Otology Flashcards

1
Q

Describe what you see in the picture
What is the most likely diagnosis. Give a brief description of what has occurred

How is this treated?

A

This is a clinical picture of the left ear showing subcutaneous collection affecting the left pinna especially the area of the antihelix and crura (sup and inf) of the antihelix,

This clinical picture is consistent to an auricular hematoma usually secondary to trauma. Collection of blood formed between cartilage and pericondrium (which supplies blood to cartilage due to shearing of these BV due to trauma) leading to ischemia and necrosis.
Diagnosis: Cauliflower ear typically seen in rugby players

Tx: Urgent incision and drainage before suturing bolster dressing (splinting in book) on pinna to prevent it from re-opening

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2
Q

Auricular haematoma formation is due to the shearing of perichondrial blood vessels supplying the cartilage. What is the main complication of this if it is not treated promptly

A

Cauliflower ear from avascular necrosis

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3
Q

On otoscopy, you find this clinical picture. The patient has been complaining of pain. On examination this finding is pulsatile. What is the finding in this image?

A

The finding is a pulsatile (given) red mass behind a normal tympanic membrane

This is actually Globus Tympanicum which is a vascular tumour in the middle ear

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4
Q

You see this patient in the OPD as you clock in to start your shift. What diagnoses come to your head (give 2)

A

Mastoiditis (from AOM…)
Bat ears (congenital)

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5
Q

In the field of ENT what red flags are you always looking to rule out?

A

Unilateral symptoms
Presence of eye symptoms
Weight loss
Dysphagia
Chest infection (aspiration)

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6
Q

What is vertigo?

A

Sensation that the environment around you is spinning

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7
Q

Define Odynophagia

A

Pain from swallowing referred to the ears via the glossopharyngeal nerve (CNIX)

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8
Q

Define Globus sensation

A

sensation of a lump in the throat

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9
Q

Define Stridor

A

High pitched wheeze caused by airway obstruction

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10
Q

Define Torticollis
Whatis the etiology of torticollis in children and adults?

A

Children: Positional torticollis due to SCM (shorter)
Adult: Spasm or viral infection causing irritation to cervical ligaments or vigorous movement

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11
Q

During inspection in an ENT exam, what are you looking for?
Other than looking at the patient what other actions must be performed? Go crazy with detail for bonus points

A

Make a show by looking around the patient and raising their hair for the back of their neck esp for scars. You would be looking for scars, asymmetry, swellings, masses erythema, rashes, tophi…

Ask patient to smile for symmetry and facial nerve palsy
Ask patient to swallow for thyroid gland (give glass of water if needed)
Ask patient to stick tongue out for thyroglossal duct cyst
ET function (Valsalva test)
Inspect oral cavity via tongue depressor and otoscope (light) around the tongue looking at Dentition!! and symmetry of structures
Inspect Nasal cavity for polyps, masses, swellings, DNS…
Check eyes for signs of cysts, abscess, eye movements.

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12
Q

Describe the image as you would on your finals

A

I see two clinical pictures each demonstrating a patients ear, one on each side. The ear looks healthy with no evidence of any obvious asymmetry, swellings, or disease. There appears to be 3 white/skin colored lesions around .5cm diameter along the pinna. This seems to be consistent with tophi which appear peripherally and on extensor surfaces usually including the ear. Colchicine may be used to treat this acutely and allopurinol preventatively to lower uric acid levels in the body

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13
Q

This picture is given to you on an exam. Have at it

How is this managed?

A

This is a clinical picture of a male paediatrics patient. They appear to have a nice symmetrical face with no obvious signs of weakness. No masses, scars, or swellings are evident. They appear to have prominent ears due to an absent antihelix.

Prominant ears is surgically managed via Pinnaplasty or Otoplasty (cosmetic procedures) !prior to the child commencing school! to prevent bullying and psychological sequelae

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14
Q

This picture is given to you on an exam. The clinical context is a patient in the pediatric outpatient department for a follow up.
What is this finding associated with (not disease or syndrome)

A

This is a clinical picture of a paediatric patient’s left ear. The external ear itself looks healthy and does not have any evident swelling, erythema, lesions, or masses. The main finding is what appears to be a sinus present pre-auricularly. Pre-auricular sinus. They are associated with deafness and may become easily infected.

