Paediatric ENT Flashcards

1
Q

Why is ET dysfunction and its complications (AOM, Glue ear…) more common in the paediatric population?

A

1) ET in children is shorter and straighter => tighter
2) Recurrent URTIs => Increased secretions and congestion
3) Adenoid Hypertrophy => Less space => exacerbation. Dissolves in adults => resolved

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

You are asked to see a paediatric patient with some otological problem. Give 6 Ddx for Otological problems in paediatric population

A

1) Glue Ear
2) Acute Otitis Media
3) Acute Mastoiditis
4) Foreign Body
5) Hearing Loss
6) Congenital (absent antihelix, prominent ears)
7) Neoplastic (V. unlikely)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

A 7 year old boy presents to the OPD for a checkup. You note his ears are prominent. What is the most common reason for his prominent ears?
What is general workup and treatment for this patient?

A

Most common: Antihelix > Deep Concha, protruding lobule

Workup: Check hearing in examination and Pure tone audiometry
Tx: Pinnaplasty/Otoplasty

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Adenoids are lymphoid tissue located at the superior posterior wall of the nasopharynx. It typically increases in size until 7 years old before starting to shrink.
What paediatric conditions may arise from adenoid hypertrophy?
If adenoids are found to be the cause or an exacerbating factor to any of these conditions, what is the tx?

A

As it is a lymphoid tissue, any infection may lead to its hypertrophy => obstruction

OSA (snoring, sweaty sleeps..)
ET dysfunction => Retracted TM, Glue Ear, Recurrent OM

Tx: Adenoidectomy may be escalated to adenotonsillectomy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

You are asked to see a paediatric patient with some rhinological problem. Give 7 Ddx for Rhinological problems in paediatric population

A

1) Epistaxis
2) Allergic Rhinitis (seasonal or perennial)
3) Adenoid hypertrophy
4) Sinusitis (w/orbital complications)
5) Choanal Atresia
6) Foreign Body
7) Neoplastic

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

A mother in the post-natal ward complains that their neonate is unable to feed. She says that they latch on well but then it is just a constant crying then blue.
What is the most likely diagnosis? Why?
How will you confirm your diagnosis and manage the patient?
What syndrome is this associated with?

A

Inability to feed + Cyclical crying + cyanosis. Infants are obligate nose breathers for the first 3 months of life and hence with this presentation, most likely diagnosis = Choanal atresia

Diagnosis and tx:
1) Observe mucus/fogging of fogging-metal spatula
2) Pass NG tube -> secure with nasopharyngeal tube
If still unable then
3) Fiberoptic scope
4) CT head

CHARGE syndrome

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

When passing an NG tube for choanal atresia, you do not seem to be able to pass it far enough. What are your next steps?

A

If still unable then
3) Fiberoptic scope
4) CT head

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

What Oropharyngeal pathologies may the paediatric population present with?

A

Acute Tonsillitis
Glandular fever
Quinsy/Peritonsillar abscess, paratonsilar abscess, retropharyngeal abscess
Obstructive sleep apnea
Congenital: Cleft Palate

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Blood supply to the tonsils are branches of which artery?

Venous drainage is via the lingual and pharyngeal veins. These veins anastomose with what major vein?

A

External Carotid Artery

Internal Jugular vein

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

What is the general workup of acute tonsillitis?

A

It is the same workup regardless if bacterial or viral.
Throat culture + FBC w differentials showing leukocytosis with neutrophils vs lymphocytes
Must rule out EBV via Monospot test
LFTs (raised in infectious mononucleosis)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

How could you differentiate the presentation of bacterial vs viral tonsillitis?

go through the score and include how to analyze results

A

Centor Score FACT

Fever
Absence of Cough
Cervical lymphadenopathy (Tender)
Tonsillar Exudate

0-2 Do not treat with antibiotics
3-4 Treat with antibiotics

Viral: Mild pyrexia, sore throat, adenoid hypertrophy - typically resolves <48hrs
Bacterial: High-grade pyrexia, severe sore throat, adenoid hypertrophy !Lasting >48 hours

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Similar to Sinusitis, A viral infection typically precedes a bacterial superinfection in Acute Tonsillitis.
What are the viral and bacterial pathogens involved?
What is the workup of Acute Tonsillitis

A

What are the viral and bacterial pathogens involved?
Viral: Adenovirus, Parainfluenza virus
Bacterial: S.Pneumoniae, H.Influenzae, and M. Catarrhalis (same as AOM and Sinusitis)

What is the workup of Acute Tonsillitis:
In all cases same cuz of risk of infectious mononucleosis
=> Full hx and examination (check for splenomegaly)
=> Throat culture + FBC (increased UCC mostly neutrophils vs lymphocytes) + procalcitonin + CRP…
=> Monospot test + LFTs

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

How would you treat acute tonsillitis?
What are the complications of acute tonsillitis?

