s10 - TMJ Disorders Flashcards

1
Q

What type of joint is the temporomandibular joint (TMJ)?

A

A diarthrodial synovial joint.

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2
Q

What are the two main compartments of the TMJ?

A

The superior and inferior compartments.

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3
Q

What type of motion occurs in the inferior compartment of the TMJ?

A

Hinge (ginglymoid) motion.

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4
Q

What type of motion occurs in the superior compartment of the TMJ?

A

Translatory (arthrodial) movement.

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5
Q

What is the function of synovial fluid in the TMJ?

A

Lubrication and nutrition of avascular joint structures.

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6
Q

What are the main bony components of the TMJ?

A

The mandibular condyle and temporal bone (glenoid fossa and articular eminence).

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7
Q

What type of cartilage covers the TMJ surfaces?

A

Dense fibrous connective tissue (not hyaline cartilage).

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8
Q

What are the three regions of the articular disc?

A

Anterior band, intermediate zone, and posterior band.

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9
Q

What structure stabilizes the articular disc to the condyle during function?

A

The superior head of the lateral pterygoid muscle.

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10
Q

What is the role of the retrodiskal tissue in TMJ function?

A

It provides vascular supply and elasticity, limiting excessive anterior translation of the disc.

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11
Q

What are the two primary types of TMJ movement?

A

Rotation and translation.

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12
Q

Which part of the TMJ is responsible for rotational movement?

A

The inferior joint compartment (between condyle and disc).

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13
Q

Which part of the TMJ is responsible for translatory movement?

A

The superior joint compartment (between disc and temporal bone).

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14
Q

How much pure hinge movement is possible in normal mandibular function?

A

About 2.5 cm at the incisal edges.

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15
Q

What is the maximum forward translation movement of the condyle?

A

Approximately 1.5 cm.

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16
Q

What structure prevents excessive posterior movement of the condyle?

A

The inner horizontal portion of the temporomandibular ligament.

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17
Q

What happens to the condyle-disc complex during mandibular opening?

A

It rotates first, then translates forward along the articular eminence.

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18
Q

How does lateral excursion of the mandible occur?

A

One condyle rotates while the other translates forward (Bennett movement).

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19
Q

What are the main arteries supplying the TMJ?

A

Superficial temporal and maxillary arteries.

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20
Q

Which artery supplies the anterior region of the TMJ?

A

The masseteric artery.

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21
Q

What is the function of the venous plexus in the retrodiskal tissue?

A

It fills and empties during jaw movements to maintain joint stability.

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22
Q

What nerve primarily innervates the TMJ?

A

The auriculotemporal nerve.

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23
Q

Which additional nerves contribute to TMJ innervation?

A

Masseteric nerve and posterior deep temporal nerve.

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24
Q

What type of nerve fibers are most common in the TMJ?

A

Vasomotor and vasosensory fibers involved in pain and synovial fluid production.

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25
Q

What is the function of the collateral (diskal) ligaments?

A

They attach the disc to the condyle and restrict excessive movement.

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26
Q

What is the primary role of the capsular ligament?

A

It encloses the TMJ and retains synovial fluid.

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27
Q

What is the function of the outer oblique portion of the temporomandibular ligament?

A

It limits excessive downward movement of the condyle.

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28
Q

What is the function of the inner horizontal portion of the temporomandibular ligament?

A

It restricts posterior movement of the condyle to protect the retrodiskal tissue.

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29
Q

What is the function of the sphenomandibular ligament?

A

It acts as a pivot point for mandibular movements.

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30
Q

What is the function of the stylomandibular ligament?

A

It limits excessive mandibular protrusion.

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31
Q

What type of tissue composes the articular disc?

A

Dense fibrous connective tissue.

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32
Q

Why is the articular disc avascular and non-innervated?

A

To withstand high mechanical stress without pain.

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33
Q

What are the three main bands of the disc?

A

Anterior band, intermediate zone, and posterior band.

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34
Q

What structure holds the articular disc in place?

A

The capsular ligament and collateral ligaments.

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35
Q

What are the two main components of the retrodiscal tissue?

A

The superior and inferior retrodiscal lamina.

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36
Q

What is the function of the superior retrodiscal lamina?

A

It contains elastic fibers that retract the disc posteriorly.

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37
Q

What is the function of the inferior retrodiscal lamina?

A

It prevents excessive forward displacement of the disc.

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38
Q

How are TMJ disorders generally classified?

A

As nonarticular (muscle-related) or articular (joint-related).

