Q2: Pathology (Review of Clark Lectures) Flashcards
Primary gait deviations
caused by a direct factor
Secondary gait deviations
caused in order to compensate for a primary deviation
Gait deviations from Steppage Gait
Primary - Foot Drop (Paralytic Equinas)
Secondary - Excessive knee and hip flexion
Gait deviations from Plantarflexor Paralysis
Primary - Uncontrolled tibial progression
Secondary - shortened contralateral step
CP - Etiology (congenital)
85-90% are due to congenital problems (mostly idiopathic)
placenta detachment
uterine rupture
disrupted oxygen supply
CP - Etiology (acquired)
infection: meningitis or encephalitis
TBI: MVA or abuse
reduced blood flow to brain
CP - (T/F) Cerebral Palsy is progressive and gets worse with time
False
CP - Four Types
spastic
ataxic
athetoid
mixed
CP - Spastic
most common
70-80% of diagnosed cases
hypertonia
spasticity
joint stiffness
damage to the cerebral cortex
CP - Ataxic
occurs in 10% of diagnosed cases
difficulty with balance
poor muscle coordination
cognitive functioning impairment
hypotonia
damage to cerebellum
CP - Athetoid
dyskinetic
5% of cerebral palsy diagnoses
hypo and hypertonia combination
postural impairments
motor functioning difficulties
poor/uncontrolled movements
damage to basal ganglia
CP - Mixed
combination of 2 or more CP types
damage occurs in multiple areas of brain
CP - Measurement of Motor Function Scale
Gross Motor Function Classification
CP - Measurement of Communication Impairment System
Communication function classification system
CP: LL Ox Intervention
Foot orthoses
SMOs
AFOs
FRAFOs
Positional AFOs
Decontracture orthoses
CP: UE Ox Intervention
hand, wrist, and elbow static positioning orthoses can help to decrease contractures