passmed year 5 Flashcards

1
Q

Which of the following options confirms that the chest drain is located in the pleural cavity?

A

Chest drain swinging: Rises in inspiration, falls in expiration

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2
Q

reduced breath sounds and dullness to percussion

A

pleural effusion

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3
Q

Auscultation reveals normal heart sounds, bilateral coarse crackles, and a widespread polyphonic wheeze. She has a past medical history of pertussis as a toddler and recurrent pneumonia as a teenager.

A

bronchiectasis

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4
Q

penumonia caused by aspiration

A

klebsiella

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5
Q

klebsiella normally in what zones of the lung

A

upper

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6
Q

target O2 sats in asthma

A

94-98%

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7
Q

mx of brochiectasis

A

hysical training (e.g. inspiratory muscle training) - has a good evidence base for patients with non-cystic fibrosis bronchiectasis
postural drainage
antibiotics for exacerbations + long-term rotating antibiotics in severe cases
bronchodilators in selected cases
immunisations
surgery in selected cases (e.g. Localised disease)

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8
Q

tx in high altitude cerebral oedema

A

dexamethasone

acetazolamide in prevention

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9
Q

COPD standby medication:

A

NICE recommend offering a short course of oral corticosteroids and oral antibiotics to keep at home

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10
Q

30-40 year old with basal emphysema and abnormal LFTs

A

alpha 1 antitrypsin deficiency

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11
Q

Infective exacerbation of COPD

A

first-line antibiotics are amoxicillin or clarithromycin or doxycycline

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12
Q

Bibasal fine end-inspiratory crepitations

A

PF

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13
Q

Decrease in pO2/FiO2 in poorly patient with non-cardiorespiratory presentation →

A

ARDS

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14
Q

persistent mouth ulcer

A

think SCC

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15
Q

ramsay hunt syndrome tx

A

Oral aciclovir and prednisolone

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16
Q

dix hallpike manouvre reproduces what

A

rotatory nsytagmus

17
Q

bird lung is a type of what allevolitis

A

Extrinsic allergic alveolitis

18
Q

extrinsice allveolitis affects the upper and middle lobes what is the mx

A

avoid precipitating factors

19
Q

Fine end-inspiratory crepitations are seen in

A

fibrosis

sudden opening of small airways during inspiration that were held closed in the previous expiration.

20
Q

Coarse crackles heard when

A

fluid in the lungs

21
Q

klebsiella caused what extra associated signs

A

lugn abcess or empyema

22
Q

what marker might be raised in chronic copd due to another conditition - 2secondary deu to the copd

A

hematocrit

polycythaemia

23
Q

what is haematocrit

A

the ratio of the volume of red blood cells to the total volume of blood.

24
Q

why are high urea levels a poor prognostic sign in CAP

A

This is because elevated blood urea nitrogen levels indicate dehydration or reduced renal perfusion, both of which can lead to increased mortality

25
Q

two paraneoplastic feature of adenocarcinoma of the lung

A

gynaecomastia
hypertrophic pulmonary osteoarthropathy (HPOA)

26
Q

4 things causing lower zone fibrosis - rest all therefore all upper by default

A

idiopathic pulmonary fibrosis

most connective tissue disorders (except ankylosing spondylitis) e.g. SLE

drug-induced: amiodarone, bleomycin, methotrexate

asbestosis

27
Q

occupational asthma caused by things such as working in bakery what should GP advise

A

Advise the patient to keep a diary of peak flow readings both in and out of work

28
Q

treatment of alpha-1 antitrypsin deficiency treatment

A

Lung volume reduction surgery

removes the worst affected part of the lungs in order to improve airflow and alveolar gas exchange in the remaining portion of the lung.

29
Q

describe brochiectassis presentation or features in an individual

A

a productive cough with copious amounts of purulent sputum, occasional haemoptysis, wheezing, and is often related to a history of childhood respiratory infections.

30
Q

nasogastric tubes are safe to use under what ph

A

Nasogastric tubes are safe to use if pH <5.5 on aspirate

31
Q

smoking hx

A

cancer over tb

32
Q

pancoast tumour dx test

A

ct if cant see

33
Q

3 stages of churg strauss - pANCA

A

There are three stages to the presentation of Churg-Strauss disease.

The initial phase is characterised by allergy with many patients having a history of asthma or allergic rhinitis. This inflammation of the nasal passages can lead to the development of nasal polyps.

The second phase is eosinophilia

The third stage is the vasculitis itself which affects small and medium-sized blood vessels and therefore resulting in damage to many organs. Given this patient’s kidney failure, it appears to be affecting his renal blood supply.