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15
Q

Give some congenital ear deformities you may spot on inspection
What would your workup be once you spot any of these deformities including curative treatment

A

1) Prominent ears (Bat ears): Absent anti-helix! others include deep concha anc protruding lobule
2) Anotia/Microtia
3) Pre-auricular sinus

Workup: Check hearing by obtaining a Pure Tone Audiogram and tympanogram. If hearing not affected, this becomes cosmetic. Pinnaplasty/otoplasty would be the curative surgery.

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16
Q

This picture is given to you on an exam. Have at it

A

This is a clinical picture of a patient’s right eat. The external ear itself looks healthy apart from a lesion on the pinna. The most common neoplastic lesion is an SCC due to the exposure to sunlight. (may also be a BCC)

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17
Q

When holding an otoscope, it is typically held like a pencil. Why does the doctor typically rest their pinky on the patient’s face?
The otoscope is typically held by the hand that is on the same side as the ear that is being examined (right hand right ear). What does the other hand do?
Once going in make sure you angle it upwards to see the attic or the pars flaccida. What is the pars flaccida?
You see a light, what is that light and where is it located?

A

For stability especially in the pediatric population who may jerk in any direction suddenly from potential pain

The other hand holds the pinna up and out and back for adults, down and out and back in pediatrics

The pars flaccida is the thinnest portion of the tympanic membrane and hence the weakest => commonest point of perforation and retraction. It is composed of 2 layers as it is missing the third, middle fibrous layer that the pars tensa has

The light is the light reflex, typically located on the anterior inferior part (=> towards front of patient) of the TM. It is missing in disease and grommets (inserted here)

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18
Q

While using an otoscope, this image is what you see. Explain what you see

A

This is a picture of the external auditory meatus obtained via otoscopy. There appears to be a non-erythematous swelling of the walls of the EAM. This is consistent with Exostosis or Surfer’s ear secondary to chronic exposure to cold weather/water. It is a bony overgrowth that may lead to recurrent infections and wax compaction leading to conductive hearing loss

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19
Q

While using an otoscope, this image is what you see. Explain what you see
Clinical context: 2 year old child presents with severe otalgia, 2 days after going to the community pool for a swim with the family. You conduct otoscopy but cannot move further due to the pain.
How are you expecting the Tympanic membrane to look if you were able to go further?
What is the most likely diagnosis?

A

This is a picture of the external auditory meatus obtained via otoscopy. There appears to be evidence of extensive swelling of the erythematous wall of the EAM leading to stenosis. There also appears to be a yellow/white cottage cheese discharge. Pressing on the walls of the EAM may lead to more production and can further confirm the likely diagnosis of acute otitis externa.

This is basically a skin infection, the tympanic membrane should be normal

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20
Q

How would you test ET tube function?

(Honours, Contraindications)

A

Valsalva test (pinch nose, deep breath, close mouth, exhale through nose). This will test if the ET is limited by effusions/discharge or perforation.
Normally the tympanic membrane would just move outwards. If there is a
Hissing sounds -> Perforation
Crackling sound -> Discharge/effusions

Contraindications: Atrophic sclerosis with risk of rupture or presence of infection in nasopharynx (may make it go to middle ear)

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21
Q

The examiner gives you this on the exam. What is the main finding of this image?

A

Tympanosclerosis (say full thing like its an exam)

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22
Q

While using an otoscope, this image is what you see. Explain what you see

A

This is an image of the tympanic membrane of the left ear obtained via otoscope. This tympanic membrane appears to be retracted highlighted mainly at the Pars Flaccida. There is a brown/yellow tinge giving evidence of effusions in the middle ear consistent with glue ear or otitis media with effusions. Other differentials would be chronic supporative otitis media with cholesteatoma (complication of glue ear)

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23
Q

While using an otoscope, this image is what you see. Explain what you see

A

This is an image of the tympanic membrane obtained via otoscopy. There is a red, bulging tympanic membrane (should have reduced movement and a history of conductive hearing loss and severe otalgia until perforation - obs you dont have to be saying that here). There is an absent light reflex in the anterior inferior quadrant and I cannot visualize the handle of the malleus. I suspect the bulging to be as a result of an infective process such as otitis media => filled with pus.

