Schizophrenia: Paper 3 Flashcards

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1
Q

What is Schizophrenia?

What can it lead to?

A

A group of disorders whereby people interpret reality abnormally.

It can result in a combination of hallucinations, delusions and extremely disordered thinking and behaviour that can impair daily functioning.

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2
Q

What are positive symptoms?

A

Positive symptoms enhance the typical experience of schizophrenic sufferers and occur in addition to their normal experiences such as hallucinations and delusions.

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3
Q

What are negative symptoms?

A

Negative symptoms take away from the typical experience of schizophrenic sufferers, and so represents a ‘loss’ of experience e.g. speech poverty and avolition.

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4
Q

What are auditory hallucinations and what type of symptom it?

A

It is a positive symptom in which the individual may hear voices in the person’s mind either talking each other or to the person.

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5
Q

What are delusions, can you give all the types of delusions?

Is it a positive or negative symptom?

A

Positive symptom!

Delusions are false beliefs which remain despite confirmation or otherwise by logic.

This means that people with delusions cannot distinguish between their private thoughts and external reality such as:

  • Delusions of Grandeur, which refers to a false impression of one’s own importance.
  • Delusions of Persecution, an irrational belief that others are trying to plot against or kill the individual.
  • Delusions of Control, the false belief that another person, group of people or external forces like aliens are trying to take control of one’s thoughts, feelings, impulses or behaviours.
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6
Q

What is affective flattening?

What type of symptom is it?

A

It is a negative symptom!

A reduction in the range and intensity of emotional expression such as having a lack of body language.

For example, fewer body and facial movements.

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7
Q

What is anhedonia?

Is it a positive or negative symptom?

If you think you’re cold can you tell me the difference between two different types of anhedonia, and why one is perhaps more reliable than the other?

A

It is a negative symptom!

Anhedonia refers to a loss of pleasure or interest in all or almost all activities or a lack of reactivity to normally pleasurable stimuli.

Physical anhedonia refers to the inability to experience physical pleasures such as from food whereas social anhedonia refers to the inability to experience pleasure from interpersonal situations such as interacting with other people.

Some may say that physical anhedonia is considered a more reliable symptom of schizophrenia because it does not overlap with other disorders like depression unlike social anhedonia.

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8
Q

What is avolition?

Is it a positive or negative symptom?

A

It is a negative symptoms!

Avolition refers to a reduction of interests and desires as well as an inability to initiate and persist in goal-directed behaviour such as not taking showers.

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9
Q

What is speech poverty?

Is it a positive or negative symptom?

A

It is a negative symptom!

Speech poverty is a lessening of speech fluency and productivity.

It is thought to reflect slowing or blocked thoughts. For example, fewer words may be produced in a given time on a task of verbal fluency.

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10
Q

What are the main differences between classification of schizophrenia in the DSM-5 and the ICD-10?

A

For the DSM 5 - there most be at least one positive symptom. It is used in America.

Whereas…

ICD-10 - Two or more negative symptoms are sufficient for diagnosis. It is used worldwide.

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11
Q

Discuss issues of reliability and validity associated with the classification and/or diagnosis of schizophrenia. (16 marks)

A
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12
Q

What is Diagnostic Reliability?

A01: Classification and/or diagnosis of schizophrenia

A

Diagnostic Reliability means that a diagnosis of schizophrenia must be repeatable.

This means that different clinicians using the same system (such as DSM-5) should arrive at the same diagnosis:

  • for the two clinicians studying the same schizophrenic patient (inter-rater reliability).
  • for testing schizophrenic patients at two different points in time (test-retest reliability).
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13
Q

What is cultural bias in schizophrenic and how can this limit the reliability and validity of diagnosing schizophrenia?

A01: Classification and/or diagnosis of schizophrenia

A

Cultural bias in schizophrenia refers to how the cultural assumptions of psychiatrists have an impact on their diagnosis of people from different cultural backgrounds or origins with Schizophrenia.

If diagnosis is not consistent among different cultures, the reliability of diagnosis of schizophrenia can be questioned.

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14
Q

What is validity and how does this relate to the diagnosis in schizophrenia?

A01: Classification and/or diagnosis of schizophrenia

A

Validity refers to the extent that the diagnosis of schizophrenia represents something that is real and distinct from other disorders and the extent that a classification system such as DSM 5 measures what it sets out to measure.

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15
Q

How can gender bias relate to the diagnosis of schizophrenia?

A01: Classification and/or diagnosis of schizophrenia

A

Gender bias in the diagnosis of schizophrenia refers to the accuracy of the diagnosis being dependent on the individual’s gender.

This is because the accuracy of diagnostic judgements can vary for a number of reasons such as clinicians basing their judgements on stereotypical beliefs held about a gender.

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16
Q

How do different manuals diagnose schizophrenia differently?

A01: Classification and/or diagnosis of schizophrenia

A

There are different diagnosing manuals that look for different characteristics meaning that diagnosis may depend on the manual used.

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17
Q

What is co-morbidity, and how can it affect diagnosis of schizophrenia?

A01: Classification and/or diagnosis of schizophrenia

A

Co-morbidity describes people who suffer from two or more mental disorders.

Psychiatric co-morbidities are common among patients with schizophrenia.

This is a problem which could decrease the validity when diagnosing schizophrenia because co morbidity makes it more difficult to confidently diagnose schizophrenia.

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18
Q

Cultural bias: Tendency to over diagnose members of other cultures.

Do you know any research support for this statement?

A03: Classification and/or diagnosis of schizophrenia

A

Research shows that there is a tendency to over diagnose members of other cultures.

For example, Cochrane found that the incidence of schizophrenia in the West Indies is similar to the UK at 1% but found that people of Afro-Caribbean origin are seven times more likely to be diagnosed with schizophrenia when living in the UK

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19
Q

Why is this a problem for diagnosing schizophrenia?

(to achieve a second mark)

A03: Classification and/or diagnosis of schizophrenia

A

This is a problem because the reason for this may be due to the original construction of the diagnosis system in the West now being applied to culturally different people means that the validity of diagnosing schizophrenia can be questioned because it does not account for cultural differences, thus leading to an inaccurate diagnosis of schizophrenia.

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20
Q

High-inter-rater reliability…

A03: Classification and/or diagnosis of schizophrenia

A

One strength of the diagnosis of schizophrenia is that using the DSM classification appears to have high inter-rater reliability over time.

This is a strength because research support from Beck found 54% concordance between experienced practitioners diagnoses when assessing 153 patients using the DSM classification system, while Soderberg reported an 81% concordance rate of schizophrenia.

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21
Q

How does this support the reliability of diagnosing schizophrenia?

A03: Classification and/or diagnosis of schizophrenia

A

This supports the notion that schizophrenia can be diagnosed consistently over time and thus diagnosis of schizophrenia has become more reliable.

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22
Q

Gender bias in diagnosing schizophrenia

A03: Classification and/or diagnosis of schizophrenia

A

This is a limitation because men have consistently been diagnosed with schizophrenia more than woman.

Research support to explain this comes from Cotton et al who found female patients with negative symptoms of schizophrenia can typically function better than men- and therefore escape diagnosis of schizophrenia.

Moreover, Loring and Powell found that some behaviours which was regarded as psychotic in males were not regarded as psychotic in females.

