Breathlessness prep for sim Flashcards
what are you looking for in A in a breathless patient
signs of obstruction - stridor or choking
intervene with airway manoeuvres or adjunct if necessary
what are you looking for in B in a breathless patient
RR , resp effort , SPo2
breath sounds - wheeze and crackles
administer oxygen if needed and vent support
what are you looking for in C in a breathless patient
pulse, BP, crt , JVP , cyanosis, cold peripheries, ABG/VBG , IV fluids and meds
what are you looking for in D andE in a breathless patient
AVPU or GCS temp, pedal oedema, calf swelling and trauma
what investigations might you consider in a patient with breathlessness
if abnromal HR - ecg
suspected pe - ctpa - fluid pre and post - creatiine
abnromal chest exam
system causes of breathlessness
respiratory
cardiovascular
others
sudden onset breathlessness
PE
anaphylaxis
ACS
pneumothorax
gradual onset breathlessness
infection
pulmonary oedema
pleural effusion
pathophysiology behind asthma
bronchial hypersensitivity and inflammation leading to reversible bronchoconstriction causing recurrent sx cough, wheeze, chest tightness and dyspnoea
how do we dx asthma
obstructive spirometry with positive bronchodilator reversibility - ratio less than 70
peak flow readings over 2-4 weeks with 20% variability
FeNO level of over40ppb in adults anad 35 in children 5-16
management of severe asthma attack
A-E - target sats 94-98
oxygen driver nebulised bronchodilator therapy - salbut 5mg and ipratropium 0.5mg
repeat doses 15-30min intervals
ocntinuous if no repsonse
give pred 30mg )D for 5 days or IV hydrocortisone 100mg 6hrly
magenesium sulphate 2g IV over 20mins - senior
procalcitonin to guide abx
life threatening asthma features
hypoxia
hypercapnia
altered consciousness
ABG showing fall in ph
hypotnesion
silent chest
confusion
PEFR less than 33%
follow up long term mx after asthma attack
early follow up with GP post discharge
identify and avoid triggers
optimise preventative treatment
self management plan
pathophysiology of COPD
chronic infalmmation leads to strctural lung changes - destruction of alveoli, mucus hypersecretion.
airflow obstruction and air trapping
impaired gas exchange
features of COPD
cough
sputum
dyspnoea
reduced exercise tolerance
sometimes fever
auscultation - reduced air entry , wheeze and rhonchi