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24
Q

While using an otoscope, this image is what you see. Explain what you see

A

This is an image of the tympanic membrane of the left ear obtained via otoscopy. The handle of the malleus is evident along with the pars flaccida and pars tensa. There is a small perforation in the location of the light reflex on the anterior inferior margin. It seems to be dry with no evidence of discharge as it would in AOM. Here it may be a more chronic otitis media or trauma or cholesteatoma.

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25
Q

You have conducted the Rinne and Weber test and now want to conduct the Whisper test.
Explain the Whisper test and its escalations

A

Explain the test to the patient. Start by testing the hearing via normal conversation infront of them and ask them to repeat
Stand arms length (60cm) behind the patient and rub the tragus of the non-tested ear to mask noise. Whisper bisyllabic # (twenty). (12db). If they cannot hear, escalate.
Conversation voice (50db)
Loud (75db)
Move 15 cm closer (=>45cm) and whisper (35db)
Conversational (55db)

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26
Q

What are the 3 ossicles of the ear? Which portion of the ear are they located

A

Malleus, incus, stapes
Middle ear

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27
Q

The stapes connects indirectly to the inner ear via a structure. What is this structure and what is the other connection between the middle and inner ear?

A

Oval window closed by stapes footplate
Round window closed by membrane

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28
Q

What is the main function of the Eustachian Tube?

A

Equalize pressure in the middle ear

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29
Q

Describe the structures of the inner ear.

A

The inner ear is divided into anterior and posterior parts, the anterior being the cochlear and posterior being the vestibular
The cochlear (hearing) portion is composed of the Scala tympani, Scala vestibuli, and cochlear duct
The vestibular (balance) portion is composed of the utricle, saccule, and 3 semicircular canals

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30
Q

State differentials of the outer ear (as many as you can)

A

Congenital: Pinna disorders, preauricular sinus
Trauma: e.g. Battle’s sign (post-auricular ecchymosis from basilar skull fracture), temporal bone fracture, Cauliflower ear
Infectious: AOE, Perichondritis, pinna cellulitis, mastoiditis, erysipelas,
Neoplastic: SCC
Wax impaction

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31
Q

CSF leak is a major complication of a temporal bone fracture due to its proximity to the Tegmen in the middle ear. What are the 2 ways a CSF leak may manifest in a patient presenting to the ED with temporal bone trauma?

A

Otorrhoea => CSF in middle ear through perforated tympanic membrane
Post-nasal drip of CSF from middle ear through ET into post-nasal space (classic bending over)

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32
Q

What is seen in this image? Explain
What is your workup for this patient including
1) What you are looking out for in history and physical exam
2) imaging and its possible findings
What are the main ENT-related complications of this?

A

This is a clinical picture of a patient’s left ear which appears to have post-auricular ecchymosis consistent with Battle’s sign, basilar skull fracture, temporal bone fracture.

This is considered a head injury and should be worked up as such ABCDE especially if major
1) if you get like 90% its a 5/5 definitely
Main: GCS!!, nausea, vomiting, headache, (increased ICP)
Base of skull fracture => Check for orbital ecchymosis or raccoon eyes
Tympanic perforation => otoscopy
CSF leak (Otorrhoea vs post-nasal drip)
Facial Nerve function
Hearing (tuning fork, rinne, whisper, Pure Tone Audiogram, tympanogram)
Tinnitis
Vertigo
Otalgia
Cuts/lacerations
Auricular hematoma

2) Non-contrast CT brain (contrast later if needed). It will show left temporal bone fracture and air bubbles around fracture medially +/- orbital emphysema. May also show hematoma.

