Public Health (Respiratory) Flashcards

1
Q

Are the numbers of diagnoses of asthma increasing or decreasing?

A

Increasing.

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2
Q

Are the numbers of hospitalisations with asthma increasing or decreasing?

A

Decreasing

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3
Q

What are the risk factors for asthma?

A

Family history of atopic disease, co-existence of atopic disease, male (pre-pubertal) or female (childhood to adulthood), bronchiolitis in infancy, parental smoking (including perinatal), low birthweight and/or premature birth.

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4
Q

In what sex and where is lung cancer more prevalent?

A

Males, Scotland in the UK.

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5
Q

What socioeconomic correlation is there with lung cancer?

A

Correlation with deprivation and mortality.

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6
Q

What are the sources of information when we assess the burden of morbidity?

A

Hospital admissions/discharges, bed occupancy, GP contacts.

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7
Q

Why when looking at statistics of illnesses does setting matter?

A

As some conditions are managed more commonly in primary care whereas others are managed in hospitals.

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8
Q

What does it mean when we say there is a dose response relationship in smoking and lung cancer?

A

If you smoke more you will have a greater risk.

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9
Q

How can health inequalities be decreased?

A

Governments making legislative changes.

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10
Q

What was the effect of the Irish smoking ban on the respiratory health of bar workers in Dublin pubs?

A

Statistically significant improvements in pulmonary function tests, significant reduction in self-reported symptoms.

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11
Q

Why can we not just ban tobacco?

A

There is a lot of tax revenue, would add to illicit trade.

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12
Q

What is Scotland and England’s most popular quitting aid?

A

E-cigarettes.

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13
Q

What is the clinical presentation of influenza (what are the signs/symptoms)?

A

Fever (high, abrupt onset), malaise, myalgia (sore muscles), headache, cough, prostration (knocked flat).

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14
Q

What is the aetiology (causes)?

A

Classical flu (A+B), flu-like illnesses (parainfluenza viruses, many other viruses), haemophilus influenzae (bacterium, not primary cause of flu, may be a secondary invader).

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15
Q

Describe the surface antigens of the flu virus?

A

2 different types (hemaghlutin, neuraminidase).

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16
Q

How do we name viruses/flu?

A

Virus type/geographic origin/strain number/year of isolation (virus subtype).

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17
Q

What are the 2 complications of flu (both pneumonias)?

A

Primary influenzal pneumonia (seen most during pandemic years, can be disease of young adults, high mortality). Secondary bacterial pneumonia (more common in elderly, debilitated, pre-existing disease, cause of mortality in all influenza epidemics).

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18
Q

Describe the 2 types of therapy for flu.

A

Symptomatic (bed rest, fluids and paracetamol). Antivirals (oseltamivir, zanamivir, 2% of population were prescribed during 2009 pandemic, prescribed when flu is circulating, risks of complications, use in prophylaxis).

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19
Q

What are the differences between epidemics and pandemics?

A

Epidemics: in winter, seen in association with minor mutations in surface proteins of virus (antigenic drift). Pandemics: rare, unpredictable, influenza A, antigenic shift, animal reservoir/mixing vessel required.

20
Q

Define endemic, epidemic and pandemic.

A

Endemic: occurs naturally in population. Epidemic: outbreak of an unexpected size (more than one area/country for flu). Pandemic: global distribution of disease.

21
Q

Describe phases 1-6 of a pandemic.

A

1-3: predominantly animal infections. 4: sustained human-to-human transmission. 5-6: widespread human infection.

22
Q

Describe post peak and post pandemic levels.

A

Post peak: possibility of recurrent events. Post pandemic: activity at seasonal levels.

23
Q

Give 2 examples of flu pandemics in the past.

A

2009 swine flu epidemic, 1918 post world war flu epidemic.

24
Q

What are the internal, external and social causes of lung disease?

A

Internal: genetic and uterine development. External: smoking, exposure to agents. Social: deprivation, cultural norms.

25
Q

Give some examples of occupational lung diseases.

A
Hypersensitivity pneumonitis (extrinsic allergic alveolitis) e.g farmer's lung, bird breeder's lung, cheese workers lung, mollusc shell hypersensitivity, paprika splitter's lung. 
Pneumoconiosis e.g. asbestosis, silicosis, coal worker's lung, berylliosis.
26
Q

What is the pathological changes in chronic hypersensitivity pneumonitis and what is the treatment focused on?

A

Extensive fibrosis with honeycombing and air-trapping. Treatment focused on antigen avoidance.

27
Q

What is asbestosis and what is it caused by?

A

Fibrotic lung disease, cause by inhalation of asbestos fibres.

28
Q

What are the different types of asbestos?

A

White (safest), brown and blue.

29
Q

Is there a synergystic effect between smoking and asbestos exposure causing lung cancer?

A

Yes, there is some interaction between the smoking and the asbestos that causes greater cancer rates.

30
Q

What industries are involved in asbestos exposure?

A

Shipbuilding, mining, heat insulation and building.

31
Q

What are the indoor and outdoor causes of lung disease?

A

Indoor: asbestos, mould, cooking smoke, passive smoking, nanparticles.
Outdoor: air/traffic pollution.

32
Q

What can mould in housing cause?

A

Respiratory problems, a lot more childhood asthma.

33
Q

What is the definition of fuel poverty?

A

Where over 50% of your household budget is spent on heating.

34
Q

What was introduced to decrease the cost of heating on an island and has it been effective?

A

A subsidy, no.

35
Q

Why is overcrowding greater in Perth and Kinross than in Dundee?

A

Eastern European immigrants living in caravans for fruit picking.

36
Q

What groups is overcrowding more common in?

A

Minority ethnic groups, poorer areas.

37
Q

What can help solve the problem of cooking smoke?

A

Types of cooking stoves.

38
Q

What makes up photochemical smog and where can it be found?

A

Ozone, nitrogen oxide, nitrogen dioxide, volatile organic compounds (VOCs) all produced from cars.

39
Q

What makes up olden days “London” smog and what event did it affect?

A

Fly ash, sulphur dioxide, sodium chloride, calcium sulphate particles (all from high sulphur coal).

40
Q

What are the most common hazardous materials in air pollution?

A

SO2, NOs, particular matter, O3, volatile organic compounds, persistent organic compounds (POPs), benzene, CO, lead and heavy metals.

41
Q

What negative effects can traffic fumes have on people’s health?

A

Reduced lung growth in adolescence, increased rates of asthma and COPD, increased rates of respiratory symptoms like wheeze/cough/breathlessness.

42
Q

What effects outside the lungs can inhaled substances have?

A

Low birth weight, appendicitis, stroke, neurological/neurobehavioural outcomes e.g. neurodegenerative disease, cognitive decline, depressions and suicide.

43
Q

What are dioxins and what problems do they cause?

A

A subset of persistent organic compounds. Associated with reproductive and immunological problems. Bioaccumulates both in people and animal fat.

44
Q

Why is there a cycle of smoking in deprived areas?

A

There are more cigarette shops.

45
Q

What makes you more likely to live next to hazardous landfills? (environmental justice)

A

If you are poorer.

46
Q

What are the causes of water scarcity?

A

Global population increase, growing demand for food, more agriculture needing more water, increased allocation for cities/agriculture/industries, increased tension/conflict/environmental strain.

47
Q

What effect does climate change have on water scarcity?

A

More frequent droughts, intensified storms and flooding, destroys crops, contaminates freshwater and damages the facilities used to store and carry that water.