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23
Q

How does this limit the reliability of diagnosing schizophrenia?

A03: Classification and/or diagnosis of schizophrenia

A

Therefore, the reliability of the diagnosis of schizophrenia is low because men and women with the same symptoms may get different diagnoses, meaning that diagnosis is not consistent.

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24
Q

How can different manuals used show that there is lack of validity in diagnosing schizophrenia?

A03: Classification and/or diagnosis of schizophrenia

A

If there was consistency, then they would’ve diagnosed the same number of people using both diagnostic tools, and yet, they varied.

This shows a lack of validity as different diagnostic tools will diagnose people differently, so one patient may be diagnosed as schizophrenic under the DSM, but not the ICD.

This means that some people will be misdiagnosed, or not diagnosed at all, and therefore have the potential to miss out of the appropriate treatment

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25
Q

Contradictory research support for inter-rater reliability

A03: Classification and/or diagnosis of schizophrenia

A

Cheniaux et al (2009) found that inter-rater reliability amongst two psychiatrists was low.

One diagnosed 26 out of 100 patients with schizophrenia using the DSM, and 44 out of 100 using the ICD. The other diagnosed 13 using the DSM and 24 using the ICD.

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26
Q

How can co-morbidity undermine the usefulness of diagnosing schizophrenia?

A03: Classification and/or diagnosis of schizophrenia

A

Research support from Buckley found that 50% of the patients diagnosed with schizophrenia also were diagnosed with depression.

This is a limitation because it undermines the usefulness of diagnosis and classification systems of schizophrenia.

This is because if half as many patients with schizophrenia also have severe depression it suggests that schizophrenia could be one single condition, affecting the validity of diagnosing schizophrenia.

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27
Q

Research support from Loring and Powell for existence of gender and cultural bias

A03: Classification and/or diagnosis of schizophrenia

A

Loring and Powell sent 290 psychiatrists two identical case studies, the psychiatrists changed the gender and race of the case studies to different genders and races and a control situation in which no gender or race is disclosed.

The researchers found an over diagnosis of black case studies and under-diagnosis of female case studies.

When the race and gender of the psychiatrist was the same, an accurate diagnosis was achieved.

This suggests the existence of both gender and cultural bias in psychiatrists diagnosis of schizophrenia.

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28
Q

Outline and Evaluate the Biological Explanations of Schizophrenia. (16 marks)

A
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29
Q

How are twin studies used to explain why people inherit schizophrenia?

A01: Biological Explanations of Schizophrenia

A

One biological explanation is that schizophrenia is inherited/passed on through twin studies.

By comparing the concordance rates between MZ and DZ twins we can determine the likelihood of genetic inheritance of schizophrenia.

If MZ twins have a higher concordance rate than DZ twins, we can determine that schizophrenia was inherited.

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30
Q

What does the Dopamine Hypothesis theory propose about people with schizophrenia?

A01: Biological Explanations of Schizophrenia

A

The Dopamine Hypothesis theory suggests that symptoms of schizophrenia are associated with an imbalance of the dopamine neurotransmitter across the brain.

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31
Q

What is the difference betweem ‘hyperdopaminergia’ and ‘hypodopamingeria’?

A01: Biological Explanations of Schizophrenia

A

Excessive amounts of dopamine (this is called hyperdopaminergia)

in speech areas such as the Broca’s area could lead to positive symptoms such as auditory hallucinations.

WHEREAS…

Lower levels of dopamine (this is called hypodopaminergia) in areas like the frontal cortex lead to negative symptoms such as avolition or speech poverty.

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32
Q

Drugs been developed to treat schizophrenic symptoms…

Tell me how amphetamines treat schizophrenic symptoms…

A01: Biological Explanations of Schizophrenia

A

Amphetamines increases dopaminergic activity by stimulating nerve cells containing dopamine, flooding the synapse.

It has been found that giving normal individuals large doses of dopamine releasing drugs such as amphetamines leads to schizophrenic symptoms such as hallucinations and delusions.

Thus, amphetamines have been used to treat schizophrenics suffering from negative symptoms.

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33
Q

Drugs been developed to treat schizophrenic symptoms…

Tell me how antipsychotics treat schizophrenic symptoms…

A01: Biological Explanations of Schizophrenia

A

Antipsychotics decreases dopaminergic activity.

By reducing the activity in the neural pathways of the brain that use dopamine as the neurotransmitter, this leads to the elimination of these positive schizophrenic symptoms such as hallucinations.

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34
Q

Socially sensitive, perhaps other approaches may be more suitable in explaining schizophrenia…

(2 marks)

A03: Biological Explanations of Schizophrenia

A

A biological approach to explaining schizophrenia, such as a genetic basis assumes that schizophrenia is inevitable, or biologically determined.

Potentially making sufferers feel disempowered when diagnosed.

Other more psychological approaches such as the cognitive approach has a soft determinist perspective suggesting that mental processes can be altered or managed via free will to control the disorer.

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35
Q

How has the development of psychotic drugs helped the economy?

A03: Biological Explanations of Schizophrenia

A

This has positive implications for the economy because it means patients can return to their homes, saving the NHS money on hospitalisations for schizophrenia, reducing the burden on the NHS (with already very long waiting times).

It also allows patients to go back to work and contribute to the economy.

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36
Q

Biological reductionism is highly scientific and has practical applications…

A03: Biological Explanations of Schizophrenia

A

Explaining schizophrenia at the basic cellular and chemical level has the advantage of the scientific principle of parsimony.

This has allowed for the development of antipsychotic drugs, which are the primary course of treatment for this condition.

These drugs are indispensable treatment for schizophrenia and the fact that they have dramatically improved the lives of schizophrenic sufferers and their families is a real strength of the dopamine hypothesis.

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37
Q

Research support for implications of dopamine in causing schizophrenia through effectiveness of drugs…

A03: Biological Explanations of Schizophrenia

A

Barlow & Durand (1995) report that chlorpromazine is effective in reducing schizophrenic symptoms in about 60% of cases. The drug blocks dopaminergic activity receptors and reduces its levels.

Clearly shows that dopaminergic activity is implicated in schizophrenia because the study demonstrates that using a dopaminergic activity blocking drug like chlorpromazine is effective in treating schizophrenia in many cases.

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38
Q

Genetic mutations support the genetic explanation of schizophrenia

A03: Biological Explanations of Schizophrenia

A

This is because it is possible for people to develop schizophrenia even in the absence of a family history of the disorder.

This could be through mutation in the paternal DNA in the sperm because of mutagenic agents.

Brown et al found a relationship between paternal age (which is linked with an increased risk of mutation) and the risk of developing schizophrenia. He found that in men under 25 the risk was 0.7% and men over 50 was 2%.

Therefore, the older the father, the higher the risk that the child develops schizophrenia, supporting the theory of the genetic explanation

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39
Q

Other factors such as those in the environment may have played a role in schizophrenia…

A03: Biological Explanations of Schizophrenia

A

The fact that the concordance rates are not 100% means that schizophrenia cannot be solely explained by genes and therefore the environment could have also played a role in the development of schizophrenia.

This means that the biological explanation of schizophrenia does not give a full explanation of the schizophrenia disorder.

