Post-activation, Potentiation Flashcards
A theory that purpose that the contractile history of a muscle influences the mechanical performance of subsequent muscle contraction.
Post-activation and potentiation
Contractile history
- Warm-up
- Prior exercise
- Intensity
- Duration
- Frequency
- Specificity
Decrease in force production, contraction velocity and power
Fatigue
Enhanced force production, contraction velocity and power
Potentiation
- CNS regulates exercise performance to prevent catastrophic physiological failure
- Sensory feedback from working muscles
- Accumulation of ammonia
- Increases in serotonin in the brain
Central Fatigue
- Accumulation of fatigue causing metabolites in muscle fibers (lactate, inorganic phosphates, and ammonia)
- Depletion of ATP-producing fuels (Phosphagen and glycogen)
Peripheral fatigue
The magnitude of skeletal muscle force production is determined by;
- Motor unit recruitment
- Muscle twitch characteristics
Motor nuit recruitment
Size principle
- Smallest (type 1) –> largest (type 2)
- higher frequency = higher force
Mechanisms of Potentiation
-Post-activation potentiation;
1) Improved sensitivity of actin-myosin complex to calcium
2) increased excitability of motor neurons and/or the quantity of released neurotransmitters
Appears to be a means for acutely increasing force and power production, and thus, athletic performance
Post-activation potentiation
Is the most established method for eliciting a PAP effect, but it lacks specificity and presents logistical issues
Traditional resistance exercise
Are more practical and specific methods for stimulating PAP, but appropriate prescription is unknown.
Plyometrics and resisted jumping/sprinting
sensitivity of actin-myosin complex
- Calcium in sarcomere binds with troponin and calmodulin
- Calmodulin (messenger protein) activates regulatory light chains(RLC)
- RLC’s cause myosin head t move away from thick filament backbone
- Quicker transition from relaxation to a force producing state (i.e; rate of force production)
Motor neuron excitability
- Acetylcholine (ACH) is stored at the terminal end of motor neuron’s axon
- Calcium allows ACH to be released into synaptic gap (cleft)
- More calcium increases probability of ACH release
- Greater stimulus for muscle contractions
- 10-second isometric maximal voluntary (MVC) contraction increased peak torque 71% at 5 seconds-post MVC
- By 30 to 60 seconds post-MVC, peak torque decreased to +44% and +31%, respectively
- Potentiation continued to gradually decrease, but was still +12% at 300 seconds-post MVC
Hamada et al. (2000)