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39
Q

What are common examples of nonarticular TMJ disorders?

A

Myofascial pain dysfunction syndrome and occlusal trauma.

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40
Q

What are common examples of articular TMJ disorders?

A

Internal derangements, degenerative joint disease, and ankylosis.

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41
Q

What are the two main types of disc displacement?

A

Disc displacement with reduction and without reduction.

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42
Q

What are the primary symptoms of TMJ internal derangement?

A

Joint pain, clicking, locking, and limited jaw movement.

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43
Q

What are the three main classifications of TMJ hypomobility?

A

Trismus, pseudoankylosis, and true ankylosis.

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44
Q

What are common odontogenic causes of trismus?

A

Myofascial pain, malocclusion, and erupting teeth.

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45
Q

What are common infectious causes of trismus?

A

Pterygomandibular and lateral pharyngeal infections.

46
Q

What are common traumatic causes of trismus?

A

Mandibular fractures and muscle contusions.

47
Q

What are common psychological causes of trismus?

A

Hysteric trismus.

48
Q

What are common pharmacological causes of trismus?

A

Phenothiazine-induced muscle rigidity.

49
Q

What are common neurological causes of trismus?

A

Tetanus and neurological disorders affecting mastication.

50
Q

What is the primary imaging modality for assessing TMJ ankylosis?

A

CT scan for detailed anatomical evaluation.

51
Q

What are the two main types of TMJ hypermobility?

A

Subluxation and dislocation.

52
Q

How is subluxation different from dislocation?

A

Subluxation is self-reducing, while dislocation requires external reduction.

53
Q

What are common intrinsic causes of TMJ hypermobility?

A

Yawning, vomiting, wide biting, and seizure disorders.

54
Q

What are common extrinsic causes of TMJ hypermobility?

A

Trauma, intubation, endoscopy, and dental extractions.

55
Q

What are connective tissue disorders associated with TMJ hypermobility?

A

Hypermobility syndromes (e.g., Ehlers-Danlos syndrome).

56
Q

What is the initial management of a TMJ dislocation?

A

Manual reduction before muscle spasm develops.

57
Q

What are general non-surgical management options for TMJ hypermobility?

A

Diet modification, short dental appointments, and bite blocks.

58
Q

Why should TMJ dislocation be reduced quickly?

A

To prevent muscle spasm and difficulty in reduction.

59
Q

What is the role of sclerosing agents in chronic dislocation?

A

They induce fibrosis but may cause joint damage.

60
Q

What surgical techniques can be used for severe TMJ dislocation?

A

Plication procedures, zygomatic arch down-fracture, and tendon sacrifice.

61
Q

How is botulinum toxin (Botox) used in TMJ hypermobility?

A

It relaxes overactive muscles to prevent recurrent dislocation.

62
Q

What is the long-term concern with untreated TMJ hypermobility?

A

It may lead to joint degeneration and internal derangement

63
Q

What is the most common nonarticular TMJ disorder?

A

Myofascial pain dysfunction syndrome (MPDS).

64
Q

What is the occlusal trauma theory for MPDS?

A

It suggests that malocclusion causes abnormal muscle function leading to pain.

65
Q

What is the psychogenic theory for MPDS?

A

It suggests that stress and psychological factors contribute to TMJ pain.

66
Q

What are the primary signs and symptoms of MPDS?

A

Referred pain, limited range of motion, muscle tenderness.

67
Q

What are common trigger points in TMJ myofascial pain?

A

Masseter, temporalis, and lateral pterygoid muscles.

68
Q

What is the definition of internal derangement of the TMJ?

A

An abnormal positional and functional relationship between the disc and articulating surfaces.

69
Q

What is the most common direction of TMJ disc displacement?

A

Anterior displacement.

70
Q

What are the main etiological factors for TMJ disc displacement?

A

Trauma, functional overloading, joint laxity, and degenerative joint disease.

71
Q

What is the role of the lateral pterygoid muscle in TMJ disc displacement?

A

Hyperactivity may contribute to anterior disc displacement.

72
Q

What are the two types of disc displacement?

A

With reduction and without reduction.

73
Q

What are the clinical signs of disc displacement with reduction?

A

Joint clicking, pain, and deviation during mouth opening.

74
Q

What are the clinical signs of disc displacement without reduction?

A

Limited mouth opening (closed lock) and restricted lateral movement.

75
Q

Why is pain a major complaint in TMJ internal derangement?

A

Due to inflammation and strain on retrodiscal tissues.

