Central Vestibular System & Peripheral System Disorders- Lecture 12 Flashcards

1
Q

vestibular nuclei complex

A

primary processor of vestibular input

connects incoming afferent info and motor output neurons

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2
Q

what does the vestibular nuclei complex have

A

4 major nuclei in the pons

extends into the medulla

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3
Q

what does the vestibular nuclei complex process

A

vestibular sensory input and extra vestibular proprioceptive, visual, tactile and auditory info

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4
Q

cerebellum

A

monitors vestibular performance and readjusts the central processing

major recipient of vestibular info from the nuclei

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5
Q

what do both the cerebellum and nuclei process

A

info in association with somatosensory, proprioceptive and visual input

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6
Q

vertebrobasilar arterial system

A

provides supply for both the peripheral and central vestibular system

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7
Q

when might clinical syndromes with vestibular components appear

A

after occlusions of the basilar artery and its branches

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8
Q

VSR

A

vestibular spinal reflex

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9
Q

vestibular spinal reflex

A

adjusts posture when the head is moved

anticipates a loss of balance and adjusts posture to maintain balance

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10
Q

abnormal VSR

A

difficulty with static or dynamic balance and the feeling of being off balance

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11
Q

age related changes with the vestibular system

A

increased difficulty with eyes closed activities

decreased ability to detect head position and motion

reduced number of receptor cells

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12
Q

BPPV

A

benign paroxysmal positional vertigo

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13
Q

what is BPPV

A

positional vertigo of short duration (up to one minute) which occurs when the pt lie down, sits up, rolls over in bed, looks up or bends over

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14
Q

what is BPPV caused by

A

otoconia from the otolith that have become misplaced within the semicircular canals

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15
Q

Hallpike-Dix position

A

20-30 degrees of cervical extension

45 degrees of rotation

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16
Q

what does the Hallpike-Dix position elicit

A

symptoms of vertigo within 1-40 secs

not lasting more than 60 sec

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17
Q

why does BPPV occur

A

head trauma

degeneration of the vestibular system

following ear surgery

dental work

virus

idiopathic

link to vitamin D deficiency

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18
Q

what will be seen in the dix hallpike position with BPPV

A

torsional nystagmus

pt’s will habituate with repetition

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19
Q

what else can BPPV be seen in

A

roll test position

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20
Q

roll test position

A

pt supine

30 degrees of flexion and 45 degrees of rotation

nystagmus will not be torsional

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21
Q

how can BPPV resolve

A

w/o treatment

usually b/w 6 mo-1 yr

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22
Q

how can BPPV be treated

A

epley/CRt within 1-3 visits

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23
Q

labyrinthitis

A

inflammation of the labyrinth including cochlea

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24
Q

what is labyrinthitis d/t

A

upper respiratory infection

flu

bacterial or viral infection occurring up to 2 weeks prior to onset of symptoms

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25
acute symptoms of labyrinthitis
sudden onset of vertigo lasting days w/ nausea and vomiting common pt may be bed ridden or hospitalized secondary to severe symptoms
26
labyrinthitis med
antivert or meclizine may be used initially to surpress the vestibular system decrease vertigo
27
when should meds be stopped --> labyrinthitis
prior to coming to therapy in order for compensation to occur
28
chronic sx --> labyrinthitis
imbalance and dizziness
29
when are symptoms worse --> labyrinthitis
head movement busy visual stimuli stress fatigue weather changes altitude changes illness
30
pts that recover on their own --> labyrinthitis
80%
31
pts that need treatment --> labyrinthitis
prolonged vestibular rehab can speed up compensation
32
vestibular neuritis
inflammation of the vestibular nerve
33
sx of vestibular neuritis
same as labyrinthitis except hearing is spared
34
acoustic neuroma
slow growing benign tumor on CN8 overtime will compress on structures adjacent to it
35
first sx of acoustic neuroma
hearing loss tinnitus decreased vestibular fxn with impaired balance
36
acoustic neuroma has
no dizziness CNS is able to gradually adapt in response to slow onset of sx and slow growing tumor
37
what is recommended for acoustic neuroma
surgery to remove tumor risk of facial palsy and/or permanent hearing loss
38
gamma knife procedure --> acoustic neuroma
used for smaller tumors
39
stereotactic radiotherapy --> acoustic neuroma
used for small tumors
40
how do pts recover --> acoustic neuroma
without tx vestibular rehab is successful for those who seek treatment
41
meniere's/endolymphatic hydrops is caused by
alteration in production, flow, absorption or reabsorption of the inner ear fluids
42
underlying origin of meniere's/endolymphatic hydrops
unknown may be influenced by infection, abnormal response of the immune system or genetic tendency
43
sx of meniere's/endolymphatic hydrops
vertigo lasting 20 min-12 hrs sensory neural hearing loss nausea vomiting diarrhea
44
sensory neural hearing loss
predominantly low frequency tinnitus or fullness in ears
45
when do pts have episodes --> meniere's/endolymphatic hydrops
one episode every hew years to weekly or daily episodes
46
what occurs after an episode --> meniere's/endolymphatic hydrops
may or may not be residual sx overtime residual sx may worsen --> become symptoms were able to treat
47
when does rehab become ineffective --> meniere's/endolymphatic hydrops
no sx in between episodesq
48
tx for meniere's/endolymphatic hydrops
diet changes various surgeries gentamycin injections endolymphatic shunt
49
endolymphatic shunt
tube inserted into endolymphatic sac to facilitate drainage
50
superior canal dehiscence
thinning or opening in the bone overlying the superior (anterior) semicircular canal
51
symptoms of SCD
vertigo oscillopsia caused by loud noise and activities that increase intracranial pressure sensitivity to sound fatigue fullness in the ear
52
what does SCD create
a third area affected by pressure fluid w/in the canal is moved by stimuli from pressure or sound
53
what is SCD caused by
developmental abnormality of thin, fragile bone over the superior canal
54
when do symptoms present SCD
following trauma or slow erosion of the bone over time
55
how is SCD diagnosed
CT symptoms clinical exam
56
how is SCD treated
surgery to reface the bone or plugging of the canal
57
perilymphatic fistula
rare condition a tear or defect in the oval or round window that separates the inner and middle ear changes in pressure that affect the middle ear will also affect the inner ear
58
when does perilymphatic fistula occur
following trauma or following rapid changes of intracranial or atmospheric pressure
59
sx perilymphatic fistula
vertigo nausea imbalance
60
when do symptoms increase perilymphatic fistula
pressure changes or altitude exertion
61
tx perilymphatic fistula
strict bed rest or surgery do not get better with therapy
62
ototoxicity
damage to hair cells of inner ear
63
what causes ototoxicity
drugs --> primarily gentamicin and other "mycin" drugs esp if meds are delivered by IV for greater than 6 days
64
pt may not have --> ototoxicity
total loss of vestibular fxn
65
sx ototoxicity
imbalance ataxia oscillopsia gaze instability with decreased or absent VOR
66
sx will be worse w/ --> ototoxicity
increased loss of hair cells
67
when will a pt have vertigo --> ototoxicity
damage is asymmetrical
68
vestibular rehab --> ototoxicity
focused on strengthening residual fxn or on substitution if there is total bilateral loss
69
TBI/head trauma can cause
both central and peripheral problems
70
sx -->TBI/head trauma
dependent on the area involved and the severity of central or peripheral injury
71
sx could include --> TBI/head trauma
dizziness, vertigo, imbalance, headache, migraine, short term memory deficit and motion sensitivity
72
vestibular rehab --> TBI/head trauma
prolonged