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40
Q

Taking an interactionist approach may be necessary…

A03: Biological Explanations of Schizophrenia

A

Alternatively, the diathesis stress model may be a more suitable explanation to explain schizophrenia as it combines both biological and psychological factors in the development of schizophrenia, providing a more holistic approach to explaining schizophrenia.

This is because this explanation considers not only the development of schizophrenia determined by a biological predisposition but also the environmental stressors within an individual’s lives that trigger this vulnerability, causing schizophrenia.

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41
Q

Gottesman and Shield: Research support from twin studies

A03: Biological Explanations of Schizophrenia

A

Gottesman reviewed cases of schizophrenia and found a concordance rate of 48% for MZ twins and 17% for DZ twins.

This suggests that schizophrenia has a genetic aspect because MZ twins had a significantly higher concordance rate than DZ twins.

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42
Q

Research support from effectiveness of antipsychotic drugs.

A03: Biological Explanations of Schizophrenia

A

Leucht et al carried out a meta-analysis of 212 studies which analysed the effectiveness of different antipsychotic drugs compared with a placebo.

They found all drugs tested were significantly more effective than the placebo in the treatment of positive and negative symptoms.

The effectiveness suggests that dopamine does play an important role in the development of schizophrenia.

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43
Q

Research support from Cognitive Neuroscience

A03: Biological Explanations of Schizophrenia

A

Kesler et al (2000) used MRI and PET scans to compare schizophrenic and non-schizophrenic participants. They found differences in dopamine levels suggesting that dopamine is an important factor in the onset of schizophrenia.

This study supports the dopamine strand as the finding shows that dopamine levels are different in non-schizophrenics and schizophrenics.

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44
Q

Outline and discuss the neural correlates explanation of schizophrenia. (8 marks)

A
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45
Q

What are neural correlates?

A01: Neural correlates explanation of schizophrenia

A

Neural correlates are patterns of structure or activity in the brain that occur in conjunction with schizophrenia

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46
Q

What are ventricles and how can this relate to schizophrenia?

A01: Neural correlates explanation of schizophrenia

A

Ventricles are fluid filled cavities (i.e. holes) in the brain that supply nutrients and remove waste.

It has been found that the ventricles of a person with schizophrenia are on average about 15% bigger than normal (Torrey, 2002).

Researchers have also found that many people with schizophrenia, particularly those displaying negative symptoms have enlarged ventricles.

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47
Q

What is the difference betweem ‘hyperdopaminergia’ and ‘hypodopamingeria’?

How can they produce schizophrenic symptoms?

Tested again, to see if you remember previous flashcard :)

A01: Neural correlates explanation of schizophrenia

A

Excessive amounts of dopamine (this is called hyperdopaminergia)

in speech areas such as the Broca’s area could lead to positive symptoms such as auditory hallucinations.

WHEREAS…

Lower levels of dopamine (this is called hypodopaminergia) in areas like the frontal cortex lead to negative symptoms such as avolition or speech poverty.

Current understanding of the role of dopamine in schizophrenia is that both hyperdopaminergia and hypodopaminergia may be at work in different brain areas to produce schizophrenia

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48
Q

Biological determinism & social sensitivity.

A03: Neural correlates explanation of schizophrenia

A

A neural correlates explanation of schizophrenia is an example of biological determinism as it assumes that specific brain structures and/or dopaminergic activity in the brain play a key role in the onset of schizophrenia

This suggests that the dopamine hypothesis does not account for freewill and could potentially make sufferers feel disempowered when diagnosed as they feel that they cannot do anything about their illness.

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49
Q

Most research into neural correlates is correlational…

A03: Neural correlates explanation of schizophrenia

A

The research evidence for neural correlates takes (as the name suggests) a correlational approach to mapping brain regions to schizophrenia which means that it lacks a cause-effect explanation

There is no acknowledgement of the role of the environment to a neural correlates-based explanation of schizophrenia which means that it lacks external validity

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50
Q

Neural correlates explanation is highly scientific…

A03: Neural correlates explanation of schizophrenia

A

One strength is that the biological explanation of schizophrenia using neural corelates uses highly scientific procedures and standardised procedures.

These machines take accurate readings of brain regions such as the frontal and pre-frontal cortex, the basil ganglia, the hippocampus and the amygdale.

This suggests that if this research was tested and re-tested the same results would be achieved, high test-retest reliability

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51
Q

Research support for enlarged ventricles

A03: Neural correlates explanation of schizophrenia

A

Supporting evidence for the brain structure explanation comes from further empirical support from Suddath et al. (1990).

He used MRI (magnetic resonance imaging) to obtain pictures of the brain structure of MZ twins in which one twin was schizophrenic.

The schizophrenic twin generally had more enlarged ventricles and a reduced anterior hypothalamus. The differences were so large the schizophrenic twins could be easily identified from the brain images in 12 out of 15 pairs.

This suggests that there is wider academic credibility for enlarged ventricles determining the likelihood of schizophrenia developing.

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52
Q

Discuss psychological explanations of schizophrenia (16 marks)

A
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53
Q

What do explanations based on family dysfunction explanations claim?

A01: Psychological explanations of schizophrenia

A

Explanations based on family dysfunction claim that schizophrenia is caused by abnormal patterns of communication within the family.

It is argued that poor relationships and communication in families increase the risk of developing schizophrenia.

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54
Q

What are mixed messages in relation to schizophrenia?

A01: Psychological explanations of schizophrenia

A

Parent gives contradictory messages by verbally communicating a message but non-verbally rejecting another message.

When the child ‘disobeys’, the parent punishes the child by withdrawing their love.

As a result of these mixed messages from parents, the child fears voicing objections about their unfair treatment and becomes confused and doubtful.

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55
Q

Can you provide an example of ‘Mixed Messages’ and what psychologists have suggested about how it leads to the development of schizophrenia?

A01: Psychological explanations of schizophrenia

A

An example of mixed messages would be telling a child that they need to be more independent and then the parent becomes overprotective and critical of their child’s attempts to be independent.

Bateson suggested that this upbringing affects the child’s internal reality resulting in disorganised thinking and paranoia.

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56
Q

What is expressed emotion in relation to schizophrenia?

A01: Psychological explanations of schizophrenia

A

Expressed emotion refers to communication patterns which involve high levels of negative emotion expressed to the schizophrenic patient from their carers, which contribute to schizophrenia.

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57
Q

Can you provide an example of expressed emotion, and research to support expressed emotion impacting schizophrenic behaviour?

A01: Psychological explanations of schizophrenia

A

Examples of behaviours shown by families with high expressed emotions can be:

  • Exaggerated involvement. This is when families indicate that the sufferer is a burden through self-sacrifice such as by reminding the sufferer what an inconvenience they are all the time.
  • Over-involvement in a patients life. This lack of freedom could lead to the development of positive symptoms such as hallucinations and is a major cause of relapse.

Research support for Hooley found that 705 of schizophrenic sufferers from families with expressed emotion relapse while only 30% with low expressed emotions relapse when the patient returns after being hospitalised.

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58
Q

Practical applications

A03: Psychological explanations of schizophrenia

A

One strength of the research into expressed emotion is that it has practical applications.

Family therapy has been demonstrated to reduce expressed emotion in families and to be an effective intervention for reducing relapse rates

The fact that family therapy for psychosis is effective shows that psychological factors are involved in schizophrenia thus supporting the argument that family dysfunction causes schizophrenia.

