Chapter 6 Flashcards
Premature Ventricular Complexes (PVC)
AKA premature ventricular extrasystole, ventricular premature beat, or premature ventricular depolarization
Rhythm: irregular; if the PVC is interpolated, PVC will be regular
Rate: normal range
P waves: absent, with retrograde conduction to the atria, may appear after QRS
PR interval: none because the ectopic beat originates in the ventricle
QRS duration: 0.12sec or more (wide)
Causes: acid base imbalance, acute coronary syndromes, digitalis toxicity, stimulants, normal variant, medications, ventricular aneurysm, hypoxia, heart failure, exercise, emotion stress
What to do about it: most do not require treatment, focusing on treatment of potentially reversible causes, ambulatory monitoring, beta blockers, calcium blockers
Fusion beat
Result of an electrical impulse from a supraventricular site discharging at the same time as an ectopic site in the ventricles.
Because fusion beats result from both supraventricular and ventricular depolarization, these beats do not resemble normally conducted beats, nor do they resemble true ventricular beats
Three or more sequential PVCs are termed a run, a salvo or burst, and three or more PVCs that occur in a row at a rate of more than 100bpm is considered a run of VT
Uniform and multiform premature ventricular complexes
PVCs that look alike in the same lead and begin from the same anatomical site are called uniform PVCs
PVCs that look different from one another in the same lead are called multiform PVCs
Unifocal and Multiform
Unifocal - arise from the same anotomic site within the ventricles
Multiform - arise from different anotomic sites
Unifocal and Multiform
Unifocal - arise from the same anotomic site within the ventricles
Multiform - arise from different anotomic sites
Interpolated premature ventricular complexes
PVC occurs between two normally conducted QRS complexes without interfering with the normal cardiac cycle
Does not have a full compensatory pause; it is squeezed between two normally conducted QRS complexes (the R-R intervals between sinus beats remain the same) and does not disturb the next ventricular depolarization or SA node activity
R on T premature ventricular complexes
R on T PVC occurs when the R wave of a PVC falls on the T wave of the preceding beat
Because ventricular depolarization is not yet complete during the last half of the T wave, it is possible that a PVC that occurs during this period will precipitate VT or VF
R on T phenomenon refers to the start of a ventricular tachydysrythmia because of an improperly timed electrical impulse on the T wave
Ventricular escape beats or rhythms
Escape beats are late
Occurs after a pause in which a supraventricular pacemaker failed to fire
Protective mechanism safeguarding the heart from more extreme slowing or even asystole; because it is protective, you do not want to administer any medication that would wipe out the escape beat
Rhythm: irregular
Rate: normal
P waves: absent or with retrograde conduction to the atria
PR interval: none because VEB originate from the ventricles
QRS duration: 0.12sec or more (Wide)
Causes: acute coronary syndrome, digitalis toxicity, metabolic imbalance. SA node and AV junction fail to initiate electrical impulse
What to do about it: pulse oximeter, 12 lead ECG, atropine
Idioventricular rhythm
Three or more ventricular escape beats occur in a row at a rate of 20 to 40bpm. QRS is wide
Rhythm: regular
Rate: 20 to 40bpm
P waves: absent or with retrograde conduction to the atria
PR interval: none
QRS: 0.12sec or greater (wide)
Accelerated Idioventricular rhythm (AIVR)
Exist when three or more ventricular beats occur in a row at a rate of 41 to 100bpm
Rhythm: regular
Rate: 41 to 100bpm
P waves: absent or with retrograde conduction to the atria
PR interval: none
QRS duration: 0.12sec or more (wide)
Cause: acute MI, interventional coronary artery procedures. Acute myocarditis, cardiomyopathies, cocaine toxicity, digitalis toxicity, electrolyte imbalance, hypertensive heart disease, subarachnoid hemorrhage
What to do about it: generally does not require treatment. Symptomatic: apply pulse oximeter and administer oxygen, 12 lead ECG, IV access, stimulate SA node and improve AV conduction, atropine.
Ventricular tachycardia (VT)
Exists when three or more sequential PVCs occur at a rate of more than 100bpm
VT may occur with or without pulse
Occur as a short run that lasts less than 30sec
Monomorphic ventricular tachycardia
When the QRS complexes of VT are of the same shape and amplitude
Also called ventricular flutter
Rhythm: regular
Rate: 101 to 250bpm
P waves: usually not seen
Causes: acid base imbalance, acute coronary syndromes, cocaine or methamphetamine abuse, electrolyte imbalance, structural heart disease, trauma, tricyclic antidepressant overdose
What to do about it: beta blocker, ambulatory monitoring or use of an implemented cardiac monitor may be ordered
Polymorphic ventricular tachycardia (PMVT)
QRS complexes vary in shape and amplitude from beat to beat and appear to twist from upright to negative or negative to upright and back, resembling a spindle
Rhythm: regular or irregular
Rate: 150 to 300bpm
P waves: none
PR interval: none
QRS duration: 0.12 sec or more (wide)
Torsades de pointes (TdP)
Causes: long QT interval. QT interval may be congenital, acquired or idiopathic
What to do about it: different depending on the patient
Ventricular Fibrillation
Chaotic rhythm that begins in the ventricles. No organized ventricular depolarization. Ventricular muscle quivers and there is no effective myocardial contraction and no pulse. No normal looking waveforms are visible.
Rhythm: rapid and chaotic with no patter or regularity
Rate: cannot be determined because there are no discernible waves or complexes to measure
P waves: none
PR interval: none
QRS duration: none
Cause: acute coronary syndromes, dysthymias, electrolyte imbalance, environmental factors, hypertrophy, increased sympathetic nervous system activity, proarrhythmic effect of anti arrhythmic and other medications, severe heart failure, structural heart failure, structural heart disease, vagal stimulation
What to do about it: patient in VF is unresponsive, apneic, and pulseless. CPR. Medication intervention.
Defibrillation
The delivery of an electrical current across the heart muscle over a very brief period to terminate an abnormal rhythm
Unsynchronized countershock or asynchronous countershock because current delivery has no relationship to the cardiac cycle