Rapid EKG I Flashcards
SA node
sinus node
-pacemaking automaticity
upper posterior wall of right atrium
contraction
wave of Na ions
AV node
between tricuspid and mitral valve
-only way electricity from atrium to ventricles
-conduction is slowed - to allow ventricular filling
slow conduction through AV node
calcium ions
purkinje fibers
His bundle
R and L bundle branches
-conduction Na ions
terminal purkinje fibers
spread out beneath endocardium
-go toward epicardium
Q wave
first downward deflection
R wave
upward deflection
S wave
downward deflection following R wave
QS wave
no R wave (no upward)
ST segment
end of QRS to beginning of T
ventricular repolarization
occurs ST segment and S wave
QT interval
beginning of QRS to end of T
-indicator of repolarization
myocyte contraction
Ca ion inflow
repolarization of myocytes
K outflow
cell-to-cell fast conduction
Na ion movement
AV node slow conduction
Ca ions
small square
0.04s
large square
0.2s
lead I
0 degrees
lead II
+60 degrees
lead III
+ 120 degrees
lead AVF
+90 degrees
lead AVL
-30 degrees
lead AVR
-150 degrees
inferior limb leads
II, III, AVF
lateral limb leads
I, AVL
center of horizontal plane
chest leads - from AV node
right chest leads
V1 and V2
left chest leads
V5 and V6
IV septum leads
V3 and V4
symapthetics
NE to beta1 adrenergic
- increased SA node pacing
- increased conduction rate
- increased inotrophy
- increased irritability of foci**
parasympathetics
ACh to cholinergic receptors
- decreased SA node pacing
- decreased conduction rate
- decreased inotropy
- decreased irritability of atrial and junctional foci**
decreased irritability of atrial and junctional foci
PS innervation
constriction of arteries
sympathetic to alpha1
syncope
severe vagus response
- dilated arteries and bradycardia
- loss of consciousness
vaso-vagal syncope
vagal maneuvers
gag or carotid massage
- increase PS
- inhibit irritable foci
- inhibit AV node (longer refractory)
standing
sympathetic response
- baroreceptors sense low BP
- vasoconstriction
- and SA node stimulation
orthostatic hypotension
failure of compensatory sympathetic response when standing
neuro-cardiogenic syncope
in elderly
- prolonged standing
- stimulates sinus pacing
- vasoconstriction falls
- paradoxical PS reflex
- slows SA node and causes vasodilation