PSY1004 SEMESTER 2 - WEEK 4 - PART 1 Flashcards

1
Q

when does perinatal complications occur

A

period just before and after birth

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2
Q

what % congenital defects does genetics account for

A

10-15%

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3
Q

outline risk of inherited gene disorders, give examples of autosomal gene disorders and how they can be screened

A

risk dependent on father status
autosomal gene disorders eg: sickle cell disease, cystic fibrosis
able to detect in prenatal screening of maternal blood plasma (non-invasive) but cannot treat only terminate

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4
Q

how common is chromosomal abnormality, survival rate and what causes this

A

1/200 live births (eg; down syndrome), accounts for 50-70% first trimester miscarriage
whole chromosome/missing parts/duplicated, normally caused by error in separation of chromosomes into appropriate daughter cells during meiotic division

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5
Q

name 6 environmental risk factors for foetal developments

A

radiation, air pollution, viral infections, domestic violence, SES, maternal malnutrition

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6
Q

environmental risk factors to foetal development - what could radiation cause

A

serious birth defect and cancer

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7
Q

environmental risk factors to foetal development - air pollution

A

potential links autism

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8
Q

environmental risk factors to foetal development - can can viral infection cause

A

eg: COVID-19, increase risk of premature births, stillbirth, low birth weight, but limited evidence of neurodevelopmental impact

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9
Q

environmental risk factors to foetal development - how many mothers experience domestic violence in pregnancy

A

7-8%, high rate miscarry

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10
Q

environmental risk factors to foetal development - name key components of how SES can influence neural development

A

cognitive stimulation, education, language environment, nutrition, parenting quality, environmental toxin (smoking)

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11
Q

environmental risk factors to foetal development - what can maternal malnutrition cause

A

premature birth, low-birth weight, spina bifida (NTD), reduced if folic acid

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12
Q

what can low birth weight link to

A

increased risks for future cardiovascular disease, risk of future breast cancer, mental illness

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13
Q

name 3 maternal risk factors in foetal development

A

age, teenage pregnancies, stress and support

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14
Q

how can age of mother be a maternal risk factor

A

increased risks of chromosomal abnormality (downs syndrome risk 1/2000 at 20, 1/200 at 37)

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15
Q

how can age of father be a risk factor for foetal development

A

factor for social functional impairment eg: autism, sz due to sperm DNA methylation abnormalities

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16
Q

outline how teenage pregnancy can be maternal risk factor for foetal development

A

underdeveloped pelvic cradle causing birth complication
correlation between SES and teenage pregnancies causes maternal malnutrition, poor quality health care, stressors like unsupportive environments

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17
Q

what is psychological stress during pregnancy associated with (maternal risk factors to foetal development)

A

premature deliveries, low birth weight, foetal neurobehavioural development

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18
Q

what are protective factors against maternal psychological stress

A

sympathetic family and partner, and adequate housing

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19
Q

what are teratogens

A

risk factors that have massive impacts in prenatal development and affect foetus via mother

20
Q

name some teratogen examples

A

drugs, environmental contaminent, infection, x-ray, radiation, smoking, alcohol

21
Q

teratogens- explain smoking as both a environmental factor and maternal factor

A

environmental factor = second-hand smoke
maternal factor = first hand smoke

quantity/timing important
stopping isnt easy (addiction), especially in unplanned pregnancy = NHS nicotine replacement therapies important

22
Q

what can nicotine result in for foetal development

A

abnormal placenta growth, increasing rate of spontaneous abortion, stillbirth, neonatal death and low birth weight

23
Q

what problems can maternal smoking cause in children

A

lung problems
childhood athsma
developmental, learning and behavioural problems
short-attention span

24
Q

what is leading cause of intellectual disabilities

A

alcohol

25
Q

what physical developmental abnormalities can FAS cause

A

facial deformity, underdeveloped upper jaws, widely spaced eyes, congential heart disease, abnormally small head, joint abnormalities

26
Q

what brain damage can fetal alcohol syndrome cause

A

corpus callosum, hippocampus, basal ganglia
can result in neurological and not physiological problem
most impact during first GWs

27
Q

teratogenic risk factors- what can caffeine cause

A

increased spontaneous abortion and low birth weight

28
Q

teratogenic risk factors- what can marijuana cause

A

low birth weight, premature deliveries, infant startles easily, tremors, sleep-cycle problems

29
Q

teratogenic risk factors- what can cocaine cause

A

stillborn, premature, low birth weight, stroke, birth defect, irritable, uncoordinated, slow learner

30
Q

teratogenic risk factors- what can methadone & heroin cause

A

born addiction, premature, underweight, respiratory problems, tremors, irritable, difficulties with attention, motor control

31
Q

teratogenic risk factors- what can rubella (viral) cause

A

congential heart disease, deaf, cataracts, intellectual disabilities

32
Q

teratogenic risk factors- what can syphilis (bacteria) cause

A

liver damage, impaired hearing and vision, teeth and bone deformities

33
Q

explain the teratogenic principles

A

infant susceptibility dfepends on foetal development stage, normal variance in susceptibility across population and mother physiological state
can impact infant little/not at all

34
Q

define transatal learning

A

learning that occurs during prenatal period but is remembered during postnatal period

35
Q

give an example for prenatal learning 8 GW

A

touch is the first sense developing

36
Q

give an example for prenatal learning 9-24 GW

A

smell, continue to develop post-birth

37
Q

give an example for prenatal learning 12-28 GW

A

taste combined with smell, producing olfaction

38
Q

give an example for prenatal learning 18-20 GW

A

good hearing

39
Q

give an example for prenatal learning 21-28 GW

A

vision, continuing to develop post-birth

40
Q

explain habituation paradigms when studying transnatal learning

A

present foetus or infant with auditory cues multiple times until response plateu (sucking, HR), infant beings to respond to stimuli again (dishabituation). present again and if learning occurred then less trials needed for habituation

41
Q

give a research study for prenatal auditory learning (Morokuma et al, 2004) using HR to study habituations, and which GW is important in development

A

32-34GW, more variable habitual response but no dishabituation
35-37GW, foetus can habituate/dishabituate
shows 35GW an important stage in learning

42
Q

give research evidence for transnatal auditory learning via mothers voice

A

infant prefer prenatal version of mothers voice (low filter), 2 hr neonate react with more movement to mothers voice>strangers voice suggesting learn about sound inutero
can use sucking rate, suck more to activate recording but not found in preterm neonate (pre 37-GW)

43
Q

outline research evidence for mothers voice in transnatal learning (Beebe & Lachmann, 2015) - cat in hat

A

read story x2 day last 6GW, change in rate of sucking after turning off tape, and infant modified sucking rates to activate recording of familiar stories

44
Q

what positive impacts can playing recordings of HB show in neonates

A

greater weight gain and less crying

45
Q

explain language discrimination in infants

A

4 days post birth discriminates mothers language with a preference

46
Q
A