Phosphorus Flashcards

1
Q

Phosphorus: lab

A

2.4-4.4mg/dL

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2
Q

Phosphorus is the primary anion of

A

ICF

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3
Q

phosphorus is ___ most abundant in the body

A

second most abundant element in the body (after calcium)

most found in bones and teeth

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4
Q

phosphorus is metabolically active and essential to

A

muscle function
RBC
nervous system

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5
Q

phosphorus is involved in

A
acid-base buffering system
mitochondrial formation of ATP 
cellular uptake and use of glucose
metabolism of carbohydrates, proteins and fat
inversely related to calcium
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6
Q

what maintains serum phosphorus levels and balance

A

PTH

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7
Q

what is the major route for phosphorus secretion?

A

kidneys (regulation requires adequate renal functioning)

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8
Q

when PTH levels are too low, the kidneys

A

reabsorbs additional phosphorus

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9
Q

high serum phosphate levels cause

A

a low calcium concentration in serum

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10
Q

low serum Ca levels stimulate release of PTH which

A

dec reabsorption of phosphorus

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11
Q

what is absorption influenced by?

A

vit D

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12
Q

what is phosphorus needed for?

A

production of ATP

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13
Q

maintains WBC functioning

A

phagocytosis

platelet function

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14
Q

hyperphosphatemia: Pts at risk

A
high serum phosphate
acute kidney injury or chronic kidney disease(cant excrete phosphate)
dec excretion 
hypoparathyroidism
enemas- P containing
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15
Q

hyperphosphatemia: assessment

A

inc nerve impulse transmission

low Ca S&S

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16
Q

hyperphosphatemia: assessment- mild

A

asymptomatic

17
Q

hyperphosphatemia: assessment- severe

A

low serum Ca levels with high serum phosphate levels

tetany. muscle cramps, paresthesias, seizures

18
Q

hyperphosphatemia: assessment- inc levels of phosphate readily precipitate with Ca

A

found in soft tissues: joints, arteries, skin, kidneys, and corneas and produce organ dysfunction- RF

19
Q

hypophosphatemia: pt at risk

A

low serum phosphate

20
Q

hypophosphatemia: pt at risk- dietary changes

A
malnutrition/malabsorption
chronic alcoholism (dec dietary intake, diuresis)
21
Q

hypophosphatemia: pt at risk- GI abnormalities

A

vomiting, anorexia
chronic diarrhea
intestinal malabsorption

22
Q

hypophosphatemia: pt at risk- hormonal influence

A

hyperparathyroidism

23
Q

hypophosphatemia: pt at risk- med influence

A

aluminum containing antacids dec P

diuretics can dec P

24
Q

hypophosphatemia: assessment- CV

A

tissue hypoxia

dsrhythmias

25
Q

hypophosphatemia: assessment risk of infection & musculoskeletal

A

weakness, fx, rhabdomyolysis (rapid breakdown of skeletal muscle-> myoglobin-> acute RF)

26
Q

hypophosphatemia: assessment- CNS

A

CNS depression: AMS (altered mental status); Confusion

seizures with severe dec P

27
Q

hyperphosphatemia: Nsg Dx

A
dec CO r/t dysrhythmias 
ineffective breathing pattern r/t respiratory muscle weakness 
activity intolerance r/t NM weakness
administer phosphate binding meds 
dietary restriction: dairy products
avoid P laxatives and enemas
28
Q

hyperphosphatemia: Nsg Dx- treat hypocalcemia

A

providing hydration and instituting measures to correct Ca levels assist with returning phosphorus levels to normal

29
Q

hypophosphatemia: Nsg Dx

A
impaired physical mobility
dec CO 
injury 
infection 
DC/change med
30
Q

hypophosphatemia: Nsg Dx: prevent infection

A

hand washing, mask, WBC

31
Q

hypophosphatemia: Nsg Dx: P replacement

A

dairy products

IV Phosphate- assess for hypcalcemia

32
Q

hypophosphatemia: Nsg Dx: Dec Ca intake

A

avoid antacids and Ca supplements

33
Q

hypophosphatemia: Nsg Dx: monitor

A

respirations- rate and depth

HF