A

How would you treat acute tonsillitis?
Viral: Antipyretics, cough suppressants, !hydration
Bacterial (>48 hours): penicillin or clarithromycin (avoid amoxicillin => infectious mononucleosis) + viral tx + analgesia
Surgical: (if indications met) Tonsillectomy/Adenotonsillectomy

What are the complications of acute tonsillitis?
Suppurative: Quinsy (Peritonsilar abscess), Parapharyngeal abscess, retropharyngeal abscess,
Non-suppurative: Post-strep => rheumatic fever, scarlet fever and post-strep glomerulonephritis + OSA

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

IMP: What are the indications for a tonsillectomy?

A

1) Recurrent disabling sore throat due to acute tonsillitis
a) 7 or more in preceding year
b) 5 or more in preceding 2 years
c) 3 or more in preceding 3 years
2) Suspected neoplasm (e.g. asymmetrical tonsils)
3) 2+ peritonsillar abscess
4) Causing dysphagia/OSA
5) Part of Staged procedure

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Parents always ask if removing the tonsils will have an effect on their child’s immunity. There is no evidence to prove that => it has no effect on that. With that, what are the actual complications of tonsillectomy?

Bonus (no effect on grade): What exam findings are found post-tonsillectomy?

A

Bleeding (Primary <24hrs post secondary >24)
Palatal dysfunction
TMJ dislocation
Teeth damage
Pulmonary: pneumonia, barotrauma…
Voice changes/hoarsness
Typical: Scarring dehydration, hemorrhage, anesthetic risk, DVT…

Post-tonsillectomy:
Eschar (dead tissue that sloughs off)
Foul smelling
palatal oedema

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

A patient bleeds so much post-tonsillectomy. This is a recognised complication and is common enough. Management is how you would manage every bleed exactly. Once stabilised there is just one thing that makes this different what is it?

A

Hydrogen peroxide gargles

17
Q

NOT IN BOOK: How would you differentiate glandular fever from typical acute tonsillitis when seeing a patient with enlarged palatine tonsils?

A

FBC increased WCC with increased leukocytes (vs neutrophils)
Monospot test +vs (vs -ve)
Abnormal LFTs (vs normal)
Rash w/Ampicillin
!!Hepatosplenomegaly on Exam
Grey Slough on grossly enlarged tonsils (picture)

Lymphadenopathy is incorrect as other viruses may cause that (correct if differentiating vs bacterial in general)

18
Q

NOT IN BOOK: What is Glandular fever/infectious mononucleosis?
What is the typical presentation of a patient with Infectious mononucleosis including exam findings)?
What is your workup for this patient?
How would you manage this patient?

A

Glandular fever is a group of symptoms used to describe a patient with symptoms of EBV infection. It is exacerbated by ampicillin (or amoxicillin or coamoxiclav) which leads to a large maculopapular rash throughout the body (little teaching note)

Acute sore throat and fever with acute tonsillitis (pain, pyrexia…)
Cervical lymphadenopathy!
Grossly enlarged tonsils with grey slough!
Maculopapular Rash with ampicillin!
!Hepatosplenomegaly on exam?

Workup:
Throat culture + FBC w differentials showing leukocytosis with neutrophils vs lymphocytes
Must rule out EBV via Monospot test
LFTs (raised in infectious mononucleosis)0

Management: Supportive: hydration, antipyretics, analgesic
Honors: Avoid contact sports for 4-6 weeks to avoid ruptured spleen/liver (from hepatosplenomegaly)

19
Q

What is seen in this image. Describe it
Give 2 differentials

A

It is a large maculopapular throughout the back of the patient.
This may be consistent with infectious mononucleosis or Rubella infection

20
Q

What is trismus?