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33
Q

This is handed to you in an exam. Explain what you see

A

DO the full thing: Must include
Perichondritis. Inflammation of the cartilage. Redness and oedema on pinna but not lobule as lobule has no cartilage
Caused by Pseudomonas Aeruginosa, Staph, strep

34
Q

This is handed to you in an exam. Explain what you see

A

Must include:
Pinna cellulitis affecting all of pinna and lobule with a more diffuse border (as opposed to erysipelas)
Caused by Pseudomonas Aeruginosa, Staph, strep

35
Q

This is handed to you in an exam. Explain what you see
What else would you check for after seeing this finding

A

Must include:
Erysipelas: Intense redness on entire ear including pinna, lobule, concha and preauricular space. Typically well demarcated and may appear on the face and nose => check
Typically caused by Group A Strep (Pyogenes).
Very tender
Also Check for lymphadenopathy

36
Q

This is handed to you in an exam. Explain what you see

A

Must include:
Full introduction of image
Subperiosteal abscess typically resulting from progression of orbital cellulitis which can occur from ethmoidal sinusitis especially in infants suffering from recent URTI (wow if you get all that).

37
Q

This is handed to you in an exam. Explain what you see
What is it associated with?
What is the treatment for this patient?

A

Must include
Post-auricular swelling => Mastoiditis. Associated with AOM, subperiosteal abscesses (Ethmoidal sinusitis/sinusitis), Temporal lobe abscess, brain abscesses.
Caused by M. Catarrhalis, S.pneumonia, H. Influenzae (same as AOM and acute sinusitis)
IV antibiotics +/- Mastoidectomy + Grommet insertion

38
Q

How would you describe a normal tympanic membrane

A

!in tact, mobile
All features present (Handle of the malleus, light reflex, pars flaccida, pars tensa)

39
Q

How would you manage stenosis (including wax impaction) of the EAM?

What if there is an infective cause?

A

Wick (hard sponge we insert to open)
Canal Toilet (Microsuction to get rid of debris - you saw prof o neil do)

If infective cause -> Wick laced with topical antibiotic

40
Q

How would you deal with an insect in the ear?

A

Kill then remove

41
Q

Acute Otitis Externa: (define it)
Main causative organisms:
RFs:
Main symptoms and examination findings:
Full Treatment:
Complications:

A

Otitis Externa: Infection of skin of EAC w/typically unaffected middle and inner ear. This is typically bacterial as opposed to most other things in the middle and inner ear, and nose.
main causative organisms: Pseudomonas Aeruginosa, Staph aureus, Fungi (like cellulitis)

RFs: Water exposure, scratching, skin conditions (atopic dermatitis, psoriasis, seborrheic dermatitis), Immunocompromised, Post-URTI
foreign body

Main symptoms and examination findings:
Sx: Otalgia, Otorrhoea (cottage cheese white/yellow discharge), Itchiness
Exam: Conductive hearing loss,
red oedematous outer ear
Otoscopy:
Red oedematous skin of EAM, Debris (especially when pressing on walls),
!Normal tympanic membrane (in tact, mobile).

Full Treatment:
Analgesia
Topical steroid drops
Topical Antibiotics
If Stenosed: Wick or Canal Toilet laced with antibiotics

Complications:
Ear Canal Stenosis (treated above)
Perichondritis
!!!Necrotizing Otitis Externa

42
Q

In ENT if you ever see pus what do you do?

A

Always send it to the lab

43
Q

What is another word for Necrotizing Otitis Externa? How does it occur?
What is the main RF for this to occur?
What is the main causative organism?
What examination findings would indicate this?

A

What is another word for Necrotizing Otitis Externa?
Malignant otitis externa

How does it occur?
It is a serious complication of Acute Otitis externa where the inflammation becomes necrotic => Granulation tissue

What is the main RF for this to occur?
Immunocompromised individuals particularly diabetics and HIV

What is the main causative organism?
Pseudomonas Aeruginosa

What examination findings would indicate this?
Severe Otalgia, Granulation tissue in EAM on otoscopy, Cranial nerve palsy on neurological examination

44
Q

What is wax in the ear secreted by?
How would you treat wax impaction?

A

Secreted by sebaceous and Ceruminous glands of EAC
Wax impaction leads to conductive hearing loss and is treated by syringing and microsuction.

45
Q

A patient has conductive hearing loss and extensive wax impaction is seen on otoscopy. You opt for syringing and/or microsuction. what are the contraindications to this?

A

CI: Recent/current otitis/infection, hx of surgery in external or middle ear, tympanic perforations

46
Q

State differentials of the middle ear (as many as you can)
What is the Common causative mechanism of all of these?
What are the common RFs of this mechanism and by definition all of the stated differentials?