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59
Q

Ignores individual differences as expressed emotions does not always lead to schizophrenia.

A03: Psychological explanations of schizophrenia

A

However one limitation is that not all patients who live in high expressed emotion families relapse, and not all patients who live in low expressed emotion homes avoid relapse.

Research has shown individual differences in stress response to high expressed emotional behaviour.

For example, vulnerability may be physiological as one quarter of patient studies showed no physiological responses to stressful comments made by their relatives.

This is a limitation because it shows that not all patients are equally vulnerable to high levels of expressed emotion within the family.

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60
Q

Ignores other factors in determining schizophrenic behaviour.

(2 marks)

A03: Psychological explanations of schizophrenia

A

The family explanation for schizophrenic only deals with one aspect of the disorder (faulty communication) and in doing so it ignores the compelling evidence for the role of biology in the onset of the disorder.

{insert Gottesman and Shield study as supporting evidence)

This is a weakness as it fails to provide a complete explanation for schizophrenia and over emphasises the role of nurture at the expense of nature.

61
Q

Perhaps taking an interactionist approach to explaining schizophrenia is more appropriate…

A03: Psychological explanations of schizophrenia

A

Alternatively, the diathesis stress model may be a more suitable explanation to explain schizophrenia as it combines both biological and psychological factors in the development of schizophrenia, providing a more holistic approach to explaining schizophrenia.

This is because this explanation considers not only the development of schizophrenia determined by a biological predisposition but also the environmental stressors such as expressed emotion and mixed messages within an individual’s lives that trigger this vulnerability, causing schizophrenia.

62
Q

Supporting research of the influence of family dysfunction from family studies.

A03: Psychological explanations of schizophrenia

A

Tienari who studied the biological children of schizophrenic mothers who had been adopted.

It was found that 5.8% of those adopted into psychologically healthy families developed schizophrenia, compared to 36.8% of children raised in dysfunctional families.

This suggests that the interpersonal family environment has a significant impact on the development of schizophrenia is genetically vulnerable people.

63
Q

Meta analysis supporting family dysfunction in causing schizophrenia…

A03: Psychological explanations of schizophrenia

A

Butzlaff and Hooley showed using a meta-analysis of 27 studies that relapse into schizophrenia is significantly more likely in families that have issues with expressed emotion.

64
Q

Socially sensitive, how?

A03: Psychological explanations of schizophrenia

A

It is socially sensitive to suggest that schizophrenia is caused by the family, parents are already dealing with the difficulty of their relatives behaviour.

Theorists may be adding additional stress and anxiety by making the family think they are responsible.

65
Q

Impossible to demonstrate cause and effect, how?

A03: Psychological explanations of schizophrenia

A

It could be that schizophrenia of the child and the associated behaviour is the cause of the family dysfunction.

66
Q

We do not know for sure that family dysfunction causes schizophrenia.

There is research support contradicting the importance of double bind (mixed messages)…

A03: Psychological explanations of schizophrenia

A

For example, research from Ringuette and Kennedy contradicts the importance of double bind.

A content analysis of letters sent to psychiatric patients from mothers had found no difference in the number of double bind communications in patients with schizophrenia compared to patients with other psychiatric conditions.

Therefore, it was concluded that double bind led to poor mental health not schizophrenia and thus limits the importance of double bind communication in the development of schizophrenia opposing the argument that family dysfunction causes schizophrenia.

67
Q

Discuss the cognitive explanation of schizophrenia (16 marks)

A
68
Q

What is Firth’s attention theory and what type of symptoms does it focus on?

A01: Cognitive Explanations of Schizophrenia

A

Firth’s (1979) “Attention-deficit theory” suggests that schizophrenia is due to a faulty attention system unable to filter preconscious thoughts and gives too much significance to information that would usually be filtered out, thus overloading the mind.

This accounts for positive symptoms such as hallucinations and delusions.

69
Q

What is meta-representation, can you fully outline how dysfunction in it leads to schizophrenia?

A01: Cognitive Explanations of Schizophrenia

A

Meta representation is the cognitive ability to reflect on thoughts and behaviour. This allows us insight into our own intentions and goals and interpret the actions of others.

Dysfunction in meta representation would disrupt our ability to recognise our own actions and thoughts as being carried out by ourselves rather than someone else.

This would explain hallucinations of voices and delusions like thought insertion (the experience of having thoughts projecting into the mind by others)

70
Q

What is central control, and how can faulty central control lead to schizophrenia?

A01: Cognitive Explanations of Schizophrenia

A

Central control = cognitive ability to suppress automatic responses while performing deliberate actions instead.

Disorganised speech and thought disorders could result from the inability to suppress automatic thoughts and speech triggered by thoughts (faulty central control)

For example, sufferers with schizophrenia tend to experience derailment of thoughts and spoken sentence because each word triggers associations and the patient cannot suppress automatic responses to these

71
Q

Research support for faulty cognitions…

A03: Cognitive Explanations of Schizophrenia

A

Sarin and Wallin (2014) reviewed recent research relating to the role of cognitive biases and found supporting evidence for the claim that the positive symptoms of schizophrenia have their origin in faulty cognition.

They found that patients with hallucinations were found to have impaired self-monitoring and tended to experience their own thoughts as voices.

This research illustrates that schizophrenia sufferers have cognitive biases not present in normal controls.

72
Q

Most research is correlational…

A03: Cognitive Explanations of Schizophrenia

A

There is a problem of causality with the cognitive approach because the approach assumes that cognitive dysfunction is the cause of schizophrenia, when it may be that schizophrenia causes the cognitive dysfunction.

Therefore it is impossible to claim that the cognitive explanation is valid as we cannot establish a casual relationship because we cannot identify whether cognitive dysfunction is a cause or symptom of schizophrenia.

73
Q

Stirling et al - Research support for deficits in central control.

A03: Cognitive Explanations of Schizophrenia

A

Stirling et al (2006) conducted the Stroop test on 30 patients with schizophrenia and 18 control patients. The task involved naming the ink colours without saying the word.

This is difficult as there is desire to say the words that needs to be controlled. Stirling found that patients with schizophrenia took twice as long to name the colours as controls.

This shows that patients with schizophrenia do have dysfunctional thought processing such as deficits in Central control tasks, supporting Firth’s ideas

74
Q

Firth supported his ideas with cognitive neuroscience studies.

A03: Cognitive Explanations of Schizophrenia

A

Firth (1992) supported these ideas with biological/cognitive neuroscience studies. 30 schizophrenic patients with various symptoms had PET scans. The scans indicated a reduction in the blood flow in the frontal cortex of patients with negative symptoms like Avolition and inability to suppress automatic thoughts.

Scans also showed increased activity in an area of the temporal lobe responsible for the retrieval of memories (Para hippocampal) with patients with reality distortion

This suggests that there are biological differences in schizophrenics brain regions associated with theorised cognitive processes.

75
Q

Practical applications of cognitive explanation…

A03: Cognitive Explanations of Schizophrenia

A

Yellowless et al. (2002) developed a machine that produced virtual hallucinations, such as hearing the television telling you to kill yourself or one person’s face morphing into another’s.

The intention is to show schizophrenics that their hallucinations are not real. This suggests that understanding the effects of cognitive deficits allows psychologists to create new initiatives for schizophrenics and improve the quality of their lives.

