Brainscape's Knowledge GenomeTM


Top Flashcards (Certified)
All Flashcards

Neruo 1 - Test 3 > Primary Impairments - Lecture 11 > Flashcards

Primary Impairments - Lecture 11 Flashcards

1
Q

motor recovery is initially

A

flaccid

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

motor recovery: there is a development of

A

spasticity

hyperreflexia

mass patterns of movement

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

as recovery continues –> motor recovery

A

spasticity and synergies decline

advanced motor patterns are possible

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Brunnstrom Stages of recovery

A

6 stages

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

stage 1

A

flaccidity

no mvt

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

stage 2

A

minimal voluntary mvt

associated rxns

spasticity begins to develop

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

stage 3

A

voluntary control of mvt synergies

spasticity peaks in severity

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

stage 4

A

mastery of some mvt synergies

spasticity peaks in severity

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

stage 5

A

difficult mvt combos are learned

synergies lose their dominance

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

stage 6

A

spasticity disappears

isolated joint movement and coordination achieved

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

flexion synergy components UE strongest component

A

elbow flexion

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

strongest component of flexion synergy components LE

A

hip flexion

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

strongest component of extension synergy components

A

shoulder ADD

forearm pronation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

strongest component of extension synergy component

A

hip ADD

knee extension

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

why do we experience paresis

A

decrease # of fxning agonist motor units

recruitment order may be altered

decreased firing rates

denervation changes in corticospinal tract

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

why do we experience paresis (2)

A

atrophy of mm fibers

contraction time increases with increased fatigability

inappropriate co-contract of mm

mechanical changes in soft tissue

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

incoordination is the result of

A

cerebellar or BG involvement

proprioceptive losses

motor weakness

ataxia (esp w/ cerebellar disorders)

impaired stretch reflex response)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

impaired stretch reflex response

A

normally allows automatic adaptation of mm to postural movement changes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

aphasia

A

brocas

wernikes

global

conduction

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

dysarthria

A

impairment of speech production secondary to damage to the CNS or PNS

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

what does dysarthria cause

A

oral motor weakness, paralysis or incoordination of the motor speech system

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

what does dysarthria affect

A

respiration

phonation

articulation

resonance

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

dysarthria has deficits in

A

swallowing

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

dysarthria has lesions affecting

A

CN 9 & 10

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
CN lesions cause
delayed triggering of swallow reflex decreased pharyngeal peristalsis decreased lingual control
26
cognitive/behavioral deficits
in orientation attention processing speed conceptual abilities executive fxn memory
27
cognitive/behavioral deficits cause
emotional liability (R) CVA (L) CVA generalizations
28
emotional liability
unstable or changeable emotional state
29
emotional liability is characterized by
pathological rapid change from laughing to weeping w/ only slight provocation
30
(R) cva
difficulty grasping the whole idea or the overall organization of a pattern or activity
31
what is R cva described as
indifferent, quick, impulsive, euphoric pt overestimates their ability while minimizing the problem
32
R CVA major issue
safety
33
L cva
difficulty with processing ingo in sequential/linear manner
34
L cva described as
slower, negative, cautious, uncertain, depressed, anxious
35
generalizations
does not apply to everyone
36
Visual perception disorders
body scheme/body image disorders spatial relation syndrome agnosias apraxia
37
body scheme/body image disorders
somatagnosia unilateral neglect right-left discrimination finger agnosia anosognosia
38
somatagnosia
lack of awareness of the body structure and the relationship of body parts in oneself or others
39
somatagnosia --> difficulties
performing transfers following direction
40
somatognosia lesion
dominant parietal lobe or posterior temporal lobe
41
unilateral neglect
spatial neglect inability to register and to integrate stimuli and perceptions from one side of the body and the environment
42
unilateral neglect types
personal peri personal extrapersonal
43
where does unilateral neglect typically affect
left side of the body sensory loss compounds the problem
44
unilateral neglect lesion
non-dominant parieto-occipital area
45
right left discrimmination
inability to identify R or L side of one's own body or the examiner
46
RL discrimmination lesion
parietal lobe or either hemisphere
47
finger agnosia
inability to identify the fibers of one's own hand correlates highly with poor dexterity
48
finger agnosia lesion
parietal lobe of either hemisphere
49
anosognosia
severe condition including denial, neglect and lack of awareness of the presence or severity of one's paralysis
50
anosognosia lesion
non-dominant parietal lobe
51
spatial relation deficits
figure - ground discrimination form consistency spatial relations position in space spatial memory topographical disorientation depth and distance perception vertical disorientation
52
figure ground discrimmination
inability to visually distinguish a figure from the background lesion: non-dominant parietal lobe
53
form consistency
inability to perceive or to attend to subtle differences in form and shape lesion: non-dominant parieto-temporo-occipital region
54
spatial relations
inability to perceive the relationship of one object in space to another object or to oneself difficulty crossing midline
55
spatial relations lesion
non-dominant parietal lobe
56
position in space
inability to perceive and to interpret spatial concepts such as up, down, under, over, in, out, etc
57
position in space lesion
non-dominant parietal lobe
58
spatial memory deficit
impaired memory of location of objects/places
59
spatial memory lesion
non-dominant parietal lobe
60
topographical disorientation
difficulty in understanding and remembering the relationship of one location to another unable to trace path/route
61
topographical disorientation lesion
non-dominant occipitoparietal lobe
62
depth and distance perception
inaccurate judgement of direction, distance and depth lesion: non dominant occipital lobe
63
vertical disorientation
distorted perception of what is vertical causes imbalance and distorted midline orientation
64
vertical disorientation lesion
non-dominant parietal lobe
65
agnosia
inability to recognize familiar objects using one or more sensory modalities while often retaining the ability to recognize the same object using other sensory modalities
66
types of agnosia
visual auditory tactile
67
visial
visual object agnosia simultanagnosia propagnosia color agnosia
68
visual object agnosia
inability to recognize and name common objects
69
simultanagnosia
inability to perceive the whole or the "big picture" only sees 1 element of an object at a time decreases visual span --> tubular vision
70
prosopagnosia
facial agnosia inability to recognize familiar faces
71
color agnosia
difficulty recognizing names of colors
72
motor praxis
ability to plan and execute coordinated movements
73
apraxia
disorder of voluntary learned movement inability to perform purposeful movements
74
where is apraxia seen
L hemisphere lesions more than R often accompanied by aphasia
75
lesion --> apraxia
premotor frontal cortex of either hemisphere left inferior parietal lobe corpus callosum
76
types of apraxia
ideomotor ideational
77
ideomotor
breakdown b/w concept and performance movement is not possible upon command by occurs automatically
78
oral apraxia
subtype of ideomotor apraxia buccofacial ms cannot produce purposeful movement
79
ideational
failure in conceptualization of the task purposeful movement is not possible either automatic or on command
80
apraxia like syndromes
not true apraxias more associated with R hemisphere lesions
81
like syndromes
constructional apraxia dressing apraxia
82
constructional apraxia
difficulty in recognizing parts to a whole secondary faulty spatial analysis and conceptualization of the task
83
dressing apraxia
inability to dress oneself properly owing to a disorder in body scheme or spatial relations

Neruo 1 - Test 3 (12 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2025 Bold Learning Solutions. Terms and Conditions