76
Q

What systemic conditions can contribute to TMJ disc displacement?

A

Rheumatoid arthritis and connective tissue disorders.

77
Q

What are the primary imaging modalities used to diagnose TMJ internal derangement?

A

MRI (for disc position) and CT scan (for bony changes).

78
Q

What is functional overloading in TMJ disorders?

A

Excessive stress on the TMJ due to parafunctional habits like bruxism and clenching.

79
Q

What are the two main types of bruxism?

A

Awake bruxism and sleep bruxism.

80
Q

What are the common triggers for awake bruxism?

A

Stress, concentration, and emotional states.

81
Q

Why is sleep bruxism harder to manage than awake bruxism?

A

It occurs subconsciously during sleep and cannot be stopped voluntarily.

82
Q

What are the key clinical signs of bruxism?

A

Tooth wear, jaw pain, morning headaches, and enlarged jaw muscles.

83
Q

How does sleep bruxism differ from awake bruxism in terms of treatment?

A

Sleep bruxism often requires occlusal splints, while awake bruxism can be managed with behavior modification.

84
Q

What is the primary purpose of a night guard in bruxism treatment?

A

To protect teeth from wear and reduce TMJ strain.

85
Q

What systemic condition is often associated with sleep bruxism?

A

Obstructive sleep apnea.

86
Q

What are the two broad categories of TMJ internal derangement treatment?

A

Extrajoint therapy and intrajoint therapy.

87
Q

What are common forms of extrajoint therapy?

A

Splint therapy, therapeutic manipulation, physical therapy, and pharmacotherapy.

88
Q

What is the goal of splint therapy in TMJ disorders?

A

To stabilize the bite and reduce joint stress.

89
Q

What is the role of physical therapy in TMJ treatment?

A

It helps to reduce pain and improve jaw function.

90
Q

What is the most commonly used pharmacotherapy for TMJ pain?

A

NSAIDs, muscle relaxants, and corticosteroids.

91
Q

What are two primary types of intrajoint therapy?

A

Arthroscopy and arthrocentesis.

92
Q

How does arthroscopy help in TMJ disorders?

A

It allows visualization and treatment of joint pathology.

93
Q

What is arthrocentesis, and how does it work?

A

A minimally invasive procedure using sterile fluid to flush the joint and reduce inflammation.

94
Q

What is the main advantage of arthrocentesis over open surgery?

A

It is less invasive, has a shorter recovery time, and avoids major surgical risks.

95
Q

What is the primary indication for TMJ surgical treatment?

A

Severe pain and dysfunction that do not respond to conservative treatments.

96
Q

What dietary modifications are recommended for TMJ disorders?

A

Soft diet, avoiding hard or chewy foods, and eliminating gum chewing.

97
Q

How do NSAIDs help in TMJ pain management?

A

They reduce inflammation and relieve pain.

98
Q

What is the role of muscle relaxants in TMJ disorders?

A

They reduce muscle tension and spasms.

99
Q

What are the main goals of occlusal appliance therapy?

A

To redistribute occlusal forces and reduce joint strain.

100
Q

What is the purpose of jaw exercises in TMJ therapy?

A

To improve mobility and strengthen jaw muscles.

101
Q

What is transcutaneous electrical nerve stimulation (TENS) used for in TMJ therapy?

A

To relieve pain by stimulating nerve activity.

102
Q

How does ultrasound therapy help TMJ disorders?

A

It provides deep tissue heating to improve blood circulation and reduce pain.

103
Q

What is the primary role of heat therapy in TMJ treatment?

A

To relax muscles and increase blood flow.

104
Q

How does cryotherapy (cold therapy) help in TMJ disorders?

A

It reduces inflammation and muscle soreness.

105
Q

What is the purpose of trigger point injections in TMJ therapy?

A

To relieve pain from muscle spasms and hyperirritable trigger points.

106
Q

What is the main difference between passive and active jaw exercises?

A

Passive exercises involve assisted movement, while active exercises are performed by the patient.

107
Q

What is the most effective occlusal splint type for TMJ disorders?

A

A full-arch hard acrylic splint.

108
Q

What is the main mechanism of botulinum toxin (Botox) in TMJ treatment?

A

It blocks neuromuscular transmission to relax overactive muscles.

109
Q

What is arthrocentesis, and why is it considered a contemporary treatment?

A

A minimally invasive lavage technique to reduce joint inflammation.

110
Q

What is the purpose of intra-articular drug delivery in TMJ disorders?

A

To provide prolonged drug action within the joint space.