Rhinology Flashcards

Nasal obstruction , epistaxis, polyposis, Sinusitis, Allergic rhinitis, vasomotor/non-allergic rhinitis, Deviated Nasal Septum

1
Q

What is Epistaxis?
Is it typically unilateral or bilateral?

A

Nose bleed
Epistaxis is typically unilateral but may be bilateral. It is important to take the whole history of it into consideration. Unilateral epistaxis consistently may be a JNA (if young male for ex). But if someone constantly picks their nose, probably in the long hx itll be bilateral, once from each side. Another thing is how much blood? Could be it sphenopalatine or is it the little’s area. Always think of the bigger picture. Unilateral epistaxis is considered a red flag but is also the normal presentation!

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2
Q

A patient presents with bleeding from the nose. What are the most common sources?

A

Most common: Little’s area or Kiesselbach’s plexus (90%) followed by sphenopalatine artery

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3
Q

What arteries form Little’s area or Kiesselbach’s plexus

A

This plexus is the anastomosis of the external and internal carotid arteries

External carotid -> Imax (internal maxillary) -> SPA - Sphenopalatine + GPA - Greater Palatine
External carotid -> Facial artery -> SLA - Superior labial artery

Internal carotid -> Ophthalmic artery -> Anterior and posterior ethmoidal arteries

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4
Q

A patient presents with a large volume of nasal bleed or epistaxis. What artery is the most likely source?

A

Sphenopalatine artery

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5
Q

A 16 year old male presents to the OPD with long-standing epistaxis unilaterally. what is the most likely diagnosis?

A

JNA tumour (juvenile nasopharyngeal angiofibroma)

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6
Q

List the causes/RFs of Epistaxis (5)

A

Digital trauma/nose picking
Mucosal irritation: Allergic rhinitis, recurrent sinusitis, cold, coughs, flu)
Neoplastic (JNA in young male)
Drugs : Anti-coag, warfarin, heparin, aspirin - CVD patients!!!)
Coagulopathies (e.g. VWF, hemophilia)
HHT - Hereditary Hemorrhagic Telangiectasia
HTN

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7
Q

Your nephew begins to have a nose bleed. How would you manage them?

A

Pinch anterior 1/2 of nose (cartilaginous part) for 10 minutes x2, with head tilted slightly forward. If this does not stop the bleed, I will probably have to take them to the hospital for silver nitrate cautery

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8
Q

What artery is most commonly embolized in the last-line treatment of epistaxis?

A

Sphenopalatine artery most commonly cause of major epistaxis => embolizing the Imax or internal maxillary would be best

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9
Q

State your management escalations for Epistaxis starting with first aid.

A

1) Pinch anterior 1/2 of nose (cartilaginous part) for 10 minutes x2, with head tilted slightly forward.
2) Silver Nitrate Cautery
3) Nasal Packing
4) Artery Ligation
5) Embolization of Imax

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10
Q

An adolescent male presents to the OPD reporting nasal bleed on the left side happening more often in the past 2 months. They report weight loss but arent sure if it is from starting to practice on a new college football team. What is the most likely diagnosis?

A

JNA (juvenile nasopharyngeal angiofibroma). Unilateral epistaxis in an adolescent male.

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11
Q

List the causes of Nasal Obstruction
List the common symptoms of Nasal Obstruction

A

Categorize!!!
Congenital: Choanal atresia, Craniofacial abnormalities (high arched palate)
Anatomical: Deviated septum, turbinate hypertrophy, fracture
Inflammatory: Polyps, Rhinosinusitis, allergic rhinitis, sarcoidosis, wegner’s granulomatosis
Neoplastic: SCC, JNA, inverted papilloma
Foreign body

Symptoms of Nasal Obstruction:
NASEEM: Nasal obstruction, Anosmia, Sinusitis, Epistaxis, External Deformity, Middle ear infection + Headaches, breathing difficulties, sleeping difficulties…

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12
Q

What congenital syndrome is most associated with high arched palate?

A

Marfan’s Syndrome

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13
Q

What is a spur?

A

Shelf-like septal deformity that touches the lateral wallL

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14
Q

Deviated Nasal Septum: There are 4 types including anterior dislocation info one of the chambers, C-shaped dislocation, S-shaped deformity (affecting bothchambers), or spurs (shelf).
State 3 causes
State 5 Symptoms
How would you manage a patient with DNS

A

Causes: Trauma, Congenital (high arched palate/Marfan’s), Family Hx, Idiopathic

Symptoms: Most commonly asymptomatic
NASEEM: Nasal obstruction, Anosmia, Sinusitis, Epistaxis, External Deformity, Middle ear infection + Headaches, breathing difficulties, sleeping difficulties…

Management: Conservative: No tx indicated if no sx
Surgical: Septoplasty or Submucosal Resection

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15
Q

What is septoplasty?

