Rhythm Mechanisms Flashcards
(34 cards)
Sinus Rhythm
Normal heart rhythm - normal electrical activity starting in the SA node, heads down the normal conduction pathway through the atria, antrioventricular (AV) node and bundle, right and left bundle branches, and pukinje fibers.
Fires at rate of 60-100bpm
Rhythm: R-R and P-P intervals are regular
Rate: 60-100bpm
P waves: positive, precedes each QRS complex, all look alike
PR interval: 0.12 to 0.20 sec and constant
QRS duration: 0.11 sec or less
Sinus Bradycardia
SA node fires at a rate slower than expected for the patients age
Heart rate is 60bpm or less
Rhythm: R-R and P-P intervals are regular
Rate: Less than 60bpm
P waves: positive, precedes each QRS complex, all look alike
PR interval: 0.12 to 0.20 sec and constant
QRS duration: 0.11 sec or less
Causes: prolonged standing, stimulation of the vagus nerve, coughing, vomiting, straining to have a bowel movement, sudden exposure of the face to cold water.
Other causes: Disease of the SA node, hyperkalemia, hypokalemia, hypothermia, hypothyroidism, hypoxia, increased intracranial pressure, inferior myocardial infraction, medications (calcium blockers, digitalis, beta-blockers, amiodarone, and sotalol), sleep apnea, post heart transplant.
What to do about it: assessment (no symptoms, no treatment needed). If symptomatic, assess patients oxygen saturation level and determine if signs of increased breathing effort are present. Give supplemental oxygen, establish IV access and obtain 12 lead ECG. Atropine is drug of choice for SB
Sinus tachycardia
SA node fires at a rate faster than normal for the patients age. Begins and ends gradually.
Rhythm: R-R and P-P intervals are regular
Rate: ventricular rate over 100bpm (max 220bpm)
P waves: positive, precedes each QRS complex, all look alike
PR interval: 0.12 to 0.20 sec and constant
QRS duration: 0.11 sec or less
Causes: acute myocardial infraction, anemia, anxiety, fear, caffeine, dehydration, drugs, exercise, fever, heart failure, hyperthyroidism, hypoglycemia, hypoxia, infection, medications, pain, panic attack, pericarditis, pneumothorax, postural changes, pregnancy, shock, sepsis
What to do about it: what is the underlying cause? (Fluid replacement, pain, removal of offending medication or substances, reducing fever). Lifestyle changes.
Sinus arrhythmia
SA node fires irregularly. Sinus arrhythmia usually occurs at a rate of 60-100bpm (most common type of sinus arrhythmia)
Rhythm: irregular and often phasic with breathing (heart rate increases during inspiration, R-R intervals shorten. heart rate decreases with expiration, R-R intervals lengthen)
Rate: 60-100bpm
P waves: positive, precedes each QRS complex, all look alike
PR interval: 0.12 to 0.20 sec and constant
QRS duration: 0.11 sec or less
Causes: occurs with phases of breathing and changes in intrathoracic pressure. Heart rate increases with inspiration and decreases with expiration.
Non respiratory sinus arrhythmia may result from effects of medication or carotid sinus pressure
What to do about it: usually does not require treatment unless accompanied by a slow heart rate that causes hemodynamic compromise. IV atropine may be indicated to treat.
Sinoatrial block
Sinoatrial block is called sinus exit block. SA node’s pacemaker cells initiate an impulse, but it is blocked as it exits the SA node, resulting in periodically absent PQRST complexes.
Rhythm: Irregular because of the pause - pause is exact distance of P-P intervals (even multiple) between it
Rate: normal but varies due to pause
P Wave: when present, positive and upright, precedes QRS complex and look alike
PR interval: when present 0.12-0.20sec and constant
QRS duration: 0.11sec or less
Causes: hypoxia, damage or disease to the SA node from CAD, myocarditis, or acute MI; carotid sinus sensitivity; increased fatal tone on the SA node and medication
What to do about it: depend on number of sinus beats blocked. No symptoms, patient is observed. SA block episodes are frequent, IV atropine, temporary pacing, or insertion of permanent pacemaker.