A

1) Tympanic Membrane retraction
2) Acute Otitis Media
3) Glue Ear - Otitis Media with Effusion
4) Chronic Suppurative Otitis Media without cholesteatoma
5) Chronic Suppurative Otitis Media with Cholesteatoma

Majority of diseases in the middle ear stem from Eustachian Tube Dysfunction

RFs:
1) Craniofacial Abnormalities (cleft palate) => chromosomal abnormalities
2) Immunodeficiencies
3) Adenoid Hypertrophy
4) Nasal Allergy (e.g. Allergic rhinitis)
5) Parental smoking
6) Bottle feeding
7) Daycare attendance
8) Reduced mucociliary clearance e.g. CF, Primary ciliary dyskinesia, Kartagener Syndrome
9) Any infection including sinusitis, URTI…

47
Q

NOT IN BOOK. Just to read
What is the first part of the TM that gets retracted?
What are the stages of TM retraction?
What is TM retraction a sign of?
TM retraction in a child with mild hearing loss. What is the most likely diagnosis?

A

Pars Flaccida (not part of classification) because it is missing middle fibrous layer

Sade classification
I - Slight retraction of Pars Tensa
II - Retraction onto Incus
III - Non-adherent retraction onto Promontory
IV - Adherent to Promontory
V - Perforation

TM retraction is a sign of ET dysfunction
Glue ear: Chronic Otitis Media with effusions

48
Q

How do infections in the middle ear cause intracranial complications

A

1) Via Mastoid - mastoiditis (similar to how sinusitis especially frontal sinusitis causes intracranial abscesses)
2) Via Tegmen (roof) => CSF leak (similar to how sinusitis especially ethmoidal sinusitis goes through the bone -or vessels- to cause orbital cellulitis and abscesses

49
Q

What is the medical and surgical management of Acute otitis media

A

Management:
Medical: Analgesia, Antipyretic, nasal decongestants, Oral antibiotics (if severe, IV and admit)
Surgical:
If severe - Myringotomy

50
Q

What is a Myringotomy

A

Myringotomy is a surgical procedure that involves making a small incision in the eardrum (tympanic membrane) to drain fluid or relieve pressure

51
Q

When is a Grommet indicated in otitis media?

A

If recurrent

52
Q

What is petrositis?
What is it most commonly caused by?

A

Infection and inflammation of the petrous part of the temporal bone

Typically caused by Acute otitis media or its main complication mastoiditis

53
Q

Acute Otitis Media - Pathophysiology:
Main causative organisms:
RFs - As that of ET dysfunction
Main symptoms and examination findings:
How is this managed?

A

Viral infection typically causes inflammation (e.g. sinuses) causing ET dysfunction leading to Otitis Media => may occur after URTI. A bacterial superinfection then typically occurs.

Main causative organisms: Viral (85%), Bacterial (15%) S.pneumonia, H.influenza, M. Catarrhalis (same as sinusitis)

Main symptoms and examination findings:
Sx: Otalgia, Pyrexia, nausea
Otoscopy: Bulging red, immobile TM. Erythematous, missing light reflex…, !may be perforated with discharge.
Exam: Conductive hearing loss

Management:
Medical: Analgesia, Antipyretic, nasal decongestants, Oral antibiotics (if severe, IV and admit)
Surgical:
Myringotomy
If severe - Myringotomy
If Recurrent otitis media -> Grommet placement

54
Q

List the complications of Acute Otitis Media

A

Complications: Categorize!
Intracranial: Via mastoiditis - meningitis, extra/subdural abscess, brain abscess, sigmoid sinus thrombophlebitis, otitis hydrocephalus

Extracranial: Tympanosclerosis, TM perforation => otorrhoea, Hearing Loss, !!Mastoiditis, !!Subperiosteal abscess,
!!Facial Nerve paralysis,
Labyrinthitis (nausea, vomiting, hearing loss, vertigo, tinnitus), Petrositis, Chronic Suppurative Otitis Media

55
Q

What is the most common cause of conductive hearing loss in children?

What does this mean in terms of their development?

