renal transplant Flashcards

1
Q

what is a chronic kidney disease

A

long term condition where kidneys can no longer filter waste and excess fluids

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2
Q

stages

A
  1. normal function -signs of kindey damage
    2.midly decreased kidney function (60-89
    3.moderate decreased kidney function (30-59
  2. severe damage (15-29
    5.kidney failure ( less than 15
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3
Q

symtoms of kidney failure

A

fatigue, itch, urine output, fluid overload, vomiting, anorexia, nausea

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4
Q

causes

A

-diabetes
-HBP
-Glomerulonephritis
-polycystic kidney disease
-heart disease/obesity

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5
Q

how to fix this

A

prevent not cure
-meds-control hbp, blood sugar, cholestrol
-dietry changes- reduce salt, protein, and potassium intake
-dialysis
-transplant- not a lot to be done
control syptoms
negative QoL

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6
Q

what is a acute kidney injury

A

sudden decline in either days or hours
and is usually reversible

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7
Q

what causes AKIs

A

prerenal-reduced blood flow
intrintic-direct damage to kindeys-infection
postrenal - obstruction of urine flow

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8
Q

symptoms

A

-decreased urine output
-swelling
-fever
-back pain
-fatigue

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9
Q

what can be done?

A
  1. address underlying issue e.g imrpoving blood flow
  2. stopping nephrotoric medications
  3. draining obsturction
  4. temprorary dialysis \
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10
Q

positives of transplants

A

-patient returns to normal QoL
-normla life expecatany

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11
Q

negatives of transplant

A

-potential surgical complications
-fialure to work
-psot op thrombosis
-wound deniscience
-risk of infection-immunosuppression
-weakining of the immune system- bacterial, UTI, pneumonia

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12
Q

types of kidney transplants

A

-DCD
-DBD
-paired exchange
-alteristic

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13
Q

incomatible transplants

A

HLA- Human leukocyte antigen
-proteins on cell surface that identify self cells
-markers do not match
-increase chance of rejection
ABO- blood group incompatibility
ABO antibodies attack donor kidney’ blood vessles
-increaase chsnce of rejection

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14
Q

sources

A

Nationial kidney foundation
national institute of diabetes, digestive and kidney diseases
bloom and reese, 2007

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15
Q

expansion on causes:diabetes

A

diabetic nephropathy:
-high blood sugar damages blood vessels
-longterm damages nephrons
-causes kidneys to work harder=fibrosis

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16
Q

hypertension

A

damages all blood vessles and kidney ones
reduces ability to filter waste products

17
Q

glomerulonephritis

A

inflammation of glomeruli (filters)
inflammation can lead to infection and autoimmunity

18
Q

polycystic kidney disease

A

genetic disroder were cyts develop innthe kidneys
damamges surrounding kindey tissues

19
Q

heart disease/obesity

A

related to hypertension and diabetes
poor circulation= kidney ischeamia