A

Inability to fully open the mouth

21
Q

Quinsy/peritonsillar abscess is a clinical diagnosis. What are the cardinal features to make this diagnosis?

A

Deviated uvula
Peritonsilar swelling
Trismus
Deviated Uvula

22
Q

You are asked to examine this patient. Describe what you see and your most likely diagnosis.
What is the typical presentation and clinical features?
how would you workup this patient?
How would you manage this patient?

A

This is oral cavity of a patient presenting with acute tonsillitis. The tongue is depressed with a tongue depressor and there is a clear shift to of the uvula to the left (patient’s) as there is clear enlargement of the right tonsil. This finding is consistent with a peritonsillar abscess or Quinsy

Presentation: Severe !!unilateral sore throat, dysphagia, and odynophagia
Clinical features: Deviated uvula
Peritonsilar swelling
Trismus
Deviated Uvula
Hot potato voice

Workup:
=> Full hx and examination (check for splenomegaly)
=> Throat culture + FBC (increased UCC mostly neutrophils vs lymphocytes) + CRP….
=> Monospot test + LFTs (dont forget to rule out EBV)

Tx: (surgical emergency => ABCDE)
Medical: IV Antibiotics (Penicillin)
Surgical: Draingage +/- Tonsillectomy (it is an indication by itself even)

23
Q

When draining a peritonsillar abscess (Quinsy), where is the incision made?

A

1/2 way between the uvula and the site of the first wisdom tooth

24
Q

Which oropharyngeal abscess is most likely to have neck stiffness?

A

Retropharyngeal

25
Q

Unlike peritonsillar abscesses, retropharyngeal and parapharyngeal abscesses are not clinical diagnoses.

Which is more dangerous? Why?

What are the common clinical features of these abscesses?

What is the best imaging modality to confirm the diagnosis?

How would you treat them?

A

Retropharyngeal is more dangerous because it can cause a stridor especially in children => emergency

Clinical features: Sore throat, odynophagia, lateral neck mass
Specific for retro: Stridor and neck stiffness

Imaging -> CT

Urgent drainage under general anaesthetic

26
Q

For retropharyngeal and parapharyngeal abscesses, Ct is the best scan. For retropharyngeal specifically, a lateral X-ray can be performed as well. What will it show?

In the management of these abscesses why is drainage performed under general anaesthesia?

A

Lateral neck X-ray will show soft tissue widening at the level of C2 to C6

Drainage performed under general anaesthesia
1) presentation mostly in kids
2) Prevent spread to mediastinum causing mediastinitis

27
Q

How would you manage a patient with OSA?

A

Overnight oximetry or cardiopulmonary/full polysomnography
tx: Conservative: Weight loss, dietitian referral, allergens at home (dust, pets, dolls, carpet)
Surgical: Adenotonsillectomy

28
Q

What is the most common congenital (1) and acquired causes (2) of stridor in the paediatric population

On first glance of a child, what clinical feature would lead you to directly suspect airway obstruction/stridor?

A

Congenital: Most common cause of chronic stridor - Laryngomalacia (others include congenital vocal cord paralysis, subglottic stenosis, laryngeal web)

Acquired: Croup (laryngotracheitis), Epiglottitis, Foreign body

Tripod positioning, leaning forward

29
Q

What is laryngomalacia?
What is it the most common cause of?
What type of stridor is exhibited?
How would you manage a patient with laryngomalacia?

A

Congenital softening of tissues in larynx => floppy => obstruction => chronic stridor (most common cause) => cyanotic episodes in infants

Supraglottic tissues are softening => Inspiratory stridor

Laryngomalacia is typically self-resolving within 2 yrs
If recurrent => Supraglottoplasty

30
Q

What is the most common pathogen causing Epiglottitis?
What is the typical presentation of a patient with epiglottitis?
When examining or treating a patient with epiglottitis what is important to keep in mind?
How would you manage your patient?

A

H. Influenzae (now rare cuz of vaccine)

Acutely unwell, febrile child in tripod position, leaning forwards => Drooling, wont cough or swallow (dysphagia), agitated.

When examining or treating a patient with epiglottitis (esp children), do not irritate with tongue depressor or IV lines until sedated or secured airway.

It is an emergency => inform ENT and anesthetics to secure airway
+ IV antibiotics (cefotaxime, ceftriaxone)