A

Glue Ear - (Chronic) Otitis Media with Effusion

!Common cause of speech and language delay

56
Q

Glue Ear - (Chronic) Otitis Media with Effusion
Definition:
Peak Age:
Clinical Features including otoscopy:

What investigations must be performed?

Treatment?

Complications:

What are the RF? Oh right the same as otitis media and all otology which is ET dysfunction

A

Glue Ear - (Chronic) Otitis Media with Effusion
Definition: Chronic accumulation of non-purulent fluid in middle ear without sx of infection

Peak Age: 2-5yrs

Clinical Features including otoscopy:
Commonest cause of conductive hearing loss in children (30-40dBs) - Speaking louder
!Common cause of speech and language delay
Tinnitus
Balance issues
Otoscopy: Dull immobile, retracted TM with brown/yellow tinge on TM from fluid in middle ear

Investigations:
Pure tone audiometry
Tympanometry

Treatment:
Watchful waiting for at least 3 months -> Grommet insertion if the effusion is still persistent
Consider Adenoidectomy if recurrent

Complications:
1) Tympanosclerosis
2) Atelectasis
3) Retraction Pockets
4) Erosion of ossicles
5) Cholesteatoma!!

57
Q

If pure tone audiometry cannot be performed cuz the children is not cooperating or too young, what will you do instead?

A

Otoacoustic emissions (testing outer hair cell response as part of cochlear function)
ABR - Auditory brainstem response (brain’s response to sound via electrodes)

58
Q

What is Autoinflation?

A

It is a treatment used for Glue ear or otitis media with effusion. It involves using a balloon in nose, close other nostril and pump air in => air goes through eustachian tube and reverts the retration.

59
Q

What is a grommet and its role?
What are 2 other terms for grommet?
Where is it inserted?
What are it’s indications?
What are it’s complications?

A

A grommet is a tube-like device inserted at the point of the light reflex which is in the anterior inferior quadrant and lasts approximately 9 months before being extruded on its own. It’s role is to equalize the pressure and take the role of the Eustachian tube.

Other terms: Tympanostomy tube, ventilation tubes

Indications: AOM, Glue ear

Complications: Discharge/otorrhoea, Tympanosclerosis,
Residual perforation
Tube blockage
Early extrusion

Important note: there is a new one which is a T-shaped tube so that it stays there for longer

60
Q

You insert a grommet or T-shaped tube and notice more bleeding than usual. what is the reason

A

High Dehiscent jugular bulb

61
Q

What are the complications of any ear surgery?

A

Vertigo, tinnitus, hearing loss, failure, Taste disturbance (chorda tympani), facial nerve injury, cholesteatoma, !!!Tegmen (=>CSF leak, meningitis, brain abscess, reduced GCS) !!!otalgia, infection/recurrent infection, death (GA)

62
Q

What is the age cutoff for Pure Tone Audiogram testing?
How do you screen for hearing loss in an infant? How does it work? What is the main drawback?
If someone is positive on the screening test, what is the next step (for an infant still)? How does it work? How would you interpret the results.

A

Infant=> cannot give input => Cannot produce audiogram. Children <4 cannot reliably perform the test
Screening: Otoacoustic Emissions test. It tests for an echo via a small microphone-like device. Inner hair cells receive sound waves from an sound played by examiner. That sends signals to the brain via Cochlear nerve. The brainstem will then reflex by contracting the external hair cells leading to formation of the echo picked up by device.
Drawback? Does not assess function of the vestibulocochlear nerve (CNVIII) . => ABR

After this, Auditory Brainstem reflex which is the reflex to focus on a sound, uses the same concept as above but here, surface electrodes are placed on the scalp to detect electrical signals originating from the brainstem and along CNVIII. Results are obtained in a series of waves. The 5th wave is what is important as its presence signifies that 1,2,3,4 have already passed without issues.

63
Q

A patient born with a cleft palate and a history of ear infections presents to the OPD with hearing loss with discharge but no pain
What is the most likely diagnosis?

A

Chronic Suppurative Otitis media (can be with but needs more indications). A patient with cleft palate (=> ET dysfunction => recurrent otitis media) and history of otitis media.

