PSY1003 SEMESTER 2 - WEEK 9 Flashcards

1
Q

describe behavioural sleep characteristic

A

reduced motor activity or response to stimulations, stereotypic positions and easily reversible

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2
Q

what was Freuds interpretation of dream

A

triggered by unacceptable, and repressed wishes

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3
Q

explain activation-synthesis hypothesis

A

dreams due to cortex attempting to make sense of random brain activities

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4
Q

explain frequency and amplitude of EEG when alert, or relaxed

A

alert- high frequency (15-30Hz), low amplitude, ‘beta’
relaxed- medium frequency (8-12Hz), medium amplitude, ‘alpha’

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5
Q

during first stage of sleep, describe EEGs

A

transition from wake-sleep, muscles still active, gentle eyes movements
theta
3.5-7.5Hz

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6
Q

in stage 2/3, what happens to EEG

A

progressively get lower in frequency but higher in amplitude

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7
Q

in stage 3/4 what happen to EEG

A

delta, high amplitude, <3.5Hz
slow-wave, sleep walking/talking

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8
Q

how long does the sleep cycle last

A

90min

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9
Q

describe carosel animal studies in sleep deprivation

A

when EEG indicates rat asleep, floor move rat into water
dies after several days (post mortem show extreme stress)

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10
Q

what does sleep deprivation have little effects on

A

logical deduction, critical thinking, physical strength, motor performance

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11
Q

what does sleep deprivation have large effect on

A

EF = assimilating info, updating plan or strategy, innovative lateral insight thinking, reference memory

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12
Q

what can 3-4hr of deprivation in one night cause

A

increased sleepiness, disturbance on mood written tests, poor performance in vigilance test

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13
Q

what can 2-3 days of continuous deprivation cause

A

microsleep (nap of 2-3s)

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14
Q

what is impact of sleep deprivation on physiology

A

reduced temp, increase BP, decreased immune function, hormone change, metabolic change

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15
Q

explain why sleep deprivation can increase sleep efficiency

A

post-dep, most of lost stage4 regained. slow-wave sleep increase
short sleepers get as much slow-wave as long
nap without slow-wave doesn’t impact sleep
little sleepiness if repeatedly woke in REM

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16
Q

how do you study REM sleep deprivation, and what 2 consistent effects have shown

A

wake those in REM, then let them go back to sleep
1. go more rapidly to REM as dep increase, so need to wake them more frequently
2. REM rebound- spend more time in REM post-dep for first 2,3 nights

17
Q

explain impact of sleep on depression

A

insomnia and hypersomnia
get less slow-wave sleep but no memory disruption
once taking anti-depressants, may not get REM sleep for years

18
Q

how may sleep deprivation improve depression

A

total sleep deprivation for 1 night improve symptoms for 40-60% but no-one knows why

19
Q

explain link between sleep deprivation and alzheimers

A

risk factor
sleep used as a waste clearing system- CSF moves through brain and remove waste products (eg; amyloid-B)

20
Q

what is the recuperation theory of sleep

A

restoring homeostasis, energy levels to clear toxins

21
Q

what is adaptation theory of sleep

A

internal 24hr timing mechanisms, programmed to sleep at night regardless of what happens during day

22
Q

explain purpose of REM being to process explicit memory, but issues with this

A

process explicit memory?- inconsistencies, antidepressants REM-blocking don’t impact memory

23
Q

explain default theory for purpose of REM

A

difficult to remain in non-REM
if wake sleeper in REM, stay awake for 15min
showed no sleepiness/REM rebounds next day suggesting no need for REM sleep if wakefulness sustitues

24
Q

explain why purpose of REM may be brain development

A

70% REM newborn
30% REM 6 months
continues to decrease in adults

25
Q

what are circadian rhythms

A

24hrs, endogenous, persist without environmental cues, modulated by external environment which can muddle them “zeitgebers”

26
Q

give example of a zeitegeber

A

daylight, artificial lights, jet lag

27
Q

define internal desynchronisation

A

when housed in constant lab environment, sleep-wake and body temp cycles break away from one another

28
Q

what does hypothalamu control

A

circadian rhythm, body temp, hunger, thirst

29
Q

what is SCN (medial hypothalamus)

A

has direct projection from retina
major internal clock, dampens circadians, regulate sleep timings

30
Q

what does lesioning SCN result in

A

disrupt circadian rhythm but not impact time spent asleep, stops circadian periodicity of sleep cycles
transplant SCN into different mammal results in animal adopting different sleep-wake cycle

31
Q

when are SCN neurons active

A

SCN neuron inactive at night
starts firing at dawn
steady firing pace during day

32
Q

what is pons clocks responsible for

A

basic rest-activity cycle (BRAC)
cycles of REM sleep, slow wave sleep

33
Q

explain reticular formation

A

set of interconnected nuclei located throughout brainstem

34
Q

what is pontine reticular formations

A

brain region without clearly defined border in pon centre

35
Q

what do lesions at midcollicular level of reticular formation cause

A

damage, but leave sensory fibre intact so produce cortical EEG indicative of continuous slow-wave sleep

36
Q

what cells mediates circadian rhythms

A

retinal ganglion photoreceptors cells