A

Procedure whereby most deviated parts of septum are removed and repositioned. Used in the tx of deviated nasal septum

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16
Q

Submucosal resection is a procedure used in the treatment of

A

Deviated nasal septum (septoplasty typically chosen first)

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17
Q

Define Nasal Polyposis
What are the clinical features of Nasal polyposis?
What are some findings expected on examination?
How would you manage a patient suffering from

A

Inflammatory process where the now oedematous mucosa of the (most commonly ethmoid) sinus prolapse into nasal cavity leading to nasal obstruction

Clinical features: Nasal Obstruction ( NASEEM + Headaches, breathing difficulties, sleeping difficulties…) + Allergy part: !sneezing, itchy nose

Exam: Pale, boggy (soft and wet), oedematous mucosa (not inflamed or erythematous, just oedematous)

Management:
Conservative: Adherence asthma medications (if asthmatics) or antihistamines (for allergic rhinitis)
Medical: Steroids (Spray 6/12, Drops 2-3/52, PO 10 days)
Surgical: If refractory to medical management -> Endoscopic Polypectomy (as part of FEES - Functional Endoscopic Sinus Surgery)

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18
Q

What population characteristically have Nasal polyposis?

A

Asthma population, Allergic rhinitis

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19
Q

What is Samter’s Triad?

A

Triad encompassing pattern of symptoms in individuals
1) Polyposis
2) Asthma
3) Aspirin Sensitivity!!

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20
Q

Are Nasal Polyps typically unilateral or bilateral?

A

Bilateral => If you find a unilateral polyp it is important to be suspicious of a neoplastic process

21
Q

You are called to see a patient in A&E suffering from recurrent sinusitis and otitis media due to nasal obstruction. On exam you see a polyp in the left nostril and none in the right. What is your general workup

A

Polyps are typically bilateral but it is unilateral here => workup for cancer. Normal polyps dont really have a workup unless checking if patient has asthma cuz of samter’s triad

Always biopsy, CT sinus, and MRI brain/skull base

22
Q

What is the most common location for a nasal polyp?
What is the most involved sinus in polyposis? Where does it drain?

A

Middle Turbinate/Middle meatus
Remember it is a cyst-like sac => from drainage from all 3 sinuses => most likely there at the osteomeatal complex

The most involved sinus in polyposis is the Ethmoidal sinus which drains into 2 places
Post. Ethmoidal -> Superior meatus
Ant. Ethmoidal -> Middle Meatus

23
Q

The nasal cavity is composed of 3 turbinates. Each of which has a meatus. What drains into each?

A

Superior Meatus: Post-ethmoidal
Middle Meatus/Osteomeatal complex: Ant. Ethmoidal, frontal, maxillary
Inferior Meatus: Nasolacrimal duct

24
Q

Give the Diagnostic Definition of Sinusitis
i.e. Definition that encompasses diagnostic criteria of sinusitis

A

Nasal Obstruction(/congestion/nasal discharge) (encompassing RF)
+ At least 1: Facial pain, hyposmia, anosmia (clinical features)
+ Endoscopic/CT findings showing polyposis, obstruction, or discharge in the osteomeatal complex (Diagnosis)

25
Q

What are sinuses?
Humans have 4 paired sinuses. List them along with where they drain.

A

Sinuses are hollow spaces in bone around the nasal cavity. They typically only have one opening => easily obstructed especially when mucosal layer is inflamed (oedematous => polyposis and obstruction)

Maxillary sinus -> osteomeatal complex, middle meatus
Frontal Sinus -> osteomeatal complex, middle meatus
Ethmoidal Sinus Ant. ->osteomeatal complex, middle meatus. Post. Superior meatus
Sphenoid Sinus -> Sphenoethmoidal recess. Close to sup. Meatus.

26
Q

What is a turbinate? What is its main function?

A

Bone structures covered by mucosa. They regulate, warm, and humidify airflow. That is why when you breathe through your mouth, it becomes so dry so fast

27
Q

What causes sinusitis?
List Clinical features for sinusitis (not complications)
What is the gold standard to diagnose Sinusitis?
How would you manage a patient with acute Sinusitis

A

Typically, it is a vital infection => viral rhinosinusitis (98%). This can then be superinfected with bacterial => Bacterial rhinosinusitis. Alternatively, in the immunocompromised population, fungal sinusitis may also occur.