76
Q

Effectiveness of CBT shows that faulty cognition is involved in schizophrenia.

A03: Cognitive Explanations of Schizophrenia

( 2 marks)

A

As cognitive behavioural therapy can be an effective treatment this indicates that cognitive factors are involved in the disorder.

The effectiveness of cognitive behavioural therapy for psychosis (CBTp) was demonstrated in the National Institute for Health and Care Excellence review of treatments for schizophrenia.

This review found consistent evidence that, when compared with treatment by antipsychotic medication, CBTp was more efficient in reducing symptom severity and improving levels of social functioning.

This supports the view that faulty cognition has an important causal influence in the development of schizophrenia.

77
Q

Cognitive explanation of schizophrenia is too reductionist

A03: Cognitive Explanations of Schizophrenia

A

One weakness of the cognitive model is that it is reductionist. The reason for this is because the approach does not consider other factors such as genes.

It could be that the problems caused by low neurotransmitters creates the cognitive deficits.

This suggests that the cognitive approach is oversimplistic when considering the explanation of schizophrenia and is not addressing the root cause (imbalance in neurotransmitters) according to the biological explanation of schizophrenia.

78
Q

Cognitive explanation of schizophrenia is highly scientific and replicable.

A03: Cognitive Explanations of Schizophrenia

A

Much of the research into cognitive explanations is scientific and replicable.

For example, McGuigan (1966) found that the vocal cords of patients with schizophrenia were tense during the time they experienced auditory hallucinations. This suggests that they were mistaking their own inner speech for someone else’s voice.

Thus supporting cognitive explanations of schizophrenia as they are high in internal validity.

79
Q

Does not account for other factors that lead to schizophrenia.

A03: Cognitive Explanations of Schizophrenia

A

The cognitive approach is limited because it can only explain the positive symptoms experienced by schizophrenics, but offers no explanation for negative symptoms, such as avolition.

Therefore, it is likely that there are other factors that play a role in causing schizophrenia that account for the presence of the negative symptoms.

80
Q

Discuss the use of an interactionist approach in explaining and treating schizophrenia. (16 marks)

A
81
Q

What does the interactionist approach propose about schizophrenia, and can you explain the model associated with it?

A01: Interactionist Approach and Treatment of Schizophrenia

A

The interactionist approach to schizophrenia is the idea that the interaction of multiple factors cause schizophrenia.

An interactionist model for schizophrenia is the diathesis stress model. The diathesis-stress model is the psychological concept that a disorder is due to the interaction between predisposed vulnerability (this is the diathesis) and an environmental trigger later in life such as stress.

82
Q

What had a diathesis previously been considered to be, and how has this changed?

A01: Interactionist Approach and Treatment of Schizophrenia

A

Diathesis in schizophrenia is often considered to be a genetic vulnerability, which leads to dopamine imbalance.

However, recent research now takes into consideration non genetic diathesis such as psychological trauma such as child abuse. This is thought to also influence brain development, creating a diathesis.

83
Q

What is a stressor and how does it lead to schizophrenia?

A01: Interactionist Approach and Treatment of Schizophrenia

A

A stressor in schizophrenia refers to later negative environmental experiences such as high levels of stress.

This emotional event triggers schizophrenia along with having a diathesis.

84
Q

Why are treatments combined in the interactionist treatment of schizophrenia?

A01: Interactionist Approach and Treatment of Schizophrenia

A

Combinations of CBT and drug therapies are seen as a particularly effective interactionist treatment for schizophrenia as they address both the diathesis and the trigger for the condition.

Biological therapies such as drugs allow patients to reduce their symptoms so that they can engage in psychological therapies that give them skills to change their underlying faulty cognitions.

85
Q

Gottesman - Findings from twin studies supports the need for interactionist approaches.

A03: Interactionist Approach and Treatment of Schizophrenia

A

Gottesman found a concordance rate of 48% for MZ twins and 17% for DZ twins.

This suggests a role for both biological genetic factors as the MZ twins have a higher concordance rate than DZ twins,

But as the concordance rates is far les than 100% for MZ twins, this suggests that there must be some psychological experience triggered in one twin but not the other, supporting the interactionist approach.

86
Q

Tienari - Findings from family studies supports the need for interactionist approaches.

A03: Interactionist Approach and Treatment of Schizophrenia

A

Studied the biological children of schizophrenic mothers who had been adopted.

It was found that 5.8% of children adopted in psychologically healthy families developed schizophrenia compared to 36.8% of children raised in dysfunctional families.

This research supports the influence of biological factors, due to high rate even in psychologically healthy families, but the even higher figure for dysfunctional families suggests a psychological trigger is needed.

87
Q

Research shows combination therapy is effective.

A03: Interactionist Approach and Treatment of Schizophrenia

A

Both meta-analysis by Pharoah, and Jauhar showed that patients responded better to combination therapy (CBTp with Family Therapy) than with just antipsychotics alone.

They found that these patients had lower relapse rates and less rehospitalisation in dealing with their schizophrenia. Jauhar had also found that combination therapy reduced both negative and positive symptoms.

This relative success of combination therapy compared to antipsychotics alone suggests combination therapy is more effective, supporting the interactionist approach.

88
Q

Further research support for combination therapies effectiveness.

A03: Interactionist Approach and Treatment of Schizophrenia

A

Tarrier et al had 315 patients randomly allocated to either:

  • Combination of drugs and CBT
  • Combination of drugs and supportive counselling
  • Control of drugs only

He found patients in the two combination groups show lower symptom levels.

This is a strength because it helps highlight the clear practical advantage to adopting an interactionist approach to treatment in form of superior treatment outcomes and therefore demonstrates the importance of taking an interactionist role.

89
Q

Interactionist approach is holistic and provides specific treatments.

A03: Interactionist Approach and Treatment of Schizophrenia

A

One strength is that this approach is holistic as it recognises the compelx nature of the disorder.

This is a strength because it identifies that schizophrenic patients have different triggers such as genes and therefore avoids being reductionist by not reducing complex mental disorder to a single causal factor.

This means that schizophrenic patients can receive different and individualised treatments for their schizophrenia which will be more effective, because it targets the specific problem that each person with schizophrenic has and how to effectively treat it.

90
Q

Interactionist treatments may be unrealistic

A03: Interactionist Approach and Treatment of Schizophrenia

A

Moreover, holistic explanations appear to be impractical as they tend not to lend themselves to rigorous scientific testing and become vague and speculative as they become more complex.

For example, if we accept there are many factors contributing to schizophrenia, it is difficult to establish which is most influential and which to use as a basis for therapy.

Therefore, in practical terms, this complexity suggests that lower-level explanations may be more applicable when seeking solutions for real-world problems, as they allow for a more focused and measurable approach.

91
Q

We do not know for sure whether diathesis and stressors interact with one another.

A03: Interactionist Approach and Treatment of Schizophrenia

A

The fundamental mechanism by which a negative psychological event actually triggers a complex biological response resulting in symptoms is still uncertain.

This reduces validity in the interactionalist approach as a full explanation for schizophrenia.

92
Q

Sometimes, the interactionist approach may not suit all patients.