64
Q

Chronic Suppurative Otitis Media (without Cholesteatoma)
Clinical Features including otoscopy:
2 most common Pathogens:
Tx not in book

A

Clinical Features including otoscopy:
Recurrent Discharging ear >3/12 with perforation
Otorrhea, Conductive hearing loss, (NO OTALGIA),
Pathogens: P.auroginosa, S.aureus

65
Q

NOT IN BOOK: In a tympanoplasty, what is used as a graft?
What must the patient be informed about post-op to avoid complications?
What are the complications of tympanoplasty?

A

Temporalis fascia or Tragal cartilage used as graft as scaffold for tympanic membrane regrowth

Advice: Waterproof ear until dressing comes out and avoid flying for 6 weeks

Complications: Chorda Tympani injury (taste disturbance), Tinnitus, vertigo, cholesteatoma!, Hearing loss, failure of procedure, death (GA), pain, infection…

66
Q

What is the stereotypical otoscope finding for congenital cholesteatoma?

A

Presents as a white anterior attic mass behind an in tact tympanic membrane in the first year of life

67
Q

CSOM with cholesteatoma:
Define Cholesteatoma:
Typical presentation:
Typical otoscope finding:
Complications:

Main diagnostic investigation

Treatment: Medical + surgical

A

Define Cholesteatoma: Presence of benign, !keratinizing! squamous cell epithelium in the middle ear (which may be infected)

Typical presentation: Progressive, gradual onset sensorineural hearing loss,
vertigo,
Painless foul-smelling otorrhea
Facial Nerve Palsy

Otoscope: Presents as a white anterior attic mass behind an in tact tympanic membrane in the first year of life (if congenital)

Complications: Mastoiditis -> subperiosteal abscess -> Brain abscess and/or meningitis

Main investigation: HRCT Temporal bone

Treatment:
Medical: Antibiotic drops (non-ototoxic)
Surgical: Mastoidectomy +/- urgent mastoid exploration if inracranial complications (meningitis, intracranial abscess)

68
Q

Cholesteatoma acts as a local tumour, leading to the erosion of ossicles and inner ear leading to tinnitius and vertigo. It may become infected and also spread into the mastoid air cells leading to mastoiditis, sub periosteal, and brain abscesses. It may be acquired or congenital. state the hypothesis for each

A

Acquired: Retraction pockets from ET dysfunction (e.g. glue ear) leading to chronic infection within epithelial tumour
Congenital: Embryonic squamous epithelium trapped in middle ear

69
Q

What type of hearing loss is most associated with CSOM with cholesteatoma?

A

Sensorineural hearing loss (may begin as conductive but it erodes into the inner ear causing sensorineural)

70
Q

What type of hearing loss is most associated with CSOM without cholesteatoma?

A

Conductive hearing loss as with everything not affecting the inner ear

71
Q

A patient with acute otitis externa presents to the ED. During otoscopy you notice a discharge. Describe it

A

White, cottage-cheese like

72
Q

A patient with acute otitis media presents with discharge. Describe it.
Is the patient likely to be in pain? why?

A

Mucoid discharge
The TM has perforated => discharge. The accumulation of pus behind the ear is what would cause the otalgia. This would be relieving

73
Q

While examining a patient in the ED, you notice a blood-stained discharge from the AEM. What is your ddx

A

CSF leak => trauma
Neoplasm
Acute necrotizing otitis externa

74
Q

While examining a patient’s ear in OPD, you notice foul-smelling discharge. What is your most likely diagnosis?

A

OPD => not acute => unlikely to be acute otitis media
foul-smelling => cholesteatoma (CSOM)

75
Q

While examining a patient in OPD, you notice a pale, yellow effusion. What is your ddx

A

CSOM w/out cholesteatoma, w/cholesteatoma
Glue ear

76
Q

What is the main function of the middle ear?

A

Amplify mechanical longitudinal waves to move endolymph in inner ear.

77
Q

What is the most common method for infections to spread in ENT to the orbit?

A

Lamina Papyracea

78
Q

What nerve is responsible for referred pain to the ear?

A

CN IX - Glossopharyngeal nerve

79
Q

What ear pathology is most associated with the need for speech and language therapy?

A

Glue ear

80
Q

A mother complaining of her child speaking louder indicates what?

A

Hearing loss