Clinical features
1) Nasal obstruction: NASEEM + Headaches, breathing difficulties, sleeping difficulties…
2) Sinusitis: pyrexia, !!!Facial pain/pressure, Anosmia, sneezing

Diagnosis:
Endoscopy (or CT) findings showing polyposis, mucopurulent discharge, or oedema/mucosal changes in osteomeatal complex

Add these 3 together = Diagnostic definition of sinusitis

Management of acute sinusitis:
Advice: Hydration, humidification, warm compress (good for any nasal problem)
Viral: Antipyretic + Decongestants + Mucolytics + Analgesics
Bacterial: Viral + 10-14 days Amoxicillin/Clarithromycin/C-amox

28
Q

Typically, it is a vital infection => viral rhinosinusitis (98%). This can then be superinfected with bacterial => Bacterial rhinosinusitis. List the organisms responsible for acute and chronic bacterial rhinosinusitis

A

Acute (<12 weeks) = S. Pneumonia, H. Influenzae, M. Catarrhalis (same as AOM)
Chronic (>12 weeks) = Anaerobes, staph aureus

29
Q

List RFs for Sinusitis

A

1) Obstructive: Nasal obstruction e.g. polyps, neoplastic, DNS
2) Allergic -> Allergic rhinitis, asthma (both give polyps)
3) Environmental -> Smoking exposure, occupational, chemical, medications
4) Congenital -> Anatomical abnormalities (craniofacial abnormalities), high arched palate, DNS, Choanal atresia
5) Immunodeficiency -> Diabetes, HIV, Chemotherapy, radiotherapy, splenectomy, autoimmune drugs…
6) Reduced mucocilliary clearance: CF, primary cilliary dyskinesia, kartagener syndrome (increased stasis => infection)

30
Q

What is Otia?

A

Obstruction of a sinus opening, typically from inflammation

31
Q

How would you treat a patient with severe bacterial rhinosinusitis?

A

Antipyretic + Decongestants + Mucolytics + Analgesics + 10-14 days Amoxicillin/Clarithromycin/C-amox + Prednisolone PO
Also for patients with recurrent rhinosinusitis

32
Q

How would you treat a patient with recurrent bacterial rhinosinusitis on a background of allergic rhinitis?

A

Antipyretic + Decongestants + Mucolytics + Analgesics + 10-14 days Amoxicillin/Clarithromycin/C-amox + Antihistamine/nasal steroid

33
Q

What would warrant ENT referral for sinusitis AKA for surgery?

A

1) 3-4 infections per year (multiple)
2) Failure to improve after 2-3 weeks on antibiotics
3) Nasal Polyposis on exam (part of same surgery)
4) Complications of sinusitis

34
Q

List the complications of sinusitis
Which complication is the most common in adults?
What population do most of these complications occur in?

A

1) Orbital Complications (orbital cellulitis, abscess, and cavernous thrombosis)
2) Intracranial complications (meningitis, frontal lobe abscess)
3) Osteomyelitis
4) Acute Otitis Media
5) LRTI (most common adults)
6) Mucocele formation (-> obstruction)

Sinusitis and its complications are common in paediatric population

35
Q

How long does a patient have to have sinusitis to be considered chronic sinusitis?

A

Sinusitis lasting more than 12 weeks

36
Q

Orbital complications of sinusitis are one of the more severe ones to keep an eye out for.
What is the relation of the orbit to sinusitis?
What sinus is most associated to orbital complications of sinusitis? What population is that seen most in?
What classification is used to grade the extent of these complications? List the complications.
What clinical features are associated with these complications?
How are these complications treated?

A

Howdoes Sinusitis lead to orbital complications?
Bacteremia occurs via veins and bones (!Lamina Papyracae) of the sinuses

What sinus is most associated to orbital complications of sinusitis? What population is that seen most in?
Ethmoidal sinuses seen typically in the paediatric population

What classification is used to grade the extent of these complications? List the complications.
Chandler’s Classification:
I - Preseptal/orbital cellulitis
II - Orbital Cellulitis
III - Preseptal/orbital abscess
IV - Orbital abscess
V - Cavernous Sinus Thrombosis

What clinical features are associated with these complications?
Lid swelling/redness +/- proptosis of the eye
Opthalmoplegia
Vision disturbances (color goes first)
Cavernous sinus disturbance (CNII, III, IV, Va, VI)

How are these complications treated?
IV antibiotics +/- nasal toilet
Drain abscess if abscess

37
Q

The cavernous sinus runs behind the sphenoid sinus, next to the optic chiasm (note that). What runs in the cavernous sinus?
How would you diagnose cavernous sinus thrombosis as a complication of sinusitis?

A

Internal carotid artery, CN2,3,4,5A,6
From this you should understand how to add all these sx to a patient that has this affected
CT sinus +/- MRI brain

38
Q

What intracranial complications may arise from Sinusitis?
What sinus is most associated with these complications?
How would you manage a patient with these complications?