A03: Interactionist Approach and Treatment of Schizophrenia

A

The interactionist approach may not suit all patients: some patients may have difficulty understanding or interpreting the side-effects of their drugs which CBT sessions could actually make worse with the emphasis they place on challenging dysfunctional thought processes i.e. the effects of the drug are exacerbated by the effects of the CBT

93
Q

Positive economic implications in the long term.

A03: Interactionist Approach and Treatment of Schizophrenia

A

Using the interactionist approach may prove to be cost-effective (i.e. good for the economy) if it is more successful than other treatments as the patient will improve more quickly and is less likely to relapse, saving money on health services and getting the patient back into the workplace more quickly

94
Q

Outline and evaluate drug therapies in the treatment of schizophrenia. (16 marks)

A
95
Q

What are antipsychotics?

Do you know the two types of antipsychotics used to treat schizophrenia?

A01: Biological approach in treating schizophrenia

A

Antipsychotic drugs also known as neuroleptics are used to control psychosis, such as hallucinations and delusions. They are taken in pill form or injected.

There are two types of antipsychotics used to treat schizophrenia, typical antipsychotics (first generation), and atypical antipsychotics

96
Q

What are typical anti-psychotics?

Could you provide an example and tell me how it treats schizophrenia?

A01: Biological approach in treating schizophrenia

A

Typical anti-psychotics (1st Gen) are drug therapies that have been used since the 1950s that have become less popular due to its severe side effects and only treating positive symptoms.

Example:

One example is the drug Chlorpromazine.

The drugs work as dopamine antagonists, which reduce dopamine activity by blocking dopamine receptors at the synapse.

This calms the dopamine system in the brain, which reduces positive symptoms such as hallucinations and delusions in schizophrenic patients.

97
Q

What are the side effects of anti-psychotics?

A01: Biological approach in treating schizophrenia

A

Side effects:

  • It has a sedative effect (feelings of sleepiness)
  • As these drugs affect all dopamine receptors in the brain equally, they produce side effects such as dry mouth, constipation, lethargy, confusion, involuntary muscle movements
98
Q

What are atypical anti-psychotics?

Could you provide an example and tell me how it treats schizophrenia?

A01: Biological approach in treating schizophrenia

A

Atypical anti-psychotics (2nd Gen) are a drug therapy that started to be used in 1970’s to avoid the more severe side effects of typical anti-psychotics.

Example:

Block dopamine receptors but also act on other neurotransmitters such as glutamate and serotonin.

This means that antipsychotics also reduce the negative symptoms such as avolition, alongside positive symptoms.

99
Q

What are the side effects of atypical anti-psychotics?

A01: Biological approach in treating schizophrenia

A

Side effects of atypical antipsychotics

  • Weight gain, cardiovascular problems etc.
  • However, they are less likely to cause involuntary movements as a side-effect.
100
Q

Research support for effectiveness of anti-psychotics: meta-analysis from Leucht

A03: Biological approach in treating schizophrenia

A

Leucht (2013) reviewed 212 studies in a meta-analysis on the effectiveness of biological antipsychotic drug treatments that work via normalising levels of dopamine.

Treatment of symptoms with drugs was found to be much more effective than a placebo.

This suggests that the effectiveness of these treatments is due to dopamine being involved in the underlying mechanisms/causes of schizophrenia, and also generally shows that these drugs are able to treat schizophrenia.

101
Q

Further research support for effectiveness of antipsychotics: meta-analysis from Bagnall

A03: Biological approach in treating schizophrenia

A

Bagnall (2003) meta-analysed 232 studies to compare the effectiveness of a range of atypical drugs to each other and to typical antipsychotic drugs.

Bagnall found that atypical drugs were more effective than typical in treating overall symptoms, had fewer movement disorder side effects, and fewer people left the drug treatment early, suggesting that its side effects were more tolerable.

However, clozapine was found to be the most effective in reducing negative symptoms and treating people who were resistant to other drugs.

This suggests that atypical antipsychotics have improved treatment of people with schizophrenia.

102
Q

Research conducted by Tarrier shows for the need for taking a holistic approach.

A03: Biological approach in treating schizophrenia

A

Tarrier placed patients randomly into routine care (anti-psychotics) or routine care and CBT.

Patients in the combined treatment had a significant improvement in the severity and number of positive symptoms as well as fewer days in hospital receiving care.

This suggests that drug therapy alone is not the most effective treatment plan and an interactionalist approach to treating schizophrenia along side CBT is a better option.

103
Q

Psychology and the economy: How do anti psychotics help benefit the economy?

A03: Biological approach in treating schizophrenia

A

This has positive implications for the economy because it means patients can return to their homes, saving the NHS money on hospitalisations, reducing the burden on the NHS (with already very long waiting times).

It also allows patients to go back to work and contribute to the economy.

104
Q

Has real life applications as it has helped sufferers…

A03: Biological approach in treating schizophrenia

A

The use of drug treatments has resulted in the end of long-term institutionalisation in mental hospitals for those with schizophrenia. Now people can be treated at home and perhaps have enhanced quality of life as they have additional independence.

105
Q

Why is it argued that financially drug therapies are better than alternative treatments?

A03: Biological approach in treating schizophrenia

A

Drug therapies are often cheaper to prescribe than providing hospital treatment or psychological therapies such as CBT and family therapy.

This means that more people can be treated with the same amount of money.

106
Q

Why may drug therapies be ineffective in the long term?

A03: Biological approach in treating schizophrenia

A

Drug therapies may only be suppressing symptoms and not treating the underlying problem, as symptoms quickly return when the drugs stop.

There is a lack of evidence showing the long-term benefits of medication as most studies look at the short-term effects.

107
Q

Why can suffer side effects such as those from 1st gen anti-psychotics limit the treatment of schizophrenia?

A03: Biological approach in treating schizophrenia

A

One limitation is that it has severe side effects.

This is a limitation because long term use can result in tardive dyskinesia which manifests as involuntary facial movements such as blinking and lip smacking.

While they may be effective, the severity of the side effects means the costs outweigh the benefits therefore they are not an appropriate treatment.

108
Q

Why are both antipsychotic types still questionable in treating schizophrenia?

A03: Biological approach in treating schizophrenia

A

Both types of drugs have side effects, and due to the severity of the side effects, two thirds of people stop taking them, resulting in symptoms returning and a ’revolving door’ effect of people leaving treatment and returning when their symptoms become too serious to control.

109
Q

How do anti-psychotic drugs have ethical issues?

A03: Biological approach in treating schizophrenia

A

Drugs are often prescribed and forcefully given to those experiencing severe schizophrenia symptoms.

There are ethical issues with this as the patients cannot give informed consent.

This is particularly problematic given the severe side effects.

110
Q

Outline and evaluate cognitive behavioural therapy as used in the treatment of schizophrenia. (16 marks)

A
111
Q

What procedures does the treatment take to try to address schizophrenia and how does it attempt to change people suffering from schizophrenia?

A01: Cognitive approach to treating schizophrenia

A
  • Treatment involves getting the patient to challenge their irrational beliefs about their experiences and their distorted perceptions so that they can modify their hallucinations and delusions.
  • It also attempts to change negative attitudes and to get them to seek help when the schizophrenic symptoms become severe rather than withdraw from society.
112
Q

Can you fully outline a CBT used to treat schizophrenia?

(I chose personal therapy, your school may choose CBTp, but I like personal therapy more…)

A01: Cognitive approach to treating schizophrenia

A

One CBT is personal therapy which focuses on the hallucinations and delusions which distresses sufferers.

