sars Flashcards
complication of covid
acute resp failure
pnuemonia
ARDS
acute liver injury
cardiac
2 infection
AKI
septic shock
DIC
blood clots
multisustem inflammaotry syndrome
chronic faitgue
rhabdo
sx ARDS
dyspnoea
elevated respiratory rate
bilateral lung crackles
low oxygen saturations
ARDS what is it
caused by the increased permeability of alveolar capillaries leading to fluid accumulation in the alveoli, i.e. non-cardiogenic pulmonary oedema
what are key investigations for ARDS
and what will these find
A chest x-ray and arterial blood gases are the key investigations.
Criteria (American-European Consensus Conference)
acute onset (within 1 week of a known risk factor)
pulmonary oedema: bilateral infiltrates on chest x-ray (‘not fully explained by effusions, lobar/lung collapse or nodules)
non-cardiogenic (pulmonary artery wedge pressure needed if doubt)
pO2/FiO2 < 40kPa (300 mmHg)
pulmonary wedge pressure
integrated measurement of the compliance of the left side of the heart and the pulmonary circulation
norma 4-12
elevated in Ventricular failure and mitral stenosis for example
tx ards
oxygenation/ventilation to treat the hypoxaemia
general organ support e.g. vasopressors as needed
treatment of the underlying cause e.g. antibiotics for sepsis
certain strategies such as prone positioning and muscle relaxation have been shown to improve outcome in ARDS
ITU