestosterone to stimulate external genital growth - so there’s ambiguous genital patterns like CAH, or just get female looking genitalia.

But, if they’re producing testosterone, but NOT dihydrotestosterone, their internal sex organs are male thanks to the Wolffian (just need testosterone) and Anti-mullerian hormones (from testes).

+Once they hit puberty, testes start producing enough testosterone that produces dihydrotestosterone, it changes the secondary sex characteristics of the external genitalia to the MALE pattern.

35
Q

What are sex hormones synthesized from?

What’s the order of synthesis?

A

Cholesterol to
Progesterone to
Testosterone

> Testosterone is converted into Estradiol by way of Aromatase - how males get Estradiol.

36
Q

What is the function of Aromatase?

A

To convert Testosterone into Estradiol.

37
Q

If the hypothalamus releases GnRH, which tells the pituitary gland to produce FSH and LH - what tells the hypothalamus to produce the GnRH?

A
  • Melatonin; it inhibits GnRH
  • Light inhibits Melatonin
  • When Melatonin levels are high (more dark than light; puberty is inhibited bc GnRH, FSH, and LH is inhibited).
38
Q

Why do we see growth spurts in the summer?

A

Melatonin levels are lower - so GnRH, FSH, and LH is higher - allowing kids to grow.

39
Q

Describe day 1 of the menstrual cycle.

A

-Day 1: The anterior pituitary gland release of FSH (follicle stimulating hormone) -

40
Q

How is the interaction of the FSH after day 1 of the menstrual cycle?

  • Corpus Luteum
  • Estradiol
  • Progesterone
A

Day 1 - the FSH is active, and now the follicles grow. One follicle matures exceptionally faster and releases Estradiol in proportion to how large they are - Estradiol has inhibitory effect on growth of other follicles.

This signals back to LH and LH shoots up and causes the rupture of the mature Ovum from ovary (Ovulation). This ruptured follicle becomes Corpus Luteum.

Corpus Luteum release Estradiol AND NOW Progesterone.

The Estradiol and Progesterone prep the uterine wall thicken in prep for implantation.

If fertilization doesn’t occur, the corpus luteum stops producing Estradiol and Progesterone.

The uterus then sheds its lining and cycle repeats - and if embryo does attach, it tells the Hypothalamus to continue to leach out Estradiol and Progesterone throughout pregnancy.

41
Q

Why do women have fluctuations in mood?

A

Fluctuations in estradiol correlate with mood.

Higher estrogen = Positive mood

Lower estrogen = Negative mood

42
Q

How is estrogen levels positively correlated with Serotonin levels?

A

Estrogen affects serotonin levels; sex differences between depression - females are more likely to be diagnosed than males.

43
Q

Why does exclusive breastfeeding with no menstruation correlated with reduced fertility?

A

-Higher lactation rates and on-demand suckling keeps hormone levels high enough to stop ovulation

44
Q

What is the difference between Estrus and Menstrual cycle?

A

Estrus cycle is a specific time when females are receptive to males vs. those on menses are always participating in sex activity.

45
Q

What’s the difference between receptive and proceptive?

A

Proceptive is seeing how likely someone is to initiate an act - Around ovulation (highest estradiol and testosterone levels), proceptiveness is higher.

46
Q

Does estrogen or testosterone have higher influence on female sexual interest?

A

Testosterone; proceptive levels are due to testosterone.

47
Q

Testosterone drives ____.

Estradiol drives ________.

A

Testosterone drives sex.

Estradiol drives conception.

48
Q

Why are females less likely to get degenerative diseases?

When are they better at cognitive tasks?

When are they better at spacial tasks?

A

Estrogen - has neuroprotective effects - also more plastic than males.

Dendritic spines in hippocampus and prefrontal cortex are increased when estradiol levels are high

  • Cog tests show that higher estradiol levels = better memory, verbal fluency, manual dexterity, and decision tasks.

Better at spacial tasks when testosterone levels are high (ovulation), and worse when estrogen levels are high (right before ovulation).

49
Q

When is estrogen’s biggest peak?

A

Before ovulation and pre-menstrual.

50
Q

Why did the scientists beard grow longer after going out to the mainland?

A

His anticipation of sex activity with women and competition.

51
Q

Females and males in Long-term marriage have what levels of testosterone? Why do we theorize this?

A

Lower testosterone levels - no longer competing for sex, more availability.

52
Q

T or F. Testosterone predicts sex frequency.

A

False - you might be more interested, but you aren’t having more sex, obvi.

53
Q

Who will do better during a mental rotation task: A female who is ovulating or an 80 y.o male?

A

A female who is ovulating; she has higher testosterone levels than the 80 y.o man.

54
Q

What is a legit reason to use steroids?

A

+Muscular dystrophies

+Ashtma - to build up muscles of diaphragm.

55
Q

What masculinizes the male brain?

A

Estradiol resulting from aromatization masculinizes the brain.

56
Q

Why don’t we all have male brains, since Estradiol makes the male brain… and our prenatal environment is full of estrogen?

A

We don’t because the placenta has enzymes that block out the mom’s estradiol from affecting the fetus.

57
Q

What happens when a female fetus is exposed to synthetic estrogen?

How do females id themselves when exposed to DES?

A

DES masculinizes the female brain and is not blocked by the placenta.

Higher likelihood to id themselves as lesbian but not males.

58
Q

If an XX individual gets prenatal exposure to androgens, but given estrogen and progesterone at puberty - what will happen?

A

Not normative female sexual behavior because the substrate of the organizational effect is not there; essentially an XY.

You need the correct substrate for activational effects.

59
Q

Does the brain differ bw males and females?

A

Yes, there are areas in the hypothalamus…

Commissures are thicker in females = more connections than males.

60
Q

How are sexual orientations mediated?

A

Hormonally

61
Q

What type of sexual orientation and behavior would a female who was exposed to high levels of prenatal androgens engage in?

A

They would exhibit male sex behaviors and approach females - more bi and lesbian behaviors… but majority are not exposed to prenatal androgens so it’s difficult to say this is the reason behind it.

62
Q

Explain how otoacoustic emissions show the difference bw lesbian women and straight women.

A

Lesbian women appear to have otoacoustic emissions that fall between heterosexual male and female.

63
Q

Which area of the brain is important for performance on sexual motivation in both males and females?

What NT is important for this?

A

Dopamine activity in the MPOA

64
Q

What levels of dopamine and serotonin is good for sex drive?

A

High levels of dopamine but low levels of seratonin

65
Q

What does bursts of oxytocin do?

A

Social recognition - we are more friendly towards people.

66
Q

Why are prairie voles monogamous vs. the ho-bags meadow and mountain voles?

A

Prairie voles are more sensitive to vasopressin.

67
Q

What are some pheromonal influence on humans?

A

Although odorless - higher attractiveness rating by men of odor of sweat from women during ovulation.

Higher test/est = more attractive

Amygdala activation when smelling first-time sky-divers

68
Q

Describe the differences between the following:

Sex
Gender
Gender role
Gender identity

A

Sex is biological characteristics

Gender is cultural conception

Gender role is social

Gender id is due to person’s subjective feeling

69
Q

What is sex assignment based on now?

A

Genotype test - because of prenatal hormones in the brain that have been adopted.

70
Q

How many percent of men and women have engaged in same sex activity since puberty?

How many describe themselves as gay or bisexual?

A

9% of men and 4% of women engaged in sam sex activity.

Only 2.8% men and 1.4% women say they’re gay or bi.