  • Identify delusions – the therapy works on positive symptoms such as hearing voices and tries to change the perception about these symptoms feeling out of their control (hearing things for example)
  • Analysis and challenge of evidence on which delusions are based. Tests the reality of the evidence. For example, questions as to whether the beliefs are to be true are raised.
  • Helps with relaxation strategies – identify situations they found stressful and help them develop coping strategies such as relaxation.
  • Helps them recognise signs of relapse or when the delusions have returned.
113
Q

CBT requires engagement, but when in some cases this can’t be done..

A03: Cognitive approach to treating schizophrenia

A

CBT requires engagement from the patient, but negative symptoms like avolition may lead the patient to be unwilling to take part, or positive symptoms lead to distrust of the therapist, meaning that it may not be a practical treatment for severely schizophrenic individuals.

In these cases, anti-psychotic medication can be used first to reduce the severity of the symptoms, but this would reduce the effectiveness of using CBT as a sole treatment for schizophrenic symptoms.

114
Q

Research support for CBT being arguably a better treatment than anti-psychotics

A03: Cognitive approach to treating schizophrenia

A

Sensky (2000) showed that patients who had resisted drug treatments had a reduction in positive and negative symptoms when treated with CBT.

They also continued to improve even 9 months after treatment had ended.

This suggests that psychological therapy can be effective even in cases when drugs are not.

115
Q

Long & expensive process

A03: Cognitive approach to treating schizophrenia

A

A problem with CBT is that it’s a lengthy process.

It requires commitment from the patient and the clinician, a suitable setting, and 6 to 12 weeks or more.

It’s expensive compared to simply using antipsychotic drugs and patients often end treatment early, meaning that it is impractical for some schizophrenics.

116
Q

Research support about effectiveness of CBT.

A03: Cognitive approach to treating schizophrenia

A

Research evidence by Kingdon and Turkington (1991) found that 35 out of 65 patients with schizophrenia (54%) in a five-year follow-up were free of symptoms when normalising and standard CBT techniques were used.

This evidence shows that more than 50% of patients were eventually free of symptoms, which indicates that CBT is useful in treating patients with schizophrenia. Therefore, this supports the use of CBT as a form of treatment for schizophrenia

117
Q

Research support for improvement in patients with schizophrenia (hospital stays)

A03: Cognitive approach to treating schizophrenia

A

Research evidence by Drury et al looked at CBT in an acute phase and found that CBT patients showed fewer psychotic symptoms at week 7 of a 12-week intervention than those in a comparison group.

The CBT group average (median) stay in hospital was 49 days compared with 108 days for the comparison group which had another therapy involving activities and informal support.

This is strong evidence to support the use of CBT as a form of treatment for schizophrenia as it has been proven to show improvements in patients with schizophrenia in a short amount of time.

118
Q

CBT appears to be cost-effective in the long term

A03: Cognitive approach to treating schizophrenia

A

However, in the long-term CBT appears to be cost effective.

Elizabeth Kuipers et al. (1998) analysed the economic impact of offering CBT to individuals with schizophrenia in addition to using antipsychotic medication.

The researchers reported that the costs involved in delivering CBT were likely to be offset by the reduced utilisation of service costs in the future.

119
Q

What implications does this research support from the previous flashcards imply on the impact of CBT on the economy?

A03: Cognitive approach to treating schizophrenia

A

This suggests that, although the use of CBT may initially be more costly, in the longer term those costs are likely to be recouped because the individuals with schizophrenia are less likely to need emergency psychiatric services.

This has economic implications for the NHS because this money used on re-hospitalisation can be saved for other health treatments, reducing the burden on the NHS.

It also allows patients to go back to work quicker and contribute to the economy.

120
Q

Why do patients feel like CBT is ‘better’?

A03: Cognitive approach to treating schizophrenia

A

CBT does not provide the side effects of drug therapies, making it a preferred treatment plan for many patients and arguably the better treatment.

High face validity.

121
Q

Maybe using a combination of treatments would be more appropriate..

A03: Cognitive approach to treating schizophrenia

A

As there is strong evidence that relapse is related to stress and expressed emotion within the family, it seems likely that CBT should be employed alongside family therapy in order to reduce the pressures on the individual patient.

122
Q

Outline and evaluate family therapy as used in the treatment of schizophrenia. (16 marks)

A
123
Q

How do family therapies attempt to improve schizophrenic behaviour and how does it differ from other treatments above?

A01: Psychological approach to treating schizophrenia

A

Family dysfunction can increase the risk of relapse of schizophrenic patients, so family therapies attempt to improve the home situation of the person with schizophrenia to reduce this risk.

This is a family-centred therapy, intended to change the behaviour of the whole family, not just the person with schizophrenia and is thus a different solution to other treatments.

124
Q

What is the main aim of family therapy?

A01: Psychological approach to treating schizophrenia

A

One of the main aims of family therapy is to educate the family on the symptoms of schizophrenia (psychoeducation) in order to help them to be more understanding of the patient’s behaviour.

125
Q

What are the other aims of family therapy?

A01: Psychological approach to treating schizophrenia

A

The other aims of family therapy are to:

  • Reduce the conflict in the family by addressing anger within the family,
  • To reduce the stress caused by caring for the schizophrenic person,
  • To reduce the self-sacrifice by getting carers to consider and find ways to make time for their own needs,
  • To improve communication by considering how to limit expressed emotion, and by improving problem solving skills within the family by predicting problems and having solutions ready.
126
Q

Does not treat what caused schizophrenia…

A03: Psychological approach to treating schizophrenia

A

Family therapy is about improving symptoms aiding the homelife of the family, ultimately avoiding admission to amental health facility.

This is not a cure for schizophrenia, but simply controls symptoms to reduce relapse and thus readmission, therefore it does not treat the root cause of schizophrenia.

127
Q

What did Pharoah et al find in his meta-analysis that shows the effectiveness of family therapy…

A03: Psychological approach to treating schizophrenia

A

Pharaoh et al. (2003) meta – analysis found family interventions help the patient to understand their illness and to live with it, developing emotional strength and coping skills, thus reducing rates of relapse.

128
Q

What research supports the view of using an interactionist treatment…

A03: Psychological approach to treating schizophrenia

A

Research suggests that a combination of drugs and family therapy may be a more effective form of treatment.

A study by Anderson et al. (1991) found a relapse rate of almost 40% when patients had drugs only, compared to only 20 % when Family Therapy or Social Skills training were used, and the relapse rate was less than 5% when both were used together with the medication.

129
Q

How does this research reduce readmission into the hospital in the short term?

A03: Psychological approach to treating schizophrenia

A

Leff (1985) looked at the aftercare of patients who had been hospitalised with schizophrenia.

Of those provided with standard outpatient care, 50% had relapsed within 9 months, compared to only 8% of those who received family therapy.

After two years this had risen to 50% of those who received the therapy and 75% of those with standard outpatient care.

This suggests that family therapy is helpful in reducing readmission in the short term, however, families may not maintain positive behaviour patterns in the longer term.

130
Q

How does family therapy have economic benefits?

A03: Psychological approach to treating schizophrenia

A

One strength is that family therapy has economic benefits as it is highly cost effective because it reduces relapse rates, so the patients are less likely to take up hospital beds and resources.