Sinus arrest
Disorder of impulse formation. SA node’s pacemaker cells do not initiate an electrical impulse for one or more beats, resulting in absent PQRST complexes.
Rhythm: irregular; more than one missing PQRST and is not the same distance as other P-P intervals
Rate: normal
P Waves: when present, positive and upright, precedes QRS, look a like
PR intervals: when present, 0.12-0.20sec and constant
QRS duration: 0.11sec or less
Causes: damage to or disease of the SA node from CAD, acute MI or rheumatic disease, carotid sinus pressure, a sudden increase of parasympathetic activity on the SA node, stimulation of the pharynx, obstructive sleep apnea, medications
What to do about it: signs and symptoms are observed from carotid sinus sensitivity, vagal stimulation, removal of tight clothing are option. If hemodynamic compromise present, IV atropine, temporary pacing or both.
Atrial dysrhythmias: mechanisms
Reflect abnormal electrical impulse formation and conduction in the atria.
Result from altered automaticity, triggered activity, or reentry.
Altered automaticity and triggered activity are disorders of impulse formation.
Reentry is a disorder in impulse conduction
Include:
(altered automaticity) premature atrial complexes (PAC), multifocal atrial tachycardia (MAT), and atrial fibrillation (AFib)
(Reentry) atrial flutter, AVNRT, and AVRT
Premature beats
Premature beats are identified by the following site of origin:
-Premature atrial complexes (PACs)
-Premature junctional complexes (PJCs)
-Premature ventricular complexes (PVCs)
Premature patterns
Couplet - two premature beats in a row
Runs or Bursts - three or more premature beats in a row
Bigeminy - every other beat is a premature beat
Trigeminy - Every third beat is a premature beat
Quadrigeminy - every fourth beat is a premature beat
Premature atrial complex (PAC)
Early beat occurring before the next expected beat
Rhythm: irregular - early beat
Rate: usually within normal range
P waves: premature, positive (upright) that differ in shape from sinus P waves. Early P waves that may or may not be following QRS complex
PR interval: normal or prolonged depending on prematurity of beat
QRS duration: 0.11sec or less (may be wide or absent)
Causes: altered automaticity or reentry. Other - acute coronary syndromes, atrial enlargement, digitalis toxicity, electrolyte imbalance, emotional stress, heart failure, mental and physical fatigue, caffeine, drugs, medications
What to do about it: correcting underlying cause - correcting electrolyte imbalances, reducing stress, reducing or eliminating stimulants, treating heart failure
Noncompensatory pause
Follows a PAC, representing the delay during which the SA node resets its rhythm for the next beat
Compensatory pause
If the period between the complex before and after a premature beat is the same as two normal R-R intervals
How to find out if it’s a noncompensatory or compensatory pause?
To find out whether the pause after a premature complex is compensatory or noncompensatory, measure the distance between R-R intervals of 3 normal beats. Then compare that measurement with the distance between the R-R intervals of 3 beats, one of which includes the premature complex.
Pause is noncompensatory if the period between complex before and after a premature beat is less than two normal R-R intervals
Pause is compensatory if the period between the complex before and after premature beat is the same as two normal R-R intervals
Abberrantly conducted premature atrial complexes
If a PAC occurs very early, the right bundle branch can be slow to respond to the impulse. The impulse travels down the left bundle branch with no problem. Stimulation of the left bundle brace subsequent results in stimulation of the right bundle branch. The QRS will appear wide (greater than 0.11sec) because of the delay in ventricular depolarization.