A

Meningitis
Intracranial abscess (epidural… not hematomas)
!Frontal lobe abscess

Frontal sinus most associated => frontal lobe abscess. Frontal lobe contains many veins that penetrate post. of sinus

Tx: Aggressive antibiotics (3rd gen cephalosporins, cefotaxime, ceftriaxone)
Surgical drainage of abscess
Topical vasoconstriction

39
Q

What is “Potts Puffy Tumor”?
What sinus is most involved?

A

It is osteomyelitis of the frontal bone. Similar to how the frontal sinus may cause intracranial complications (abscess, meningitis) due to its proximity, the location of the sinus within the frontal bone makes this more likely.

40
Q

What is allergic rhinitis? What are the 2 types?
List clinical features and exam findings of Allergic rhinitis
What are some conditions that allergic rhinitis increases the risk for?
What population is this condition common in?
How would you diagnose allergic rhinitis?
State the full treatment ladder for allergic rhinitis (5)

A

Allergic rhinitis is inflammation of the nose due to allergens in the environment causing Type 1 hypersensitivity. (you didnt actually need to say all that). It may be Seasonal or perennial.

Clinical features:
Nasal obstruction: NASEEM + Headaches, breathing difficulties, sleeping difficulties…
Allergy: Sneezing and itching, watery eyes, Conjunctival swelling, headache
Exam findings: Pale, oedematous nasal mucosa, Darcyocystitis, swollen turbinates, oedematous mucosa, conjunctival swelling

Allergic rhinitis is common in asthma populations and can increase the risk of !Sinusitis, ET dysfunction (AOM, conductive hearing loss, glue ear…), and !Nasal polyps

Diagnosis:
1) Full hx and exam showing clinical features of allergic rhinitis
2) Paediatrics: RAS test - Radioallergosorbent test
Adults: Skin prick test
3) Bloods: Showing increased serum-specific IgE and Increased eosinophils on FBC

Tx ladder:
1) Prevention: avoidance, face mask in pollen season, remove carpets, pets, dolls, avoid smoking at home! etc..
2) Antihistamines (topical or oral)
3) Nasal steroid spray (Fluticasone)
4) Antileukotrienes
5) Immunotherapy by immunologist

41
Q

What is Non-Allergic Rhinitis?
Patients presenting with non-allergic rhinitis typically present similarly to that of allergic rhinitis. How would you differentiate to diagnose this?
What is the treatment approach?

A

It is Vasomotor rhinitis due to hyperactive nasal mucosa

Patients here typically complain of nasal blockage, clear discharge after a non-specific irritant exposure as opposed to allergic rhinitis which has an allergen.

To confirm this, the skin prick test should be negative in vasomotor rhinitis.

Another way is to inspect the nasal mucosa. It is often red and boggy here whereas it is pale and boggy in allergic rhinitis (and nasal polyps cuz same etiology)

Tx: Medical: Antihistamines
Surgical: All we can do is make more space => maybe adenectomy? (just get correct concept thats why “approach

42
Q

What is seen in this image?
What are the possible causes? (3)

A

Saddle nose dformity. It is caused by the necrosis of the cartilaginous pyramid (just cartilage) of the nose.

Wegner’s Granulomatosis/Granulomatosis with polyangitis. Chronic rhinosinusitis here leads to its eventual breakdown and collapse
Trauma
Congenital

43
Q

Features of which disease are displayed in this image?
In ENT, what clinical symptom does this mainly increase the risk of?

A

HHT - Hereditary Hemorrhagic Telangiectasia (these findings are also seen in liver disease)
It is widened hemorrhagic vessels

Epistaxis

44
Q

What is meant by vestibule? what are its parts?

A

Part of nose involved in anterior rhinoscopy involving the Septum, inferior turbinate and floor of nose

45
Q

When inspecting a patient in an ENT clinic, you would like to conduct anterior rhinoscopy
What are you inspecting and assessing?
With what instrument would you conduct this?

A

In anterior rhinoscopy, we are inspecting the vestibule (septum, inferior turbinate, and floor of nose) with a Thudicum or Killian speculum. During this inspecting, we will assess for turbinate size, septal deviation, presence of polyps, drainage, masses, foreign bodies…

46
Q

What is this?

A

This is Thudicum’s Speculum used in Anterior Rhinoscopy

47
Q

What is this?

A

This is Killian’s Speculum used in Anterior Rhinoscopy

48
Q

What device is used to inspect the nasopharynx?

A

Rigid/Flexible endoscopy

49
Q

Why would rigid endoscopy be used over flexible endoscopy

A

Rigid endoscopy allows for the other hand to be free as compared to flexible endoscopy which is a 2-hand operation to maneuver the scope