The NICE review of family therapy studies demonstrated that it was associated with significant cost savings when offered to patients alongside the standard care – relapse rates are also lower which suggests the savings could be even higher.

131
Q

Can you tell me further evidence for family therapy having economic benefits?

A03: Psychological approach to treating schizophrenia

A

Further evidence for this comes from the Schizophrenia Commission who found that family therapy saved £1004 a patient compared to the standard over three years.

132
Q

Why is family therapy effective for lower levels of rehospitalisation?

A03: Psychological approach to treating schizophrenia

A

An explanation for lower levels of rehospitalisation could be partly because family members have been trained to help sufferers manage their medication, providing further evidence for using family therapy as an effective treatment of schizophrenia.

133
Q

Family therapy is a long process…

A03: Psychological approach to treating schizophrenia

A

Therapy is long (often taking up to a year), meaning that patients and family may drop out part way through therapy at times of severe episodes as symptoms may get worse before they get better.

134
Q

What did Lobban find about treating family therapy in mild and severe cases?

A03: Psychological approach to treating schizophrenia

A

Lobban (2013) reports that other family members felt they were able to cope better thanks to family therapy.

In more extreme cases the patient might be unable to cope with the pressures of having to discuss their ideas and feelings and could become stressed by the therapy, or over-fixated with the details of their illness.

135
Q

Discuss token economies as used in the management of schizophrenia. (16 marks)

A
136
Q

What is token economy?

A01: Behaviourist/Token Economy approach to treating schizophrenia

A

Token economies are a behavioural therapy technique based on Skinner’s operant conditioning.

This is learning through reinforcement of desired behaviours.

137
Q

How do token economies work in treating schizophrenia?

A01: Behaviourist/Token Economy approach to treating schizophrenia

A

The tokens themselves are used as a kind of positive reinforcement.

They are given as an immediate reward for patients showing a predefined target behaviour (such as washing).

The tokens are then exchanged for something the patient wants such as activities or chocolate.

138
Q

What is behaviour shaping and how does it reduce symptoms of schizophrenia?

A01: Behaviourist/Token Economy approach to treating schizophrenia

A

This may involve behaviour shaping, where behaviours are progressively changed, with tokens first given for small changes in behaviour leading up to the ideal behaviour.

This helps reduce negative symptoms of schizophrenia such as social withdrawal (Eating and hygiene) by using token economies to modify their behaviour.

139
Q

How does this treatment attempt to help patients after they are finished with their treatment?

A01: Behaviourist/Token Economy approach to treating schizophrenia

A

Therefore, this treatment is designed to produce easier to manage behaviour within the mental hospital, or to prepare long stay patients for transfer into the community.

140
Q

Research support by Dickerson

A03: Behaviourist/Token Economy approach to treating schizophrenia

A

Evidence by Dickerson who found when reviewing the findings of 13 studies, token economies can be effective in improving the adaptive behaviour of people with schizophrenia.

141
Q

Are token economies effective in treating schizophrenics with severe symptoms?

A03: Behaviourist/Token Economy approach to treating schizophrenia

A

Treatment is not effective with severely unresponsive patients such as those with strong negative symptoms, so not allowing them to take part but letting the more mildly affected patients take part in token therapy could be seen as punishing them for the severity of their illness.

142
Q

Why do token economies have ethical issues?

A03: Behaviourist/Token Economy approach to treating schizophrenia

A

Some more mildly affected patients may refuse to engage in this therapy as they may find it infantilising/degrading as it is very similar in procedure to the sticker charts that small children have to encourage good behaviour, with valueless tokens used to indicate a small treat when behaviour is good, and to the clicker training that is used on dogs and other animals

143
Q

Why do token economies have low ecological validity?

A03: Behaviourist/Token Economy approach to treating schizophrenia

A

One limitation is that token economies may not generalise beyond the hospital setting. Within a psychiatric unit, inpatients receive 24-hour care so there’s better control for staff to monitor and reward patients appropriately.

However, with outpatients it becomes impossible to reward the patient every time they perform a desirable act as outpatients only receives day treatment for a few hours a day. This results in a situation where the desired behaviour becomes depended on the reinforcer- if the reinforcer stops so does the desired behaviour- this is called extinction and makes relapse likely.

This is a weakness of token economy as they may only be useful to change unwanted behaviour of hospitalised patients and be less useful for patients living in the community.

144
Q

How are studies assessing the success of token economies limited?

A03: Behaviourist/Token Economy approach to treating schizophrenia

A

Difficulties assessing the success of studies tend to be uncontrolled. When token economy systems were introduced into a psychiatric ward, typically all patients are brought into the programme rather than having an experimental group which goes through the token economy programme and a control group that doesn’t.

This means that patients improvements can only be compared to their past behaviours rather than the behaviour of a control group.

This comparison may be misleading as other factors (such as an increase in staff attention) may be causing the patients improvements rather than the token economy.

145
Q

Token economies not being effective in treating what?

Hint: (? —> Schizophrenia)

A03: Behaviourist/Token Economy approach to treating schizophrenia

A

Token economies do not directly treat symptoms of schizophrenia, they only attempt to manage negative symptoms such as poor attention, poor motivation, and withdrawal.

146
Q

Token economies could have potentially dangerous and life-threatening consequences…

A03: Behaviourist/Token Economy approach to treating schizophrenia

A

The use of token economies in the treatment of schizophrenia may have unintended consequences, potentially worsening the condition if the targeted behaviours are inappropriate or unattainable for the patient.

For instance, if a schizophrenic individual is rewarded only when they exhibit behaviour suggesting they are not experiencing hallucinations, they might become adept at concealing these symptoms without actually addressing the underlying issue.

This could create a situation where staff may find it challenging to identify and intervene when the patient acts on hallucinations or delusions, especially if the external appearance seems normal.

In such cases, the patient’s ability to effectively hide their struggles might lead to situations where they carry out dangerous actions prompted by hallucinations, putting both themselves and others at risk.

Therefore, token economies could have dangerous consequences because we do not know for sure if these symptoms have been adequately addressed if hidden.

147
Q

How is token economy potentially more ethical than other forms of treatment?

A03: Behaviourist/Token Economy approach to treating schizophrenia

A

A programme of token economy behaviour management is less potentially harmful than drug therapy so it may be more ethically valid to some extent

148
Q

Baker et al. (2018) does it treat symptoms effectively as a whole?

Tell me what he found…

A03: Behaviourist/Token Economy approach to treating schizophrenia

A
  • A longitudinal (18 months) experiment in which the independent variable was tokens awarded for either positive behaviour or tokens were awarded regardless of the patients’ behaviour: both groups improved in terms of social withdrawal but overall symptoms did not change for either group i.e. a token economy has limited power in symptom management
149
Q

How has token economies been helpful in environments like the hospital?

A03: Behaviourist/Token Economy approach to treating schizophrenia

A

One advantage of a token economy is patients becoming more independent and active, which has the knock-on effect of nurses’ increased respect for the patients, leading to the patients becoming even more motivated and developing positive self-esteem.

This indicates that token economies are an effective way of helping with institutionalisation which occurs when a patient has been in the hospital for a long time.

Also, where token economies have been used on hospital wards they have helped to create a more healthy, safe and stable environment