PACs associated with a wide QRS complex are called aberrantly conducted PACs, indicating that conduction through the ventricles is abnormal
Nonconducted (or blocked) premature atrial complexes
When a PAC occurs very early and close to the T wave of the preceding beat, only a P wave may be seen with no QRS after it, appearing as a pause. This type of PAC is called nonconducted or blocked PAC because the P wave occurred too early to be conducted
Wandering Atrial Pacemaker (WAP) aka Multiform Atrial Rhythm (MAR)
Requires at least three different p waves seen in the same lead
P waves, size, shape, and direction vary, sometimes from beat to beat
Rhythm: irregular
Rate: 60-100bpm (if rate is faster than 100bpm, its termed Multifocal Atrial Tachycardia)
P waves: size, shape, direction may change form beat to beat, may be upright, inverted or biphasic, rounded, flat, pointed, notched, or buried in QRS
PR interval: varies as pacemaker site shifts from SA node to ectopic
QRS duration: 0.11sec or less
Causes: observed in normal hearts, during sleep, can occur with underlying heart disease
What to do about it: usually resolves on its own
Multifocal atrial tachycardia
When wandering atrial rhythm is associated with ventricular rate of more than 100bpm
Rhythm: irregular
Rate: over 100bpm
P waves: size, shape, direction may change form beat to beat, may be upright, inverted or biphasic, rounded, flat, pointed, notched, or buried in QRS; at least 3 different P wave configurations
PR interval: varies as pacemaker site shifts from SA node to ectopic
QRS duration: 0.11sec or less
Causes: may involve altered automaticity or triggered activity. May be seen in older adults with severe COPD, coronary syndromes, heart failure, pneumonia, hypoxia, valvular heart disease, digoxin toxicity.
What to do about it: consult cardiologist to start treatments. Beta blocker, calcium blocker, pacemaker.
Supraventricular Tachycardia
Begin above the bundle of his. Include rhythms that begin in the SA node, atrial tissue, or AV junction
3 examples of SVT
-Atrial tachycardia (AT)
-Atrioventricular nodal reentrant tachycardia (AVNRT)
-Atrioventricular reentrant tachycardia (AVRT)
Atrial tachycardia
3rd most common type of SVT
Regular rhythm that arises from an ectopic focus in the atria at a rate faster than 100bpm and does not require the AV node’s participation to maintain the dysrhythmia
Rhythm: regular
Rate: 101-250bpm
P waves: 1 P wave precedes QRS complex; P wave differ in shape; isoelectric baseline is usually present between P waves; P waves will be negative if rhythm originates in low portion of atrium. Could be hard to distinguish P waves from T waves
PR interval: may be shorter or longer; difficult to measure because P waves could be hidden in the T waves
QRS duration: 0.11sec or less
Causes: acute illness with excessive catecholamine release, digitalis toxicity, electrolyte imbalance, heart disease, infection, pulmonary embolism, stimulant use
What to do about it: applying pulse oximeter, administering oxygen, obtaining vital signs, IV access, 12 lead ECG.
Paroxysmal supraventricular tachycardia (PSVT)
Describes a rapid, regular SVT that starts and ends suddenly
PSVT may las for minutes, hours, or days
Synchronized Cardioversion
Type of electrical therapy during which a shock is timed or programmed for delivery during ventricular depolarization
Delivering a shock during the QRS complex reduces the potential for the delivery of current during ventricular depolarization including the vulnerable period of the T wave
Atrioventricular Nodal Reentrant Tachycardia (AVNRT)
Most common SVT
Results from a reentry circuit that uses two separate pathways leading into the AV node. One pathway conducts impulses rapidly but has a long refractory period (slow recovery time). The other pathway conducts impulses slowly but has a short refractory period (fast recovery time).
Caused by single or multiple PACs.
Rhythm: regular
Rate: 150-250bpm
P waves: often hidden in QRS, can have a negative P wave after QRS; atria depolarized after the ventricles, the P wave typically distorts the end of the QRS complex
PR interval: not measurable
QRS duration: 0.11 sec or less
Causes: often seen young adults who have no structural heart disease or ischemic heart disease. Occurs in patients with COPD, CAD, valvular heart disease, heart failure and digitalis toxicity. Can be triggered by hypoxia, stress, anxiety, caffeine, sleep deprivation and medications.
What to do about it: pulse oximeter, administering supplemental oxygen, IV access, and sedation.
Preexcitation
Refers to the premature activation of the ventricles by a supraventricular impulse arising from an accessory pathway
Atrioventricular reentrant tachycardia (AVRT)
Most common type of SVT.
Number of atrial impulses reaching the ventricles may approach 300 - 350bpm, significantly increasing risk of ventricular fibrillation
